ライフサイエンスコーパス: pancreatic duct

1 12-week-old female diabetic Balb/c mice via pancreatic duct.

2 ccompanied by reverse-Z type meandering main pancreatic duct.

3 nfusion of TLCS or taurocholic acid into the pancreatic duct.

4 nd to a lesser extent in the gallbladder and pancreatic duct.

5 ated with local fibrosis and stenosis of the pancreatic duct.

6 coordinate fluid and HCO3- secretion by the pancreatic duct.

7 gnificant disruption or stenosis of the main pancreatic duct.

8 of endocrine cells usually present near the pancreatic duct.

9 uct, a stricture, or complete cut-off of the pancreatic duct.

10 roliferation in the epithelium of the common pancreatic duct.

11 or signs referrable to the biliary tract or pancreatic duct.

12 ells in the biliary tract but not the normal pancreatic duct.

13 ocus in the expected region of the accessory pancreatic duct.

14 sed mechanism is the reflux of bile into the pancreatic duct.

15 and ducts, whereas AC7 was expressed only in pancreatic ducts.

16 ency most likely due to undetected accessory pancreatic ducts.

17 d monocytes/macrophages, centered around the pancreatic ducts.

18 tions in cultured murine sealed intralobular pancreatic ducts.

19 on of beta-cells rather than neogenesis from pancreatic ducts.

20 e able to differentiate malignant and normal pancreatic ducts.

21 with an initial expansion of the developing pancreatic ducts.

22 ression in a subset of cells embedded within pancreatic ducts.

23 c liver precursors reside within or close to pancreatic ducts.

24 new beta-cells (neogenesis) derived from the pancreatic ducts.

25 hyperplastic and remained very close to the pancreatic ducts.

26 thelial cells adjacent to, or emerging from, pancreatic ducts.

27 and cyst formation in maturing nephrons and pancreatic ducts.

28 elial neoplasms involving the main and large pancreatic ducts.

29 with a standard meal to 5 dogs with ligated pancreatic ducts.

30 e model to experimental data from guinea-pig pancreatic ducts.

31 locations within the human biliary tree and pancreatic ducts.

32 and greater proportion of nondilated (<3 mm) pancreatic ducts.

33 oradic glp1r-fluorescent cells were found in pancreatic ducts.

34 reatography, 160 for deep cannulation of the pancreatic duct, 120 for stone extraction, and 60 for st

35 to 0.60) and 164 patients with a nondilated pancreatic duct (15% vs. 27%; relative risk, 0.55; 95% C

36 as well as among 136 patients with a dilated pancreatic duct (2% vs. 15%; relative risk, 0.11; 95% CI

37 ively, who were all independent and blinded; pancreatic duct abnormalities were evaluated.

38 Pancreatic duct abnormalities were observed in 60.2% of

39 an secretin (RG1068)-stimulated MRCP detects pancreatic duct abnormalities with higher levels of sens

40 s include cystic kidney disease, biliary and pancreatic duct abnormalities, skeletal patterning defec

41 pelvic pseudocyst associated with a dilated pancreatic duct and an elevated serum amylase level.

42 eatoscopy allows direct visualization of the pancreatic duct and bile duct.

43 shows promise for improved visibility of the pancreatic duct and biliary tree, compared with the conv

44 E images for image quality parameters in the pancreatic duct and common bile duct by using a five-poi

45 Pancreatic duct and endocrine lineages arise in a spatia

46 ue, while the anterior bud gives rise to the pancreatic duct and exocrine cells.

47 O(3)(-) secretion are vital functions of the pancreatic duct and other secretory epithelia.

48 oxide and Pefabloc) alone (n=7), through the pancreatic duct and preserved using the two-layer method

49 pancreatitis is the reflux of bile into the pancreatic duct and subsequent exposure to pancreatic ac

50 e overall visibility of the biliary tree and pancreatic duct and the number of ductal segments visual

51 nd distal (20 of 29 [69%] vs 13 of 29 [45%]) pancreatic duct and the proximal (25 of 28 [89%] vs 22 o

52 islets are located in close proximity to the pancreatic duct and there is a possibility of impaired c

53 Pancreatic ducts and acini from control mice and early-s

54 ixed inflammatory infiltrate centered around pancreatic ducts and ductules, combined with obliterativ

55 Hi) regulation as well as HCO3- secretion by pancreatic ducts and HCO3- reabsorption by renal proxima

56 tin and collagen IV in the basal membrane of pancreatic ducts and of cell clusters budding from the d

57 Pancreatic ducts and pancreatic duct glands harbor commi

58 PAR-2 expression on dog pancreatic ducts and PDEC was verified by immunofluoresc

59 We investigated fluid secretion by sealed pancreatic ducts and the function of Slc26a6 and the cys

60 ancreatic ducts in humans and mice occluding pancreatic ducts and thereby driving pancreatic inflamma

61 Patients with normal pancreatic ducts and those with strictures but no commun

62 s after infusion of 120 mM (5%) STC into the pancreatic duct, and duct ligation.

63 dual ductal segments of the biliary tree and pancreatic duct, and number of ductal segments visualize

64 Increased body mass index, small main pancreatic duct, and soft pancreatic texture are well-es

65 thickening of the wall, dilation of the main pancreatic duct, and tumor markers.

66 Normal bile duct, pancreatic duct, and tumor-associated metaplasias from t

67 ts with chronic pancreatitis, a dilated main pancreatic duct, and who only recently started using pre

68 ted molecule expressed in endothelial cells, pancreatic ducts, and embryonic beta cells that we previ

69 lizes to the lateral membrane in bile ducts, pancreatic ducts, and epididymis.

70 zed for appearances of pancreas, biliary and pancreatic ducts, and other findings, such as peripancre

71 Optical coherence tomography within the pancreatic duct appears to be able to differentiate mali

72 strated cell proliferation in the islets and pancreatic duct areas.

73 By contrast, the pancreatic duct arises from PDX1+ progenitors that are s

74 n reaction in total RNA from isolated common pancreatic ducts at levels 10% of those of isolated isle

75 on of secretin improves the visualization of pancreatic ducts at magnetic resonance (MR) cholangiopan

76 Ten percent (4/39) of nonneoplastic pancreatic ducts at the margins of MCN harbored mutation

77 ng WNK4 include sweat ducts, colonic crypts, pancreatic ducts, bile ducts, and epididymis.

78 unication was seen in 20 and 21 of 24 branch pancreatic duct (BPD) IPMNs with CT and MRCP, respective

79 readers for the proximal, middle, and distal pancreatic duct, but the difference was not statisticall

80 beta-cell formation from progenitors in the pancreatic ducts by selectively deleting Pdx1 from the d

81 Alternatively, 26 of 30 (86.7%) pancreatic duct CA were correctly categorized as inconsi

82 he diagnosis of PDP and differentiation from pancreatic duct CA.

83 vated receptor 2 activating peptide into the pancreatic duct can activate and sensitize pancreas-spec

84 Rats were prepared with pancreatic duct cannulation, pyloric ligation, and bile

85 but patients in TPS group had more frequent pancreatic duct cannulation.

86 Kras signaling has been shown to accelerate pancreatic duct carcinogenesis, it is unclear whether el

87 1 cases), malignant melanoma (5 of 5 cases), pancreatic duct carcinoma (4 of 4 cases), non-small cell

88 Pancreatic duct carcinoma remains a common disease with

89 eoplasms, grade 3; P: < 0.001) and in 55% of pancreatic duct carcinomas (P: < 0.0001).

90 duct epithelium in primary culture and a rat pancreatic duct cell line but had no effect on a hamster

91 has been investigated in the cystic fibrosis pancreatic duct cell line, CFPAC-1.

92 In the mPEC pancreatic duct cell line, where endogenous pNBC1 functi

93 o be expressed in the majority of neoplastic pancreatic duct cell lines and VIP stimulates growth of

94 Ngn3-dependent differentiation by infecting pancreatic duct cell lines with an Ngn3-expressing adeno

95 Immunohistochemistry, however, showed that pancreatic duct cell-specific differentiation (carbonic

96 udied two models of Ngn3 activation in adult pancreatic duct cells (low-dose alloxan treatment and pa

97 del of branching morphogenesis using primary pancreatic duct cells and identified a transient surge o

98 tion of bicarbonate and fluid secretion from pancreatic duct cells and suggested that cholecystokinin

99 idence that the bestrophins are expressed in pancreatic duct cells and, more specifically, that hBest

100 To test whether pancreatic duct cells are in vitro progenitors, they wer

101 decisions in the pancreas, and reveals adult pancreatic duct cells as a latent multipotent cell type.

102 The three actions of secretin on guinea-pig pancreatic duct cells described in this and the accompan

103 In knockdown experiments with pancreatic duct cells exposed to UV light, RNF43 appeare

104 Percentages of nonviable pancreatic duct cells in groups 1 to 5 were 3.8+/-2.7%,

105 CO(3)(-) exchanger at the apical membrane of pancreatic duct cells is now known to be SLC26A6.

106 dence as to whether Ngn3 activation in adult pancreatic duct cells may lead to duct-to-beta cell tran

107 r to normal columnar epithelial cells, these pancreatic duct cells secreted mucin constitutively and

108 tely 90% methylated in microdissected normal pancreatic duct cells using bisulfite-modified sequencin

109 port of HCO3- across the luminal membrane of pancreatic duct cells was studied by monitoring the lumi

110 BC3.To measure cotransporter activity, mouse pancreatic duct cells were grown to confluence on a poro

111 ficient mice were transplanted with cultured pancreatic duct cells, but no donor-derived hepatocytes

112 ce were transplanted with cells enriched for pancreatic duct cells, but only three of the 34 (9%) rec

113 ajor role in the secretion of bicarbonate by pancreatic duct cells, by transporting bicarbonate into

114 proproliferative signaling pathways in human pancreatic duct cells, cAMP-protein kinase A and mitogen

115 cancer cells and spares immortalized normal pancreatic duct cells, hTERT-HPNE.

116 progenitor cells with similarities to mature pancreatic duct cells.

117 mitogen-activated protein kinase, and Src in pancreatic duct cells.

118 introns 2 and 18 in CFTR expression in some pancreatic duct cells.

119 hat pNBC1 is functionally expressed in mouse pancreatic duct cells.

120 We studied pNBC1 function in mouse pancreatic duct cells.

121 proliferation of epithelial cells as well as pancreatic duct cells.

122 nzyme of polyol metabolism, in Capan-1 human pancreatic duct cells.

123 FBR1-dependent ERK phosphorylation in benign pancreatic duct cells.

124 15 patients), "halo" (nine of 15 patients), pancreatic duct changes (15 of 15 patients), and distal

125 etion of p120 catenin displayed dilated main pancreatic ducts, chronic pancreatitis, acinar to ductal

126 planar reformations were generated along the pancreatic duct, common bile duct, and major mesenteric

127 sed a model of telomerase-immortalized human pancreatic duct-derived cells (E6/E7/st) to study mechan

128 The GSI inhibited the growth of premalignant pancreatic duct-derived cells in a Notch-dependent manne

129 ic features such as solid component and main pancreatic duct diameter >=10 mm also differed (solid co

130 nfidence interval (95% CI) 1.80-3.69], small pancreatic duct diameter (per mm increase, OR: 0.68, 95%

131 Median pancreatic duct diameter was 3 mm [interquartile range (

132 IPMN type and main pancreatic duct diameter were significant predictors of

133 variables included pancreatic gland texture, pancreatic duct diameter, intraoperative blood loss, pat

134 f enhanced solid component (P = 0.014), main pancreatic duct dilatation of more than 5 mm (P < 0.001)

135 h-risk features on initial imaging (eg, main pancreatic duct dilatation, a solid component, or mural

136 d for Das-1 in multivariate regression, main pancreatic duct dilation >5 mm (odds ratio, 14.98; 95% c

137 fidence interval, 2.63-108; P < .0012), main pancreatic duct dilation >=1 cm (odds ratio, 47.9; 95% c

138 ick walls (14.4 vs 5.6%, p = 0.001) and main pancreatic duct dilation (13.4 vs 5.6%, p = 0.004) were

139 age >65 years, preoperative size >2 cm, and pancreatic duct dilation were independently associated w

140 ncreatic cancers and not expressed in normal pancreatic duct displayed a high prevalence of hypomethy

141 ts with diabetes or in those patients with a pancreatic duct disruption.

142 ning the lumen of the hepatic biliary ducts, pancreatic ducts, epididymis, sweat ducts, colonic crypt

143 ormal pancreata or in an immortalized normal pancreatic duct epithelial (HPDE) cell line.

144 Expression of P5P6 in immortalized human pancreatic duct epithelial (HPDE) cells activates downst

145 ined but was expressed in immortalized human pancreatic duct epithelial (HPDE) cells.

146 We have established a primary pancreatic duct epithelial cell culture (PDEC) system to

147 ms and mucin secretion via exocytosis in dog pancreatic duct epithelial cells (PDEC).

148 2, on well-differentiated nontransformed dog pancreatic duct epithelial cells (PDEC).

149 Pancreatic duct epithelial cells (PDECs) have been shown

150 We further show that Shh protects pancreatic duct epithelial cells from apoptosis through

151 We generated architectures of human pancreatic duct epithelial cells in a 3-dimensional base

152 y be expressed on the majority of neoplastic pancreatic duct epithelial cells in vivo.

153 nsequent decrease in Na,K-ATPase activity in pancreatic duct epithelial cells, and that this response

154 hat Shh expression enhances proliferation of pancreatic duct epithelial cells, potentially through th

155 atic cancer cells compared with normal human pancreatic duct epithelial cells.

156 nce between expansion and differentiation of pancreatic duct epithelial progenitors.

157 50>1,000 ng/mL) was observed in normal human pancreatic duct epithelium cells, fibroblasts, and human

158 The pancreatic duct epithelium is remarkable for its capacit

159 In many species the pancreatic duct epithelium secretes HCO3- ions at a conc

160 BSTRACT: A computational model of guinea-pig pancreatic duct epithelium was developed to determine th

161 re expressed in the SAGE libraries of normal pancreatic duct epithelium, and were excluded from furth

162 is the first report of AR regulation in the pancreatic duct epithelium.

163 at these early stages in the progenitors of pancreatic ducts, exocrine and endocrine cells, rather t

164 Pancreatic duct exteriorization abrogated T/HS-induced g

165 ested by diversion of pancreatic enzymes via pancreatic duct exteriorization whereas the role of the

166 ciency, or mechanical drainage of obstructed pancreatic ducts for some patients.

167 ethod to micro dissect patient-derived human pancreatic ducts from pancreatic remnant cell pellets, f

168 Pancreatic duct glands (PDGs) are gland-like outpouches

169 Pancreatic duct glands (PDGs) have been hypothesized to

170 Pancreatic duct glands (PDGs) were recently described as

171 16a and miR-216b KO mice was the presence of pancreatic duct glands (PDGs).

172 t that is gathered in gland-like outpouches (pancreatic duct glands [PDG]) of major ducts and charact

173 Pancreatic ducts and pancreatic duct glands harbor committed pancreatic proge

174 Ex vivo explants of mouse pancreatic ducts grew dense cultures that costained for

175 atients with chronic pancreatitis, a dilated pancreatic duct (>/=5 mm) and moderate pain and/or frequ

176 SA 3 OR 0.59 [0.44-0.80]), whereas a dilated pancreatic duct (>3 mm) and pancreatic ductal adenocarci

177 It remains controversial whether adult pancreatic ducts harbor facultative beta cell progenitor

178 nesis or the budding of new islet cells from pancreatic ducts has been reported, but the existence an

179 rent histologic specimens (comprising normal pancreatic ducts, hyperplasia, low-grade dysplasia, high

180 atively noninvasive technique of biliary and pancreatic duct imaging.

181 ducts in six, diffuse irregular narrowing of pancreatic duct in nine, and focal stricture of proximal

182 uct in nine, and focal stricture of proximal pancreatic duct in six.

183 l, the mean luminal diameter and area of the pancreatic duct in the control group was significantly l

184 ere we show that such aggregates form inside pancreatic ducts in humans and mice occluding pancreatic

185 nIN-2 or PanIN-3 lesions, whereas 40% of the pancreatic ducts in the control animals had PanIN-2 or P

186 Ten percent of all pancreatic ducts in the nimesulide-fed animals showed Pa

187 Infusion of Na-taurocholate into the pancreatic duct induced necrotizing pancreatitis in the

188 ced model and a model elicited by retrograde pancreatic duct infusion of sodium taurocholate).

189 , immunoreactivity was also strong in normal pancreatic ducts, intestinal smooth muscle, and several

190 septation, calcification, mural nodularity, pancreatic duct involvement, and presence of multiple cy

191 Collagenase infusion into the pancreatic duct is an essential step in human islet isol

192 n the 5-8 mM range and its infusion into the pancreatic duct is commonly used to study pancreatitis.

193 This assessment is possible only when the pancreatic duct is visualized.

194 Expansion of pancreatic ducts is accompanied by apoptosis of neighbor

195 uctal papillary-mucinous tumor (IPMT) of the pancreatic ducts is increasingly recognized.

196 KRAS2 mutations were also detected in pancreatic duct juice from patients with pancreatic canc

197 Oncogenic transformation of pancreatic ducts led to two types of neoplastic growth:

198 These data support the hypothesis that pancreatic duct lesions are neoplastic and that they rep

199 of large numbers of morphologically defined pancreatic duct lesions.

200 arcinoma libraries but not in the two normal pancreatic duct libraries.

201 Here we used long-term partial pancreatic duct ligation (PDL) as a model to study CPRD.

202 In the adult, pancreatic duct ligation (PDL) caused facultative reacti

203 ing pancreatic macrophage infiltration after pancreatic duct ligation (PDL) completely inhibits beta-

204 Here, we demonstrate that pancreatic duct ligation (PDL) does not activate progeni

205 The effect of pancreatic duct ligation (PDL) on distant organ injury f

206 Pancreatic duct ligation in a minipig model leads to exo

207 ion of exocrine pancreatic insufficiency via pancreatic duct ligation in minipigs and the long term f

208 Pancreatic duct ligation in minipigs is an excellent met

209 olization of the exocrine pancreas gland and pancreatic duct ligation in minipigs.

210 crosis, in models of pancreatitis induced by pancreatic duct ligation in the opossum and rat.

211 tion after severe pancreas injury by partial pancreatic duct ligation or partial pancreatectomy.

212 14 Goettingen minipigs underwent pancreatic duct ligation via midline laparotomy for the

213 c duct cells (low-dose alloxan treatment and pancreatic duct ligation) and lineage-traced Ngn3-activa

214 From approx. 130 weeks post pancreatic duct ligation, all animals were supplemented

215 ured pancreas, or as islet progenitors after pancreatic duct ligation.

216 cysts of the pancreas and a non-dilated main pancreatic duct (<5 mm).

217 Well-preserved pancreatic ducts maintained good distribution of collage

218 the pancreatic duct system and therefore the pancreatic duct may be difficult to visualize, represent

219 Patients with pancreas divisum and a dilated pancreatic duct may be ideally suited for this surgical

220 s of GPR31 impairs pancreatic bud fusion and pancreatic duct morphogenesis.

221 ist symptoms, cyst size >30 mm, dilated main pancreatic duct (MPD) >6 mm, mural nodule (MN) and "posi

222 tment in main-duct IPMN patients with a main pancreatic duct (MPD) diameter of >/=10 mm.

223 ed IPMNs) of the pancreatic system is a main pancreatic duct (MPD) diameter of 5.0 mm or greater on c

224 patients with chronic pancreatitis and main pancreatic duct (MPD) dilatation (>7 mm).

225 on was defined as a new IPMN, increased main pancreatic duct (MPD) size, and increased size of an exi

226 as-specific morbidity and injury to the main pancreatic duct (MPD) was noted.

227 tic phase; 2D curved reformations along main pancreatic duct (MPD) were generated.

228 high-risk of malignancy associated with main pancreatic duct (MPD)-involved intraductal papillary muc

229 otent progenitor-like cells within the major pancreatic ducts (MPDs) of the human pancreas.

230 eatic mass (84 cystic, 3 solid) or a dilated pancreatic duct (n = 5) by any of the imaging modalities

231 n these six patients included an interrupted pancreatic duct (n = 5), dilated biliary and pancreatic

232 pancreatic duct (n = 5), dilated biliary and pancreatic ducts (n = 1), atrophic distal pancreatic par

233 evealed histological involvement of the main pancreatic duct not evident in preoperative imaging.

234 head, intractable abdominal pain, and a main pancreatic duct obstruction or stricture resulting in ab

235 as present (n = 10), or evidence of complete pancreatic duct obstruction was found ar ERCP (n = 7).

236 s study was to assess safety and efficacy of pancreatic duct occlusion (PDO) with neoprene-based glue

237 basal levels of TG2 compared with the normal pancreatic ducts [odds ratio (OR), 2.439; P = 0.012].

238 by retrogradely infusing bile acids into the pancreatic duct of anesthetized mice.

239 havioral pain response when infused into the pancreatic duct of awake rats.

240 4 cm (P < 0.001), presence of a dilated main pancreatic duct of over 4 mm (P < 0.001), histopathologi

241 were not significantly different for common pancreatic ducts of Px, sham Px, and unoperated rats and

242 yl tri-oleate) or OAEE was injected into the pancreatic ducts of rats, and local and systemic severit

243 We infused the radiocontrast agent into the pancreatic ducts of wild-type mice (C57BL/6) to create a

244 retrograde infusion of taurocholate into the pancreatic ducts of wild-type, NFAT luciferase reporter

245 adiologists considered that depiction of the pancreatic duct on CT images was not sufficient to enabl

246 /6 mice by infusion of taurocholate into the pancreatic duct or by intraperitoneal administration of

247 r data suggest that Dnmt1 is dispensable for pancreatic duct or endocrine cell formation, but not for

248 Thus, islet progenitor cells in adult pancreatic ducts or in isolated islets of Langerhans hav

249 most normal blood vessels or gain access to pancreatic ducts or intestinal smooth muscle.

250 eatic remnant (P = 0.41) nor ligation of the pancreatic duct (P > 0.05) affected the risk of clinical

251 veral methods can be used to help locate the pancreatic duct postendoscopic papillectomy (endoscopic

252 hat-consistent with theory predictions-small pancreatic ducts produced exophytic growth, whereas larg

253 nvasive epithelial proliferations within the pancreatic ducts, referred to as pancreatic intraepithel

254 ed, mouse epithelial cell lines derived from pancreatic duct, renal collecting duct, salivary gland a

255 of the fluid content within the lumen of the pancreatic ducts, responsible of an increase of MR signa

256 HCO(3)(-) secretion into sealed intralobular pancreatic ducts revealed that deletion of slc26a6 enhan

257 Impaired pancreatic duct secretion is frequently observed in insu

258 Stenting of the pancreatic duct should be strongly considered after bili

259 proliferation, and/or observations in large pancreatic ducts similar to those described in the liter

260 s important to identify and ligate accessory pancreatic ducts since persistence of accessory ducts wi

261 best 2-predictor model (body mass index and pancreatic duct size) resulted in a c-index of 0.748.

262 mass index, RPV, SAT/VAT area, SM area, main pancreatic duct size, and pancreatic gland texture was d

263 s in demographics, comorbidities, pathology, pancreatic duct size, pancreas texture, baseline quality

264 s in demographics, comorbidities, pathology, pancreatic duct size, pancreas texture, or operative tec

265 ethod of transection, suture ligation of the pancreatic duct, staple size, the use of staple line rei

266 l pancreatic endocrine neoplasms can produce pancreatic duct stenosis resulting in ductal dilatation

267 dvancements in endoscopic techniques such as pancreatic duct stenting and dye-free guidewire cannulat

268 Pancreatic duct stenting protects significantly against

269 Pancreatic duct stenting reduces the risk of developing

270 nist and clinically by use of diclofenac and pancreatic duct stenting.

271 hypertension were randomized to groups with pancreatic duct stents (n = 41) or no stents (n = 39) af

272 ous findings that neither the routine use of pancreatic duct stents decreases the rate of fistula for

273 d controlled trial has shown that the use of pancreatic duct stents is associated with less incidence

274 aving idiopathic pancreatitis complicated by pancreatic duct stones, we discuss the evaluation and tr

275 ncreatic endocrine neoplasms with associated pancreatic duct stricture had prominent stromal fibrosis

276 MRCP revealed pancreatic duct strictures in two and sclerosing cholang

277 phy (MRCP), because of an enlargement of the pancreatic duct system and an increase of the fluid cont

278 luid within the stomach may overlap with the pancreatic duct system and therefore the pancreatic duct

279 MRCP, which has the aim to depict the whole pancreatic duct system, the biliary tree, the major and

280 helial progression to full malignancy in the pancreatic duct system.

281 e gland-like outpouches budding off the main pancreatic ducts that function as a progenitor niche for

282 tic fistula include a soft pancreas, a small pancreatic duct, the underlying pancreatic pathology, th

283 In pancreatic ducts, the electrogenic sodium bicarbonate co

284 r, PDX-1 protein transduced into cultures of pancreatic ducts, thought to be islet progenitor cells,

285 1-mm diameter scope can be advanced into the pancreatic duct through a duodenoscope.

286 stinguish nonneoplastic from neoplastic main pancreatic duct tissue.

287 tic AFGPs enter the intestinal lumen via the pancreatic duct to prevent ingested ice from nucleating

288 ing to the type of resection and whether the pancreatic duct was dilated at the site of transection.

289 , because BrdU incorporation in cells of the pancreatic duct was not increased.

290 tation of the terminal portion of the dorsal pancreatic duct was observed unaccompanied by pancreas d

291 In cats, the main pancreatic duct was partially obstructed and the animals

292 Ligation of the pancreatic duct was performed in 22% of cases.

293 Nonneoplastic pancreatic ducts were also microdissected from tissues a

294 ditions used here, cells from isolated human pancreatic ducts were cultured under the same conditions

295 BMI, perirenal fat thickness, pancreatic duct width on computed tomography and at oper

296 ative score [using body mass index (BMI) and pancreatic duct width] to predict POPF was tested upon a

297 secretin improves noninvasive imaging of the pancreatic duct with magnetic resonance cholangiopancrea

298 Preinfusion of the pancreatic duct with proteinase-activated receptor 2-spe

299 Jagged1-deficient mice displayed malformed pancreatic ducts with resulting acinar cell death, fatty

300 US, and diffuse irregular narrowing of main pancreatic duct, with associated multiple biliary strict