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1 e cytokines IL-1beta and IL-13 or by porcine pancreatic elastase.
3 ficantly amplified after exposure to porcine pancreatic elastase, an emphysema model, and to oxidativ
5 le elastin following hydrolysis with porcine pancreatic elastase and human neutrophil elastase were p
7 derwent surgical AAA formation using porcine pancreatic elastase and received daily beta-aminopropion
8 re perfused with saline (control) or porcine pancreatic elastase and studied on postperfusion days 1,
10 ibroblast cultures were treated with porcine pancreatic elastase, and elastase-mediated changes in bF
15 d a broad range of proteases (neutrophil and pancreatic elastases, cathepsin G, subtilisin, and tryps
16 -function mutations in the gene encoding the pancreatic elastase chymotrypsin-like elastase family me
17 st other serine proteases, including porcine pancreatic elastase, chymotrypsin, and trypsin, was also
19 se-3, an elastase-like protease, and porcine pancreatic elastase demonstrated rapid inhibition rate c
20 -selected sequences as inhibitors of porcine pancreatic elastase demonstrates a significant differenc
28 the role of Olfr2 in AAA using PPE (porcine pancreatic elastase) infusion in Olfr2-deficient (Olfr2(
29 he application of the MSCS method to porcine pancreatic elastase is discussed, and comparison of the
30 hymotrypsin A alpha (Tyr, Pro, Trp), porcine pancreatic elastase (Leu/Ala, Arg, Ala), subtilisin Carl
33 ibitor of the serine proteases chymotrypsin, pancreatic elastase, neutrophil elastase, and trypsin.
34 but pPG3 was relatively resistant to porcine pancreatic elastase or human neutrophil cathepsin G.
35 Sprague-Dawley rats to intra-aortic porcine pancreatic elastase (PPE) (12 U/mL), AAA rupture was ind
36 (rShPI-1/K13L) exhibits a novel anti-porcine pancreatic elastase (PPE) activity together with a signi
37 nalysis of the complex between 1 and porcine pancreatic elastase (PPE) and subsequently the complex b
39 We previously showed that inhaled porcine pancreatic elastase (PPE) causes bronchoconstriction in
41 human neutrophil elastase (HNE) and porcine pancreatic elastase (PPE) complexed to peptidic ligands,
42 rine models of experimental AAA: the porcine pancreatic elastase (PPE) infusion model in C57BL/6 mice
43 se inhibitor, to the serine protease porcine pancreatic elastase (PPE) using isothermal titration cal
44 owing cleavage at the P1-P1' bond by porcine pancreatic elastase (PPE), as shown by the spontaneous f
46 proteinase inhibitor (alpha1PI) with porcine pancreatic elastase (PPE), which differs from the only o
48 Moreover, commercially available porcine pancreatic elastase preparations also activated Stx2d cy
50 Cleavage of the two proteins with porcine pancreatic elastase results in a 1.6- and 2.6-fold incre
51 trum inhibitor of trypsin, chymotrypsin, and pancreatic elastase that has previously been isolated fr
52 tion of elastin by human leukocyte elastase, pancreatic elastase, thermolysin, and Pseudomonas elasta
53 frarenal, intraluminal aorta to PPE (porcine pancreatic elastase) under pressure to induce aneurysmal
57 solvent crystal structures (MSCS) of porcine pancreatic elastase were used to map the binding surface