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1 y disease leading to pain, maldigestion, and pancreatic insufficiency.
2 ly characterized by neutropenia and exocrine pancreatic insufficiency.
3 r preschoolers aged 2 to 6 years with CF and pancreatic insufficiency.
4  the parenchyma, fibrosis, pancreatitis, and pancreatic insufficiency.
5 ects were children aged 6-8 y who had CF and pancreatic insufficiency.
6  is an excellent method of inducing exocrine pancreatic insufficiency.
7 rotein and mRNA levels, thus suggesting mild pancreatic insufficiency.
8 ion of patients with deltaF508 genotypes and pancreatic insufficiency.
9 these mice died within 3 weeks of birth from pancreatic insufficiency.
10 o progressive diabetes mellitus and exocrine pancreatic insufficiency.
11 ine laparotomy for the induction of exocrine pancreatic insufficiency.
12 y progressive pulmonary decline and exocrine pancreatic insufficiency.
13    Long-term problems include recurrence and pancreatic insufficiency.
14 ue to malabsorption associated with exocrine pancreatic insufficiency.
15  cause of severe infantile isolated exocrine pancreatic insufficiency.
16 nths): 27% of patients developed an exocrine pancreatic insufficiency, 45% developed or worsened a pr
17 greater than 5% for 74 phenotypes, including pancreatic insufficiency (67%), chorea (64%), atrial sep
18  the genders in these parameters, as well as pancreatic insufficiency, age at diagnosis, mode of pres
19  direct and indirect costs, quality of life, pancreatic insufficiency, alternative pain scales, lengt
20 inherited disorder characterized by exocrine pancreatic insufficiency and bone marrow failure.
21 late had a significantly lower prevalence of pancreatic insufficiency and CF-related diabetes, and be
22 recessive disorder characterized by exocrine pancreatic insufficiency and hematopoietic dysfunction,
23 ence of epilepsy and the absence of exocrine pancreatic insufficiency and hypoplasia of nasal alae.
24 me sequencing data on 4,248 unique pwCF with pancreatic insufficiency and lung function measures were
25  5-10 y with CF, mild pulmonary disease, and pancreatic insufficiency and of 26 healthy control child
26 or (CFTR) gene mutations are associated with pancreatic insufficiency and pancreatitis.
27                                              Pancreatic insufficiency and quality of life did not dif
28 ctal metaplasia occur, culminating in marked pancreatic insufficiency and the development of pancreat
29 inct from JBS due to the absence of exocrine pancreatic insufficiency and the presence of autism, epi
30 llow-up period, especially the children with pancreatic insufficiency and those who were homozygous f
31 infection, in combination with correction of pancreatic insufficiency and undernutrition by multidisc
32 rioperative morbidity, a substantial risk of pancreatic insufficiency, and may overtreat noninvasive
33        No patients had endocrine or exocrine pancreatic insufficiency, and none required chronic anal
34 ncreaticocutaneous fistula, reinterventions, pancreatic insufficiency, and quality of life.
35 tive management, a determination of exocrine pancreatic insufficiency are scarce in the literature.
36 enital onset with sequelae of early exocrine pancreatic insufficiency as in the diseases of cystic fi
37 ciency and subjects with cystic fibrosis and pancreatic insufficiency, as compared with values in hea
38 ltisystem disorder characterized by exocrine pancreatic insufficiency, bone marrow dysfunction, and m
39 rospectively analyzed 3,328 CF patients with pancreatic insufficiency born after 1985 and recruited i
40 me assembly and are associated with exocrine pancreatic insufficiency, chronic neutropenia and skelet
41  the risk of new-onset diabetes and exocrine pancreatic insufficiency compared with distal pancreatec
42 intestinal conditions, particularly given in pancreatic insufficiency, could threaten digestibility.
43 c colitis, steatorrhea secondary to exocrine pancreatic insufficiency, dietary lactose or fructose ma
44 tants reported to be associated with CF with pancreatic insufficiency do not support HCO3- transport,
45 zing inflammatory disease that can result in pancreatic insufficiency due to acinar atrophy and fibro
46  in the use of diagnostic tests for exocrine pancreatic insufficiency (EPI) confound interpretation o
47                                     Exocrine pancreatic insufficiency (EPI) stems from a deficiency o
48 igation in a minipig model leads to exocrine pancreatic insufficiency (EPI).
49 types from an additional 3,592 patients with pancreatic insufficiency from the United States, Canada,
50                       For 1010 subjects with pancreatic insufficiency, genome-wide significant linkag
51  BMI-z(5to10) was negatively correlated with pancreatic insufficiency, history of meconium ileus, and
52 gmus, progressive cholestatic liver disease, pancreatic insufficiency, hypoglycemia, anemia, intermit
53 ts leading to fibrotic injury and ultimately pancreatic insufficiency in 85% of the CF population.
54                                              Pancreatic insufficiency in cystic fibrosis (CF), even w
55  and acinar cell loss may be responsible for pancreatic insufficiency in Jagged1-deficient mice and,
56 tional bowel disorders, microscopic colitis, pancreatic insufficiency, inflammatory bowel disease, la
57 to 6 years (mean age, 3.8 years) with CF and pancreatic insufficiency (intervention, n = 36 and contr
58                    Cystic fibrosis (CF) with pancreatic insufficiency is associated with poor absorpt
59                  Treatment of human exocrine pancreatic insufficiency is suboptimal.
60                                    Male sex, pancreatic insufficiency, meconium ileus, histamine bloc
61 ltisystem disorder characterized by exocrine pancreatic insufficiency, multilineage hematopoietic dys
62 ng was available in 4082 people with CF with pancreatic insufficiency (n = 516 with severe CFLD; n =
63 >/=1 mo, and 38% of infants were ExFM22+) or pancreatic insufficiency (n = 70; 25% of infants were Ex
64 ticocutaneous fistula, exocrine or endocrine pancreatic insufficiency, need for additional radiologic
65  syndrome (SDS) is characterized by exocrine pancreatic insufficiency, neutropenia, and a high risk o
66 king cessation, pain control, replacement of pancreatic insufficiency, or mechanical drainage of obst
67        Persons with cystic fibrosis (CF) and pancreatic insufficiency (PI) are at risk of vitamin A d
68                 Pulmonary function, exocrine pancreatic insufficiency (PI), and CF-related diabetes (
69 among children with cystic fibrosis (CF) and pancreatic insufficiency (PI).
70                           Risk of transplant pancreatic insufficiency, posttransplant lymphoprolifera
71 ting in chronic pain, exocrine and endocrine pancreatic insufficiency, reduced quality of life, and a
72 orption of fat and nitrogen in patients with pancreatic insufficiency related to cystic fibrosis.
73 is may be underappreciated, and both gut and pancreatic insufficiency represent modifiable targets in
74 sulin-treated neonatal diabetes and exocrine pancreatic insufficiency requiring enzyme replacement th
75        Two of the four patients had exocrine pancreatic insufficiency requiring replacement therapy b
76 ice are viable but have defects that include pancreatic insufficiency, similarly to UBR1-/- human pat
77 K status in subjects aged 8-25 y with CF and pancreatic insufficiency taking various vitamin K supple
78 nd adolescents with cystic fibrosis (CF) and pancreatic insufficiency, the efficacy of routine vitami
79 ille syndrome patients also display exocrine pancreatic insufficiency, the pathobiology of which is u
80 mental approach to the induction of exocrine pancreatic insufficiency via pancreatic duct ligation in
81 yvitamin D in 10 adults with CF and exocrine pancreatic insufficiency was compared with that of 10 he
82                                              Pancreatic insufficiency was managed with nutritional in
83                                     Exocrine pancreatic insufficiency was successfully induced in 12
84                              In infants with pancreatic insufficiency, weight z scores declined from
85               We observed endocrine/exocrine pancreatic insufficiency, which had not previously been
86                    Cftr (-/-) mice have mild pancreatic insufficiency, which partially explains the b
87 or (CFTR) gene typically cause pulmonary and pancreatic insufficiency while rarely causing pancreatit