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1 sles virus, the cause of subacute sclerosing panencephalitis.
2 patient who succumbed to subacute sclerosing panencephalitis, a rare but lethal consequence of measle
3 sist in the central nervous system causing a panencephalitis and should be considered in patients wit
4 ortality (eg, death from subacute sclerosing panencephalitis), and studies that did not include count
6 brain of a patient with subacute sclerosing panencephalitis, and single-cell RT-PCR was used to anal
7 es with MS and 1 case of subacute sclerosing panencephalitis, but not in IgG from noninflamed control
8 asles virus (MV)-induced subacute sclerosing panencephalitis in children, it is unclear whether the v
9 patient who succumbed to subacute sclerosing panencephalitis (SSPE) about 20 years after acute measle
15 ses, MeV persistence and subacute sclerosing panencephalitis (SSPE) occur even in the face of an inta
16 ands in diseases such as subacute sclerosing panencephalitis (SSPE) or cryptococcal meningitis have b
17 GBs) in diseases such as subacute sclerosing panencephalitis (SSPE) or cryptococcal meningitis have b
18 RNA virus, rarely causes subacute sclerosing panencephalitis (SSPE) several years after acute infecti
19 he brain of a human with subacute sclerosing panencephalitis (SSPE), a chronic encephalitis caused by
20 asles virus infection is subacute sclerosing panencephalitis (SSPE), a fatal disease of the central n
21 tion of the brain causes subacute sclerosing panencephalitis (SSPE), a progressive, relentless fatal
22 mples from patients with subacute sclerosing panencephalitis (SSPE), multiple sclerosis (MS), or othe
23 generative complication, subacute sclerosing panencephalitis (SSPE), occurs during persistent MV infe
28 n be paucisymptomatic or may manifest severe panencephalitis that evolves to a minimally conscious st
29 iciently neuroinvasive, and produces a fatal panencephalitis, whereas low-dose SFV4 produces a low-ti