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1 osomal rearrangements found in radio-induced papillary thyroid carcinoma.
2 lops distant metastases more frequently than papillary thyroid carcinoma.
3 thrombosis is an extremely rare condition in papillary thyroid carcinoma.
4 Of the 93 patients, 57 (61%) had papillary thyroid carcinoma.
5 by the active RET mutant, RET/PTC1, found in papillary thyroid carcinoma.
6 tic factor for the metastatic progression of papillary thyroid carcinoma.
7 BRAF gene mutation is frequently detected in papillary thyroid carcinoma.
8 iration of the mass suggested a diagnosis of papillary thyroid carcinoma.
9 stologic and cytologic similarities to human papillary thyroid carcinoma.
10 uced iodide uptake ability observed in human papillary thyroid carcinoma.
11 he peripheral blood of only one patient with papillary thyroid carcinoma.
12 pothesis that RET/PTC is sufficient to cause papillary thyroid carcinomas.
13 his oncogene has been detected only in human papillary thyroid carcinomas.
14 especially in BRAF-mutated and MET-addicted papillary thyroid carcinomas.
15 ngements are one of the genetic hallmarks of papillary thyroid carcinomas.
16 of the RET tyrosine kinase commonly seen in papillary thyroid carcinomas.
17 re believed to be tumor-initiating events in papillary thyroid carcinomas.
18 of the RET proto-oncogene detected in human papillary thyroid carcinomas.
19 d three different somatic mutations (23%) in papillary thyroid carcinomas.
20 s, are frequently found in radiation-induced papillary thyroid carcinomas.
21 as been reported to be associated with human papillary thyroid carcinomas.
22 revalent in radiation-induced post-Chernobyl papillary thyroid carcinomas.
23 is aberrant methylation is evident in 83% of papillary thyroid carcinoma, 11% of follicular thyroid c
25 patients were more likely to have tall-cell papillary thyroid carcinoma (6 [55%] vs 13 [18%]), multi
26 thyroid carcinomas, 13 follicular variant of papillary thyroid carcinomas, 9 follicular thyroid carci
27 tested in TPC-1 and KM12 cells, derived from papillary thyroid carcinoma and colorectal carcinoma, an
28 of cutaneous melanomas, cutaneous nevi, and papillary thyroid carcinoma and in a small fraction of o
30 e length were found in follicular variant of papillary thyroid carcinomas and follicular adenomas.
31 ion protein which is frequently expressed in papillary thyroid carcinomas and has been detected in th
32 active protein, not only in FTCs but also in papillary thyroid carcinomas and Hurthle cell carcinomas
33 (55 FA, 27 follicular thyroid carcinomas, 35 papillary thyroid carcinomas, and 22 undifferentiated th
34 reviously been implicated in pathogenesis of papillary thyroid carcinoma as a fusion partner of RET.
35 st commonly detected BRAF mutations in human papillary thyroid carcinomas (BRAF(V600E)) in thyroid fo
36 s expressed at high levels in differentiated papillary thyroid carcinomas but at low levels in anapla
37 ll lines and samples of human follicular and papillary thyroid carcinoma by reverse transcriptase-pol
38 region shared by FOXE1 and PTCSC2 in a human papillary thyroid carcinoma cell line (KTC-1) and unaffe
41 ransforming growth factor-beta1 (TGF-beta1), papillary thyroid carcinoma cells acquired increased can
42 pecificity of 96.2% (3 follicular variant of papillary thyroid carcinomas clustered with the benign l
45 mphoma, chronic myelomonocytic leukaemia and papillary thyroid carcinoma described similar rearrangem
47 n, and that PRRX1 plays an important role in papillary thyroid carcinoma EMT and disease progression.
49 d tumor DNA from 17 pediatric post-Chernobyl papillary thyroid carcinomas for mutations at three diff
53 d a remarkable increase in radiation-induced papillary thyroid carcinoma in children and young adults
59 d demonstrate that alternative exon usage in papillary thyroid carcinomas is restricted primarily to
60 ons drive differential pathway activation in papillary thyroid carcinomas, leading to different tumor
61 patients thyroidectomized for follicular or papillary thyroid carcinoma may represent the thymus.
62 ovel strategies to prevent or to treat human papillary thyroid carcinomas, MEN 2 disease, as well as
63 X) models (five colorectal carcinoma and two papillary thyroid carcinoma models) with different KRAS,
64 MDS, and both solid tumors (osteosarcoma and papillary thyroid carcinoma) occurred in recipients of D
66 epithelial cells were microinjected with the papillary thyroid carcinoma oncogene (RET/PTC1 short iso
71 ological examination of the mass confirmed a papillary thyroid carcinoma (PTC) and enlarged metastati
72 es of nonmedullary thyroid carcinoma, namely papillary thyroid carcinoma (PTC) and follicular thyroid
73 protein RET/PTC3 (RP3) that is expressed in papillary thyroid carcinoma (PTC) and thyroid epithelia
74 neoplasms commonly found among adults, with papillary thyroid carcinoma (PTC) being the most prevale
75 observed that 63 of 110 (57%) human primary papillary thyroid carcinoma (PTC) cases expressed nuclea
77 MTC and follicular tumours resembling human papillary thyroid carcinoma (PTC) depending on the found
81 mph node metastasis (LNM) from N1b to N1a in papillary thyroid carcinoma (PTC) has sparked debate due
82 Many patients undergoing thyroidectomy for papillary thyroid carcinoma (PTC) have subclinical nodal
83 nobyl nuclear power plant accident increased papillary thyroid carcinoma (PTC) incidence in surroundi
94 variants associated with the development of papillary thyroid carcinoma (PTC) that can be used to co
95 gy (TCM) is a rare and aggressive variant of papillary thyroid carcinoma (PTC) that has been associat
96 Approximately 40 percent of patients with papillary thyroid carcinoma (PTC) typically have either
97 Historically, only 40% to 50% of childhood papillary thyroid carcinoma (PTC) were known to be drive
99 600E) is the most common somatic mutation in papillary thyroid carcinoma (PTC), how it induces tumor
108 ral soluble factors that were induced by RET/papillary thyroid carcinoma (PTC)3 gene expression inclu
111 sequencing and expression analysis of eight papillary thyroid carcinomas (PTC) to comprehensively ch
117 prognostication and treatment selection for papillary thyroid carcinomas (PTCs) do not uniformly pre
119 95 sporadic thyroid tumors, of which 39 were papillary thyroid carcinomas (PTCs), 12 were follicular
120 occurs in various thyroid neoplasms such as papillary thyroid carcinomas (PTCs), follicular thyroid
121 is the most common endocrine malignancy, and papillary thyroid carcinoma represents the most common t
122 tive activation of the RET proto-oncogene in papillary thyroid carcinomas results from rearrangements
123 embers of the rearranged during transfection/papillary thyroid carcinoma (RET/PTC) fusion oncogene fa
124 ing RET chimeric oncoproteins found in human papillary thyroid carcinomas (RET/PTC) as well as RET on
125 ess risk, 15.27 per 10 000 person-years) and papillary thyroid carcinoma (SIR, 5.26 [95% CI, 3.25-8.0
126 tergenic, noncoding RNA gene (lincRNA) named Papillary Thyroid Carcinoma Susceptibility Candidate 3 (
128 id-targeted expression of ret/PTC1 developed papillary thyroid carcinomas that were minimally invasiv
130 s strongly correlated with the generation of papillary thyroid carcinomas, the most prevalent maligna
131 ssed cDNA fragments were isolated from human papillary thyroid carcinoma tissues, among them a cDNA w
132 To gain insight into the pathogenesis of papillary thyroid carcinoma, transcriptional profiles of
134 In this cohort study among patients with papillary thyroid carcinomas up to 4 cm, ATA guideline c
138 ents with oncologic diseases (breast cancer, papillary thyroid carcinoma) were injected intravenously
139 nscripts were associated with follicular and papillary thyroid carcinomas, whereas long telomeres and
141 HB/SDHD mutation carriers have renal cell or papillary thyroid carcinomas, which are also CS-related
142 es and transcriptomes of pediatric and adult papillary thyroid carcinoma, with implications for under
143 mutations in p53 were found by SSCP in 2/33 papillary thyroid carcinomas, with one missense mutation