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1 ent L1 isoforms during the natural course of papillomavirus infection.
2           Cervical cancer is caused by human papillomavirus infection.
3 ctrochemical signal corresponded to a bovine papillomavirus infection.
4  immunoregulatory control of oncogenic human papillomavirus infection.
5  surface receptor(s) is a subsequent step of papillomavirus infection.
6 hat recapitulates the establishment phase of papillomavirus infection.
7 n papillomaviruses in the context of in vivo papillomavirus infection.
8  represents the epithelial response to human papillomavirus infection.
9 ounds that might block the initial stages of papillomavirus infection.
10 pproval of vaccines to prevent initial human papillomavirus infection.
11  year post-BMT, 71% developed chronic canine papillomavirus infection.
12 ins have been shown to protect against human papillomavirus infection.
13 n against E2 protein using a rabbit model of papillomavirus infection.
14  effective strategy for immunoprophylaxis of papillomavirus infection.
15 human anogenital carcinomas show evidence of papillomavirus infection.
16 n globally, often linked to persistent human papillomavirus infection.
17 eled to recognize the signs of genital human papillomavirus infection.
18 sen rapidly, because of an epidemic of human papillomavirus infection.
19 us SCC, given their common etiology in human papillomavirus infection.
20 ctions with molluscum contagiosum; and human papillomavirus infections.
21 keratin 17, which promotes immune evasion in papillomavirus infections.
22                                        Human papillomavirus infection accounts for APOBEC3B upregulat
23 es for the prevention and treatment of human papillomavirus infection and associated diseases.
24  a role is proposed for NK function in human papillomavirus infection and cervical neoplasia.
25 nteraction is predicted to impede persistent papillomavirus infection and consequently provides a nov
26 irreversible over time with persistent human papillomavirus infection and genetic damage.
27 ighly active antiretroviral therapy on human papillomavirus infection and its related anogenital abno
28 be associated with lower genital tract human papillomavirus infection and may have implications in th
29  high total cholesterol, hypertension, human papillomavirus infection and related pre-cancerous stage
30 s have reported an association between human papillomavirus infection and subsequent cervical dysplas
31  healthy participants at risk for oral human papillomavirus infection and the agreement with oral rin
32 inue to confirm the high prevalence of human papillomavirus infection and to define the different hum
33 ccine platforms for both prevention of human papillomavirus infection and treatment of associated dis
34                              High-risk human papillomavirus infections and abnormalities on cytologic
35                                        Human papillomavirus infections and human papillomavirus-assoc
36 pendent signaling pathway in the response to papillomavirus infections and the progression of these v
37 tals, and environmental tobacco smoke, human papillomavirus infection, and inherited genetic suscepti
38  high total cholesterol, hypertension, human papillomavirus infection, and related precancerous stage
39 iabetes, hypertension, hyperlipidemia, human papillomavirus infection, and tobacco use disorder using
40 ighly active antiretroviral therapy on human papillomavirus infection; and novel human papillomavirus
41                                        Human papillomavirus infections are associated with most cervi
42  experimental data implicate cutaneous human papillomavirus infection as co-factor in the development
43 viral therapy era, raising concerns of human papillomavirus infections as a rising health burden amon
44 al neutralizing antibody can protect against papillomavirus infection at both cutaneous and mucosal s
45 ody (MPV.A4) by passive immunization against papillomavirus infections at both cutaneous and mucosal
46 third of children will have persistent human papillomavirus infection beyond 2 years.
47              Given the possible link between papillomavirus infection, bracken fern in the diet and c
48 nome integration.IMPORTANCE Persistent human papillomavirus infections can cause a variety of signifi
49                                The number of papillomavirus infections detected was greater among His
50      The study furthers our understanding of papillomavirus infection dynamics and provides potential
51 n of the association of cervical cancer with papillomavirus infection encouraged development of a vac
52 .9% (95% CI: 24.3-43.5%) for high-risk human papillomavirus infection from cervical samples and 78.6%
53 n cleavage of minor capsid protein L2 during papillomavirus infection has been difficult to directly
54 evious studies examining the pathogenesis of papillomavirus infections have relied on the use of orga
55 amous cell carcinoma (HNSCC) caused by human papillomavirus infection (HPV(+)), patients have transcr
56      The model incorporated states for human papillomavirus infection (HPV), low- and high-grade squa
57 maviruses are widely used as models to study papillomavirus infection in humans despite differences i
58 widespread testing for the presence of human papillomavirus infection in patients with HNSCC.
59 alizing antibody can protect against diverse papillomavirus infections in a time-dependent manner in
60 s (MmuPV1) provides the opportunity to study papillomavirus infections in vivo in the context of a co
61                         In addition to human papillomavirus infection, increased bcl-2 expression and
62                                        Human papillomavirus infection is associated with a higher ris
63                             Persistent human papillomavirus infection is associated with squamous cel
64                                   Most human papillomavirus infection is harmless and clears spontane
65                                        Human papillomavirus infection is one of the most common sexua
66                          However, productive papillomavirus infection is species- and tissue-restrict
67                             Persistent human papillomavirus infection is the central cause of cervica
68            The viral early-to-late switch of papillomavirus infection is tightly linked to keratinocy
69  mycobacterial, bacterial, fungal, and human papillomavirus infections, lymphedema, pulmonary alveola
70                                              Papillomavirus infection normally involves virion bindin
71             In this study, cottontail rabbit papillomavirus infection of domestic rabbits was used as
72 fidence interval (CI): 2.4, 23.2), any human papillomavirus infection (OR = 5.8; 95% CI: 3.3, 10.0),
73  included HIV-1 infection (p = 0.013), human papillomavirus infection (p=0.0013), lower CD4 T lymphoc
74 ytological assay were used to look for human papillomavirus infection, p53 mutations, loss of heteroz
75 his is analogous to the late-onset cutaneous papillomavirus infection recently described for human XS
76                             Anogenital human papillomavirus infection remains highly prevalent and pe
77 tigated the extent to which animal models of papillomavirus infection resemble human disease by compa
78 ical features, including age, sex, and human papillomavirus infection status, were collected for a ba
79 hain reaction-based tests for cervical human papillomavirus infection, tests for gonorrhea and chlamy
80 uggest that the natural progression of human papillomavirus infection, the causal agent in all cervic
81                                       During papillomavirus infection, the E5 protein localizes in th
82                            During persistent papillomavirus infection, the viral E2 protein tethers t
83                      During persistent human papillomavirus infection, the viral genome replicates as
84                                           In papillomavirus infections, the viral genome is establish
85  differences in the natural history of human papillomavirus infections; the effect of highly active a
86  main risk factor for anal cancer is a human papillomavirus infection; those with chronic immunosuppr
87                                        Human papillomavirus infection was the most significant risk f
88 tigate the potential effect of EVER genes on papillomavirus infection, we pursued in vivo infection s
89       Gastroesophageal acid reflux and human papillomavirus infection were ruled out.
90                           Herpesvirus and/or papillomavirus infections were observed in 71%, malignan
91      Interventions aimed at preventing human papillomavirus infections with vaccinations need to be e