1 We now formally demonstrate by
parabiotic,
adoptive transfer, and developmental studies
2 ants, explained this increase, as seen using
parabiotic and adoptive transfer models.
3 ls generated the entire spectrum of ILC2s in
parabiotic and shield chimeric mice, consistent with the
4 ously used selective combinations of normal,
parabiotic,
and radioablated mice to demonstrate that bl
5 Shared circulation between the
parabiotic animals was confirmed by Evans blue dye injec
6 To prevent immune reaction between the
parabiotic animals, all mice were bred to be Rag1(-/-).
7 f injury-exposed neutrophils to permeate the
parabiotic barrier and induce granulopoiesis in noninfar
8 Introducing a time-dependent
parabiotic barrier to the free movement of neutrophils i
9 rns of different blood cell types across the
parabiotic barrier.
10 mice in which remyelination was enhanced by
parabiotic coupling to a younger mouse and in multiple s
11 Parabiotic experiments are still used and were recently
12 Using a blood-perfused,
parabiotic,
isolated rabbit heart model, 45 hearts under
13 epair by generating chimeric animals through
parabiotic joining of wild-type (wt) and diabetic (db/db
14 migration of endogenous DCs to the thymus in
parabiotic mice and after painting mouse skin with fluor
15 Furthermore,
parabiotic mice and BM chimeras of nonirradiated recipie
16 ether with congenic bone marrow chimeras and
parabiotic mice as tools to differentiate LC- and blood-
17 We next induced MI in
parabiotic mice by permanent ligation of the left anteri
18 Experiments in normal, radioablated, and
parabiotic mice document the cyclical accumulation (3-5
19 Experiments using
parabiotic mice fed a high-fat diet (HFD) showed differe
20 most mucosal organs, surgical separation of
parabiotic mice revealed a tissue-resident provenance fo
21 Analysis of
parabiotic mice revealed that adipose ILC1s maintained l
22 Consequently, the observations on the
parabiotic mice support the notion that PXE is a metabol
23 This study was designed to test, using
parabiotic mice that were joined surgically, whether ste
24 Parabiotic mice were generated by surgically joining wil
25 Parabiotic mice were generated surgically by joining gre
26 Parabiotic mice were made by surgically linking C3(-/-)
27 significantly in the ischemic myocardium of
parabiotic mice with hepatectomy (0 0%, 0.1 0.1%, 0.3 0.
28 FP, were found in the ischemic myocardium of
parabiotic mice with intact liver (0.2 0.1%, 1.1 0.3%, 2
29 In
parabiotic mice with separate organs but shared blood ci
30 In
parabiotic mice with separate organs, but a shared blood
31 These kinetics were confirmed in
parabiotic mice, and in cohorts of mice in whom gating w
32 We created
parabiotic mice, joining ROSA26 and PeP3b animals, to st
33 Using
parabiotic mice, we demonstrated that, despite their int
34 Using
parabiotic mice, we determined that, after mobilization
35 Similarly, in GFP+:GFP-
parabiotic mice, we found substantial chimerism of hemat
36 Using
parabiotic mice, we further show that circulating CD8(+)
37 Here, by using
parabiotic mice, we show that a preexisting pool of CD4
38 of blood-borne HSCs using genetically marked
parabiotic mice, which are surgically conjoined and shar
39 ion, and did not equilibrate among immunized
parabiotic mice.
40 2 in both reconstitution kinetic studies and
parabiotic mice.
41 pair of ischaemic myocardium using GFP+-GFP-
parabiotic mice.
42 ocytes was determined by timed separation of
parabiotic mice.
43 used a model of vaccination and challenge of
parabiotic mice.
44 mice, together with bone marrow chimera and
parabiotic models, we found that embryonic precursor cel
45 A
parabiotic mouse model was generated by surgical skin-un
46 ertility, we established transplantation and
parabiotic mouse models to assess the capacity of circul
47 ralization factor(s) in circulation, we used
parabiotic pairing, ie, surgical joining of two mice, to
48 le pathology and microenvironment is through
parabiotic pairing, where mice are surgically sutured to
49 ells in the blood of mdx mice) 2-weeks after
parabiotic pairing.
50 tor cells from these tissues, we established
parabiotic pairings (that is, a shared circulatory syste
51 the circulation into tumors was confirmed in
parabiotic pairs of COL-EGFP mice and transgenic mice de
52 Intriguingly, in
parabiotic pairs of two WT mice, leptin infusion into on
53 r, leptin treatment resulted in death of the
parabiotic pairs.
54 OL-EGFP(+) cells in tumors developing in the
parabiotic partner lacking the fluorescent reporter.
55 ns of blood-derived monocytes from the young
parabiotic partner, and preventing this recruitment part
56 Th2 cells recirculate and seed the lung of a
parabiotic partner, conferring susceptibility to OVA cha
57 ocytes and progenitor cells derived from the
parabiotic partner, suggesting splenic progenitor cells
58 later; 10.1% of marrow HSCs derived from the
parabiotic partner.
59 they arise and in the gonads of the natural
parabiotic partners.
60 old regenerating muscle of the heterochronic
parabiotic partners.
61 eria: 1) inefficient (<20%) exchange between
parabiotic partners; 2) gated importation by the thymus;
62 Using a blood-perfused
parabiotic rabbit heart Langendorff model, myocardial ox
63 this historic study, Kurt R. Reissmann used
parabiotic rats to demonstrate the functional existence
64 /ob) confirm hypotheses generated from early
parabiotic studies that suggested the existence of a cir
65 Based on
parabiotic surgery of mice receiving systemic viral infe
66 he blood of mice overexpressing ASM, through
parabiotic surgery.
67 Using time-lapse confocal imaging,
parabiotic surgical pairing of zebrafish embryos, and bl
68 lized a combination of genetic fate mapping,
parabiotic,
transcriptional, and functional analyses and
69 rohemorrhage were found in the brains of the
parabiotic wild-type mice.
70 g-term potentiation was markedly impaired in
parabiotic wild-type mice.
71 Finally, using
parabiotic zebrafish, we show that cxcr1 acts HSPC nonau