ライフサイエンスコーパス: paracentral

1 , paracentral, or mixed (both peripheral and paracentral).

2 defined by type of VF defect (peripheral or paracentral).

3 follows: (1) attached central; (2) attached paracentral; (3) detached apex; and (4) detached base.

4 reference between central (0-3 degrees ) and paracentral (4-8 degrees ) V1 in the prosimian bush baby

5 An arrangement of two elliptical paracentral ablation islands, deep inferior and shallow

6 face OCT appearance may be used to classify paracentral acute maculopathy into distinct subtypes.

7 Paracentral acute middle maculopathy (PAMM) and retinal

8 to bilateral Purtscher-like retinopathy and paracentral acute middle maculopathy (PAMM) following a

9 cent retinal vascular occlusion illustrating paracentral acute middle maculopathy (PAMM) in a periven

10 tudy is to assess the diagnostic accuracy of paracentral acute middle maculopathy (PAMM) in the setti

11 Paracentral acute middle maculopathy (PAMM) is an optica

12 uctural OCT changes including retinal fluid, paracentral acute middle maculopathy (PAMM) lesion, and

13 cal coherence tomography (SD-OCT) finding of paracentral acute middle maculopathy (PAMM) that can be

14 To report a case of paracentral acute middle maculopathy (PAMM) that progres

15 4 mm(-1) vs 14.7 +/- 3.5 mm(-1) in eyes with paracentral acute middle maculopathy (reduction -19.4%,

16 8 mm(-1) vs 12.1 +/- 1.9 mm(-1) in eyes with paracentral acute middle maculopathy (reduction -6.0%, P

17 Thin retinas had significantly more paracentral acute middle maculopathy and were more likel

18 ciations leading to retinal vasculopathy and paracentral acute middle maculopathy include eye compres

19 of 9 patients (10 eyes) from 5 centers with paracentral acute middle maculopathy lesions and previou

20 Paracentral acute middle maculopathy lesions correspond

21 Follow-up SD OCT analysis of these paracentral acute middle maculopathy lesions demonstrate

22 Paracentral acute middle maculopathy lesions may develop

23 Paracentral acute middle maculopathy may be best evaluat

24 Paracentral acute middle maculopathy may precede macular

25 Paracentral acute middle maculopathy may represent a nov

26 Paracentral acute middle maculopathy refers to character

27 , and en face OCT images of 16 patients with paracentral acute middle maculopathy were evaluated.

28 imodal imaging findings from 8 patients with paracentral acute middle maculopathy were reviewed and a

29 with acute macular neuroretinopathy, 1 with paracentral acute middle maculopathy, and 1 with subreti

30 d type 1 SD-OCT lesions, also referred to as paracentral acute middle maculopathy, and 5 eyes (4 pati

31 he middle retinal layers, otherwise known as paracentral acute middle maculopathy, were observed in a

32 resembling other acute maculopathies such as paracentral acute middle maculopathy.

33 ere evaluated for the presence of coexisting paracentral acute middle maculopathy.

34 5 (5.2%) demonstrated evidence of concurrent paracentral acute middle maculopathy.

35 hinal cortex or the mediodorsal (MDn) or the paracentral and centrolateral (PC-CL) thalamic nuclei di

36 ticipants, with regional decreases involving paracentral and occipital regions in both PD + VH and PD

37 For identifying FECD, average En-DMT of paracentral and peripheral regions achieved 94% sensitiv

38 Compared with control participants, both paracentral and peripheral VF loss groups showed reduced

39 scicular, oval paracentral, central lateral, paracentral, and central medial nuclei), as well as the

40 erodorsal, paraventricular, central lateral, paracentral, and central medial nuclei.

41 scicular, central lateral, paracentral, oval paracentral, and central medial nuclei; in the midline t

42 dentified patterns included partial arcuate, paracentral, and nasal step defects, and the most preval

43 Using 3D thickness maps, central, paracentral, and peripheral En/DMT achieved 100% sensiti

44 Moreover, central, paracentral, and peripheral En/DMT correlated significan

45 The average En-DMT of central, paracentral, and peripheral regions was correlated highl

46 d into 5 clusters: nasal, temporal, central, paracentral, and peripheral.

47 nferior parietal lobule, and also cingulate, paracentral, and precentral gyri, compared with the non-

48 terior medial parvicellular, caudal PC, oval paracentral, and reticular thalamic nuclei.

49 t typically started in the peripapillary and paracentral areas with foveal sparing.

50 In the central and paracentral areas, however, significant numbers of CD11c

51 While central and paracentral axial curvatures correlated with birth weigh

52 lingual (B [SE] = -0.104 [0.012]; P < .001), paracentral (B [SE] = -0.086 [0.012]; P < .001), perical

53 ate functional connectivity of posterior and paracentral brain regions, whereas the presence of VH is

54 parafascicular, medial parafascicular, oval paracentral, central lateral, paracentral, and central m

55 ostral intralaminar nuclei (central lateral, paracentral, central medial nuclei) as well as to the ve

56 and lateral parafascicular, central lateral, paracentral, central medial, rhomboid, reuniens, and sub

57 ]), retina (42 of 58 [73%]), and central and paracentral cornea (47 of 58 [81%]) be included in the r

58 epithelial opacifications in the central and paracentral cornea.

59 of 24 consecutive patients with central and paracentral corneal infectious ulcers and initial visual

60 caudal middle frontal cortex (P = .001) and paracentral cortex (P = .04), whereas parietal WMH volum

61 BOLD reduction in the right insula and left paracentral cortex in response to SS.

62 in the right caudal middle frontal and right paracentral cortex was greater in non-Hispanic Black par

63 te nucleus and thalamus, and surface area of paracentral cortex, indicative of altered distribution o

64 3; 95% CI, -0.393 to -0.113; P < .001; right paracentral cortex: B = -0.263; b = -0.337; 95% CI, -0.5

65 59; 95% CI, -0.114 to -0.004; P = .03; right paracentral cortex: B = -0.346; b = -0.155; 95% CI, -0.2

66 frontal, right pars triangularis, and right paracentral cortices was also stronger among non-Hispani

67 dictors of needing PKP, including central or paracentral defect location (vs peripheral, OR = 2.98, 9

68 of 234 VFs (9.4%), stage 1b (large inferior paracentral defect) in 112 of 234 VFs (47.9%), and stage

69 t common, including stage 1a (small inferior paracentral defect) in 22 of 234 VFs (9.4%), stage 1b (l

70 ce), and less normal VF results and superior paracentral defect.

71 PRS was linked to 1.65-times higher odds of paracentral defects (adjusted odds ratio [aOR], 1.65; 95

72 was linked to 0.68-times lower odds of both paracentral defects (aOR, 0.68; 95% CI, 0.57-0.82) and p

73 as associated with 1.69-times higher odds of paracentral defects (aOR, 1.69; 95% CI, 1.23-2.3) and 1.

74 HTG GRS was linked to total VF loss, but not paracentral defects.

75 ell count for the attached central, attached paracentral, detached apex, and detached base was 1247.6

76 rns (16.2%) originated from stage X (central/paracentral, enlarged blind spot, and scatter).

77 3.03 times more likely to demonstrate a new paracentral field defect than eyes progressing first on

78 NRD2 + ME3 GRS showed a higher prevalence of paracentral field loss than the bottom 1% (72.7% vs. 14.

79 gher treated IOP and a greater prevalence of paracentral field loss.

80 One graft had a linear paracentral fixed area of interface separation correspon

81 tion produces central corneal steepening and paracentral flattening in the central 3-mm diameter.

82 with POAG eyes with peripheral VF loss, the paracentral group showed reduced peripapillary VD (38.0

83 with POAG eyes with peripheral VF loss, the paracentral group showed reduced peripapillary VD (38.0

84 neal stromal tissue samples from central and paracentral hypocellular primitive stromal interface sca

85 The deep paracentral inferior island 'hot spot' coincided with th

86 erative course was unremarkable, but a dense paracentral interface opacity was observed during the 3-

87 ases with cotton wool spots with central and paracentral involvement and with few haemorrhages should

88 epth difference between the deep and shallow paracentral islands (R = 0.96; P < 0.0001).

89 The paracentral islands were accompanied by peripheral ablat

90 emporal gyrus, superior parietal lobule, and paracentral, lateral orbitofrontal, and lateral occipita

91 scotoma and demonstrated a classic dark gray paracentral lesion with near-infrared imaging.

92 ict control-related functional activation of paracentral lobule (against smaller monetary loss), vmPF

93 The paracentral lobule and cuneus had the highest resting me

94 ), premotor cortex, midcingulate cortex, and paracentral lobule and greater rsFC with the lateral tem

95 Additionally, we found evidence that the Paracentral Lobule and Medial Frontal Gyrus, regions ass

96 strated higher functional activation of left paracentral lobule and right putamen compared to HC.

97 hat in the right postcentral gyrus, the left paracentral lobule and the precentral gyrus antidepressa

98 The thalamus, angular, caudate nucleus, and paracentral lobule contributed most to the classificatio

99 Cortical thickness in the pars orbitalis, paracentral lobule, fusiform gyrus and inferior temporal

100 entified significant recruitment of the Left Paracentral Lobule, potentially suggesting the recruitme

101 parietal lobes, temporoparietal junction and paracentral lobule, right superior temporal and parietal

102 of the precentral and postcentral gyrus, the paracentral lobule, the superior temporal gyrus, the mid

103 ation difficulty to functional activation of paracentral lobule, ventromedial prefrontal cortex (vmPF

104 ght superior frontal and parietal lobes, and paracentral lobule.

105 Finally, voxelwise GM loss in the right paracentral lobulus correlated with bowel and bladder di

106 pattern reversal compared with those without paracentral loss (58.2% vs. 29.1%; P = 0.004).

107 significantly with functional measurement of paracentral loss (PaTD, r = 0.40, P = 0.02; r = 0.45, P

108 case-control study, a group of patients with paracentral loss confirmed on 10-2 VF tests were compare

109 Twelve eyes with paracentral loss had 24-2 VF results that showed defects

110 Patients with paracentral loss were more likely to have history of pat

111 at diagnosis (peripheral loss only or early paracentral loss).

112 at diagnosis (peripheral loss only or early paracentral loss).

113 re showed decreased VD and flow in POAG with paracentral loss, supporting its importance in this glau

114 control group whose VF results were without paracentral loss.

115 e compared between patients with and without paracentral loss.

116 VF finding and explore its association with paracentral loss.

117 Paracentral, mediodorsal, lateral posterior, and medial

118 glia, thalamus, hippocampus, frontal and the paracentral motor cortex and cerebellar vermis.

119 obex was activated and projected to the oval paracentral nucleus (OPC) of the intralaminar thalamic n

120 t to the central canal, corresponding to the paracentral nucleus of Herrick, and in the lateral funic

121 cular formation, lateral geniculate nucleus, paracentral nucleus, central medial nucleus, lateral hyp

122 All 9 patients noted paracentral or cecocentral location of scotomas involvin

123 tly higher FP rates compared with those with paracentral or peripheral loss only (16.25% vs. 6.26% an

124 ation of initial visual field defect (either paracentral or peripheral).

125 ascicular, central medial, paracentral, oval paracentral, or central lateral nucleus.

126 tern reversal was categorized as peripheral, paracentral, or mixed (both peripheral and paracentral).

127 lar, medial parafascicular, central lateral, paracentral, oval paracentral, and central medial nuclei

128 lar, lateral parafascicular, central medial, paracentral, oval paracentral, or central lateral nucleu

129 and right rostral middle frontal (P < .001), paracentral (P < .001), and pars triangularis (P = .02)

130 parabrachial subnuclei projected to the oval paracentral, parafascicular, and rhomboid thalamic nucle

131 eus (MD1), VL, and the central lateral (CL), paracentral (PC), central medial, rhomboid and ventromed

132 medial parafascicular, central lateral (CL), paracentral (PC), or central medial nucleus-or one of th

133 Central, paracentral, pericentral, and the thinnest corneal thick

134 In the early stage, the paracentral, peripheral arcuate 1 and peripheral arcuate

135 ere identified, including normal and various paracentral, peripheral, and total loss patterns.

136 ptor loss with a ring of minimally preserved paracentral photoreceptor nuclear layer.

137 ccipital cortex, and greater CT in the right paracentral, posterior cingulate, and superior occipital

138 The most important factor associated with paracentral progression among eyes that reached a progre

139 ery and have comparable sensitivities in the paracentral region of the cornea.

140 ery and have comparable sensitivities in the paracentral region of the cornea.

141 e, new techniques that enlist measurement of paracentral regions are discussed, and the ability of ea

142 ients with early visual field defects in the paracentral regions compared with those in the periphera

143 c disconnection to temporal, pre-, post- and paracentral regions.

144 e, angled directly toward the superior nasal paracentral retina.

145 ionally, sex had a significant impact on the paracentral retinal volume.

146 d with metabolism in the anterior cingulate, paracentral, right orbitofrontal, and left thalamic regi

147 nges in cortical thickness (increase in left paracentral, right precuneus and right but not left supe

148 patients corresponding to the diameter of a paracentral ring of increased fundus autofluorescence.

149 Lower dorsal anterior cingulate-DLPFC/paracentral rsFC was associated with residual cognitive

150 All patients presented with an acute paracentral scotoma and demonstrated a classic dark gray

151 The patient initially presented with a paracentral scotoma in his right eye persisting for 7 da

152 The paracentral scotoma is located inferotemporally and corr

153 A 63-year-old male presented with a paracentral scotoma that began several days ago.

154 d to our centre because of a deep unilateral paracentral scotoma with the presumptive diagnosis of a

155 ult patient who suffered from acute onset of paracentral scotoma, caused by branch retinal artery occ

156 e defects (n = 12), nasal steps (n = 11) and paracentral scotomas (n = 16).

157 the acute onset of reduced visual acuity and paracentral scotomas 2 weeks after their first infusion

158 atrophy of the INL, resulting in persistent paracentral scotomas for the patient.

159 improvement, but 3 eyes (23.1%) demonstrated paracentral scotomas related to the atrophy.

160 in young healthy females with acute onset of paracentral scotomas.

161 ears of age and presented with complaints of paracentral scotomas.

162 The paracentral thalamic nucleus received an input only from

163 elevated at fixation, but there were greater paracentral than central abnormalities.

164 Paracentral total deviation (PaTD) was defined as the av

165 severe loss, which was used to calculate the paracentral total deviation (PaTD), or total deviation w

166 severe loss, which was used to calculate the paracentral total deviation (PaTD), or total deviation w

167 s were overrepresented in central but not in paracentral V1 and that isoorientation domain size tende

168 selectivity or magnitude between central and paracentral V1.

169 ory devoted to higher SFs in central than in paracentral V1.

170 V2 SNPs were associated with POAG with early paracentral VF (top SNP, rs17588172; pooled P = 1.07 x 1

171 oral wedge, partial arcuate, nasal step, and paracentral VF defects.

172 ciation between CAV1/CAV2 SNPs and POAG with paracentral VF defects.

173 ogenesis in women and in patients with early paracentral VF defects.

174 for heterogeneity = .01) for POAG with early paracentral VF loss (433 cases; quintile 5 vs quintile 1

175 7; P for trend = .02) and 0.52 for POAG with paracentral VF loss (95% CI, 0.29-0.96; P for trend < .0

176 Hg (MVRR, 1.93; 95% CI, 1.09-3.43) and early paracentral VF loss (MVRR, 2.27; 95% CI, 1.32-3.88).

177 Pattern reversal may be associated with paracentral VF loss and is not always associated with el

178 ower POAG risk, particularly POAG with early paracentral VF loss at diagnosis.

179 l OCT parameters can predict the severity of paracentral VF loss of the affected hemifield, supportin

180 POAG PRSs and NTG PRSs were associated with paracentral VF loss, whereas higher HTG GRS was linked t

181 l utility of OCTA in patients with POAG with paracentral VF loss.

182 Progression within the paracentral VF was more common in the NTG group (75% vs.

183 dorsal representation of foveal vision, with paracentral vision represented more caudally.

184 We report a novel central/paracentral visual field defect after PPV for RRD repair

185 atients are visually impaired by symptomatic paracentral visual field defects despite a normal VA.

186 enotype is potentially associated with early paracentral visual field defects in primary open-angle g

187 Central/paracentral visual field defects were seen in 9 eyes.

188 nvolves assessing defects within the central/paracentral visual field-a crucial development for diagn

189 AG groups had matched VF MD (-3.1 +/- 2.5 dB paracentral vs. -2.3 +/- 2.0 dB peripheral; P = 0.31), d

190 e POAG groups had matched VF MD (-3.1 2.5 dB paracentral vs. -2.3 2.0 dB peripheral; P = 0.31), did n

191 55), but differed in age (59.2 +/- 9.6 years paracentral vs. 67.4 +/- 6.6 years peripheral; P = 0.02)

192 = 0.55), but differed in age (59.2 9.6 years paracentral vs. 67.4 6.6 years peripheral; P = 0.02).

193 < 0.001), compared with CPTR and mean TCT of paracentral zones (0.672 and 0.481, respectively; P < 0.