ライフサイエンスコーパス: parasitic infection

1 d predicting the effects of pulse warming on parasitic infection.

2 igher background rate of eosinophilia due to parasitic infection.

3 expression was up-regulated in response to a parasitic infection.

4 to humans and an important blood-borne human parasitic infection.

5 the drug discovery pipeline for this deadly parasitic infection.

6 omponents of the innate immune system during parasitic infection.

7 egral link in the chronic immune response to parasitic infection.

8 e important in Th2 cell-mediated immunity to parasitic infection.

9 tripartite model for nematode parasitism and parasitic infection.

10 beta induction and other markers of viral or parasitic infection.

11 the CD8(+) T cells during both a viral and a parasitic infection.

12 -49 receptor-expressing NK cell subsets in a parasitic infection.

13 -helper cells may be modulated by concurrent parasitic infection.

14 arthenogenetic Daphnia magna with respect to parasitic infection.

15 diated immune responses for an intracellular parasitic infection.

16 facilitate recognition and treatment of this parasitic infection.

17 tic doses to animals to selectively arrest a parasitic infection.

18 tion of the IgE response to allergens and to parasitic infection.

19 imates for a variety of conditions including parasitic infection.

20 of visceral leishmaniasis (VL) is a zoonotic parasitic infection.

21 ease globally in parallel with a decrease in parasitic infection.

22 kk-1) upon allergen challenge or non-healing parasitic infection.

23 allergic inflammation and protection against parasitic infection.

24 the liver that can determine the outcome of parasitic infection.

25 region occurred for select diagnoses such as parasitic infections.

26 eir contribution to enhance host exposure to parasitic infections.

27 ify new treatments for asthma, allergies and parasitic infections.

28 with high peanut consumption and widespread parasitic infections.

29 skin reactivity), consumption patterns, and parasitic infections.

30 ses, IL-17E is essential for defense against parasitic infections.

31 reatment of cancer, heart disease, AIDs, and parasitic infections.

32 ulating intracellular bacterial, fungal, and parasitic infections.

33 llular infiltrate associated with asthma and parasitic infections.

34 ty-adjusted life year (DALY), is unsuited to parasitic infections.

35 reatment of cancer, heart disease, AIDS, and parasitic infections.

36 reatment of cancer, heart disease, AIDS, and parasitic infections.

37 tokine production and protective immunity to parasitic infections.

38 in parasite epidemiology and the control of parasitic infections.

39 therapeutics for the treatment of cancer and parasitic infections.

40 plain many immune features characteristic of parasitic infections.

41 n allergic diseases and the host response to parasitic infections.

42 ng and managing the area-wide elimination of parasitic infections.

43 model for the study of human and veterinary parasitic infections.

44 novel drug targets and vaccine candidates in parasitic infections.

45 d an APOL3 variant involved in resistance to parasitic infections.

46 tor used for the prevention and treatment of parasitic infections.

47 tion of eosinophils in allergic diseases and parasitic infections.

48 eripheral blood T cells during bacterial and parasitic infections.

49 ment of eosinophils in allergic diseases and parasitic infections.

50 e and may confer physiological protection in parasitic infections.

51 cancers associated with bacterial, viral, or parasitic infections.

52 an attractive drug target for the control of parasitic infections.

53 diated disorders such as allergy, atopy, and parasitic infections.

54 angiogenesis and cancer to inflammation and parasitic infections.

55 in the initial suppression of bacterial and parasitic infections.

56 er, metabolic diseases as well as fungal and parasitic infections.

57 sights into these remarkable determinants of parasitic infections.

58 individuals, including the susceptibility to parasitic infections.

59 ownstream target of Rboh-mediated ROS during parasitic infections.

60 s of lower gastrointestinal bleeding such as parasitic infections.

61 allergic inflammation and protection against parasitic infections.

62 ment of T-cell malignancies and/or bacterial/parasitic infections.

63 uding solid and hematologic malignancies and parasitic infections.

64 uring Th2-mediated immune responses, such as parasitic infections.

65 kers, is a common occurrence in HIV and some parasitic infections.

66 both human and veterinary medicine to treat parasitic infections.

67 eactive protein levels and low prevalence of parasitic infections.

68 idation set including individuals with other parasitic infections.

69 rfusion injury, enteroinvasive bacterial and parasitic infections.

70 s a possible source for symptom disparity in parasitic infections.

71 lt in flare-ups of many bacterial, viral and parasitic infections.

72 ron metabolism and the etiology of anemia in parasitic infections.

73 odulatory functions in allergic diseases and parasitic infections.

74 mmune responses during viral, bacterial, and parasitic infections.

75 n diverse diseases from cancer to eukaryotic parasitic infections.

76 rological diseases and disorders, as well as parasitic infections.

77 nt specimens with other viral, bacterial, or parasitic infections.

78 including aggressive cancer, arthritis, and parasitic infections.

79 Fungal allergy (82 subjects: 21%), parasitic infection (61 subjects: 16%) and severe eosino

80 ival were less likely to screen positive for parasitic infections (9.6% versus 12.2%; adjusted preval

81 herpes viruses, mycobacteria, and fungal and parasitic infections, a single arm of the immune respons

82 in the past year fatal viral, fungal, and/or parasitic infections accounted for 16% and 34% of the mo

83 s bacterial peptidoglycan, viral infections, parasitic infections, activated Rho GTPases, and endopla

84 ted for centuries as chronically disfiguring parasitic infections affecting millions of people across

85 ed exposure to stressors (viruses, bacteria, parasitic infections, alcohol, drugs) or genetic mutatio

86 ory states arising from viral, bacterial and parasitic infection, allergic and asthmatic reactions, a

87 The pathological process was recognised as a parasitic infection, although its features were differen

88 particularly Th2-related responses including parasitic infection and allergic inflammation.

89 yet plays a critical role in defense against parasitic infection and allergic reactions.

90 complexity of the interrelationship between parasitic infection and allergy.

91 response able to clear bacterial, viral, and parasitic infection and an ability to efficiently store

92 Parasitic infection and fungal allergy often with severe

93 ation of IFN-gamma-mediated immunity to this parasitic infection and identify a novel function for th

94 How host cells sense intestinal parasitic infection and initiate the appropriate immune

95 result of incomplete surgery due to extended parasitic infection and liver anathomical changes due to

96 complexity of the murine host response to a parasitic infection and suggest that an outbred human po

97 rinary and human medicine for the control of parasitic infection and was the joint focus of the 2015

98 s that regulate both protective responses in parasitic infections and allergic airway inflammation.

99 The inverse association between parasitic infections and allergy at an ecological level

100 imarily destructive and are only relevant in parasitic infections and asthma.

101 at high levels in the T cells of people with parasitic infections and asthma.

102 icle, we review the current understanding of parasitic infections and basophils and discuss their put

103 Chronic inflammatory conditions such as parasitic infections and cancer trigger a metabolic repr

104 ological conditions, including bacterial and parasitic infections and cancer.

105 retroviruses (ERVs) are remnants of ancient parasitic infections and comprise sizable portions of mo

106 pathophysiological basis of the link between parasitic infections and epilepsy, and we consider preve

107 ne treats and prevents several bacterial and parasitic infections and has been proposed as a possible

108 have been shown to function in recovery from parasitic infections and in antibody responses.

109 4-dependent, Th2-mediated responses in other parasitic infections and in models of allergic asthma, t

110 reports on the exact roles for IL-17 during parasitic infections and limited knowledge about cellula

111 Eosinophil recruitment is a hallmark of parasitic infections and many forms of cancer, and eosin

112 This approach may find useful application in parasitic infections and more broadly in any disease sta

113 However, the role of p53 in parasitic infections and the intestinal type 2 immunity

114 for their ability to facilitate clearance of parasitic infections and their association with asthma a

115 potential, particularly for the treatment of parasitic infections and tuberculosis.

116 rnivore scavenging strategies under risks of parasitic infection, and (iii) conducting a literature-r

117 to neurodegenerative disease, bacterial and parasitic infection, and cancer among others.

118 d into tissues during allergic inflammation, parasitic infection, and certain malignancies.

119 te that CD4+ TRM cells form in response to a parasitic infection, and indicate that optimal protectiv

120 ied a novel inhibitory function for IL-17 in parasitic infection, and our results demonstrate importa

121 52 days) elapsed before the first documented parasitic infection, and the median intervals from arriv

122 y recruited into the CNS in response to this parasitic infection, and they were able to target the in

123 on exhibited elevated pathogen burden during parasitic infection, and this could be rescued by transf

124 g, 34 serum samples from patients with other parasitic infections, and 15 normal human serum samples.

125 , atopic dermatitis, allergic rhinitis, some parasitic infections, and cancer.

126 ector functions during bacterial, viral, and parasitic infections, and contributed to innate response

127 Despite, poor sanitation, diarrhea, parasitic infections, and environmental enteric dysfunct

128 admission included malnutrition and wasting, parasitic infections, and haematological disorders in th

129 autoimmune diseases, inflammatory diseases, parasitic infections, and HIV.

130 eptors have not been investigated to date in parasitic infections, and little is known about the role

131 loped because they are already used to treat parasitic infections, and there is interest in their use

132 ly credible gastrointestinal illness [HCGI], parasitic infections, anemia, growth).

133 tion of drugs against bacterial, fungal, and parasitic infections, antineoplastic agents against canc

134 The pathological processes resulting from parasitic infection are known to have important impacts

135 ch anti-viral innate immune responses affect parasitic infection are largely unknown.

136 Helminth parasitic infections are a major global health and socia

137 Chronic parasitic infections are a major risk factor for cancer

138 Parasitic infections are a potent stimulus for the gener

139 The majority of parasitic infections are caused by Giardia duodenalis, E

140 Parasitic infections are common, but how they shape ecos

141 capable of preventing viral, bacterial, and parasitic infections are currently under development.

142 Moreover, parasitic infections are important causes of anemia in t

143 better management of malaria and intestinal parasitic infections are likely to reduce the incidence

144 d optimal host defence against bacterial and parasitic infections are mediated by mast cells.

145 It most commonly occurs in parasitic infection as focal eosinophilic myositis but c

146 s review focuses on studies that use enteral parasitic infections as a tool to investigate the functi

147 iety of noxious environmental substances and parasitic infections at epithelial barrier surfaces.

148 pic cytokine involved in allergic reactions, parasitic infections, autoimmune inflammation, and cance

149 Parasitic infections belong in many countries to the neg

150 ignature appear during viral, bacterial, and parasitic infections, but also arise during humoral auto

151 number of enteric infections, including many parasitic infections, but also likely provide protection

152 Hosts are likely to respond to parasitic infections by a combination of resistance (exp

153 may lead to new therapies for microbial and parasitic infections, cancer, and neurodegenerative dise

154 Hydatid disease is a parasitic infection caused by Echinococcus granulosus an

155 Malaria, a parasitic infection caused by Plasmodium parasites and t

156 Cysticercosis is a parasitic infection caused by the larval stages of the p

157 Schistosomiasis or snail fever is an endemic parasitic infection caused by various trematodes of the

158 Three parasitic infections - cerebral malaria, Taenia solium c

159 Parasitic infections challenge vertebrate health worldwi

160 nological rationale underpinning therapeutic parasitic infection, describe recently initiated trials,

161 atopoietic growth factors or emerging during parasitic infections, display tolerogenic properties.

162 Parasitic infections due to climate changes should also

163 effect of stunting, diarrhoeal disease, and parasitic infections during infancy on cognitive functio

164 ous congenital infections, the mechanisms of parasitic infections during pregnancy remain poorly unde

165 incidence and natural history of intestinal parasitic infections during the PCVs' >2-year overseas s

166 y, the mechanisms by which mosquitoes resist parasitic infection (e.g., immune-mediated killing) have

167 mass drug administration (MDA) against these parasitic infections emphasise treatment of school-aged

168 The current studies suggest acute parasitic infections exacerbate allergic symptoms, where

169 Furthermore, individuals with parasitic infections exhibited a significantly higher li

170 Parasitic infections frequently result in highly polariz

171 eosinophils was decreased in the presence of parasitic infection (GM = 0.9 vs. 5.7%, p = 0.02).

172 Once a parasitic infection has taken hold in the brain, therape

173 in monocyte and macrophage functions during parasitic infections has not been explored.

174 Various viral, bacterial, and parasitic infections have been associated with catatonia

175 a chronic inflammatory disease diabetes and parasitic infection helminth as illustrativedisease pair

176 ntestinal type 2 immunity to protect against parasitic infections, highlighting the role of p53 as a

177 In response to parasitic infection, hosts may evolve defences that redu

178 ective innate immunity against bacterial and parasitic infections; however, its role in host immunity

179 rovided insight into innate immunity against parasitic infections; however, species-specific differen

180 adpoles leads to increased susceptibility to parasitic infection in adult frogs, in the absence of su

181 Malaria is the most important parasitic infection in people, accounting for more than

182 undisputable, we know very little about how parasitic infection in reproductive females might influe

183 utaneous leishmaniasis (CL) is a very common parasitic infection in subtropical areas worldwide.

184 rovide possible explanations for the role of parasitic infection in susceptibility and resistance to

185 to treat Chagas disease, the most important parasitic infection in the Americas.

186 ted to the development of host resistance to parasitic infection in the resistant breed.

187 Giardiasis, the most common enteric parasitic infection in the United States, causes an esti

188 n response to Toxoplasma gondii profilin and parasitic infection in vivo.

189 -1+ monocytes in the control of disseminated parasitic infection in vivo.

190 possible fates in a relevant murine model of parasitic infection in vivo.

191 This is the first evidence of a parasitic infection in which down-regulation of CD8+ T c

192 across age, sex, anthropometric indexes, and parasitic infections in a representative sample of 3595

193 in protection against bacterial, viral, and parasitic infections in humans and animals.

194 chronic infection is one of the most common parasitic infections in humans and can be reactivated to

195 Parasitic infections in humans are diagnosed incidentall

196 Toxoplasma gondii is one of the most common parasitic infections in humans.

197 T cell differentiation and susceptibility to parasitic infections in murine models, we compared T cel

198 tervention addressing 2 of the most virulent parasitic infections in pregnant women in sub-Saharan Af

199 osomiasis are the world's two most important parasitic infections in terms of distribution, morbidity

200 risk of common bacterial, viral, fungal, and parasitic infections in the general population of indivi

201 faces and serves to protect against helminth parasitic infections in the intestinal tract.

202 tudies toxoplasmosis, one of the most common parasitic infections in the world, caused by Toxoplasma

203 It is one of the most widespread human parasitic infections in tropical and subtropical regions

204 Chronic immunomodulating parasitic infections including schistosomes and malaria

205 at can protect against bacterial, viral, and parasitic infections, including lethal influenza and mal

206 th a broad-spectrum activity against various parasitic infections, including malaria, toxoplasmosis a

207 vaccines are available for human use for any parasitic infections, including the helminthic disease s

208 Many parasitic infections, including those of humans, are cau

209 o Leishmania major (Lm) or Toxoplasma gondii parasitic infections, indicating that exogenously delive

210 DACs may show benefits in diseases (cancers, parasitic infections, inflammatory conditions) where AR-

211 Intestinal parasitic infections (IPIs) are largely overlooked in Ma

212 Parasitic infection is a major factor negatively affecti

213 The direct benefit to females of avoiding parasitic infection is proposed to lead to the linkage b

214 uation of patients with suspected intestinal parasitic infection is proposed.

215 Parasitic infection is uncommon in the United States, bu

216 Furthermore, the role of CRIg during parasitic infection is unknown.

217 Although parasitic infection is usually detrimental, it can be be

218 addition, use of nephrotoxic drugs to treat parasitic infections is associated with acute kidney inj

219 information on DC-NK cell interaction during parasitic infections is available.

220 Serologic testing for chronic parasitic infections is often necessary.

221 An important feature of many chronic parasitic infections is the ability of the invading path

222 Malaria, a devastating parasitic infection, is the leading cause of death in ma

223 s and those chronic and debilitating (mostly parasitic) infections known as neglected tropical diseas

224 d neoplastic (chronic lymphocytic leukemia), parasitic infection (Leishmania major), and infectious d

225 Parasitic infections like malaria (Plasmodium falciparum

226 At entry persons with parasitic infections may be asymptomatic, and stool exam

227 This review discusses T cell exhaustion in parasitic infection, mechanisms of development, and a po

228 etion in autoimmune, transplant disease, and parasitic infection models.

229 ost defense in viral, bacterial, fungal, and parasitic infections, myeloid CLRs have been implicated

230 r regulation of immune responses, such as in parasitic infection, necessitate further characterizatio

231 Toxoplasma gondii, the most common parasitic infection of human brain and eye, persists acr

232 sent potential mechanistic pathways by which parasitic infection of mother and/or infant may lead to

233 Repeated parasitic infection of TCR beta-/- mice, but not of T ce

234 It is the most prevalent parasitic infection of the central nervous system and on

235 Neurocysticercosis, a parasitic infection of the human central nervous system

236 or immune intervention in ocular allergy and parasitic infections of the eye.

237 ing the model, we linked negative effects of parasitic infection on host survival, fecundity, and fee

238 tion problems, particularly the influence of parasitic infections on child health and nutrition.

239 Effective management and control of parasitic infections on farms depends on their early det

240 munity that can either be protective against parasitic infection or cause tissue damage in allergy an

241 patients affected by Th2 pathologies such as parasitic infections or atopic dermatitis often suffer f

242 ffector cells useful in the immunotherapy of parasitic infections or neoplasms.

243 A systemic bacterial, viral, or parasitic infection other than malaria or typhoid fever

244 esults are negative, O&P can be performed if parasitic infections other than G. lamblia, E. histolyti

245 ory antibody responses within the context of parasitic infections, our results suggest that PfGARP mi

246 ietary factors such as chronic inflammation, parasitic infections, overweight, and genetic hemoglobin

247 l established risk factors for CC, including parasitic infections, primary sclerosing cholangitis, bi

248 sed by granulomatous disorders, DNA viruses, parasitic infections, pulmonary amyloidosis, vascular ca

249 Reduced parasitic infection rates in the developed world are sus

250 ationship was mostly explained by fungal and parasitic infections rather than by viral and bacterial

251 lthough this is an expanding field, maternal parasitic infections remain understudied.

252 Surviving a parasitic infection requires the generation of a control

253 Malaria and babesiosis are significant parasitic infections, requiring timely diagnosis to avoi

254 disruptions in mucosal interfaces induced by parasitic infection, respectively.

255 ypanosoma cruzi as examples of bacterial and parasitic infection, respectively.

256 -human primates have consistently found that parasitic infections result in less movement and reduced

257 vitamin D insufficiency, and eradication of parasitic infections) risk factors.

258 sease settings such as bacterial, viral, and parasitic infections, sepsis, obesity, trauma, stress, a

259 dation of immune mechanisms underpinning the parasitic infections, some of which are parasite-specifi

260 ve disease, including hallmark signatures of parasitic infection such as elevated immunoglobulin G4 (

261 wever, the role of NK cells in extracellular parasitic infections such as filarial infections is not

262 Parasitic infections such as filariasis, schistosomiasis

263 The incidence of parasitic infections such as malaria, leishmaniasis, and

264 otic enzyme and an attractive drug target in parasitic infections such as malaria.

265 In areas endemic for multiple parasitic infections, such as sub-Saharan Africa, there

266 ogen clearance in many bacterial, viral, and parasitic infections, such as through Toll-like receptor

267 Leishmania braziliensis is a parasitic infection that can result in inflammation and

268 Schistosomiasis is a parasitic infection that is widespread in sub-Saharan Af

269 Malaria is a global parasitic infection that leads to substantial illness an

270 n people are at risk of acquiring malaria, a parasitic infection that produces more than 200 million

271 Visceral leishmaniasis (VL) is a parasitic infection that results in approximately 26 000

272 caused by Trypanosoma brucei gambiense is a parasitic infection that usually progresses to coma and

273 l burden of high E4 prevalence combined with parasitic infections that can also reduce cognitive perf

274 e habitat for intermediate hosts of tropical parasitic infections that cause disease and suffering.

275 sis may have potential utility for targeting parasitic infections that cause important neglected trop

276 ons of cell death on the outcome of numerous parasitic infections that impact the brain, meninges, an

277 e been proposed by which hosts can withstand parasitic infections, the role of multiple infections an

278 e natural antimicrobial mixture (AG) reduced parasitic infections through mechanisms that reduced pat

279 Ecologists typically see parasitic infections, through their association with dis

280 rheal episodes, the continued acquisition of parasitic infections throughout the PCVs' >2-year stay i

281 rol patients who had other parasitic and non-parasitic infections to a weak cation exchange chip, and

282 major human diseases ranging from viral and parasitic infections to autoimmunity and cancer.

283 ania infantum, is a persistent intracellular parasitic infection transmitted by the bite of infected

284 is the cause of a potentially fatal emerging parasitic infection transmitted by ticks.

285 Murine neurocysticercosis is a parasitic infection transmitted through the direct inges

286 In several parasitic infections, Treg maintain equilibrium to ensur

287 lations with a high prevalence of intestinal parasitic infections, two independently collected stool

288 d is induced in response to inflammation and parasitic infections, upon exposure to microbiota, and i

289 d mortality associated with these intestinal parasitic infections warrant the development of rapid an

290 ed modelling showed that only positivity for parasitic infections was a significant predictor of redu

291 Susceptibility to having 1 or more parasitic infections was significantly associated with p

292 dazole, an oral drug currently used to treat parasitic infections, was used as a positive control.

293 y intake, nutritional status, and intestinal parasitic infection were collected.

294 n thus offers an example of an intracellular parasitic infection where CD8(+) T-cell immunity can be

295 f reproductive age are currently at risk for parasitic infection, whereas many pregnant, chronically

296 s suggests an innate immune response against parasitic infections, whereas the other gene expression

297 iotic used in veterinary medicine to prevent parasitic infections which also displays potent inhibito

298 r without Schistosoma mansoni coinfection, a parasitic infection with T helper (Th) 2 immune bias.

299 In parasitic infection with Trichinella spiralis, the immun

300 The physiological effects of such events on parasitic infections within endotherms are poorly studie