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1 is caused by the release of ATP and ACh from parasympathetic nerves.
2 ine and ATP are excitatory cotransmitters in parasympathetic nerves.
3 ized rats after bilateral transection of the parasympathetic nerves.
4 ll secretion in vivo is under the control of parasympathetic nerves.
5 pathetic nerves induce a greater effect than parasympathetic nerves.
6 erve to oppose airway contraction induced by parasympathetic nerve activation in the airways.
7  cerebral circulation, and interactions with parasympathetic nerve activity are unique features to th
8 ollowing pharmacological blockade of cardiac parasympathetic nerve activity in eight young subjects,
9 ectrophysiological recordings showed reduced parasympathetic nerve activity in the basal state and no
10 derstand how known age-related reductions in parasympathetic nerve activity influence CBR response la
11 rves innervating the larynx can alter airway parasympathetic nerve activity, transection of the super
12        The following conclusions were drawn: parasympathetic nerves and M(1), M(2), and M(3) muscarin
13 at renal afferents travel in sympathetic and parasympathetic nerves and that this information may con
14  limitations of the terms 'sympathetic' and 'parasympathetic' nerves and functions and suggest a way
15 mplementary signals derived from beta-cells, parasympathetic nerves, and increased islet blood flow.
16                                              Parasympathetic nerves are a vital component of the prog
17                           This suggests that parasympathetic nerves are capable of re-innervating an
18 tissue was essentially devoid of sensory and parasympathetic nerves at all time points.
19 stent with a pathway where VIP released from parasympathetic nerves binds to VIPRs types I and II, ac
20  protein released from eosinophils to airway parasympathetic nerves blocks inhibitory M(2) muscarinic
21 bitory M2 muscarinic receptors on the airway parasympathetic nerves causes vagally mediated bronchoco
22 atory nucleus, which activates tarsal muscle parasympathetic nerves, elicited large contractions at 2
23        Inhibitory M2 muscarinic receptors on parasympathetic nerve endings in the lungs decrease rele
24 of inhibitory M2 muscarinic receptors on the parasympathetic nerve endings is likely to contribute to
25 vealed that the densities of sympathetic and parasympathetic nerve fibers in tumor and surrounding no
26 lex arrangement of sensory, sympathetic, and parasympathetic nerve fibers that contain classical tran
27 ered to the epicardial fat pad that projects parasympathetic nerve fibers to the AVN in 12 dogs durin
28 c nervous system consists of sympathetic and parasympathetic nerve fibres, primarily located in the e
29   We propose that neurturin will protect the parasympathetic nerves from damage and improve organ reg
30                                              Parasympathetic nerves from the pterygopalatine ganglia
31 is mediated in part through the influence of parasympathetic nerves from the pterygopalatine ganglia.
32  demonstrate an unexpected role for SOX2 and parasympathetic nerves in generating the acinar lineage
33   The goal of this study was to determine if parasympathetic nerves in the anterior fat pad (FP) can
34 of inhibitory M2 muscarinic autoreceptors on parasympathetic nerves in the lung, potentiating vagally
35 of inhibitory M2 muscarinic autoreceptors on parasympathetic nerves in the lungs, and airway hyperres
36  inhibitory M(2) muscarinic receptors on the parasympathetic nerves, increasing acetylcholine release
37 holinergic and nonadrenergic, noncholinergic parasympathetic nerves innervating the airways are subje
38                 It may be concluded that the parasympathetic nerves innervating the cerebral circulat
39 equired for salivary gland development, with parasympathetic nerves interacting with the surrounding
40 active intestinal peptide (VIP), secreted by parasympathetic nerves, is a surprising player in direct
41 inhibitory M2 muscarinic receptors (M2Rs) on parasympathetic nerves limit acetylcholine release.
42  muscarinic receptors (M(2)Rs) on the airway parasympathetic nerves limit acetylcholine release.
43                                 Intratumoral parasympathetic nerves may have a dichotomous role in ca
44                                 Intratumoral parasympathetic nerves may have a dichotomous role in ca
45 addressed the hypothesis that noncholinergic parasympathetic nerves modulate airway smooth-muscle (AS
46       These results show that noncholinergic parasympathetic nerves modulate ASM tone.
47 suggest that nitric oxide (NO) released from parasympathetic nerves participates in vasodilatation of
48 exly activated by bradykinin, noncholinergic parasympathetic nerves partly reversed histamine-induced
49                    Here, we demonstrate that parasympathetic nerves regulate tubulogenesis in the dev
50 indicate that cholinergic and noncholinergic parasympathetic nerves regulating airway caliber in guin
51 active intestinal polypeptide-immunoreactive parasympathetic nerves revealed fibers associated predom
52 ceptor-mediated prejunctional enhancement of parasympathetic nerve-smooth muscle neurotransmission, w
53 creasing heart rate and stroke volume, while parasympathetic nerve stimulation instantly slows heart
54             The influence of sympathetic and parasympathetic nerve stimulations on salivary secretion
55 classical' transmitters from sympathetic and parasympathetic nerves supplying various peripheral targ
56                         Transvascular atrial parasympathetic nerve system modification by RFCA abolis
57 tocytes and cholangiocytes are innervated by parasympathetic nerve terminals originating from the dor
58 are triggered by stochastic ATP release from parasympathetic nerve terminals rather than being myogen
59 t is innervated by sensory, sympathetic, and parasympathetic nerves that actively stimulate cancer gr
60 ce of ACh was sympathetic nerves rather than parasympathetic nerves that are the normal source of ACh
61 d airway hyperreactivity are mediated by the parasympathetic nerves that release acetylcholine onto m
62 ctrical field stimulation was used to excite parasympathetic nerves to increase action potentials, Ca
63 n, including 'hardwiring' of sympathetic and parasympathetic nerves to lymphoid organs.
64 asma glucose = 1.9 +/- 0.1 mmol/l) activated parasympathetic nerves to the pancreas as assessed by in
65 ities in the distribution of sympathetic and parasympathetic nerves, which powerfully regulate impuls
66 er smooth muscle contraction is triggered by parasympathetic nerves, which release ATP and acetylchol