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1 5-hydroxyvitamin D, phosphorus, calcium, and parathyroid hormone).
2 ts increase is associated with a decrease in parathyroid hormone.
3 (receptor activator of NFkappaB ligand) and parathyroid hormone.
4 There was no effect modification by parathyroid hormone.
5 nd action of fibroblast growth factor 23 and parathyroid hormone.
6 blood pressure or arterial pressure and with parathyroid hormone.
7 clic AMP responded to external cues, such as parathyroid hormone.
8 hyroid tissue causing excessive secretion of parathyroid hormone.
9 ized by hypercalcemia and elevated levels of parathyroid hormone.
10 er characterized by autonomous production of parathyroid hormone.
11 or inappropriately normal concentrations of parathyroid hormone.
14 tolerability of once-daily recombinant human parathyroid hormone 1-84 (rhPTH[1-84]) in adults with hy
16 f 39 residues (TIP39), via its receptor, the parathyroid hormone 2 receptor (PTH2R), modulates fear m
17 Small interfering RNA-mediated silencing of parathyroid hormone 2 receptor (PTH2R), the receptor for
19 racteristics, dietary intakes, fasting serum parathyroid hormone, 25-hydroxyvitamin D [25(OH)D], and
21 ge mean+/-SD single-pass renal extraction of parathyroid hormone (44.2%+/-10.3%) that exceeded the ex
24 signature of the arrestin pathway-selective parathyroid hormone analog [d-Trp(12), Tyr(34)]bovine PT
27 in chronic kidney disease and the effects of parathyroid hormone and FGF23 on these associations.
28 ant role for vitamin D and related hormones, parathyroid hormone and fibroblast growth factor 23 (FGF
29 splant patients significantly reduced intact parathyroid hormone and increased fibroblast growth fact
30 -dihydroxyvitamin D (1,25(OH)2D), FGF23, and parathyroid hormone and performed quantitative echocardi
32 ts of histologic analysis, as well as intact parathyroid hormone and serum calcium values obtained 1
34 alphas also inhibited internalization of the parathyroid hormone and type 2 vasopressin receptors.
35 rations <20 ng/mL who did not also have high parathyroid hormone and women with high parathyroid horm
36 luding C-terminal fragment of FGF-23, intact parathyroid hormone, and 1,25(OH)(2)D(3), were measured
38 ulated by numerous factors, including BMP-2, parathyroid hormone, and 1alpha,25-dihydroxyvitamin D3 (
39 ium, phosphorus, 25-hydroxyvitamin D, intact parathyroid hormone, and 24,25-dihydroxyvitamin D did no
41 ong hormones, including 1,25(OH)2D3 (1,25D), parathyroid hormone, and fibroblast growth factor 23 (FG
42 he HDF cohort had lower beta2-microglobulin, parathyroid hormone, and high-sensitivity C-reactive pro
43 eomalacia, hypophosphatemia, increased serum parathyroid hormone, and increased levels of the phospha
44 ar, serum osteocalcin, total calcium, intact parathyroid hormone, and increased serum C telopeptide.
47 ffect on basal bone resorption, it inhibited parathyroid hormone- and ovariectomy-induced OC activati
48 er levels of serum calcium, phosphorous, and parathyroid hormone; and nutritional vitamin D, cinacalc
49 terminal propeptide (PINP), osteocalcin, and parathyroid hormone as well as a transient decrease in t
50 oxyvitamin D, vitamin D-binding protein, and parathyroid hormone as well as bone mineral density (BMD
51 substantial single-pass renal extraction of parathyroid hormone at a rate that exceeds glomerular fi
52 More biochemical surveillance particularly a parathyroid hormone-based protocol, fine-tuned supplemen
53 hly expressed in osteocytes, is regulated by parathyroid hormone both in vitro and in vivo, and prote
54 nine, lower hematocrit, and increased intact parathyroid hormone but did not demonstrate any differen
55 FGF23, despite having high concentrations of parathyroid hormone, but administration of exogenous 1,2
56 evels, serum calcium homeostasis biomarkers (parathyroid hormone, calcium, and 25-hydroxyvitamin D),
57 relationships), yet within each quintile of parathyroid hormone concentration, blacks had significan
59 whether lower 25-hydroxyvitamin D and higher parathyroid hormone concentrations are associated with i
64 intervals (CIs) for 25-hydroxyvitamin D and parathyroid hormone concentrations with incident hyperte
65 perparathyroidism, and might be predicted by parathyroid hormone concentrations, even in individuals
69 operative success, defined by intraoperative parathyroid hormone criteria, and complication rates wer
71 th the use of dual-stable isotopes, 25(OH)D, parathyroid hormone, estradiol, calcitriol, and urinary
72 a common endocrine disease characterized by parathyroid hormone excess and hypercalcemia and caused
73 minal aortic calcification, serum phosphate, parathyroid hormone, FGF23, and 24-hour urinary phosphat
74 etermined the single-pass renal clearance of parathyroid hormone, fibroblast growth factor 23, vitami
75 /- 10.3 ng/mL; P < 0.0001) and a decrease in parathyroid hormone (from 21.4 +/- 10.4 to 12.9 +/- 7.1
76 splantation, persistent hyperparathyroidism (parathyroid hormone > 130 ng/L) and bone turnover marker
78 Compared with parathyroid hormone <33 pg/ml, parathyroid hormone >/=65 pg/ml was associated with a si
79 th low urine output, low serum albumin, high parathyroid hormone, high ferritin, and the use of bioin
81 at 1000 IU/d increases 25(OH)D and decreases parathyroid hormone in children with average vitamin D i
85 der these circumstances both agents enhanced parathyroid hormone-induced osteoblast differentiation a
89 ients with hypercalcemia and elevated intact parathyroid hormone (iPTH) concentration were eligible i
90 ium concentration is within normal range but parathyroid hormone is elevated in the absence of any ob
91 aemia caused by autonomous overproduction of parathyroid hormone, is common, especially in postmenopa
92 x, and creatinine clearance, but with intact parathyroid hormone less than 100 pg/mL, were included a
93 ltiply by 0.25; P = .15), preoperative serum parathyroid hormone level (mean [SD], 114.5 [56.8] vs 13
94 rogression were age, baseline total or whole parathyroid hormone level greater than nine times the no
95 Regarding modifiable factors, higher average parathyroid hormone level was associated with greater ri
96 walking, and higher average log-transformed parathyroid hormone level were independently associated
97 ent risk was attenuated after adjustment for parathyroid hormone level, suggesting that parathyroid h
101 ncreased sessional dialysis dose and lowered parathyroid hormone levels (from 51 +/- 67 to 24 +/- 37
103 lerated) or nonparicalcitol therapy on serum parathyroid hormone levels (primary outcome), mineral me
104 itamin D receptor activator, decreased serum parathyroid hormone levels and proteinuria in patients w
105 6-month paricalcitol supplementation reduced parathyroid hormone levels and proteinuria, attenuated b
108 s induced by aldosteronism in which elevated parathyroid hormone levels raise the risk of adverse car
109 baseline, median (interquartile range) serum parathyroid hormone levels significantly declined on par
112 mice had significantly lower serum FGF23 and parathyroid hormone levels, and higher renal 1-alpha-hyd
113 irmed by exploratory surgery, intraoperative parathyroid hormone levels, and pathologic features.
114 by increased serum 1,25-(OH)2-vitamin D3 and parathyroid hormone levels, decreased serum calcium leve
115 response or a hormonal response to decreased parathyroid hormone levels, we subjected osteocytes to a
120 5 (Gdf5), the transcription factor Erg, and parathyroid hormone-like hormone (Pthlh), and selection
121 orylation and TGF-beta-induced expression of parathyroid hormone-like hormone (PTHLH), leading to ost
122 , and Ihh target genes Patched 1 (Ptch1) and parathyroid hormone-like peptide (Pthlh) were down-regul
127 asive parathyroidectomy using intraoperative parathyroid hormone monitoring remains the standard appr
128 y invasive parathyroidectomy, intraoperative parathyroid hormone monitoring via a reliable protocol i
133 nce interval (CI) 1.1-2.8], increasing serum parathyroid hormone (OR 1.1 per 10 pg/mL 95% CI 1.05-1.1
134 ccurred in serum calcium, phosphorus, intact parathyroid hormone, or C-reactive protein levels, cinac
135 not be explained by hypocalcemia, changes in parathyroid hormone, or fibroblast growth factor 23.
136 , levels of calcium, phosphorus, calcitriol, parathyroid hormone, or fibroblast growth factor-23 were
137 not explained by urine phosphate excretion, parathyroid hormone, or fibroblast growth factor-23.
138 citol treatment significantly reduced intact parathyroid hormone (P<0.001) and alkaline phosphatase (
139 etabolic pathway (e.g., 25-hydroxyvitamin D, parathyroid hormone, phosphorus) had little impact.
140 nine, free thyroxine, free triiodothyronine, parathyroid hormone, prolactin, N-terminal pro-brain nat
141 um metabolism was defined as elevated intact parathyroid hormone (PTH) (>62 pg/mL) accompanied by a v
142 Intermittent administration of a fragment of Parathyroid hormone (PTH) activates osteoblast-mediated
145 cium through processes that are regulated by parathyroid hormone (PTH) and 1alpha,25-dihydroxyvitamin
149 a/c and levels of plasma phosphate, calcium, parathyroid hormone (PTH) and fibroblast growth factor 2
150 echanisms through which Pi intake stimulates parathyroid hormone (PTH) and fibroblast growth factor-2
151 coupled receptors (GPCRs) and natively binds parathyroid hormone (PTH) and parathyroid hormone relate
152 tissue development, has two native agonists, parathyroid hormone (PTH) and PTH-related protein (PTHrP
156 Previously, we have shown that intermittent parathyroid hormone (PTH) bone anabolic therapy involves
157 ic whites, but whether adding information on parathyroid hormone (PTH) can help explain the higher ca
158 3 patients receiving hemodialysis with serum parathyroid hormone (PTH) concentrations higher than 500
159 ravenous calcimimetic etelcalcetide on serum parathyroid hormone (PTH) concentrations in patients rec
163 and urine levels of minerals, and levels of parathyroid hormone (PTH) in healthy postmenopausal wome
164 ], 1,25-dihydroxyvitamin D [1,25(OH)2D], and parathyroid hormone (PTH) in maternal circulation and co
165 he PT-Dicer(-/-) mice did not increase serum parathyroid hormone (PTH) in response to acute hypocalce
166 ide (PTHrP), prevented the decrease in serum Parathyroid Hormone (PTH) induced by lactation, but ampl
176 yroidism is characterized by increased serum parathyroid hormone (PTH) level and parathyroid cell pro
177 ce and impact of HPT, defined as an elevated parathyroid hormone (PTH) level, after renal transplanta
179 ibited reduced serum inorganic phosphate and parathyroid hormone (PTH) levels and decreased bone form
180 nvestigated the association between baseline parathyroid hormone (PTH) levels and major cardiovascula
182 l quantitative computed tomography (HRpQCT), parathyroid hormone (PTH) levels, and bone turnover mark
183 ned assessment of 25-OH-D with its regulator parathyroid hormone (PTH) may be required for optimal ev
185 otic-treated or germ-free mice, we show that parathyroid hormone (PTH) only caused bone loss in mice
188 isciplinary analytical characterization of a parathyroid hormone (PTH) peptide active pharmaceutical
191 osteoblastic cell-specific activation of the parathyroid hormone (PTH) receptor attenuates BCR-ABL1 o
193 alloproteinase-13 (MMP-13) transcription and parathyroid hormone (PTH) regulates HDAC4 to control MMP
194 aR) modulates renal calcium reabsorption and parathyroid hormone (PTH) secretion and is involved in t
195 ition of the abundant let-7 family increased parathyroid hormone (PTH) secretion in normal and uremic
196 , whether they had any functional effects on parathyroid hormone (PTH) secretion in parathyroid neopl
202 absence of ascorbate and were stimulated by parathyroid hormone (PTH) to undergo rapid osteoclast fo
204 was the primary outcome, and serum calcium, parathyroid hormone (PTH), 1,25-dihydroxyvitamin D [1,25
205 ogenesis, confluent BMSCs were cultured with parathyroid hormone (PTH), 1,25-dihydroxyvitamin D3 (1,2
209 on tubular reabsorption of phosphate (TRP), parathyroid hormone (PTH), bone alkaline phosphatase (BA
211 xamined the prospective associations between parathyroid hormone (PTH), circulating 25-hydroxyvitamin
212 parathyroid chief and oxyphil cells produce parathyroid hormone (PTH), express the calcium-sensing r
213 y regulated in a highly reciprocal manner by parathyroid hormone (PTH), fibroblast growth factor 23 (
214 mber 1 (CYP27B1), whose gene is regulated by parathyroid hormone (PTH), fibroblast growth factor 23 (
215 BD) parameters including calcium, phosphate, parathyroid hormone (PTH), fibroblast growth factor 23 (
216 ercalcemic patients underwent measurement of parathyroid hormone (PTH), had documentation of hypercal
218 transferrin saturation (TSAT) concentration, parathyroid hormone (PTH), IV vitamin D dose, cinacalcet
219 ata defined by their pretransplant levels of parathyroid hormone (PTH), low PTH (>65 to </=300 pg/mL;
221 enes involved in signaling through Wnt, FSH, parathyroid hormone (PTH), oxytocin, and calcineurin/nuc
222 vels of fibroblast growth factor-23 (FGF23), parathyroid hormone (PTH), phosphate, and 25-hydroxyvita
223 ced bone deposition with decreased levels of parathyroid hormone (PTH), which is a key regulator of b
224 over and pronounced osteoblast resistance to parathyroid hormone (PTH), which is indicated by decreas
225 des: glucagon-like peptide-1 (GLP-1) and the parathyroid hormone (PTH), which respectively help contr
226 itory effects of TCN on lipopolysaccharide-, parathyroid hormone (PTH)-, and prostaglandin E2 (PGE2)-
227 al to Cyp27b1 that mediates unique basal and parathyroid hormone (PTH)-, fibroblast growth factor 23
228 nique role of osteal macrophages in bone and parathyroid hormone (PTH)-dependent bone anabolism using
229 urinary calcium excretion in the context of parathyroid hormone (PTH)-dependent hypercalcemia, but t
230 m MS analysis, we characterized the sites of parathyroid hormone (PTH)-induced NHERF1 phosphorylation
231 m baseline in blood and urine markers of the parathyroid hormone (PTH)-vitamin D-fibroblast growth fa
237 e anabolism through direct activation of the parathyroid hormone (PTH)/PTH-related protein receptor (
238 ostasis and is tightly regulated through the parathyroid hormone (PTH)/PTHrP receptor (PTH1R) signali
254 , Ctsk deletion in osteocytes increased bone Parathyroid Hormone related Peptide (PTHrP), prevented t
258 pression in chondrocytes strictly depends on parathyroid hormone-related peptide (PTHrP) signaling pa
259 r translocation of Gli2 and transcription of parathyroid hormone-related peptide (PTHrP), a key regul
261 ATDC5 chondrogenic cells is downregulated by parathyroid hormone-related peptide through transcriptio
262 targets (plasminogen activator inhibitor-1, parathyroid hormone-related peptide, connective tissue g
263 ix protein 1, a direct targeting molecule of parathyroid hormone-related peptide, negatively regulate
264 ells are regulated by autocrine signaling by parathyroid hormone-related protein (PTHrP) and its para
266 K14-PTHrP transgenic mice [which overexpress parathyroid hormone-related protein (PTHrP) in their dev
269 Here we addressed the hypothesis that the parathyroid hormone-related protein (PTHrP) potentiates
272 nscription factor Gli2 as a key regulator of parathyroid hormone-related protein (PTHrP), which is pr
273 egulates endochondral ossification in both a parathyroid hormone-related protein (PTHrP)-dependent an
274 ivation, increased Indian hedgehog (Ihh) and parathyroid hormone-related protein (Pthrp, also known a
275 sion data suggested that the Indian Hedgehog-parathyroid hormone-related protein signaling axis was i
276 as been made in determining the roles of the parathyroid hormone-related protein, Indian hedgehog, fi
277 or the quantification of a cancer biomarker (parathyroid hormone-related protein, PTHrP) in a real cl
279 cancer cachexia, we show that tumour-derived parathyroid-hormone-related protein (PTHrP) has an impor
280 een the groups, patients who did not mount a parathyroid hormone response to vitamin D deficiency had
282 as phosphates, fibroblast growth factor 23, parathyroid hormone, sclerostin, or vitamin D and their
283 ices with isoproterenol, norepinephrine, and parathyroid hormone similarly increased NCC phosphorylat
284 roblast growth factor (FGF-2)-knockout mice, parathyroid hormone stimulation and myeloablative treatm
285 ndomized to receive 20 mug recombinant human parathyroid hormone (teriparatide) or placebo for 18 mon
287 ositively by 1,25(OH)2D3, retinoic acid, and parathyroid hormone through both intergenic and intronic
290 calcium, phosphate, 25-hydroxyvitamin D, and parathyroid hormone), various inflammatory markers (C-re
291 me BP, plasma and urine electrolytes, renin, parathyroid hormone, vitamin D, and response to oral glu
294 nal 25-hydroxyvitamin D [25(OH)D] and intact parathyroid hormone were measured at entry to care (mean
298 is defined by a high concentration of intact parathyroid hormone when circulating 25-hydroxyvitamin D
299 high parathyroid hormone and women with high parathyroid hormone whose 25(OH)D concentrations were >2
300 actor 23 was, on average, lower than that of parathyroid hormone with greater variability across indi