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1 pothalamic nuclei (dorsomedial, lateral, and paraventricular nuclei).
2 ons in the hypothalamic supraoptic (SON) and paraventricular nuclei.
3 l amygdala but had normal c-fos responses in paraventricular nuclei.
4 bral cortex, and hypothalamic supraoptic and paraventricular nuclei.
5 ted in the hypothalamic supraoptic (SON) and paraventricular nuclei.
6 lar cells (MNCs) in the supraoptic (SON) and paraventricular nuclei.
7 ons including the arcuate, ventromedial, and paraventricular nuclei.
8 amin D regulates glucose homeostasis via the paraventricular nuclei and energy homeostasis via the ar
9 magnocellular neurones of the supraoptic and paraventricular nuclei and is released from the posterio
10 area (MBA), arcuate nucleus/median eminence, paraventricular nuclei and piriform cortex.
11 amic arcuate, ventromedial, dorsomedial, and paraventricular nuclei and the area postrema and the nuc
12 (the arcuate, ventromedial, dorsomedial, and paraventricular nuclei and the lateral area).
13  showed an intense phosphoCREB signal in the paraventricular nuclei and ventromedial hypothalamus in
14 ary, neurons in the hypothalamic arcuate and paraventricular nuclei, and catecholaminergic neurons in
15 terior hypothalamic nuclei, locus coeruleus, paraventricular nuclei, and the rostral ventral medulla,
16 ceptor (MC-R) populations in the arcuate and paraventricular nuclei are considered probable sites of
17 ates hypothalamic neurons of the arcuate and paraventricular nuclei are consistent with the results r
18 including the perifornical, dorsomedial, and paraventricular nuclei, as well as in the paraventricula
19      In the hypothalamus, the supraoptic and paraventricular nuclei demonstrated the densest AMGBi.
20 ed GPCR101 mRNA expression in supraoptic and paraventricular nuclei from late pregnancy to late lacta
21 num vasculosum of the lamina terminalis, and paraventricular nuclei in the forebrain, and the tractus
22 urocortin-ir perikarya in the supraoptic and paraventricular nuclei, in the central and periaqueducta
23 halamic NPY, particularly in the arcuate and paraventricular nuclei, may contribute to the altered fo
24 ntromedial nuclei, thalamic centromedial and paraventricular nuclei of dietary obese rats versus cont
25 -regulated in the microdissected arcuate and paraventricular nuclei of the fasted rat.
26 found a vasopressinergic projection from the paraventricular nuclei of the hypothalamus (PVN) to the
27 ic sites, including the central amygdala and paraventricular nuclei of the hypothalamus and thalamus.
28 n of Insr protein product in the arcuate and paraventricular nuclei of the hypothalamus, which was as
29 tion of the suprachiasmatic, supraoptic, and paraventricular nuclei of the hypothalamus.
30  magnocellular neurons in the supraoptic and paraventricular nuclei of the hypothalamus.
31 containing neurons within the supraoptic and paraventricular nuclei of the hypothalamus.
32 sely in the agranular insular cortex and the paraventricular nuclei of the thalamus and hypothalamus
33 ncipal circadian clock, and the reuniens and paraventricular nuclei of the thalamus, each independent
34 os expression in the oxytocin neurons of the paraventricular nuclei of WT but not HDC-KO mice.
35 ys (P<0.01) and c-fos mRNA expression in the paraventricular nuclei (P<0.01), but not in the suprachi
36 neurons were present in the infundibular and paraventricular nuclei, paraolfactory gyrus, posterior h
37 uclei (SON), and magnocellular region of the paraventricular nuclei (PVN) - areas previously shown to
38             Senktide also induced Fos in the paraventricular nuclei (PVN) and supraoptic nuclei (SON)
39 cleus (VMN) and parvocellular portion of the paraventricular nuclei (PVN) of glucose-infused rats sho
40                                       In the paraventricular nuclei (PVN) of the hypothalamus, DOC pr
41 ed (P<0.05) in both dorsal and median raphe, paraventricular nuclei (PVN), ventromedial nuclei (VMH),
42 pressin neurones of the supraoptic (SON) and paraventricular nuclei (PVN).
43 SFO), median preoptic (MnPO), supraoptic and paraventricular nuclei (SON and PVN), area postrema (AP)
44 n preoptic nucleus (MPN), the supraoptic and paraventricular nuclei (SON and PVN), but did not influe
45 icle (AV3V) region and in the supraoptic and paraventricular nuclei (SON and PVN).
46 he vasopressin neurons of the supraoptic and paraventricular nuclei (SON and PVN).
47 cluding the olfactory epithelium, bulb, peri/paraventricular nuclei, suprachiasmatic nucleus, arcuate
48  olfactory bulb, hypothalamic supraoptic and paraventricular nuclei, the medial habenula, and certain
49 s, including the hypothalamic supraoptic and paraventricular nuclei, the thalamic paraventricular nuc
50  neurones of the hypothalamic supraoptic and paraventricular nuclei to induce uterine contractions du
51 L-R in the lateroanterior, ventrolateral and paraventricular nuclei was significantly decreased in th