ライフサイエンスコーパス: partial epilepsy

1 in adult patients with medically intractable partial epilepsy.

2 izing the epileptogenic region in refractory partial epilepsy.

3 he presurgical evaluation of drug-refractory partial epilepsy.

4 indicative of the site of seizure origin in partial epilepsy.

5 nterictal SPECT images from 15 patients with partial epilepsy.

6 mporal areas following surgical treatment of partial epilepsy.

7 for at least part of the heritability of the partial epilepsies.

8 l electrodes in patients with drug-resistant partial epilepsies.

9 s the first-choice drug for the treatment of partial epilepsies.

10 to genetic susceptibility shared across the partial epilepsies.

11 of a consecutive series of 59 patients with partial epilepsy and a history of PIP, and 94 control pa

12 ed members in Family A (KCNA1 G724C) exhibit partial epilepsy and myokymia but no ataxic episodes, su

13 history of PIP, and 94 control patients with partial epilepsy and no history of PIP evaluated as inpa

14 ata were obtained, 96 had well characterized partial epilepsy and were enrolled, along with 27 contro

15 This disorder is the most common refractory partial epilepsy, and also the one most often treated su

16 erating seizures in patients with refractory partial epilepsy are referred to as the epileptogenic zo

17 onitoring with scalp EEG who had intractable partial epilepsy based on ictal electro-clinico-anatomic

18 eurotransmitter disturbances associated with partial epilepsy, detected and depicted (but not underst

19 oman with a history of medically intractable partial epilepsy, does not have an anatomical lesion but

20 sment, about 30% of patients with refractory partial epilepsy due to hippocampal sclerosis fail to be

21 isease risk, the role of common variation in partial epilepsies has not yet been explored in a well-p

22 Partial epilepsies have a substantial heritability.

23 individuals over three generations, two had partial epilepsy in addition to EA1.

24 10q24 may suggest it as a candidate gene for partial epilepsy in families where the disease has been

25 ight on mechanisms underlying a rare form of partial epilepsy in humans, autosomal dominant nocturnal

26 In partial epilepsies, interictal epileptic spikes (IESs) a

27 The genetic architecture of the partial epilepsies is likely to be very complex, reflect

28 e aim of this study was to determine whether partial epilepsy is associated with abnormalities in hip

29 PIP in partial epilepsy is associated with relatively broadly a

30 (b) an ictal SPECT image from a patient with partial epilepsy matched to itself after modification to

31 Temporal lobe epilepsy is the commonest partial epilepsy of adulthood.

32 TCI is demonstrable in many cases of benign partial epilepsy of childhood, a disorder once thought t

33 of a common form of human epilepsy, complex partial epilepsy of temporal lobe origin, has establishe

34 ances underlying and revealing mechanisms of partial epilepsy pathophysiology.

35 s taking gabapentin compared with 14 complex partial epilepsy patients, matched for antiepileptic dru

36 eatures (ADPEAF), an unusual inherited human partial epilepsy phenotype.

37 p brain stimulation of anterior thalamus for partial epilepsy recently was approved in Europe and sev

38 ne receptor subunit genes cosegregate with a partial epilepsy syndrome known as autosomal dominant no

39 h influence risk for epilepsy shared amongst partial epilepsy syndromes, in 3445 patients and 6935 co

40 usion are established localizing findings in partial epilepsy, their relative value is disputed.

41 RS and DBS in patients with refractory focal/partial epilepsy was conducted using PubMed and Cochrane

42 Seven patients with intractable partial epilepsy were studied.

43 have a modest role in predisposition to the partial epilepsies when considered across syndromes in E

44 ries of 20 adults with medically intractable partial epilepsy, where high-resolution brain MRI disclo

45 LGI1 have been linked to autosomal dominant partial epilepsy with auditory features (ADPEAF), an unu

46 e for a predisposition to autosomal dominant partial epilepsy with auditory features (ADPEAF).

47 of the causative gene in autosomal-dominant partial epilepsy with auditory features (ADPEAF, MIM 600

48 as adjuvant therapy in the US and Europe for partial epilepsy with or without secondary generalizatio

49 A key feature of partial epilepsy with pericentral spikes is a characteri

50 we describe a new family epilepsy syndrome, partial epilepsy with pericentral spikes, which we map t