ライフサイエンスコーパス: paternally-expressed gene

1 ene, rather than from absent expression of a paternally expressed gene.

2 r-Willi syndrome results from the absence of paternally expressed genes.

3 y expressed genes and repression of silenced paternally expressed genes.

4 for bidirectional activation of a number of paternally expressed genes.

5 cal crosses, resembling either maternally or paternally expressed genes.

6 idespread relaxation of imprinting of mostly paternally expressed genes.

7 olute carrier family 38 member 4), and Peg1 (paternally expressed gene 1)--both MBD1 and H3K9me3 bind

8 actor receptor-bound protein 10), Peg1/Mest (paternally expressed gene 1/mesoderm-specific transcript

9 The retrotransposon-derived paternally expressed gene 10 (PEG10) protein is ordinari

10 es the domesticated gag-pol retrotransposon 'paternally expressed gene 10 (PEG10)' in human cells and

11 teins, retrotransposon gag-like 8 (RTL8) and paternally expressed gene 10 (PEG10).

12 The product of the imprinted gene paternally expressed gene-10 (PEG10) has been reported t

13 -/- cells showed aberrant methylation of the paternally expressed gene 3 (Peg3) tumor suppressor gene

14 Notably, allelic imbalance in paternally expressed gene 3 (PEG3) was associated with i

15 tment of breast carcinoma xenografts induces paternally expressed gene 3 (Peg3), an imprinted gene en

16 We found that decorin induced paternally expressed gene 3 (Peg3), an imprinted tumor s

17 hin 3 maternally methylated imprinted genes: paternally expressed gene 3 (PEG3), insulin-like growth

18 mTOR, but requires an autophagic regulator, paternally expressed gene 3 (PEG3).

19 receptor 2 (VEGFR2) signaling that requires paternally expressed gene 3 (PEG3).

20 ICR (Imprinting Control Region) of the Peg3 (Paternally Expressed Gene 3) domain contains an unusual

21 ation status of the CpG islands of the PEG3 (Paternally expressed gene 3) imprinted domain in the mou

22 Peg3 (paternally expressed gene 3) is an imprinted gene locali

23 Peg3 (paternally expressed gene 3) is the first imprinted gene

24 (H19 fetal liver mRNA), p57(Kip2), Peg3/Pw1 (paternally expressed gene 3), and Zac1 (zinc finger-bind

25 d domain), located 25 kb downstream of PEG3 (paternally expressed gene 3).

26 ithin 30 kb of a known imprinted gene, Peg3 (paternally expressed gene 3).

27 ssed genes, activated expression of silenced paternally expressed genes and resulted in methylation o

28 er-Willi syndrome (PWS) is caused by loss of paternally expressed genes at an imprinted locus on chro

29 oter regions and transcriptional termini and paternally expressed genes at promoters and gene bodies,

30 Imprinted expression of both maternally and paternally expressed genes changes in F(1) hybrid with t

31 IPW, a paternally expressed gene cloned from this region, is no

32 d to decrease the length of gestation, while paternally expressed genes extend the length of gestatio

33 ernally expressed genes (Igf2r, H19) and two paternally expressed genes (Igf2, U2af1-rs1) in ES cells

34 Mash2, Kvlqt1, and p57(Kip2), as well as two paternally expressed genes, Igf2 and Ins2, and assess th

35 e previously identified Peg1/Mest as a novel paternally expressed gene in the developing mouse embryo

36 sults from loss of function of 10 clustered, paternally expressed genes in a 1.5-Mb region of chromos

37 -Willi syndrome (PWS) is caused by a loss of paternally expressed genes in an imprinted region of chr

38 y the inactivation or deletion of imprinted, paternally expressed genes in chromosome band 15q11.2.

39 nomic alterations that inactivate imprinted, paternally expressed genes in human chromosome region 15

40 genetic disorder caused by the deficiency of paternally expressed genes in the chromosome 15q11-q13.

41 (PWS) is caused by the loss of expression of paternally expressed genes in the human 15q11.2-q13 impr

42 enomic disorder resulting from deficiency of paternally expressed genes in the human chromosome 15q11

43 the two parental alleles of SNRPN and other paternally expressed genes in the region by using a chro

44 Transcription of paternally expressed genes in the region depends upon an

45 portant for regulation of SNRPN and of other paternally expressed genes in the region.

46 e required for the genomic imprinting of two paternally expressed genes, Ins-2 (encodes insulin-2) an

47 The paternally expressed gene insulin-like growth-factor 2 (

48 MAGEL2 is a maternally imprinted, paternally expressed gene, located in the Prader-Willi r

49 The paternally expressed genes NDN, IPW, PWCR1 and MAGEL2 we

50 determined that Pw1 is identical to Peg3, a paternally expressed gene of unknown function (and will

51 Surprisingly, paternally-expressed genes of the non-classical gene imp

52 PWS is caused by the loss of one or more paternally expressed genes on chromosome 15q11-q13, whic

53 osome 6q, believed to contain an exclusively paternally expressed gene or genes that cause transient

54 d the expression and methylation status of a paternally expressed gene Peg3, in germ cells from sex-r

55 stigate the role of the maternally imprinted/paternally expressed gene, Peg3, in several aspects of b

56 wn (CypA-KD) P19 cells, we observed a silent paternally expressed gene, Peg3.

57 mplete abolition of the transcription of two paternally expressed genes, Peg3 and Usp29, causing the

58 In contrast, no paternally expressed genes (PEGs) are DE.

59 ressed genes (MEGs) and approximately 29% of paternally expressed genes (PEGs) in C. rubella were com

60 associated with DNA methylation-independent paternally expressed genes (PEGs) in human morulae.

61 PWS patients lack the expression of paternally expressed genes (PEGs) in the 15q11-13 locus

62 the mechanism by which PWS-IC activates the paternally expressed genes (PEGs) using transgenes that

63 ic crosses, whereas approximately 90% of 272 paternally expressed genes (PEGs) were found only in one

64 plant Arabidopsis thaliana, most notably the paternally expressed genes (PEGs), which are upregulated

65 162 maternally expressed genes (MEGs) and 95 paternally expressed genes (PEGs), which were associated

66 and H3K36me3 peaks mostly co-localized with paternally expressed genes (PEGs), while endosperm-speci

67 54 maternally expressed genes (MEGs) and 46 paternally expressed genes (PEGs).

68 This disorder is caused by the absence of paternally expressed gene products from chromosome 15q11

69 mprinting disorder caused by a deficiency of paternally expressed gene(s) in the 15q11-q13 chromosoma

70 ched for coexpressed pairs of maternally and paternally expressed genes, showed accelerated expressio

71 LK1 (delta, Drosophila, homolog-like 1) is a paternally expressed gene that encodes for a transmembra

72 located adjacent to a cluster of imprinted, paternally expressed genes that are known to be positive

73 The largest proportion of paternally expressed genes was at 7 DAP, mainly due to t

74 PWS involves loss of function of multiple paternally expressed genes, while mutations in a single

75 in the pituitary gland, among them, Dlk1, a paternally expressed gene whose membrane-bound and secre