ライフサイエンスコーパス: pathogen-host interaction

1 refore may be useful tools in evaluating the pathogen-host interaction.

2 ree hosts, indicating the uniqueness of each pathogen-host interaction.

3 highlight the functionality of paraCell for pathogen-host interaction.

4 changes in cellular architecture that favor pathogen-host interactions.

5 of cellular targets that enhances subsequent pathogen-host interactions.

6 facilitate a comprehensive understanding of pathogen-host interactions.

7 ion dynamics, expanding our understanding of pathogen-host interactions.

8 o be linked to key cellular processes and/or pathogen-host interactions.

9 tly been demonstrated to play vital roles in pathogen-host interactions.

10 hat serve as tractable models for studies of pathogen-host interactions.

11 antigen expression and morphology following pathogen-host interactions.

12 affeensis provide an important interface for pathogen-host interactions.

13 e bacterial cell and the nature of bacterial pathogen-host interactions.

14 at was prominent in proteins associated with pathogen-host interactions.

15 the host cell may provide new insights into pathogen-host interactions.

16 regulation of gene expression, virulence and pathogen-host interactions.

17 to understanding colonization, movement, and pathogen/host interactions.

18 een successfully applied for (i) identifying pathogen-host interactions and (ii) predicting near-resi

19 understand the function of glycoproteins in pathogen-host interactions and cancer progression.

20 ental tissue and are therefore used to study pathogen-host interactions and other complex biological

21 generally applied to investigate additional pathogen-host interactions and to provide mechanistic in

22 lays the foundation for rational studies on pathogen-host interactions and vaccine development.

23 to reveal insights into new virulence genes, pathogen-host interactions, and the molecular basis of h

24 The studies reported here suggest that pathogen-host interactions are finely orchestrated by ET

25 ies, and enemy-victim (e.g. predator-prey or pathogen-host) interactions are particularly common.

26 ocesses, such as transmembrane signaling and pathogen-host interactions, are initiated by a protein r

27 far been used exclusively for investigating pathogen-host interactions, but they should be easily ad

28 ology, and controlled vocabularies to curate pathogen-host interaction data, at the level of the host

29 The pathogen-host interaction database (PHI-base) is a web-a

30 Using the Pathogen-Host Interaction database (PHI-base), our previ

31 ed proteins and secondary metabolism and the pathogen-host interaction database genes are highly enri

32 ies interactions, using data curated for the Pathogen-Host Interactions database (PHI-base) as a case

33 The Pathogen-Host Interactions database (PHI-base) catalogue

34 Since 2005, the Pathogen-Host Interactions Database (PHI-base) has manua

35 The Pathogen-Host Interactions Database (PHI-base) has, sinc

36 To elucidate pathogen-host interactions during early Lyme disease, we

37 A molecular understanding of this pathogen-host interaction has potential to inform the de

38 ed effectors (T3SEs) play important roles in pathogen-host interactions, identifying them is crucial

39 outcomes, but the interpretive complexity of pathogen-host interactions impedes identification of cri

40 y responders to infection that contribute to pathogen-host interactions in diverse ways.

41 al proteases, expanding the understanding of pathogen-host interactions in immune responses, specific

42 esents an innovative approach to investigate pathogen-host interactions in multiple airway conditions

43 treatments and may form a basis for modeling pathogen-host interactions in other emerging infectious

44 e structures provide a remarkable example of pathogen-host interactions in which a unique microbial m

45 An elucidation of such pathogen-host interaction, including determination of th

46 eplication, and the role of these regions in pathogen-host interactions is discussed.

47 This initial pathogen/host interaction is efficiently antagonized by

48 To facilitate broader exploration of how pathogen-host interactions might impact transmission and

49 echanisms that have been identified by which pathogen-host interactions might influence rejection, in

50 in the mga promoter significantly alters the pathogen-host interaction of these asymptomatic carrier

51 dicate that we need to better understand the pathogen-host interactions of persister MRSAs in vivo.

52 in vitro platform for investigating enteric pathogen-host interactions, particularly for pathogens l

53 Recent insights into pathogen-host interactions, pathogenesis, inflammatory p

54 Because genes modulated by pathogen-host interactions potentially encode putative v

55 e maintaining orthologs of most known fungal pathogen-host interaction proteins, stress response circ

56 ession patterns characterizing each phase of pathogen-host interaction provides avenues for targeted

57 ion on genes proven to affect the outcome of pathogen-host interactions reported in peer reviewed res

58 abundance during human infections, mitigate pathogen-host interactions, scavenge free ETEC toxins, a

59 Due to their relevance in pathogen-host interactions, significant computational ef

60 Here, we identify a novel pathogen-host interaction that promotes gut inflammation

61 mucins at epithelial surfaces to facilitate pathogen-host interactions that culminate in toxin deliv

62 of these results for modelling evolution in pathogen-host interactions that lack gene-for-gene deter

63 resent within the same bat and the different pathogen-host interactions that may regulate the presenc

64 We have highlighted an emerging principle in pathogen-host interactions: that the cytokine repertoire

65 in (Kgp) virulence factor is involved in the pathogen-host interaction through the production of cyto

66 hat CEACAMs play a multifaceted role in ETEC pathogen-host interactions, transiently favoring the pat

67 enient model to study type IV pilus-mediated pathogen-host interactions under physiological condition

68 rkedly expanded our understanding of the key pathogen-host interactions underlying GAS necrotizing fa

69 Genes associated with pathogen-host interactions were identified, including a