ライフサイエンスコーパス: pauci-immune

1 y ANCA-GN, but significantly less frequently pauci-immune (77 vs. 100%) and more commonly associated

2 ls that we termed "hyper-immune CD20Hi" and "pauci-immune CD20Lo." Of note, subjects had only one inf

3 Pauci-immune crescentic glomerulonephritis (PICGN) is a

4 found that a dose of 1600 microg/kg induced pauci-immune crescentic glomerulonephritis and lung hemo

5 Renal biopsy demonstrated a pauci-immune crescentic glomerulonephritis with 14 cellu

6 sis (not collapsing glomerulopathy variant), pauci-immune crescentic glomerulonephritis, acute inters

7 dings of thrombotic microangiopathy, one had pauci-immune crescentic GN, and another had global as we

8 ct phenotypes based on cellular composition (pauci-immune, diffuse and lymphoid), suggesting that dis

9 Pauci-immune focal necrotizing GN (piFNGN) is usually as

10 Thus, anti-MPO IgG alone was able to cause pauci-immune glomerular necrosis and crescent formation

11 a: prior diagnosis of ANCA-GN (biopsy proven pauci-immune glomerulonephritis) and positive serology f

12 ti-neutrophil cytoplasmic antibodies-related pauci-immune glomerulonephritis.

13 eutrophil cytoplasmic antibodies-associated (pauci-immune) glomerulonephritis, anti-glomerular baseme

14 pecific disease entities: immune-complex GN, pauci-immune GN, antiglomerular basement membrane GN, mo

15 ial who had renal involvement (biopsy proven pauci-immune GN, red blood cell casts in the urine, and/

16 antineutrophil cytoplasmic antibody-positive pauci-immune GN.

17 atients, high AXL levels are associated with pauci-immune histology and low disease activity and inve

18 culating anti-MPO resulted in development of pauci-immune NCGN in all mice and pulmonary capillaritis

19 perimental evidence indicate that ANCA cause pauci-immune necrotizing and crescentic glomerulonephrit

20 strongly associated with the development of pauci-immune necrotizing and crescentic glomerulonephrit

21 lation of approximately 80% of patients with pauci-immune necrotizing and crescentic glomerulonephrit

22 tis), the Churg-Strauss syndrome, idiopathic pauci-immune necrotizing and crescentic glomerulonephrit

23 Excluding pauci-immune necrotizing and crescentic glomerulonephrit

24 tinct forms of small vessel vasculitides and pauci-immune necrotizing glomerulonephritis.

25 y fatal autoimmune disorder characterized by pauci-immune necrotizing vasculitis affecting small- to

26 Anti-PR3 treated mice had mild pauci-immune proliferative glomerulonephritis, with infi

27 s of plasma exchange in patients with AAV or pauci-immune rapidly progressive glomerulonephritis and

28 polyangiitis (EGPA), that share features of pauci-immune small-vessel vasculitis and the positivity

29 In populations pauci-immune to malaria, risk of severe malaria increase