ライフサイエンスコーパス: pentose

1 sugar enones (dihydropyranones) derived from pentoses.

2 d for seven out of eight possible isomers of pentoses.

3 f a variety of C-nucleosides and fluorinated pentoses.

4 nic acids) to that of the simple ring-opened pentoses.

5 l hexose-pentoses, and monoterpendiol hexose-pentoses.

6 ation of the C(5) osone, D-xylosone (D-threo-pentose-2-ulose), showing that this transposition may be

7 tic oligomannose structures, the presence of pentose and deoxyhexose residues on their N-glycans.

8 and aldarate metabolism (P=9.0x10(-6)), and pentose and glucuronate interconversions (P=3.0x10(-6))

9 phenylpropanoid and flavonoid biosynthesis, pentose and glucuronate interconversions and starch and

10 of microorganisms to efficiently co-ferment pentose and hexose sugars, especially glucose and xylose

11 site and prevents release of the UDP-4-keto-pentose and NADH intermediates.

12 etric amounts of C2 metabolites from hexose, pentose and triose phosphates without carbon loss.

13 monoterpenol glucosides, monoterpenol hexose-pentoses, and monoterpendiol hexose-pentoses.

14 and purification of nine SIL-IS for hexose-, pentose-, and triose-phosphates, UDP-glucose, and adenos

15 due to the relative stability of diglycoside pentose anthocyanins.

16 Specifically, the 12 pentoses are much more structurally similar to one anoth

17 Pentoses are readily converted at room temperature, whil

18 confirm the existence of an SBP pathway for pentose assimilation in cellulolytic clostridia.

19 h on xylose, demonstrating its relevance for pentose assimilation.

20 atalyze retro-aldol reactions of hexoses and pentoses at these moderate temperatures, are shown to be

21 re and smaller energetic differences between pentose conformations than between hexose conformations.

22 inor reaction products such as furfural from pentose dehydration.

23 fficient route towards biologically relevant pentose derivatives is described.

24 de range of oligosaccharidic combinations of pentose, hexose, deoxyhexose and hexuronic acid were acc

25 Many components of pentoses, hexoses and disaccharides were identified usin

26 cid of lipid components.) Many components of pentoses, hexoses and disaccharides were identified usin

27 olyol derived from xylose, the most abundant pentose in lignocellulosic biomass.

28 ides and arabinose, the second most abundant pentose in nature.

29 Here, we show that non-fermentable pentoses inhibit growth and end-product formation during

30 UDP-4-keto-D-glucuronic acid and UDP-4-keto-pentose intermediates.

31 outes to convert biomass-derived hexoses and pentoses into valuable C2, C3, and C4 products such as g

32 hexose isomers, the chiral separation of the pentose isomers was significantly more challenging.

33 solute configuration determination of all 12 pentose isomers, including the d and l enantiomers for a

34 ously described xylans, this xylan carries a pentose linked 1-2 to the alpha-1,2-d-glucuronic acid (G

35 s, they must possess a pathway that connects pentose metabolism with the rest of metabolism.

36 ified by the addition of essential genes for pentose metabolism.

37 eals a significant increase in the levels of pentose monophosphate during hypoxia, which provides ins

38 ions, revealed that the allosteric inhibitor pentose monophosphate increases PYK structural dynamics,

39 Here we show that the competitive binding of pentose monophosphate inhibitors or the activator glucos

40 Intriguingly, pentose monophosphates were found to share the same bind

41 pplied to the identification of any isolated pentose monosaccharide using only microgram quantities a

42 ids, which together sustain the nonoxidative pentose pathway for purine synthesis.

43 stasize even in the absence of the oxidative pentose pathway, a main NADPH production route.

44 stem and its main reducing power source, the pentose pathway, as demonstrated by a 50 and 70% drop in

45 H) activity, the key enzyme of the oxidative pentose pathway.

46 ed by one or two CQAs covalently linked with pentose (Pent) residues (1-12) were identified, along wi

47 xtracellular matrix remodeling and/or aging, pentoses/pentitols, glycolytic intermediates, and lipid

48 s a cyclic route that includes the oxidative pentose phosphate (OPP) pathway and the glucose-6-phosph

49 rhof-Parnas (EMP) pathway] and the oxidative pentose phosphate (OPP) pathway.

50 -Benson-Bassham (CBB) cycle, glycolysis, the pentose phosphate (PP) pathway and the tricarboxylic aci

51 e Entner-Doudoroff pathway and the oxidative pentose phosphate (PP) pathway.

52 ose catabolic pathways including glycolysis, pentose phosphate (PP), glycogenolysis, and polyols to t

53 irects the glycolytic intermediates into the pentose phosphate (PPP) and serine pathways.

54 hich may increase metabolic flux through the pentose phosphate (PPP) pathway, enabling increased nucl

55 glycolysis, in association with a decreased pentose phosphate activity and an increased ATP producti

56 in diabetic CPCs showed dysregulation of the pentose phosphate and glycero(phospho)lipid synthesis pa

57 Glycolysis shifted towards the pentose phosphate and glycerol-3-phosphate dehydrogenase

58 ndance of enzymes in the glycolytic pathway (pentose phosphate and pyruvate metabolism) was further v

59 of compounds, multiple metabolites from the pentose phosphate and tryptophan metabolic pathways were

60 after PH, and glucose use was shifted to the pentose phosphate cycle and oxidative metabolism.

61 ogenesis and reduced glucose use through the pentose phosphate cycle and oxidative metabolism.

62 egulation of glycolysis and the nonoxidative pentose phosphate cycle.

63 tradiol led to a significant upregulation in pentose phosphate flux and glycolytic intermediates in H

64 lycolysis by increasing glutamine uptake and pentose phosphate flux to generate energy and biomass.

65 s with HKCs showing consistently lower basal pentose phosphate flux.

66 Genes involved in glycolysis (PFKP), pentose phosphate pathway (G6PD), and defense against li

67 xamined the role of the plastidial oxidative pentose phosphate pathway (OPPP) in embryo development.

68 showing that HEXOKINASE1-mediated oxidative pentose phosphate pathway (OPPP) metabolism is required

69 rk on the irreversible part of the oxidative pentose phosphate pathway (OPPP) revealed comparable eff

70 Fluxes through the oxidative pentose phosphate pathway (OPPP) were the principal sour

71 The oxidative pentose phosphate pathway (oxiPPP) contributes to cell m

72 that inhibition of the oxidative arm of the pentose phosphate pathway (oxPPP) is required for antima

73 a dependence on the oxidative branch of the pentose phosphate pathway (oxPPP), and oxPPP inhibition

74 A sudden increase in pentose phosphate pathway (PPP) activity, the fastest kn

75 dehydrogenase (G6PD) is a key enzyme in the pentose phosphate pathway (PPP) and plays an essential r

76 R-ABL-ERK signaling in H929 cells drives the pentose phosphate pathway (PPP) and RNA biosynthesis, wh

77 analysis identified the up-regulation of the pentose phosphate pathway (PPP) and the antioxidant defe

78 two cytosolic NADPH-producing pathways, the pentose phosphate pathway (PPP) and the NADP-dependent m

79 ADP(+)/NADPH ratio controls flux through the pentose phosphate pathway (PPP) and the polyol pathway,

80 and miR-206 to direct carbon flux toward the pentose phosphate pathway (PPP) and the tricarboxylic ac

81 d and carbohydrate metabolites, particularly pentose phosphate pathway (PPP) and tricarboxylic acid (

82 d the flux allocation between glycolysis and pentose phosphate pathway (PPP) as potential mechanisms

83 al crosstalk between the PI3K/AKT signal and pentose phosphate pathway (PPP) branching metabolic path

84 The importance of the pentose phosphate pathway (PPP) during S. aureus infecti

85 domestic livestock that exclusively use the pentose phosphate pathway (PPP) for hexose catabolism, w

86 y mediating fluxes through glycolysis or the pentose phosphate pathway (PPP) in an oxidative stress-d

87 increased glucose flux through the oxidative pentose phosphate pathway (PPP) in erythrocytes.

88 Survival upon nutrient stress or pentose phosphate pathway (PPP) inhibition depends on co

89 The oxidative pentose phosphate pathway (PPP) is crucial for cancer ce

90 emonstrate that a metabolic shift toward the pentose phosphate pathway (PPP) is necessary for NET rel

91 Here we add the pentose phosphate pathway (PPP) of T. brucei to the glyc

92 The pentose phosphate pathway (PPP) plays a critical role in

93 on increases the flux of glucose through the pentose phosphate pathway (PPP) to increase nucleotide p

94 -phosphogluconate dehydrogenase (PGD) in the pentose phosphate pathway (PPP) were found to be the NAD

95 6PD) is the first, rate-limiting step in the pentose phosphate pathway (PPP), a key metabolic pathway

96 lucose-dependent nucleotide synthesis by the pentose phosphate pathway (PPP), along with sphingolipid

97 atabolic glycolysis but increased use of the pentose phosphate pathway (PPP), and an enhanced abundan

98 rythrose-4-phosphate, an intermediate of the pentose phosphate pathway (PPP), generates 4-phosphoeryt

99 sses and pathways, including glycolysis, the pentose phosphate pathway (PPP), KCl cotransport, ATP re

100 The pentose phosphate pathway (PPP), metabolism in the citri

101 revealed that metabolites in the glycolysis, pentose phosphate pathway (PPP), pyrimidine biosynthesis

102 potential by shunting nectar glucose to the pentose phosphate pathway (PPP), resulting in a reductio

103 s revealed that glucose is funneled into the pentose phosphate pathway (PPP), which is indispensable

104 sphogluconate dehydrogenase in the oxidative pentose phosphate pathway (PPP), while 2-PG activates 3-

105 e metabolism and increased NADPH levels in a pentose phosphate pathway (PPP)-dependent manner.

106 lase (TKT), a key enzyme of the nonoxidative pentose phosphate pathway (PPP).

107 cription factors or increasing NADPH via the pentose phosphate pathway (PPP).

108 with H2O2 rapidly induces glycolysis and the pentose phosphate pathway (PPP).

109 glycolysis and especially the non-oxidative pentose phosphate pathway (PPP).

110 dative pathways and glucose flux through the pentose phosphate pathway (PPP).

111 -5-phosphate with NADP(+) as cofactor in the pentose phosphate pathway (PPP).

112 a cellular anti-oxidant response through the pentose phosphate pathway (PPP).

113 cose flux towards glycolysis relative to the pentose phosphate pathway (PPP).

114 sm in CDCP1+ CSCs is routed to the oxidative pentose phosphate pathway (PPP); multiple cycling of car

115 C-null strains show indications of oxidative pentose phosphate pathway activation as well as increase

116 Fasting stimulated pentose phosphate pathway activity and metabolism of [U-

117 etylglucosamine, S. marcescens exhibits high pentose phosphate pathway activity and nucleotide synthe

118 NADPH, a substrate for NOX, and pentose phosphate pathway activity increased with glucos

119 nic state, cells exhibiting a contrary, high pentose phosphate pathway activity state, spontaneously

120 ose uptake, glucose metabolism and oxidative pentose phosphate pathway activity were similarly repres

121 tabolites provides an important biomarker of pentose phosphate pathway activity, triacylglycerol synt

122 scription factors repress glucose uptake and pentose phosphate pathway activity, while their low numb

123 sponse in the clinic and of monitoring tumor pentose phosphate pathway activity.

124 cose and demonstrated reduced glycolysis and pentose phosphate pathway activity.

125 d an increase in the oxidative branch of the pentose phosphate pathway and (13)C incorporations sugge

126 fluxes in the pentose phosphate pathway and gluconeogenesis were stabl

127 th a shift in glucose metabolism between the pentose phosphate pathway and glycolysis, (2) interactio

128 between the oxidative steps of the oxidative pentose phosphate pathway and glycolysis.

129 ection against hydrogen peroxide insult in a pentose phosphate pathway and GSH-dependent manner.

130 inhibition of MCT1 suppressed the oxidative pentose phosphate pathway and increased levels of reacti

131 nsated by activation of anabolic metabolism (pentose phosphate pathway and lipogenesis) allowing live

132 glucose is preferentially metabolized by the pentose phosphate pathway and mitochondria, as opposed t

133 glutathione biosynthesis or in the oxidative pentose phosphate pathway and other NADPH-producing enzy

134 of genes associated with photosynthesis, the pentose phosphate pathway and primary metabolism, but lo

135 We identified the pentose phosphate pathway and pyrimidine metabolism - bo

136 gene deletion mutants from upper glycolysis, pentose phosphate pathway and the Entner-Doudoroff pathw

137 nd fatty acid-utilizing, upregulation of the pentose phosphate pathway as a source of antioxidant NAD

138 ase step, promoting carbon overflow into the pentose phosphate pathway as evidenced by the increased

139 s of NADPH reflecting a higher activation of pentose phosphate pathway as this is accompanied with hi

140 nclude up-regulated NADPH production via the pentose phosphate pathway as well as activation of the N

141 TORC1 directs increased glucose flux via the pentose phosphate pathway back into glycolysis, thereby

142 Glycolysis and the pentose phosphate pathway both play a central role in th

143 Inhibition of the pentose phosphate pathway by glucose-6-phosphate dehydro

144 Adiponectin also reversed induction of the pentose phosphate pathway by HFD.

145 ne phosphate and fructose-1,6-bisphosphate), pentose phosphate pathway components and Kreb's cycle in

146 Further, pentose phosphate pathway deregulation and impaired fatt

147 uiescent mutant 1 (NQM1), a paralogue to the pentose phosphate pathway enzyme transaldolase (TAL1), a

148 3.6% of glycolysis, and three non-oxidative pentose phosphate pathway exchange fluxes were calculate

149 is upon Gpi1 inactivation was compensated by pentose phosphate pathway flux and increased mitochondri

150 The oxidative pentose phosphate pathway flux was 3.6% of glycolysis, a

151 associated with a corresponding increase in pentose phosphate pathway flux, assessed using (13)C-lab

152 owever, LPS also induced a small decrease in pentose phosphate pathway fluxes and an increase in glut

153 e identified increased citric acid cycle and pentose phosphate pathway fluxes as consistent markers o

154 ol g(-1)h(-1) that was partly caused by high pentose phosphate pathway fluxes.

155 Embden-Meyerhof-Parnas, Entner-Doudoroff, or pentose phosphate pathway for glycolytic carbon metaboli

156 nd metabolomics revealed that glycolytic and pentose phosphate pathway genes are induced by dERR, and

157 Loss of the pentose phosphate pathway had no effect on plaque format

158 rains cell proliferation, glycolysis and the pentose phosphate pathway in a catalytic-activity-indepe

159 x from glucose-6-phosphate (G6P) through the pentose phosphate pathway in egg extracts maintains NADP

160 dogenously produced from glucose through the pentose phosphate pathway in humans.

161 te) was associated with perturbations in the pentose phosphate pathway induced initially by colistin

162 the two-step conversion of glucose into the pentose phosphate pathway intermediate 6-phosphogluconat

163 sphate cyclases are enzymes that utilize the pentose phosphate pathway intermediate, sedoheptulose 7-

164 chment of Embden-Meyerhof-Parnas pathway and pentose phosphate pathway intermediates indicated high a

165 ted glucose uptake and metabolism, increased pentose phosphate pathway intermediates, with a complime

166 rangements in the nonoxidative branch of the pentose phosphate pathway involving transaldolase that p

167 in the endosperm and suggest the amyloplast pentose phosphate pathway is a heat-sensitive step in ma

168 The pentose phosphate pathway is a key route of glucose meta

169 The pentose phosphate pathway is a major site of NADPH produ

170 er these conditions, NADPH generation by the pentose phosphate pathway is impaired, but AMPK induces

171 Although the oxidative pentose phosphate pathway is important for tumor growth,

172 establish that the carbon overflow into the pentose phosphate pathway is mainly through its non-oxid

173 ial amounts of NADPH and do so even when the pentose phosphate pathway is operational.

174 ysis, and the Krebs cycle, but the levels of pentose phosphate pathway metabolites and of many free a

175 in carbohydrate or hydroxyl acid metabolism, pentose phosphate pathway metabolites, or free fatty aci

176 f the carbon flux between glycolysis and the pentose phosphate pathway or the Kennedy pathway, respec

177 rnative route for directing glucose into the pentose phosphate pathway that bypasses hexokinase and t

178 tes shunting of glucose-6-phosphate into the pentose phosphate pathway to generate reduced glutathion

179 hosphate is then converted by enzymes of the pentose phosphate pathway to glyceraldehyde 3-phosphate

180 ydrogenase, the TCA cycle, and the oxidative pentose phosphate pathway to obtain NADPH.

181 ,6-bisphosphatase, potentially promoting the pentose phosphate pathway to produce NADPH for antioxida

182 pression of lipogenesis, glycolysis, and the pentose phosphate pathway triggered a strong growth rest

183 showed that: (i) flux through the oxidative pentose phosphate pathway varied independently of the re

184 Activation of the pentose phosphate pathway was identified from the list o

185 and ribulose 5-phosphate 3-epimerase) in the pentose phosphate pathway were overexpressed, and a gera

186 bstrates, such as sedoheptulose-6-phosphate (pentose phosphate pathway) and serine and glycine (1-car

187 te elevated activities of glycolysis and the pentose phosphate pathway, activation of macrophages wit

188 ncluding glycerol oxidation, glycolysis, the pentose phosphate pathway, and carbon sources of stored

189 ses including fatty acid esterification, the pentose phosphate pathway, and gluconeogenesis through t

190 lism, reactive oxygen species clearance, the pentose phosphate pathway, and glutathione homeostasis.

191 way, nucleotide sugars, intermediates of the pentose phosphate pathway, and lipogenesis, including pr

192 through up-regulation of glucose influx, the pentose phosphate pathway, and NAD salvaging pathways.

193 for the complete non-oxidative phase of the pentose phosphate pathway, and others predicted to media

194 rom erythrose 4-phosphate, generated via the pentose phosphate pathway, and phosphoenolpyruvate.

195 tion of glucose flux between glycolysis, the pentose phosphate pathway, and serine biosynthesis seems

196 r phosphates that constitute glycolysis, the pentose phosphate pathway, and the RNA and DNA backbone,

197 ent decrease in flux through glycolysis, the pentose phosphate pathway, and the tricarboxylic acid (T

198 LC7A11-associated cystine metabolism and the pentose phosphate pathway, and uncover an accompanying m

199 lites of glycolysis, TCA cycle, amino acids, pentose phosphate pathway, and urea cycle, from LRMS and

200 Its major source is considered to be the pentose phosphate pathway, but cytosolic NADP(+)-depende

201 te of the cells with regard to the oxidative pentose phosphate pathway, Calvin cycle, tricarboxylic a

202 tration of metabolites of glycolysis and the pentose phosphate pathway, central metabolic players in

203 Idn1 act together to shunt glucose into the pentose phosphate pathway, creating an alternative route

204 down and mitochondrial processing toward the pentose phosphate pathway, favoring anabolic over catabo

205 lytic RA T cells diverted glucose toward the pentose phosphate pathway, generated more NADPH, and con

206 d zwf1, which encode the first enzyme in the pentose phosphate pathway, have a more severe growth phe

207 Fru-2,6-BPase), and through promotion of the pentose phosphate pathway, increases NADPH production to

208 ugh lack of GLUT1 blunted glycolysis and the pentose phosphate pathway, MPhi were metabolically flexi

209 products of glycolysis, the Krebs cycle, the pentose phosphate pathway, nucleobases, UDP-sugars, glyc

210 luding glucose and glutamine metabolism, the pentose phosphate pathway, nucleotide and fatty acid bio

211 abolites revealed a reduction in glycolysis, pentose phosphate pathway, polyamines and nucleotides, b

212 nase (G6PD), the key enzyme of the oxidative pentose phosphate pathway, provides reducing equivalents

213 rate metabolism (glycolysis/gluconeogenesis, pentose phosphate pathway, pyruvate metabolism), proteas

214 but instead of increasing the glycolysis and pentose phosphate pathway, the glucose is shunted throug

215 While NQM1 appears not to function in the pentose phosphate pathway, the interplay of NQM1 with VH

216 creased concomitant with an induction of the pentose phosphate pathway, the primary source of de novo

217 ently in clinical trials, on glycolysis, the pentose phosphate pathway, the tricarboxylic acid (TCA)

218 vity and redirected glucose flux through the pentose phosphate pathway, thereby conferring a selectiv

219 ow increased flux through glycolysis and the pentose phosphate pathway, thereby establishing a critic

220 ase in TIGAR expression, which regulates the pentose phosphate pathway, treatment with the MUC1-C inh

221 ologs, RpiRc is a potential regulator of the pentose phosphate pathway, which also regulates RNAIII l

222 amental routes, glycolysis and the oxidative pentose phosphate pathway, which produces NADPH and the

223 th was potentiated by its suppression of the pentose phosphate pathway, which resulted in inhibition

224 xylic acid (TCA) cycle intermediates and the pentose phosphate pathway, which results in increased gl

225 produce NADPH from glucose is the oxidative pentose phosphate pathway, with malic enzyme sometimes a

226 as the transition from the glycolysis to the pentose phosphate pathway-a conserved first-line respons

227 and diversion of glucose metabolites to the pentose phosphate pathway.

228 metabolism in the non-oxidative part of the pentose phosphate pathway.

229 glucose-6-phosphate dehydrogenase or in the pentose phosphate pathway.

230 focused on the importance of glycolysis and pentose phosphate pathway.

231 asing dependence upon glutaminolysis and the pentose phosphate pathway.

232 iphosphatase 4 (PFKFB4), drives flux through pentose phosphate pathway.

233 y by glycolysis and to a minor extent by the pentose phosphate pathway.

234 sotope effect is only found in the reductive pentose phosphate pathway.

235 o glycolysis and to the oxidative arm of the pentose phosphate pathway.

236 te dehydrogenase (G6PDH), a regulator of the pentose phosphate pathway.

237 increase in glucose metabolic flux into the pentose phosphate pathway.

238 by high levels of nutrient flux through the pentose phosphate pathway.

239 tributor to cytosolic NADPH is the oxidative pentose phosphate pathway.

240 onance assigned to 6-phosphogluconate in the pentose phosphate pathway.

241 o alterations in glucose metabolized via the pentose phosphate pathway.

242 by the Calvin-Benson-Bassham (CBB) reductive pentose phosphate pathway.

243 trated defects in the oxidative phase of the pentose phosphate pathway.

244 metabolism, including several mRNAs from the pentose phosphate pathway.

245 pool and renders such cells dependent on the pentose phosphate pathway.

246 s the tricarboxylic acid (TCA) cycle and the pentose phosphate pathway.

247 se 6-phosphate and by intermediates from the pentose phosphate pathway.

248 ne metabolism, glutamate metabolism, and the pentose phosphate pathway.

249 disorders, we focused on the glycolytic and pentose phosphate pathways (GPPPs).

250 ediates into the hexosamine biosynthesis and pentose phosphate pathways (PPP).

251 The oxidative and nonoxidative pentose phosphate pathways and the ratio between them al

252 etabolic intermediates in the glycolytic and pentose phosphate pathways as well as abnormal mitochond

253 s to the less efficient Entner-Doudoroff and pentose phosphate pathways on algal-dominated reefs.

254 erating in a gluconeogenic fashion), and the pentose phosphate pathways to form an unforeseen metabol

255 volving tricarboxylic acid cycle, polyol and pentose phosphate pathways, leading to improved redox ba

256 nas, the Entner-Doudoroff, and the reductive pentose phosphate pathways.

257 : the Embden-Meyerhof, Entner-Doudoroff, and pentose phosphate pathways.

258 ion (glycolysis) pathways, as well as to the pentose phosphate shunt and the hexosamine biosynthetic

259 ke for aerobic glycolysis, coupling with the pentose phosphate shunt to facilitate biosynthesis of nu

260 ycolysis) with redox protection (through the pentose phosphate shunt).

261 is, and several glycolysis-related pathways (pentose phosphate, carbon fixation, aminoacyl-tRNA biosy

262 ynthesized endogenously from glucose via the pentose-phosphate pathway (PPP) in stable isotope-assist

263 allow glucose to be metabolized through the pentose-phosphate pathway (PPP), which regenerates NADPH

264 ificant increase in rate-limiting enzymes of pentose-phosphate pathway and 1-carbon metabolism in pos

265 a-dystroglycan, all consistent with enhanced pentose-phosphate pathway and 1-carbon metabolism that c

266 ppears to direct glycolytic metabolites into pentose-phosphate pathway and 1-carbon metabolism, which

267 oprotective and repair pathways, such as the pentose-phosphate pathway and 1-carbon metabolism, which

268 factor that may regulate serum urate via the pentose-phosphate pathway and MRPS7 and IDH2 that encode

269 acilitate metabolic reprogramming toward the pentose-phosphate pathway for achieving redox balance an

270 r supply from the Calvin cycle and oxidative pentose-phosphate pathway in primary metabolism to pheny

271 demonstrates an essential role of oxidative pentose-phosphate pathway reactions in peroxisomes, like

272 stressed cells protected Rpe and enabled the pentose-phosphate pathway to retain function.

273 pathways (the Embden-Meyerhof-Parnas or the pentose-phosphate pathway) and in dependence of Na(+) av

274 metabolites through the oxidative arm of the pentose-phosphate pathway.

275 idation via the tricarboxylic acid cycle and pentose-phosphate pathway.

276 nd is linked through its product Xu5P to the pentose-phosphate pathway.

277 iting enzyme in the nonoxidative part of the pentose-phosphate pathway.

278 ctate even in the presence of oxygen via the pentose-phosphate pathway.

279 drolytic enzymes and also enzymes key to the pentose-phosphate pathway.

280 upper Embden-Meyerhof-Parnas pathway nor the pentose-phosphate pathway.

281 perature are effective for the hydrolysis of pentose polymers in hemicellulose and also increase the

282 teen hexose, one fucose, one methyl, and two pentose residues; however, in this and most other strain

283 t in the hydrated pyrimidine ring but in the pentose ring, which is epimerized to arabinose in the mi

284 imate glycolysis pathway intermediates, less pentose shunt flux, increased anaplerosis from glutamine

285 uptake and utilization by glycolysis and the pentose shunt, but no changes in glutamine or fatty acid

286 s demonstrated, including selectivity toward pentoses such as arabinose, ribose, and xylose to the ex

287 yeasts capable of efficient fermentation of pentoses such as xylose remains a key challenge in the p

288 ons with the radical at the chiral C4 of the pentose sugar and the intramolecularly C1-tethered olefi

289 stry, is characterized by high levels of the pentose sugar L-xylulose in blood and urine and deficien

290 inability of many microbes to metabolize the pentose sugars abundant within hemicellulose creates spe

291 dily ferments sugars derived from cellulose, pentose sugars from xylan are not metabolized.

292 Therefore, for assimilating pentose sugars or for generating C(5) precursors (such a

293 ine and pyrimidine nucleotides, nucleosides, pentose sugars, and inorganic phosphate demonstrates tha

294 mentation, operates with multiple hexose and pentose sugars.

295 f F. succinogenes to utilize non-cellulosic (pentose) sugars for growth.

296 l up-regulated genes potentially involved in pentose transport and metabolism, which were targeted fo

297 minimal energetic differences between the 12 pentoses, two unique fixed ligand kinetic method combina

298 gs build toward the engineering of efficient pentose utilization in yeast and provide a blueprint for

299 nal insights needed for engineering improved pentose utilizing strains of C. thermocellum and reveal

300 ses (glucose, galactose, and mannose), three pentoses (xylose, arabinose, and ribose), two deoxyhexos