ライフサイエンスコーパス: peptic ulcer disease

1 s1m1, and s2m1 genotypes and development to peptic ulcer disease.

2 es to the pathogenesis of gastric cancer and peptic ulcer disease.

3 ggest a relationship between lung cancer and peptic ulcer disease.

4 oeconomic status, added salt, and history of peptic ulcer disease.

5 episodes of acute distress that can lead to peptic ulcer disease.

6 much lower than for patients with idiopathic peptic ulcer disease.

7 plications of gastrin in the pathogenesis of peptic ulcer disease.

8 rden due to chronic liver diseases (CLD) and peptic ulcer disease.

9 Endoscopy definitively diagnoses peptic ulcer disease.

10 infectious disease, autoimmune disorders, or peptic ulcer disease.

11 e considered the top-ranked risk factors for peptic ulcer disease.

12 < 0.001) were identified as risk factors for peptic ulcer disease.

13 in use are the widely known risk factors for peptic ulcer disease.

14 Germany) was used to confirm the presence of peptic ulcer disease.

15 orrelation is between chronic bronchitis and peptic ulcer disease.

16 factors in approximately 36% of people with peptic ulcer disease.

17 and included 1803 individuals with suspected peptic ulcer disease.

18 superior result for H. pylori eradication in peptic ulcer disease.

19 s requiring emergency surgery for perforated peptic ulcer disease.

20 ndrome, gastroesophageal reflux disease, and peptic ulcer disease.

21 nfected congenital anomalies, and perforated peptic ulcer disease.

22 d areas: morbid obesity, gastric cancer, and peptic ulcer disease.

23 or for the development of gastric cancer and peptic ulcer disease.

24 the bacterium in pathogenesis and relapse of peptic ulcer disease.

25 tes to the development of gastric cancer and peptic ulcer disease.

26 IDA regardless of the presence or absence of peptic ulcer disease.

27 and that contributes to the pathogenesis of peptic ulcer disease.

28 ith Helicobacter pylori is the main cause of peptic-ulcer disease.

29 6), myocardial infarction (1.34; 1.07-1.69), peptic ulcer disease (1.27; 1.03-1.58), peripheral vascu

30 y was oesophageal varices (57%), followed by peptic ulcer disease (18%) and gastritis (10%).

31 l bleeding (two [<0.1%] vs four [0.1%]), and peptic ulcer disease (32 [0.7%] vs 34 [0.8%]) were low a

32 cluded diverticulitis (5), pancreatitis (4), peptic ulcer disease (4), and cholecystitis (2).

33 by paralysis (90% increase), dementia (60%), peptic ulcer disease (53%), other neurological disorders

34 71 (36%), intestinal obstruction 11 (6%) and peptic ulcer disease 9 (5%).

35 In the US, peptic ulcer disease affects 1% of the population and ap

36 Peptic ulcer disease, although declining in prevalence,

37 Peptic ulcer disease, although declining in prevalence,

38 etermine magnitude and associated factors of peptic ulcer disease among dyspeptic patients in Northwe

39 ated with reduced risk of gastric cancer and peptic ulcer disease, among other conditions.

40 n gastric mucosa, and it is a major cause of peptic ulcer disease and a principal risk factor for gas

41 luids of a 64-year-old man with a history of peptic ulcer disease and alcohol abuse.

42 phenotypic subgroup has been associated with peptic ulcer disease and an increased bleeding tendency.

43 e H. pylori are more closely associated with peptic ulcer disease and cancer.

44 lori varies in severity from asymptomatic to peptic ulcer disease and cancer.

45 Helicobacter pylori, a cause of peptic ulcer disease and certain types of gastric cancer

46 ylori is the strongest known risk factor for peptic ulcer disease and distal gastric adenocarcinoma,

47 s and is the strongest known risk factor for peptic ulcer disease and distal gastric cancer, yet only

48 ter pylori, a human pathogen associated with peptic ulcer disease and gastric adenocarcinoma, we clon

49 A (CagA) into host cells are associated with peptic ulcer disease and gastric adenocarcinoma.

50 er pylori infection is the leading cause for peptic ulcer disease and gastric adenocarcinoma.

51 n that may contribute to the pathogenesis of peptic ulcer disease and gastric adenocarcinoma.

52 Peptic ulcer disease and gastric cancer are caused most

53 Helicobacter pylori, the main cause of peptic ulcer disease and gastric cancer in adult populat

54 Gastric diseases, including peptic ulcer disease and gastric cancer, affect 10% of t

55 an cells, contributes to the pathogenesis of peptic ulcer disease and gastric cancer, and is a candid

56 r pylori (HP), a known major risk factor for peptic ulcer disease and gastric cancer.

57 population, leading to an increased risk of peptic ulcer disease and gastric cancer.

58 worldwide problem and greatly contributes to peptic ulcer disease and gastric cancer.

59 tients is known to prevent the occurrence of peptic ulcer disease and gastric cancer.

60 tomach and contributes to the development of peptic ulcer disease and gastric cancer.

61 ects half of the world population and causes peptic ulcer disease and gastric cancer.

62 acter pylori infection of the stomach causes peptic ulcer disease and gastric cancer.

63 cells and contributes to the pathogenesis of peptic ulcer disease and gastric cancer.

64 tant virulence factor in the pathogenesis of peptic ulcer disease and gastric cancer.

65 ch and may contribute to the pathogenesis of peptic ulcer disease and gastric cancer.

66 use of most gastric diseases, including both peptic ulcer disease and gastric cancer.

67 ) that may contribute to the pathogenesis of peptic ulcer disease and gastric cancer.

68 tence increases the risk of diseases such as peptic ulcer disease and gastric cancer.

69 to persistence of the bacterium and risk for peptic ulcer disease and gastric cancer.

70 tant virulence factor in the pathogenesis of peptic ulcer disease and gastric cancer.

71 the human stomach and increases the risk for peptic ulcer disease and gastric carcinoma.

72 tric epithelial cells and has been linked to peptic ulcer disease and gastric carcinoma.

73 oton pump inhibitor used in the treatment of peptic ulcer disease and gastrosophageal reflux disease

74 class it is fraught with the risk of serious peptic ulcer disease and its complications.

75 isms by which H pylori increases the risk of peptic ulcer disease and noncardia gastric adenocarcinom

76 rove to lessen the morbidity associated with peptic ulcer disease and other benign conditions.

77 ction is a major cause of chronic gastritis, peptic ulcer diseases and cancer.

78 in various degrees of gastric inflammation, peptic ulcer disease, and a predisposition to gastric ca

79 h affects approximately 42% of patients with peptic ulcer disease, and aspirin or nonsteroidal anti-i

80 ion, psychiatric disorders, anemia, obesity, peptic ulcer disease, and cancer but a lower prevalence

81 s the risk of developing atrophic gastritis, peptic ulcer disease, and gastric adenocarcinoma.

82 its etiologic role in symptomatic gastritis, peptic ulcer disease, and gastric adenocarcinoma.

83 estive diseases including chronic gastritis, peptic ulcer disease, and gastric cancer.

84 elium is strongly associated with gastritis, peptic ulcer disease, and gastric cancer.

85 major role in the development of gastritis, peptic ulcer disease, and gastric cancer.

86 can lead to gastroesophageal reflux disease, peptic ulcer disease, and stress-related erosion/ulcer d

87 ploratory laparotomy and gastric surgery for peptic ulcer disease approximately 10 years ago.

88 The principal causes of peptic ulcer disease are Helicobacter pylori infection,

89 A small proportion of patients have peptic ulcer disease as cause and this can be treated em

90 nificant association observed in vacA s1 and peptic ulcer disease, as well as vacA s1/m2 and gastric

91 e human stomach and induces acute gastritis, peptic ulcer disease, atrophic gastritis, and gastric ad

92 of gastric diseases like gastric cancer and peptic ulcer disease attributed to Helicobacter pylori i

93 atients who underwent gastrectomy for benign peptic ulcer disease between 1960 and 1975, 163 patients

94 edical history (smoking, diabetes, bleeding, peptic ulcer disease, cancer, chronic liver disease, chr

95 ylori has been implicated in the etiology of peptic ulcer disease, chronic gastritis, gastric carcino

96 In patients with bleeding related to peptic ulcer disease, combination therapy (epinephrine i

97 tic significance among CAD patients, whereas peptic ulcer disease, connective tissue disease, and lym

98 ns of the PCSK9 T allele were also seen with peptic ulcer disease, depression, asthma, chronic kidney

99 in patients developing the complications of peptic ulcer disease (eg, obstruction and perforation),

100 (dyspepsia, gastroesophageal reflux disease, peptic ulcer disease), functional intestinal disorders,

101 chronic active gastritis, which can lead to peptic ulcer disease, gastric adenocarcinoma, and mucosa

102 a resultant decline in H. pylori-associated peptic ulcer disease, gastric cancer remains the second

103 , causing serious medical conditions such as peptic ulcer disease, gastric cancer, and respiratory an

104 ronic gastritis and plays a critical role in peptic ulcer disease, gastric carcinoma, and gastric lym

105 ses chronic gastritis and is associated with peptic ulcer disease, gastric carcinoma, and gastric lym

106 acterial pathogens, often causing gastritis, peptic ulcer disease, gastric mucosa-associated lymphati

107 s, in whom it is a key etiological factor in peptic ulcer disease, gastric mucosa-associated lymphoid

108 ndications for PPI use, including history of peptic ulcer disease, gastroesophageal reflux disease, o

109 Peptic ulcer disease has been associated with an increas

110 pylori in the pathogenensis of gastritis and peptic ulcer disease has been shown in adults and childr

111 roscopic surgery for treatment of perforated peptic ulcer disease has now been validated, with subseq

112 tentially identify high-risk individuals for peptic ulcer disease in clinical settings.

113 H. pylori infection rate among patients with peptic ulcer disease in Ethiopia.

114 tion of recombinant semaphorin 4D alleviates peptic ulcer disease in mice in vivo, demonstrating that

115 bed to prevent steroid-induced gastritis and peptic ulcer disease in patients with glioblastoma.

116 Incidence and risk factors for peptic ulcer disease in the United States have not been

117 Annually, 10 000 people die of peptic ulcer disease in the US.

118 Peptic ulcer disease is a frequent clinical problem with

119 Peptic ulcer disease is a prevalent gastric and duodenal

120 Partial gastrectomy for benign peptic ulcer disease is associated with an increased ris

121 H. pylori-induced peptic ulcer disease is associated with inadequate regul

122 Peptic ulcer disease is associated with increased hospit

123 ts for children in whom H. pylori-associated peptic ulcer disease is diagnosed.

124 he risk for development of gastric cancer or peptic ulcer disease is higher among humans infected wit

125 Upper gastrointestinal bleeding from peptic ulcer disease is not a new clinical problem.

126 Gastric surgery for benign peptic ulcer disease is not a risk factor for either sho

127 greatest effect on surgical intervention in peptic ulcer disease is the Centers for Disease Control

128 on, cholecystectomy, operative management of peptic ulcer disease, lysis of peritoneal adhesions, app

129 Compared with those with report of no peptic ulcer disease, men with gastric ulcer had an incr

130 intestinal tract, such as chronic gastritis, peptic ulcer disease, mucosa-associated lymphoid tissue

131 nistering therapy include active or inactive peptic ulcer disease, mucosa-associated lymphoid tissue

132 ic gastritis and leading in some patients to peptic ulcer disease, mucosa-associated lymphomas, and g

133 at a variety of gastric disorders, including peptic ulcer disease, neoplasia, and autoimmune gastriti

134 Of patients with peptic ulcer disease, nine of 37 (24%) had stigmata of r

135 Dumping, bile gastritis, or peptic ulcer disease occurred in three patients after PP

136 Globally, peptic ulcer disease occurs in 3.5-32% of patients with

137 lays an etiologic role in the development of peptic ulcer disease, only a small number of these child

138 lly asymptomatic but sometimes progresses to peptic ulcer disease or gastric adenocarcinoma.

139 ommon in strains isolated from patients with peptic ulcer disease or gastric cancer, rather than asym

140 face, evade host immune clearance, and cause peptic ulcer disease or gastric neoplasia in a significa

141 d with chronic gastritis and, in some cases, peptic ulcer disease or gastric neoplasms.

142 ns of Helicobacter pylori from patients with peptic ulcer disease or intestinal-type gastric cancer c

143 s of the peptide; and the role of gastrin in peptic ulcer disease pathogenesis secondary to Helicobac

144 Peptic ulcer disease, present in 56% of patients, was th

145 m2) was associated with an increased risk of peptic ulcer disease (PUD) (P = 0.003).

146 Perforated peptic ulcer disease (PUD) affects 4 million people annu

147 Gastric biopsy specimens of 68 peptic ulcer disease (PUD) and 327 chronic gastritis (CG

148 ction is prevalent worldwide and can lead to peptic ulcer disease (PUD) and gastric cancer.

149 etic factors are recognized to contribute to peptic ulcer disease (PUD) and other gastrointestinal di

150 udy was to analyse the risk of uncomplicated peptic ulcer disease (PUD) in a cohort of new users of l

151 Peptic ulcer disease (PUD) is a common and important cau

152 Despite progress in diagnosis and treatment, peptic ulcer disease (PUD) remains a common reason for h

153 Peptic ulcer disease (PUD) remains a significant yet poo

154 (GC), the impact on other diseases, such as peptic ulcer disease (PUD), dyspepsia, and gastric lymph

155 tion in 1994 of guidelines for management of peptic ulcer disease (PUD), trends in physician practice

156 i to activate neutrophils is associated with peptic ulcer disease (PUD).

157 (s1a) allele of the underlying vacA gene to peptic ulcer disease (PUD).

158 n causes of foregut inflammation, leading to peptic ulcer disease (PUD).

159 coronary heart disease (CHD) and subsequent peptic ulcer disease (PUD).

160 astrointestinal bleeding (UGIB) secondary to peptic ulcer disease (PUD).

161 cter pylori is the main etiologic factor for peptic ulcer disease, recent studies have explored a pot

162 in the pathogenesis of chronic gastritis or peptic ulcer disease remain unclear.

163 d contraindication to aspirin use, including peptic ulcer disease, renal insufficiency, and use of no

164 Medical treatment of peptic ulcer disease should target treatment of H. pylor

165 cobacter pylori, implicated in gastritis and peptic ulcer disease, Streptococcus agalactiae, implicat

166 h a higher risk of gastric adenocarcinoma or peptic ulcer disease than cag PAI-negative strains.

167 g(+)/type s1-vacA strains from patients with peptic ulcer disease than in cag-negative/s2-vacA strain

168 ding after successful hemostasis of bleeding peptic ulcer disease, the following questions should be

169 ri causes diseases ranging from gastritis to peptic ulcer disease to gastric cancer.

170 e from machine learning and risk factors for peptic ulcer disease to predict this disease efficiently

171 nes enable effective therapies to treat from peptic ulcer disease to psychiatric disorders.

172 refractory NVUGIB, the etiology of bleeding (peptic ulcer disease, unknown source, post surgical); pa

173 reflux disease was 1.71 (1.14-2.55; I2:94%), peptic ulcer disease was 1.21 (1.03-1.43; I(2):93%), eso

174 History of peptic ulcer disease was assessed at baseline in 1986 an

175 Peptic ulcer disease was believed to be caused by acid a

176 Peptic ulcer disease was diagnosed in 35% (95% CI 31.4-3

177 Perforated peptic ulcer disease was more prevalent among males (79.

178 ergency operation for bleeding or perforated peptic ulcer disease was performed to determine the asso

179 Explanatory variables associated with peptic ulcer disease were analyzed by applying logistic

180 Prevalences of heart or peptic ulcer disease were not significantly higher.

181 an early proponent of an infectious cause of peptic ulcer disease were recently discovered.

182 our patients without previous GI bleeding or peptic ulcer disease who were enrolled in a multicenter,

183 ed peptic ulcer is the worst complication of peptic ulcer disease whose burden is disproportionately