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1 f neurons that express the gastrin-releasing peptide receptor.
2 llowing recombination with an affinity clamp peptide receptor.
3 pTatC and Hcf106 is the twin arginine signal peptide receptor.
4 a melanogaster by deletion of the female sex peptide receptor.
5 or antagonist that targets gastrin-releasing peptide receptors.
6 the kappa opioid and nociceptin/orphanin FQ peptide receptors.
7 FPR1 and FPR2 as sensitive and broad signal peptide receptors.
9 , which then induces TRMP2 binding to formyl peptide receptor 1 (FPR1) and subsequent FPR1 internaliz
12 to be described on human neutrophils, formyl peptide receptor 1 (FPR1), is one such receptor that pla
15 te G protein-coupled receptor relaxin family peptide receptor 1 (RXFP1), has emerged as a potential a
17 he G protein-coupled receptor relaxin family peptide receptor 1 (RXFP1), little is known about the mo
21 n genetic variation in the NPR1 (natriuretic peptide receptor 1 gene, encoding NPR-A, atrial natriure
22 r 1 gene, encoding NPR-A, atrial natriuretic peptide receptor 1) was recently shown to affect blood p
24 m wild-type mice and mice lacking the formyl peptide receptor 1, we demonstrate that LTB(4) acts as a
25 eriments to study the capacity of the formyl peptide receptor-1 (FPR1) to desensitize chemokine recep
26 (HMGB1), respectively, as well as to formyl peptide receptor-1 (FPR1), which interacts with Annexin
27 tive properties that binds to relaxin family peptide receptor-1 (RXFP1) and has been shown to increas
29 atrial/brain natriuretic peptide-natriuretic peptide receptor-1 pathway is implicated in suppressing
30 upstream of the relaxin/insulin-like family peptide receptor 2 ( RXP2) (chromosome 13, rs60249166, o
31 Activation of the G-protein coupled formyl peptide receptor 2 (ALX/FPR2) by the lipid mediators lip
33 nnexin A1 (AnxA1) mainly acts through Formyl Peptide Receptor 2 (FPR2) inducing the resolution of inf
38 Selective agonists and antagonists of formyl peptide receptor 2 (Fpr2) suggested that Fpr2 mediated t
39 ion of a peptidomimetic antagonist of formyl peptide receptor 2 (FPR2) was explored by an approach in
41 activated the SAA1-binding receptor, formyl peptide receptor 2 (FPR2), which drove the epithelial re
44 st via the natriuretic peptide C/natriuretic peptide receptor 2 (NPPC/NPR2) system, and possibly also
46 e transmembrane guanylyl cyclase natriuretic peptide receptor 2 (NPR2) in response to the agonist C-t
47 s receptor, the guanylyl cyclase natriuretic peptide receptor 2 (Npr2), and the cGMP-dependent kinase
49 librium on chromosome 13 near relaxin family peptide receptor 2 (RXFP2) were associated with HbA(1c)
53 ulation, a GPCR called vasoactive intestinal peptide receptor 2 (VPAC2) is shed, rather than being in
55 Cs isolated from relaxin/insulin-like family peptide receptor 2 gene (Rxfp2) knockout and wild-type m
56 othelial-bound cathelicidin activates formyl-peptide receptor 2 on classical monocytes, resulting in
58 via the guanylate cyclase NPR2 (natriuretic peptide receptor 2) and not the G-protein-coupled cleara
60 2 (FPR2/ALX) and in mFPR2(-/-) (mouse formyl peptide receptor 2) mice lacking the mouse homolog of hu
62 riuretic peptide A and vasoactive intestinal peptide receptor 2, and a novel mineralocorticoid recept
67 ested whether the lipoxin A4 receptor formyl-peptide receptor 2/3 (Fpr2/3; ortholog to human FPR2/lip
68 ecific binding to the AnxA1 receptor (formyl peptide receptor 2/Lipoxin A4 receptor [FPR2/ALX]; IC50
70 -4 receptor, the Smoothened receptor, formyl peptide receptor-2 (FPR2), the relaxin receptor (LGR7),
71 rfering RNA-induced knockdown of LXA4 formyl peptide receptor-2 (FPR2/ALX) and in mFPR2(-/-) (mouse f
74 er in allergy to tropomyosins and the formyl peptide receptor 3 in allergy to lipocalins are outstand
77 concentration by binding to its natriuretic peptide receptor A (NPRA) receptor and, in turn, by acti
79 ion between ANP and its receptor natriuretic peptide receptor A and reduces intracellular cGMP levels
80 n-releasing peptide receptor and natriuretic peptide receptor A by genetic approaches or toxins in mi
81 The intracellular segment of a natriuretic peptide receptor A guanylyl cyclase failed to bind GCAPs
82 or the interaction of human anti-natriuretic peptide receptor A IgG4 with the neonatal Fc receptor, F
83 trin-releasing peptide receptor, natriuretic peptide receptor A, neuromedin B receptor, and sst2A.
84 228P), an antibody targeting the natriuretic peptide receptor A, show a previously unrecognized type
85 de WKYMVm, a selective agonist of the formyl peptide receptor, a 2-fold increase in leukocyte emigrat
86 e (ANP) binds guanylyl cyclase-A/natriuretic peptide receptor-A (GC-A/NPRA) and produces the intracel
87 delineate the mechanisms of GC-A/natriuretic peptide receptor-A (GC-A/NPRA) gene (Npr1) expression in
89 Npr1 (encoding guanylyl cyclase/natriuretic peptide receptor-A, GC-A/NPRA) gene-knockout (0-copy; Np
95 PRRT) is a type of radiotherapy that targets peptide receptors and is typically used for neuroendocri
96 oral, small-molecule calcitonin gene-related peptide receptor antagonist for acute migraine treatment
97 attack with the oral calcitonin gene-related peptide receptor antagonist rimegepant resulted in a hig
99 red, small-molecule, calcitonin gene-related peptide receptor antagonist that may be effective in acu
100 brogepant is an oral calcitonin gene-related peptide receptor antagonist under investigation for acut
101 nt, a small molecule calcitonin gene-related peptide receptor antagonist, has shown efficacy in the a
102 ts, and the gepants: calcitonin gene-related peptide receptor antagonists, in a preclinical model of
104 obably the first of a long list, as multiple peptide receptors are now recognized as potential target
105 cyclase B (GC-B), also known as natriuretic peptide receptor B or NPR2, stimulates long bone growth,
107 r-targeted agents, such as gastrin-releasing peptide receptor (BB2r)-targeted peptides, have been inv
110 the potential role of an NPR-C (natriuretic peptide receptor-C) in atrial fibrosis and the role of a
111 c NP receptors, including NPR-C (natriuretic peptide receptor-C), are cardioprotective hormones that
113 , as it was distinct from atrial natriuretic peptide receptor-cGMP-PKG-RyR2 Ser-2808 signaling and in
114 rtner, c-Fos, and therefore designed a c-Fos peptide receptor chemically modified to incorporate a th
121 revious studies showed that the delta-opioid peptide receptor (DOP-R) is dynamically regulated by exp
122 yl cyclase-A (GC-A) signaling, a natriuretic peptide receptor, exerts renoprotective effects by stimu
123 eceptor, an orthologue of the Drosophila sex peptide receptor, expressed in neighboring apical organ
125 a member of the chemotactic GPCR-7TM formyl peptide receptor family, whose principle function is in
127 dy that inhibits the calcitonin gene-related peptide receptor, for the prevention of episodic migrain
130 ogenous anti-inflammatory circuit via formyl peptide receptor (FPR) 2/lipoxin receptor (ALX) (Fpr2/3
131 ystemic administration of Boc2, a formylated peptide receptor (fpr) antagonist, abrogated the peptide
132 Innate immune chemoreceptors of the formyl peptide receptor (Fpr) family are expressed by vomeronas
134 ein-coupled receptor belonging to the formyl peptide receptor (FPR) family, conveys the biological fu
137 nduces calcium and chemotaxis through formyl peptide receptor (FPR)2/ALX, whereas its D-stereoisomer
138 canonical GRK that phosphorylated the formyl peptide receptor FPR1 and facilitated neutrophil migrati
139 r pharmacological inhibition of the N-formyl peptide receptor Fpr1 leads to increased insulin levels
147 nt work showed that G-protein-coupled formyl peptide receptors (FPRs) directly mediate neutrophil pha
149 f such receptor-ligand pairs involves formyl peptide receptors (FPRs) that have been shown to influen
152 mRNAs for annexin A1 (AnxA1) and the formyl peptide receptors [(Fprs) 1, 2, and 3], a loss of lipid
153 osmoregulatory functions in brain, where SCT peptide/receptor function is required for ANGII action,
156 radioligands targeting the gastrin-releasing peptide receptor (GRP-R) might offer a specific method f
157 ntly introduced the potent gastrin-releasing peptide receptor (GRPR) antagonist (68)Ga-SB3 ((68)Ga-DO
158 state cancer, radiolabeled gastrin-releasing peptide receptor (GRPr) antagonists have yielded promisi
159 stablish that NK1R and the gastrin-releasing peptide receptor (GRPR) are coexpressed within a subpopu
160 conjugate affinity for the gastrin releasing peptide receptor (GRPR) as determined against [(125)I-Ty
161 ause overexpression of the gastrin-releasing peptide receptor (GRPR) has been reported on various can
162 urons comprise a subset of gastrin-releasing peptide receptor (GRPR) interneurons and are thus positi
168 of evidence suggests that gastrin-releasing peptide receptor (GRPR) might be a valuable target in br
169 Consistently, ablation of gastrin-releasing peptide receptor (GRPR) neurons, which are essential for
171 spinal neurons expressing gastrin releasing peptide receptor (GRPR) primarily comprise excitatory in
172 ation, depends on CGRP and gastrin-releasing peptide receptor (GRPR) transmission because pharmacolog
174 with high affinity for the gastrin-releasing peptide receptor (GRPr), a receptor that is overexpresse
176 their affinity toward the gastrin releasing peptide receptor (GRPr), metabolic stability in blood pl
177 Because expression of the gastrin-releasing peptide receptor (GRPR), somatostatin receptor 2 (SSTR2)
186 the expression of PSMA and gastrin-releasing peptide receptors in different types of prostate cancer.
188 TP production and requires an initial formyl peptide receptor-induced Ca(2+) signal that triggers mit
189 ch as naproxen and a calcitonin gene-related peptide receptor inhibitor, olcegepant, were less effect
193 novel insights into the oxytocin/vasopressin peptide-receptor interaction, which led to the identific
194 tide GPCRs has advanced our understanding of peptide-receptor interactions and fueled interest in cor
196 ool for the validation and quantification of peptide-receptor interactions in their natural cellular
197 egions may not give a complete reflection of peptide/receptor interactions and should be combined wit
198 dditional experimental constraints to reveal peptide/receptor interactions occurring in the dynamic,
200 Cell-cell communication mediated by secreted peptides, receptor kinases, and downstream mitogen-activ
202 state-specific membrane or gastrin-releasing peptide receptor ligands for the imaging of prostate can
203 of cathelicidin are mediated through formyl peptide receptor-like 1 (FPRL1), we hypothesize that CSA
204 ule formation, suggesting that CRA2 is a CEP peptide receptor mediating both organogenesis programs.
205 ptides for itch, including gastrin-releasing peptide receptor, natriuretic peptide receptor A, neurom
206 nd 1980s, the nociceptin/orphanin FQ (N/OFQ) peptide receptor (NOP, also known as ORL-1) was discover
207 did not differentially modulate natriuretic peptide receptor (NPR)-A/B activity with respect to T223
208 helium-specific CNP(-/-), global natriuretic peptide receptor (NPR)-B(-/-) and NPR-C(-/-) animals, an
209 and the guanylate cyclase-linked natriuretic peptide receptors Npr1 and Npr2 are functionally redunda
210 iculate guanylate cyclase-linked natriuretic peptide receptors Npr1 and Npr2, and activation of prote
211 bilizing G protein-coupled receptors (formyl peptide receptor, P2Y2 purinergic receptor, and calcium-
215 maging may be used to monitor the effects of peptide receptor radiolabeled targeted therapy in patien
216 ion due to (68)Ga-DOTATATE was initiation of peptide receptor radionuclide therapy (14 patients, 27.4
217 reatic neuroendocrine tumors (GEP NET) after peptide receptor radionuclide therapy (PRRT) are still l
220 ty after internal radiation exposure through peptide receptor radionuclide therapy (PRRT) has not yet
221 ker, the inflammation-based index (IBI), for peptide receptor radionuclide therapy (PRRT) in neuroend
222 ker, the inflammation-based index (IBI), for peptide receptor radionuclide therapy (PRRT) in neuroend
225 ((177)Lu) DOTA-0-Tyr3-Octreotate (DOTATATE) peptide receptor radionuclide therapy (PRRT) is an effec
229 date, limited data are available concerning peptide receptor radionuclide therapy (PRRT) of grade 3
230 iation with (177)Lu-DOTA-octreotate (LuTate) peptide receptor radionuclide therapy (PRRT) of neuroend
236 ntraarterial administration of (90)Y-DOTATOC peptide receptor radionuclide therapy (PRRT) would incre
245 s might further improve the safety window of peptide receptor radionuclide therapy by reducing the li
249 I and III studies; delineate the position of peptide receptor radionuclide therapy in the therapeutic
252 opancreatic neuroendocrine tumors, access to peptide receptor radionuclide therapy is increasing.
253 ostics are peptide receptor scintigraphy and peptide receptor radionuclide therapy of neuroendocrine
254 ht enhance peptide receptor scintigraphy and peptide receptor radionuclide therapy of neuroendocrine
256 uroendocrine tumors (NETs) can be treated by peptide receptor radionuclide therapy using radiolabeled
259 ment in recent years was the introduction of peptide receptor radionuclide therapy with radiolabeled
261 for prospective dosimetry for (67)Cu-SARTATE peptide receptor radionuclide therapy, and the half-life
262 systemic treatment of advanced disease with peptide receptor radionuclide therapy, biotherapy, chemo
272 P = 0.002), and type of therapy (medical vs. peptide receptor radionuclide therapy: 16.0 vs. 26.0 mo;
273 quency of false-positive recommendations for peptide-receptor radionuclide therapy occurred in observ
274 , image-based recommendations for or against peptide-receptor radionuclide therapy require experience
277 E PET/CT correctly identified 3 patients for peptide-receptor radiotherapy incorrectly classified by
279 heart failure, activates the relaxin family peptide receptor (RXFP1), which is a class A G-protein-c
280 he highly conserved family of relaxin family peptide receptors (RXFPs), mediates the checkpoint funct
281 most successful examples of theranostics are peptide receptor scintigraphy and peptide receptor radio
283 SSTR antagonists such as JR11 might enhance peptide receptor scintigraphy and peptide receptor radio
288 urther reveal a feedback circuit between the peptide-receptor system and auxin response as a mechanis
290 human cancers, providing the opportunity for peptide receptor targeting via radiolabeled bombesin-bas
296 2, consistent with the idea that natriuretic peptide receptor type 2 (NPR2) signaling inhibits the ac
297 ratio of the signaling receptor, natriuretic peptide receptor type A, to the clearance receptor, nprc
298 ology for predicting binding scores of small peptide receptors vs. volatile compounds is proposed.
299 In a screen of all 49 predicted Ae. aegypti peptide receptors, we identified NPY-like receptor 7 (NP
300 and an antagonist for the gastrin-releasing peptide receptor were found to have excellent tumor-targ