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1 progenitors, at least some of which are also periarteriolar.
2 ting a fern-like pattern with sparing of the periarteriolar area in all cases.
3 ever, arteriolar endothelial cells and other periarteriolar cells expressed increasing levels of inte
4 is compared with control subjects; and (iii) periarteriolar changes, including periarteriolar space d
5 ncreasing vascular compliance, regression of periarteriolar collagen area, improvement of coronary re
6                                          The periarteriolar environment thus became particularly infl
7 o suggest a homology between this vasoactive periarteriolar fat and both periarterial and visceral fa
8 association with VEGFR2 blockade resulted in periarteriolar inflammation with macrophages, and B cell
9 ly arrays of splenic macrophages surrounding periarteriolar lymphatic sheaths and a red pulp depletio
10 ver, BDC-Idd9 T cells accumulated in splenic periarteriolar lymphatic sheaths, whereas BDC T cells we
11 cur in the marginal zone and surrounding the periarteriolar lymphocyte sheaths.
12 X40 is maximally expressed by T cells in the periarteriolar lymphoid sheath (PALS) 3 d after primary
13 20+ B cells clustered within the T cell-rich periarteriolar lymphoid sheath (PALS) and surrounding fo
14 were observed in or near the CD4 T cell-rich periarteriolar lymphoid sheath (PALS) during the entire
15 ion of Lm from the marginal zone (MZ) to the periarteriolar lymphoid sheath (PALS) was inhibited by p
16 n the splenic marginal zone, migrated to the periarteriolar lymphoid sheath (PALS) where they grew ex
17 pheral lymphoid organs and accumulate in the periarteriolar lymphoid sheath but are unable to generat
18 ls (FRCs) form a network in the T cell zone (periarteriolar lymphoid sheath, PALS) of the WP on which
19 the chemokine receptor CCR7, are home to the periarteriolar lymphoid sheath, whereas activated TH2 ce
20 s that were exclusively found in the splenic periarteriolar lymphoid sheath.
21 but C4H3+ DC rapidly accumulate in the outer periarteriolar lymphoid sheaths (PALS) and in follicular
22 on, activated splenic T cells proliferate in periarteriolar lymphoid sheaths (PALS) and subsequently
23 old increase in the T cell-rich areas of the periarteriolar lymphoid sheaths (PALs).
24 , foci of KJ1-26+ cells were observed in the periarteriolar lymphoid sheaths at day 3; these were not
25 ion at the expense of HSC-maintaining NG2(+) periarteriolar niche cells.
26  alters the distribution of HSCs from NG2(+) periarteriolar niches to LEPR(+) perisinusoidal niches.
27 ations, but not proximal to bone, adipocyte, periarteriolar, or Schwann cells.
28 have enabled highly-resolved measurements of periarteriolar oxygen gradients (POGs) within the brain
29 ail to migrate from the marginal zone to the periarteriolar region of the spleen.
30                This increased the numbers of periarteriolar Sca1(+)Cxcl9(+)LepR(+) cells with an infl
31 n the spleen, where they were located in the periarteriolar sheath, marginal zone, and interfollicula
32 fies all cells of the arteriole wall and the periarteriolar space 3-4 s prior to the arteriole dilati
33  and (iii) periarteriolar changes, including periarteriolar space dilatation, haemosiderin deposition
34 sence and/or severity of arteriolosclerosis, periarteriolar space dilatation, haemosiderin leakage, m
35 al reveal a novel function for podoplanin on periarteriolar stromal cells in the bone marrow: promoti
36       Produced by arteriolar endothelial and periarteriolar stromal cells, conditional netrin-1 delet
37 ssive lengthening stimulates the activity of periarteriolar sympathetic nerves; this activity propaga