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1       Such complex thalli are formed by both periclinal and anticlinal cell divisions, which also und
2 of both TMO5 and LHW can ubiquitously induce periclinal and radial cell divisions in all cell types o
3 owed a limited deconstruction of the initial periclinal array followed by a progressive array reorgan
4 atory gene, CYCD6;1, and regulates formative periclinal asymmetric cell divisions in endodermis and c
5 the endodermal cells the capacity to undergo periclinal cell division to repopulate the vascular stem
6 ccordingly, ectopic DOF3.4 expression drives periclinal cell division, while its downstream D3-type c
7 rs are impaired from the "symmetry-breaking" periclinal cell divisions during the transition between
8 ing transcription factor controls asymmetric periclinal cell divisions in flowering plants by governi
9 ddle cortex (MC), and cortex are produced by periclinal cell divisions that occur at different positi
10 primary root arising from impaired timing of periclinal cell divisions.
11 d from microtubules originating on the outer periclinal cell face, pointing to a cell-directed, rathe
12 verse coordinated between the anticlinal and periclinal cell faces.
13 , at the lateral walls of the GCs and at the periclinal cell walls of the central canal.
14 ight lead to chloroplast accumulation on the periclinal cell walls, whereas light intensities over 20
15  seedlings, the cortical array on the outer (periclinal) cell face creates a variety of array pattern
16 oscale and mesoscale structure of the outer (periclinal) cell wall of onion scale epidermis - a model
17 evel of layer-specific expression by using a periclinal chimera that has its L1 layer from Solanum pe
18                             We have analyzed periclinal chimeras and mericlinal sectors of jointless
19 insertion in the MADS box region to generate periclinal chimeras expressing alleles with different ac
20 ' lam1 mutant of Nicotiana sylvestris and in periclinal chimeras with lam1 and wild-type (N. glauca)
21 om revertant plants, indicated that all were periclinal chimeras with wild-type fim expression only i
22               We identified petunia mutants (periclinal chimeras) expressing the B-class MADS-box gen
23  autonomy by examining fim expression in flo periclinal chimeras.
24 er-specific DNA from regenerants marked by a periclinal chromosomal translocation revealed a similar,
25 o development and that none showed the usual periclinal division leading to the formation of the prot
26 liferation and is responsible for triggering periclinal division of subepidermal cells.
27 romotes tapetum differentiation and inhibits periclinal division once a tapetal cell is specified.
28 s of anticlinal cell divisions or failure of periclinal division suppression in the leaf blade.
29                              Moreover, extra periclinal divisions (new wall parallel to surface of th
30 re length of the root tip, but only promotes periclinal divisions at specific sites.
31 scr) and short root (shr) suppress the extra periclinal divisions characteristic of scz mutant roots.
32  thickening of epidermal cells and localized periclinal divisions contributed to the formation of a r
33 nd tissue, and (2) it is required to repress periclinal divisions in the meristem.
34            Disrupting SCR function abolished periclinal divisions in this lateral root primordia cell
35                            These protodermal periclinal divisions occur at the expense of normal anti
36 ermis and cortex arise continuously from the periclinal divisions of cells that surround the quiescen
37     The MC arises between days 7 and 14 from periclinal divisions of the endodermis.
38 ferentiate, and, instead, undergo additional periclinal divisions to form extra layers of cells.
39 he parietal layer then undergo two cycles of periclinal divisions to give rise to three wall layers.
40 cell layers1 (Xcl1) mutation causes oblique, periclinal divisions to occur in the protoderm layer.
41 rs are generated properly through successive periclinal divisions, in the ms32 mutant, tapetal precur
42 GRFs increases the meristem size and induces periclinal formative divisions in transit-amplifying cel
43 e rates of light, chloroplasts accumulate in periclinal layers perpendicular to the direction of ligh
44 ytes undergo division in both anticlinal and periclinal orientations, not only increasing cell number
45 ues, is abundant in anticlinal and the inner periclinal plasma membrane of 'outside' cells.
46 e-enriched and microtubule-free zones at the periclinal wall in neighboring cells predicted sites of
47 he manner in which cell wall polymers at the periclinal wall regulate the morphogenetic process in ep
48        Cortical microtubules adjacent to the periclinal wall were persistently enriched at the convex
49 ght, mesophyll chloroplasts spread along the periclinal walls normal to the light, maximizing absorba
50 creased with increasing cell size, while for periclinal walls, the number of PDs decreased.
51 tubule networks that span the anticlinal and periclinal walls.
52 fferential growth of both the anticlinal and periclinal walls.
53 -1), chloroplasts in pmi2 leaves move to the periclinal walls; 100 micromol m(-2) s(-1) of blue light