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1 t diabetic who survived the complications of peripancreatic abscess, enterocutaneous fistula, and art
2 al status of 56 liver lesions and 48 primary peripancreatic adenocarcinomas obtained from 48 patients
3 s (BDPs) from well-differentiated metastatic peripancreatic adenocarcinomas on histological grounds a
9 -Kettering Cancer Center with a diagnosis of peripancreatic cancer, 1363 of whom had adenocarcinoma o
14 ntation, acute rejection, and CT findings of peripancreatic edema and/or inflammatory change were sig
15 means of CT and MRI showed small amounts of peripancreatic fluid along with a limited area of intra-
17 sequently required operation to manage their peripancreatic fluid collection, 37 urgent or emergent.
22 rded a success rate of 80% (16/20) for acute peripancreatic fluid collections (APFC) and pancreatic p
24 ostoperative complications included: 6 (13%) peripancreatic fluid collections and 2 (4%) pancreatitis
26 en operative and nonoperative management for peripancreatic fluid collections and pseudocysts should
27 No previous study has examined the role of peripancreatic fluid collections and subsequent pseudocy
28 enhancing capsule develops, persistent acute peripancreatic fluid collections are referred to as pseu
30 kocytes to detect infection in pancreatic or peripancreatic fluid collections in patients with AP.
31 the diagnosis of infection in pancreatic or peripancreatic fluid collections in patients with AP.
33 e acute biliary pancreatitis and demonstrate peripancreatic fluid collections or pseudocysts until th
34 3, all patients admitted to our service with peripancreatic fluid collections or pseudocysts were mon
35 mplications included four splenectomies, two peripancreatic fluid collections, one pseudocyst, and on
39 imary endpoint: development of pancreatic or peripancreatic infection within 42 days following random
40 tween the treatment groups for pancreatic or peripancreatic infection, mortality, or requirement for
41 eatitis and the development of pancreatic or peripancreatic infection; all-cause mortality; requireme
43 of reducing the incidence of pancreatic and peripancreatic infections, although the benefits of doin
44 diffuse pancreatic enlargement, with minimal peripancreatic inflammation and absence of vascular enca
45 vation of pancreatic proenzymes and signs of peripancreatic inflammation in patients with clinically
46 treating early-stage pancreatic cancer, when peripancreatic inflammation promoted by the microbiome p
47 ancreatic ducts, and other findings, such as peripancreatic inflammation, encasement of vessels, mass
49 s dependent upon both initial priming in the peripancreatic lymph node and subsequent presentation in
51 d pancreaticoduodenectomy (removing only the peripancreatic lymph nodes en bloc with the specimen) or
52 d pancreaticoduodenectomy (removing only the peripancreatic lymph nodes en bloc with the specimen) or
53 -64) peptide were found spontaneously in the peripancreatic lymph nodes of pre-diabetic NOD mice.
55 condary lymphoid structures, most likely the peripancreatic lymph nodes, were essential for the devel
61 is of esophageal (n = 23), gastric (n = 75), peripancreatic (n = 86), or bile duct (n = 11) cancer un
63 es the need for reinterventions for residual peripancreatic necrotic collections and other complicati
64 sels, mass effect, pancreatic calcification, peripancreatic nodes, and peripancreatic fluid collectio
67 l (one of 15 patients) parenchymal swelling, peripancreatic stranding (10 of 15 patients), "halo" (ni
70 115 patients with radiologically resectable peripancreatic tumors underwent extended laparoscopy bef
71 ter informed consent, eligible patients with peripancreatic tumors were randomized during surgery eit
73 partial regression of tumor contact with any peripancreatic vascular axis was associated with R0 rese
75 improve the enhancement of the pancreas and peripancreatic vasculature, improve tumor conspicuity, a
76 of tumor, bordering pancreas, and all major peripancreatic vessels were obtained for both time inter