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1 erican women are at increased risk of having peripartum cardiomyopathy.
2 imilar to those of patients with traditional peripartum cardiomyopathy.
3 entricular function in women presenting with peripartum cardiomyopathy.
5 ne hundred women met traditional criteria of peripartum cardiomyopathy; 23 were diagnosed with pregna
6 e: idiopathic cardiomyopathy (616 patients), peripartum cardiomyopathy (51); and cardiomyopathy due t
8 have also been linked to conditions such as peripartum cardiomyopathy and chemotherapy-induced cardi
10 rdiology, we identified 44 women who had had peripartum cardiomyopathy and had a total of 60 subseque
11 action <45%) is crucial for the diagnosis of peripartum cardiomyopathy and the exclusion of other cau
14 adjusted OR [aOR], 2.12; 95% CI, 2.07-2.17), peripartum cardiomyopathy (aOR, 4.42; 95% CI, 3.79-5.13)
15 ) 1.37 (95% confidence interval 1.27-1.47)], peripartum cardiomyopathy [aOR 2.10 (1.11-3.99)], and ar
17 Anthracycline-associated cardiomyopathy and peripartum cardiomyopathy are nonischemic cardiomyopathi
18 ons with RA had higher risk of preeclampsia, peripartum cardiomyopathy, arrhythmias, acute kidney inj
19 gy, clinical presentation, and management of peripartum cardiomyopathy, as well as the current knowle
20 shed from 1966 to July 1999, using the terms peripartum cardiomyopathy, cardiomyopathy, and pregnancy
21 This Seminar summarises current knowledge of peripartum cardiomyopathy genetics, pathophysiology, dia
22 idiopathic cardiomyopathy, the patients with peripartum cardiomyopathy had better survival (adjusted
26 mation has also indicated that many cases of peripartum cardiomyopathy have genetic underpinnings.
27 sk of complications, including preeclampsia, peripartum cardiomyopathy, heart failure, arrhythmias, A
28 de spectrum of cardiomyopathies that include peripartum cardiomyopathy, hypertrophic cardiomyopathy,
29 Conversely, sNix protected against apoptotic peripartum cardiomyopathy in G(alpha)q-overexpressors.
30 view are to describe the clinical profile of peripartum cardiomyopathy in the United States and to pr
40 sequent pregnancy in women with a history of peripartum cardiomyopathy is associated with a significa
41 search in the past decade has suggested that peripartum cardiomyopathy is caused by vascular dysfunct
47 5), hypertrophic cardiomyopathy (n = 40) and peripartum cardiomyopathy (n = 69) for disease-causing P
54 this small retrospective study of women with peripartum cardiomyopathy, patients treated with immune
56 t ventricular (LV) recovery in patients with peripartum cardiomyopathy (PPCM) and to record rates of
57 c shock (CS) is a recognized complication of peripartum cardiomyopathy (PPCM) associated with poor pr
60 bsequent pregnancies (SSPs) in patients with peripartum cardiomyopathy (PPCM) have not been analyzed.
74 this study was to systematically review the peripartum cardiomyopathy (PPCM) literature and determin
77 psia is a risk factor for the development of peripartum cardiomyopathy (PPCM), but it is unknown whet
83 describe the characteristics and outcomes of peripartum cardiomyopathy (PPCMP) patients who received
84 r-specific antibodies (n=69, 57%), and prior peripartum cardiomyopathy pretransplant (n=57, 47%).
86 omen had a 15.7-fold higher relative risk of peripartum cardiomyopathy than non-African Americans (od
87 men have significantly higher odds of having peripartum cardiomyopathy that could not be explained by
88 tutes of Health (NIH) convened a Workshop on Peripartum Cardiomyopathy to foster a systematic review
89 ared the clinical outcomes of six women with peripartum cardiomyopathy treated with intravenous immun
91 U.S. studies confirmed that the frequency of peripartum cardiomyopathy was significantly higher among
92 icity remained a significant risk factor for peripartum cardiomyopathy when other risk factors were c
94 ons of pregnancy, including preeclampsia and peripartum cardiomyopathy, with a focus on pathological
95 thy was a prospective 30-center study of 100 peripartum cardiomyopathy women with LV ejection fractio