ライフサイエンスコーパス: peripheral nervous system

1 marcate an itch-specific labeled line in the peripheral nervous system.

2 an undecapeptide present in the CNS and the peripheral nervous system.

3 under normal physiological conditions in the peripheral nervous system.

4 r necessity during nerve degeneration in the peripheral nervous system.

5 ay a role in disorders of myelination in the peripheral nervous system.

6 lationships underpin the organization of the peripheral nervous system.

7 and other body parts, including central and peripheral nervous system.

8 elination is crucial for the function of the peripheral nervous system.

9 y the levels at which they accumulate in the peripheral nervous system.

10 from aberrant development or function of the peripheral nervous system.

11 degeneration specific to the spinal cord and peripheral nervous system.

12 iple organs, often including the central and peripheral nervous system.

13 as a disease-modifying agent for the injured peripheral nervous system.

14 r neurodegenerative conditions involving the peripheral nervous system.

15 g that HHV-7 may also have a tropism for the peripheral nervous system.

16 velopment of craniofacial structures and the peripheral nervous system.

17 anatomical and functional impairments in the peripheral nervous system.

18 rmally only produced by Schwann cells in the peripheral nervous system.

19 n of Notch-mediated cell fate choices in the peripheral nervous system.

20 mily, TRPM8, is a major cold receptor in the peripheral nervous system.

21 mpathetic neurons, to pattern the developing peripheral nervous system.

22 ptogenetic modulation of the spinal cord and peripheral nervous system.

23 ine (psychosine), accumulates in the CNS and peripheral nervous system.

24 then form the cranial sensory ganglia of the peripheral nervous system.

25 ence for a physiological role of NgR2 in the peripheral nervous system.

26 required for development and survival of the peripheral nervous system.

27 in a subpopulation of sensory neurons in the peripheral nervous system.

28 lling progenitor behavior in the central and peripheral nervous system.

29 n and transmission of noxious stimuli by the peripheral nervous system.

30 d for survival of neurons in the central and peripheral nervous system.

31 l connectivity and synaptic circuitry in the peripheral nervous system.

32 ammatory involvement of both the central and peripheral nervous system.

33 le is known about macrophages populating the peripheral nervous system.

34 cells are the nerve ensheathing cells of the peripheral nervous system.

35 iple other organs, including the central and peripheral nervous system.

36 the cases, indicating the involvement of the peripheral nervous system.

37 re efficient nerve impulse conduction in the peripheral nervous system.

38 n channel and drug target in the central and peripheral nervous system.

39 with a conserved role in myelination of the peripheral nervous system.

40 nscription factor for the myelination of the peripheral nervous system.

41 ures including the craniofacial skeleton and peripheral nervous system.

42 t to reach neuronal nuclei in ganglia of the peripheral nervous system.

43 nardo also performed detailed studies of the peripheral nervous system.

44 is an acute response to nerve injury in the peripheral nervous system.

45 rodevelopment and maintenance of central and peripheral nervous systems.

46 t excitatory transmission in the central and peripheral nervous systems.

47 icants, many of which target the central and peripheral nervous systems.

48 late many vital functions of the central and peripheral nervous systems.

49 of these disorders impact the central and/or peripheral nervous systems.

50 m of neurons and fibers in their central and peripheral nervous systems.

51 AL) is widely distributed in the central and peripheral nervous systems.

52 and are expressed throughout the central and peripheral nervous systems.

53 their proper targets in both the central and peripheral nervous systems.

54 isease complications in both the central and peripheral nervous systems.

55 iption of the bridge between the central and peripheral nervous systems.

56 ion and degeneration of both the central and peripheral nervous systems.

57 noninvasive gene transfer to the central and peripheral nervous systems.

58 ss of maternal Ube3a in the central, but not peripheral, nervous system.

59 nt includes nerve fibres that arise from the peripheral nervous system(1,2).

60 lieve clinical conditions by stimulating the peripheral nervous system(1-5).

61 Regenerative capability of the peripheral nervous system after injury is enhanced by Sc

62 display early and severe degeneration of the peripheral nervous system along with IF accumulation, bu

63 aS, including its ability to spread from the peripheral nervous system and along neuroanatomical trac

64 microtubule dynamics in vivo and ex vivo in peripheral nervous system and central nervous system neu

65 t birth with severe axon defects in both the peripheral nervous system and central nervous system so

66 strongly delayed axonal degeneration in the peripheral nervous system and CNS of wild-type mice of e

67 sition to migrate away and contribute to the peripheral nervous system and craniofacial skeleton.

68 chwann cells are the myelinating glia of the peripheral nervous system and dysfunction of these cells

69 an pseudorabies virus (PRV), that infect the peripheral nervous system and have to travel long distan

70 into the expression pattern of Jedi-1 in the peripheral nervous system and indicate that loss of Jedi

71 r Tomosyn-2) is expressed in the central and peripheral nervous system and is known to inhibit neurot

72 order that primarily affects the central and peripheral nervous system and muscles and in the latter

73 tools to study the brain, understanding the peripheral nervous system and other organs can similarly

74 process that involves both detection in the peripheral nervous system and perception in the CNS.

75 uded abnormal results of function studies of peripheral nervous system and special senses, conduct di

76 nalyses have expanded our knowledge of early peripheral nervous system and spinal cord development.

77 d by frataxin loss in neurons of the CNS and peripheral nervous system and that these changes are par

78 virus that can cause infections in both the peripheral nervous system and the central nervous system

79 involved in axonal degeneration in both the peripheral nervous system and the CNS, a process that ha

80 MR imaging of the peripheral nervous system and the limb-girdle muscle may

81 -/-) mice, which lack CB1 receptors from the peripheral nervous system and the spinal cord.

82 required to infect the Schwann cells of the peripheral nervous system and therefore may also be neur

83 y to defeat consecutive host barriers of the peripheral nervous system and together promote the poten

84 ntly expressed in neurons of the central and peripheral nervous systems and it clearly seems to be in

85 hR subtypes are expressed in the central and peripheral nervous systems and play a critical role in v

86 expressed throughout development of the CNS, peripheral nervous system, and kidneys.

87 key regulating functions in the central and peripheral nervous systems, and NK1R antagonists such as

88 Injuries to the peripheral nervous system are major sources of disabilit

89 privileged tissues such as the brain and the peripheral nervous system are shielded from plasma prote

90 wann cells, the principal glial cells of the peripheral nervous system, are now considered to be impo

91 nd the redox state by CIH in the central and peripheral nervous systems associated with the chemorefl

92 In the peripheral nervous system, axoglial contact at the nodes

93 In the peripheral nervous system, axonal neuregulin 1 type III

94 ry and stochastic simulations of central and peripheral nervous system axons from vertebrates and inv

95 tent and -incompetent sensory neurons in the peripheral nervous system but also allowed temporal tuni

96 investigated the role of glial cells in the peripheral nervous system by creating SMA mice selective

97 cranial nerves or nerves and roots from the peripheral nervous system by lymphoma, usually by B-cell

98 vities in the central nervous system and the peripheral nervous system by serving as ligands for the

99 oprotein mostly expressed in the central and peripheral nervous systems by different cell types in ma

100 ls (SCs) constitute a crucial element of the peripheral nervous system, by structurally supporting th

101 Mature neurons in the adult peripheral nervous system can effectively switch from a

102 neuropathies are inherited disorders of the peripheral nervous system caused by mutations in Schwann

103 u accumulation.SIGNIFICANCE STATEMENT In the peripheral nervous system, cholinergic modulation holds

104 Schwann cells myelinate distinct central and peripheral nervous system (CNS and PNS) domains along th

105 (SWCs), the myelinating glia of central and peripheral nervous system (CNS and PNS), respectively, r

106 The central and peripheral nervous system (CNS and PNS, respectively) ar

107 of psychosine results in diffuse central and peripheral nervous system (CNS, PNS) demyelination.

108 ment, many cells die in both the central and peripheral nervous systems (CNS and PNS, respectively).

109 us system (ENS) is the largest branch of the peripheral nervous system, comprising complex networks o

110 ated nerve fibers throughout the central and peripheral nervous system contain TRAAK in what is likel

111 In the peripheral nervous system, COVID-19 is associated with d

112 These include much of the peripheral nervous system, craniofacial skeleton, smooth

113 abilities secondary to central and, perhaps, peripheral nervous system damage.

114 The regenerative capacity of the peripheral nervous system declines with age.

115 n sclerotome anterior/posterior polarity and peripheral nervous system development, and these were co

116 nd migration, which in turn is essential for peripheral nervous system development.

117 iratory syndrome epidemics, cases of CNS and peripheral nervous system disease caused by SARS-CoV-2 m

118 directly contributes to the pathogenesis of peripheral nervous system diseases, including sensory ne

119 get for the treatment of various central and peripheral nervous system disorders, including Parkinson

120 ction causes a broad spectrum of central and peripheral nervous system disorders, ranging from microc

121 gical role of COMT inhibitors in central and peripheral nervous system disorders; (ii) to provide the

122 tributions to the development of central and peripheral nervous system dysfunction in LSDs.

123 During development of the peripheral nervous system, excess neurons are generated,

124 wing somatosensory neurons in the Drosophila peripheral nervous system exhibit organ sparing at the l

125 In the peripheral nervous system, extrinsic signals from the ax

126 n of HSV-1 in the CNS in comparison with the peripheral nervous system following ocular infection in

127 c arborization (da) neuron of the Drosophila peripheral nervous system for detection of thermal, mech

128 Despite the relevance of the peripheral nervous system for normal sensory and motor c

129 CIPN may involve multiple areas of the peripheral nervous system from the autonomic and dorsal

130 Here, we demonstrate that Ifit2 protects the peripheral nervous system from VSV infection as well.

131 hat Spry3 is highly expressed in central and peripheral nervous system ganglion cells in mouse and hu

132 The peripheral nervous system has remarkable regenerative ca

133 duced axonal degeneration in the central and peripheral nervous systems, has provided insight into a

134 However, its actions on the peripheral nervous system have been less characterized.

135 Several studies of VZV reactivation in the peripheral nervous system (herpes zoster) have been publ

136 et tissues, including skeletal muscle in the peripheral nervous system; however, the mechanisms by wh

137 ns that allow HSV to persist within the host peripheral nervous system, improved neuronal models are

138 ng regulates inflammation in the central and peripheral nervous system in ALS, supporting therapeutic

139 cking and neurodevelopmental deficits in the peripheral nervous system in Down syndrome.

140 be essential for myelination of axons in the peripheral nervous system in fish and mice.

141 By showing involvement of the peripheral nervous system in MS, this proof-of-concept s

142 gnosis of ALS and suggest involvement of the peripheral nervous system in the ALS pathogenetic cascad

143 is required for development of the brain and peripheral nervous system in this vertebrate embryo mode

144 ssays with neural cells from the central and peripheral nervous system in vitro and shown to be indep

145 polyneuropathy, an autoimmune disease of the peripheral nervous system in which humoral immune respon

146 aggregates occurs widely in the central and peripheral nervous systems in Parkinson's disease (PD).

147 the principal relay between the central and peripheral nervous systems in the autonomic ganglia.

148 rved in our cases, involving the central and peripheral nervous system, include deafness, optic neuro

149 ate extensively and give rise to most of the peripheral nervous system, including sympathetic, parasy

150 uronal types cultured from mouse central and peripheral nervous systems, including excitatory and inh

151 ural connectivity throughout the central and peripheral nervous systems, including the removal of sup

152 portant, as this process underlies both mild peripheral nervous system infection and severe spread to

153 In the peripheral nervous system, infiltrating monocyte-derived

154 SC receptors that detect peripheral nervous system injury remain incompletely und

155 ities for emerging classes of prostheses and peripheral nervous system interface technologies.

156 nt (16 of 17 patients [94.1%]; P = .008) and peripheral nervous system involvement (5 of 17 patients

157 0 years, 1.64; 95% CI, 1.19-2.27; P = .003), peripheral nervous system involvement (HR, 6.75; 95% CI,

158 nervous system, although dysfunction of the peripheral nervous system is a common manifestation.

159 The mature peripheral nervous system is a steady network structure

160 Schwann cell (SC) myelination in the peripheral nervous system is essential for motor functio

161 omoting axon regeneration in the central and peripheral nervous system is of clinical importance in n

162 in; however, the role of plasmalogens in the peripheral nervous system is poorly defined.

163 Myelination of the peripheral nervous system is required for axonal functio

164 ins unclear whether virus reactivated in the peripheral nervous system is transported to the CNS in a

165 ain hypersensitivity following injury to the peripheral nervous system, is common, greatly impairs qu

166 ferentiation of central precursor cells into peripheral nervous system-like Schwann cells that remyel

167 d widespread gene expression in, central and peripheral nervous system, liver, kidney and skeletal mu

168 engraft efficiently in the pool of resident peripheral nervous system macrophages.

169 widely used antitubulin cancer drugs on the peripheral nervous system may help guide clinical evalua

170 This novel POMT function in the peripheral nervous system may shed light on analogous fu

171 Central and peripheral nervous systems mediate adverse responses to

172 erivatives including neurons and glia of the peripheral nervous system, melanocytes, and bone and car

173 RS-mediated diseases specifically affect the peripheral nervous system-most commonly causing axonal p

174 In some cases, however, Schwann cells, the peripheral nervous system myelinating glia, are found re

175 ription factor SOX10, which is essential for peripheral nervous system myelination.

176 milieu may be a promising novel strategy in peripheral nervous system neuritis.

177 s heterozygosity strongly suppresses ectopic peripheral nervous system neurons in mir-279/996 mutants

178 In the peripheral nervous system, obesity-driven alterations in

179 In the peripheral nervous system of Drosophila melanogaster, di

180 ponse after misfolding and deposition in the peripheral nervous system of mutant transthyretin.

181 A on gadolinium retention in the central and peripheral nervous system of rats and to assess the func

182 that regulate visceral pain sensation in the peripheral nervous system of rats.

183 s emphasize the structural complexity of the peripheral nervous system of the siphon, and the importa

184 tion of ER stress is first quantified in the peripheral nervous system of type I diabetic rats.

185 Here, we examined the central and peripheral nervous systems of FE65-KO, FE65L1-KO and FE6

186 chronic stress-induced visceral pain in the peripheral nervous systems of rats.

187 affect multiple organs, with the central and peripheral nervous system often affected.

188 the development of cells in the central and peripheral nervous systems, olfactory epithelium and end

189 The requirement and influence of the peripheral nervous system on tissue replacement in mamma

190 investigating sleep, auditory functions and peripheral nervous system, ophthalmological studies incl

191 herapeutic agents can affect the central and peripheral nervous systems, patients must undergo a proc

192 esin acts as a signaling molecule regulating peripheral nervous system (PNS) and central nervous syst

193 of the major differences between the injured peripheral nervous system (PNS) and CNS is the pro- and

194 the context of the development of Drosophila peripheral nervous system (PNS) and neuromuscular juncti

195 entify particular neuronal cell types in the peripheral nervous system (PNS) and pinpoint specific ge

196 nt and long-term viability of neurons in the peripheral nervous system (PNS) and retina.

197 te nociceptive neuronal activity in both the peripheral nervous system (PNS) and the central nervous

198 Development and maintenance of the peripheral nervous system (PNS) are essential for an org

199 Macrophages (MPs) of the peripheral nervous system (PNS) are the main cellular ag

200 d natural incidence of ER stress in diabetic peripheral nervous system (PNS) argue for a major role o

201 protein that is constitutively expressed by peripheral nervous system (PNS) axons and Schwann cells.

202 Injury to the peripheral nervous system (PNS) causes a dramatic shift

203 nisms of how cholesterol/lipid metabolism in peripheral nervous system (PNS) contributes to the patho

204 cells (including Schwann cells; SCs) of the peripheral nervous system (PNS) could be purified and ex

205 tion presented with a broad range of CNS and peripheral nervous system (PNS) disease.

206 multiple sclerosis, and Parkinson's disease, peripheral nervous system (PNS) disorders such as chemot

207 metrical heminodes found in both the CNS and peripheral nervous system (PNS) during development.

208 Selective in vivo delivery of cargo to peripheral nervous system (PNS) has broad clinical and p

209 n, the contribution of hypoxic damage to the peripheral nervous system (PNS) has not been addressed.

210 High glucose levels in the peripheral nervous system (PNS) have been implicated in

211 ion of both central nervous system (CNS) and peripheral nervous system (PNS) in PD.

212 copy (SBSEM) is used to describe the sensory peripheral nervous system (PNS) in the tail of a cephalo

213 llowing central nervous system (CNS) but not peripheral nervous system (PNS) injury.

214 main limitation on axon regeneration in the peripheral nervous system (PNS) is the slow rate of regr

215 in these cells and in the myelination of the peripheral nervous system (PNS) is unknown.

216 Of patients presenting with pain or peripheral nervous system (PNS) manifestations, 39% were

217 chanosensory and chemosensory neurons of the peripheral nervous system (PNS) must signal to the motor

218 port that sulfated N-glycans are involved in peripheral nervous system (PNS) myelination.

219 Previous studies using cultured peripheral nervous system (PNS) neurons have demonstrate

220 viruses that establish life-long latency in peripheral nervous system (PNS) neurons of their native

221 es are neuroinvasive viruses that infect the peripheral nervous system (PNS) of infected hosts as an

222 Sensory and autonomic neurons of the peripheral nervous system (PNS) play a critical role in

223 ammals is severely limited, yet axons in the peripheral nervous system (PNS) regrow, albeit to a limi

224 Our understanding of other cells in the peripheral nervous system (PNS) remains limited.

225 N) neurons to understand whether ScNs of the peripheral nervous system (PNS) synapse with CN neurons

226 non-neuronal cells in the central (CNS) and peripheral nervous system (PNS) that nourish neurons and

227 contact with sensory neuron clusters of the peripheral nervous system (PNS), and blood cells (hemocy

228 (CNS), non-myelinating Schwann cells in the peripheral nervous system (PNS), and enteric glial cells

229 ed Big tau, is expressed mainly in the adult peripheral nervous system (PNS), but also in adult neuro

230 In the peripheral nervous system (PNS), developmental axon prun

231 immunoreactive neurons were detected in the peripheral nervous system (PNS), especially in lip and f

232 Schwann cells, the myelinating glia of the peripheral nervous system (PNS), originate from multipot

233 dendrocytes myelinate multiple axons; in the peripheral nervous system (PNS), Schwann cells (SCs) mye

234 During development of the peripheral nervous system (PNS), Schwann-cell-secreted g

235 dant not only in the shk CNS but also in its peripheral nervous system (PNS), which, as in other "mye

236 raphs of the dorsal root entry zone show the peripheral nervous system (PNS)-CNS transition of regrow

237 These peripheral nervous system (PNS)-experienced microglia ca

238 Apparent peripheral nervous system (PNS)-like and central nervous

239 ntegral to the formation and function of the peripheral nervous system (PNS).

240 ) displays this role primarily affecting the peripheral nervous system (PNS).

241 ell as axon regeneration after injury in the peripheral nervous system (PNS).

242 study pathological conditions affecting the peripheral nervous system (PNS).

243 blish lifelong latent infections in the host peripheral nervous system (PNS).

244 much less is known about macrophages of the peripheral nervous system (PNS).

245 rotein in multiple tissues, including in the peripheral nervous system (PNS).

246 nnel isoforms are primarily expressed in the peripheral nervous system (PNS): Na(V) 1.7, Na(V) 1.8 an

247 ertebrate development, the central (CNS) and peripheral nervous systems (PNS) arise from the neural p

248 h neurological disorders of both the CNS and peripheral nervous systems (PNS), yet few studies have d

249 egulate synaptic activity in the central and peripheral nervous system, presynaptically and postsynap

250 ow that Schwann cells (SCs), the glia of the peripheral nervous system, protect injured axons by virt

251 Developing neurons of the peripheral nervous system reach their targets via cues t

252 Cholinergic inputs originating from the peripheral nervous system regulate the inflammatory immu

253 Myelinating Schwann cells in the vertebrate peripheral nervous system rely on Brg1 (Smarca4) for ter

254 Development of a functional peripheral nervous system requires axons to rapidly inne

255 Brain and peripheral nervous system research conducted over the pa

256 s that efficiently transduce the central and peripheral nervous systems, respectively.

257 Understanding how the peripheral nervous system responds to electro-mechanical

258 rs that control Schwann cell myelination and peripheral nervous system responses to nerve injury.

259 Analysis of the peripheral nervous system revealed that Schwann cells an

260 In the peripheral nervous system, Schwann cells (SCs) demonstra

261 In the peripheral nervous system, Schwann cells (SCs) radially

262 tronic medicine seeks to decode and modulate peripheral nervous system signals to obtain therapeutic

263 physiology, and for treating diseases of the peripheral nervous system, such as chronic nausea, vomit

264 nal perspectives for chronic diseases of the peripheral nervous system, such as diabetic neuropathy a

265 ificant perturbations in eye development and peripheral nervous system, suggesting novel targets in a

266 (V)1.9 - are preferentially expressed in the peripheral nervous system, suggesting that their inhibit

267 Schwann cells (SCs), ensheathing glia of the peripheral nervous system, support axonal survival and f

268 ectual disability and additional central and peripheral nervous system symptoms but an absence of fro

269 e-Tooth disease (CMT) is a neuropathy of the peripheral nervous system that afflicts ~1:2500 people.

270 e stroma and pathogenic modifications to the peripheral nervous system that elevate metastatic capaci

271 Schwann cells are myelinating glia in the peripheral nervous system that form the myelin sheath.

272 we present the structural components of the peripheral nervous system that underlie its susceptibili

273 During the development of the peripheral nervous system, the large number of apoptotic

274 Unfortunately, in contrast to axons from the peripheral nervous system, the limited capacity of regen

275 ause a persistent, lifelong infection in the peripheral nervous system, the virus remains within the

276 en the aim of reading neural activity in the peripheral nervous system, this work has investigated an

277 an cause neuropathy in mice if it enters the peripheral nervous system through skin lesions; however,

278 onfirmed SH3TC2 mRNA expression in different peripheral nervous system tissues.

279 rain stimulation can be used to "bypass" the peripheral nervous system to induce multisensory illusio

280 ranging from neurons and glial cells of the peripheral nervous system to pigment cells, fibroblasts

281 chwann cells, the myelin-making cells of the peripheral nervous system to remyelinate adult CNS axons

282 The central and peripheral nervous system transcriptomes of the spider C

283 use model, in vivo viral reactivation in the peripheral nervous system, triggered by hyperthermic str

284 ly, we evaluated primary human NF1-deficient peripheral nervous system tumors and found that MNKs are

285 nding of rare human hereditary neuropathies, peripheral nervous system tumors, and common degenerativ

286 portantly, opioid receptors expressed in the peripheral nervous system undergo regulation uncommon to

287 defined neuronal subclasses within the mouse peripheral nervous system using an experimental strategy

288 Here we analyzed the role of Miz1 in the peripheral nervous system, using an early embryonic cond

289 Itch is induced chemically in the peripheral nervous system via a wide array of receptors.

290 alling factor controlling myelination in the peripheral nervous system, via signalling through ErbB t

291 y inside the tissues of both the central and peripheral nervous systems was normalized, and the neuro

292 tion require concurrent reinnervation by the peripheral nervous system, we hypothesized that cardiac

293 eurological disorder of both the central and peripheral nervous systems, we identified a homozygous p

294 They are widely expressed in the central and peripheral nervous systems where they participate in a r

295 le is known about expression of UBE3A in the peripheral nervous system, where loss of maternal UBE3A

296 k between macrophages and nociceptors in the peripheral nervous system, which may contribute to the s

297 a group of genetic disorders that affect the peripheral nervous system with heterogeneous pathogenesi

298 shown to be of particular importance in the peripheral nervous system with pharmacological and genet

299 ves our understanding of autoimmunity in the peripheral nervous system with potential relevance for h

300 Periaxin (PRX) is an abundant protein in the peripheral nervous system, with an important role in mye