ライフサイエンスコーパス: peripheral vascular resistance

1 ctivity regulates blood pressure by altering peripheral vascular resistance.

2 through modulation of cardiac output and/or peripheral vascular resistance.

3 sure, there is no decrease in either MSNA or peripheral vascular resistance.

4 een sympathetic activity and the increase in peripheral vascular resistance.

5 The effect of water is mediated by increased peripheral vascular resistance.

6 nsiveness, and vascular volume and increased peripheral vascular resistance.

7 a and a sympathetically mediated increase in peripheral vascular resistance.

8 ex from controlling these circuits to adjust peripheral vascular resistance.

9 em may be due, in part, through reduction of peripheral vascular resistance.

10 output (36%), stroke volume (30%), and total peripheral vascular resistance (45%), along with increas

11 in peripheral mean arterial pressure (PMAP), peripheral vascular resistance, AIx, aortic stiffness an

12 Women who develop PE have an increase in peripheral vascular resistance and a mild reduction in L

13 on cardiac systolic and diastolic function, peripheral vascular resistance and arrhythmogenesis.

14 ving two important functions: (a) regulating peripheral vascular resistance and blood pressure and (b

15 -Hb is a composite effect created by reduced peripheral vascular resistance and direct inhibition of

16 were improved by drug treatments that reduce peripheral vascular resistance and hypertension, consist

17 siologic parameters (e.g., decrease in total peripheral vascular resistance and increase in cardiac s

18 be a compensatory mechanism for the blunted peripheral vascular resistance and lower central volume.

19 In contrast, the decline in peripheral vascular resistance and the increase in nitri

20 s been traditionally ascribed to decrease in peripheral vascular resistance and to an antiatherogenic

21 ETA/B receptor antagonist TAK-044 decreases peripheral vascular resistance and, to a lesser extent,

22 greater effects on arterial pressure, total peripheral vascular resistance, and cardiac index, compa

23 re, cardiac output, stroke volume, and total peripheral vascular resistance by diaspirin crosslinked

24 d PDE3A gene drives mechanisms that increase peripheral vascular resistance causing hypertension.

25 in constant BP in salt resistance, renal and peripheral vascular resistance falls and is associated w

26 index and lowered mean arterial pressure and peripheral vascular resistance in patients and healthy c

27 harmacologic strategies designed to maintain peripheral vascular resistance in patients with insuffic

28 The mechanisms for elevation of peripheral vascular resistance in spontaneously hyperten

29 Instead, an increase in peripheral vascular resistance in these animals is consi

30 this study investigated how changes in fetal peripheral vascular resistance, in plasma concentrations

31 Cooling resulted in a greater increase in peripheral vascular resistance index (+515+/-160 versus

32 whereas reducing mean arterial pressure and peripheral vascular resistance index (P<0.001 for all) i

33 ed fall in cardiac output with no changes in peripheral vascular resistance may contribute to (pre)sy

34 Peripheral vascular resistance, MSNA, and norepinephrine

35 s a reduced ability of astronauts to augment peripheral vascular resistance, often resulting in ortho

36 ts of a sympathetically mediated increase in peripheral vascular resistance, parasympathetically medi

37 Peripheral vascular resistance (PVR), cardiac output (CO

38 Increased peripheral vascular resistance, rather than salt retenti

39 ine in CHF rats may be due to a reduction in peripheral vascular resistance since epicardial lidocain

40 7% +/- 3% of baseline and increases in total peripheral vascular resistance to 146% +/- 5% of baselin

41 al nervous system adjusts cardiac output and peripheral vascular resistance to changing physiological

42 nd (3) assess the contribution of changes in peripheral vascular resistance to the glucocorticoid-ind

43 en, high SNA is associated with higher total peripheral vascular resistance (TPR), and appears to be

44 ine-induced hypertension consist of elevated peripheral vascular resistance, ventricular vascular unc

45 rates between groups were not observed, and peripheral vascular resistance was significantly greater

46 Following multivariable analysis, peripheral vascular resistance was significantly higher

47 Cardiac output (and total peripheral vascular resistance) was determined by thermo

48 One of the hallmarks is a rise of peripheral vascular resistance, which largely depends on