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1  antibodies (0 of 8); 27% had neutrophils in peritubular capillaries.
2 rteries and afferent arterioles, and 40X for peritubular capillaries.
3 s constricting the descending vasa recta and peritubular capillaries.
4 epithelium and occasionally, in contact with peritubular capillaries.
5 dr/Vegfr2) is largely restricted to adjacent peritubular capillaries.
6  with a striking reduction in the density of peritubular capillaries.
7 HLA) cause graft injury identified by C4d in peritubular capillaries.
8  in association with immature glomerular and peritubular capillaries.
9 C3d) deposition was diffuse and prominent in peritubular capillaries.
10  microcirculation, especially with regard to peritubular capillaries.
11 optosis in arteries, tubules, glomeruli, and peritubular capillaries.
12                                    Injury to peritubular capillaries and capillary basement membrane
13 maging highlighted the notable dilatation of peritubular capillaries and decreased kidney blood flow
14 irus infection and were more frequent within peritubular capillaries and glomeruli from antibody-medi
15 scular cells and how these cells detach from peritubular capillaries and migrate to the interstitial
16 Additionally, we found endothelial damage in peritubular capillaries and vasa recta.
17 atient with hemoglobin SC disease who showed peritubular capillary and vasa recta thrombi and capilla
18 ne, proteinuria, DSA+, Banff C4d staining of peritubular capillaries+, and chronic interstitial fibro
19 , 0.93 for distal tubular segments, 0.81 for peritubular capillaries, and 0.85 for arteries and affer
20  had acute graft dysfunction, neutrophils in peritubular capillaries, and a concurrent positive cross
21 IgM, and C5b-9 were scored in the glomeruli, peritubular capillaries, and arterioles.
22 surfaces of endothelial cells in glomerular, peritubular capillary, and arterial renal sites of matur
23                                 Human kidney peritubular capillaries are particularly susceptible to
24 o cases was the absence of C4d deposition in peritubular capillaries as well as the absence of C1q-bi
25  The aging rats also displayed focal loss of peritubular capillaries (as noted by focally decreased R
26                                       Severe peritubular capillary basement membrane multilayering (P
27                                              Peritubular capillary basement membrane multilayering on
28 pair that manifest as multilamination of the peritubular capillary basement membrane or arteriopathy
29  found that decreased blood flow velocity in peritubular capillaries by kidney congestion and upregul
30  found that decreased blood flow velocity in peritubular capillaries by kidney congestion and upregul
31 asured in double-perfused tubules (lumen and peritubular capillaries) by manipulating the applied tra
32 dy (DSA) and histologic evidence of AMR with peritubular capillaries C4d deposition.
33 complement pathway, it was hypothesized that peritubular capillary C4d deposition might distinguish t
34           These results suggest that diffuse peritubular capillary C4d deposition without rejection i
35 teria, including capillaritis, glomerulitis, peritubular capillary C4d deposition, and donor-specific
36         In ABO-incompatible grafts, however, peritubular capillary C4d is often present on protocol b
37 ic associations and clinical implications of peritubular capillary C4d staining from long-term renal
38 nsitivity and specificity by the presence of peritubular capillary C4d staining on renal biopsy and d
39                                              Peritubular capillary C4d staining was negative in all c
40                                         Rare peritubular capillary C4d staining was present in 50% of
41 y-one patients (group A) had strong, diffuse peritubular capillary C4d staining without histologic ev
42  degree of microcirculation inflammation and peritubular capillary C4d staining.
43 tients (group B) had negative or weak, focal peritubular capillary C4d staining.
44 onents correlated with the g score, DSA, and peritubular capillary C4d+.
45 r endothelium, resulting in the formation of peritubular capillary C4d.
46 ury, with a relative loss of staining of the peritubular capillaries compared with young rats.
47                                  The loss of peritubular capillary density and caliber at week 8 clos
48 y rarefaction, which refers to a decrease in peritubular capillary density leading to hypoxic and isc
49  failed repair was inversely correlated with peritubular capillary density.
50 blast development, with secondary effects on peritubular capillary differentiation.
51 nding of the positively charged AuNPs to the peritubular capillaries during the initial phase of elim
52 h mice to visualize, analyze, and quantitate peritubular capillary dynamics after AKI.
53 kidney, TNFR1 is expressed in glomerular and peritubular capillary EC, and some tubular cells, and co
54 ely associated with CD31- and Tie-2-positive peritubular capillary endothelia, and some of the alpha
55 was also associated with more glomerular and peritubular capillary endothelial cell loss in associati
56 us vehicle, P < 0.05), a twofold increase in peritubular capillary endothelial cell proliferation (1.
57 nal unit, formed by resident macrophages and peritubular capillary endothelial cells, which monitors
58 gradation split-product 4d (C4d) staining of peritubular capillaries+, endothelial C4d staining of gl
59             Damage to tubular epithelium and peritubular capillary endothelium also was seen.
60 herefrom perturb normal interactions between peritubular capillary endothelium and pericyte-like fibr
61     Additionally, HIFD significantly reduced peritubular capillary erythrocyte congestion and improve
62 evels and contrasted with nominal changes in peritubular capillary flow and plasma creatinine.
63 ntations of renal tubules, interstitium, and peritubular capillaries from which morphometry features
64      Dynamic photoacoustic microscopy of the peritubular capillary function and tissue oxygen metabol
65 ohistochemistry and evaluated on arterioles, peritubular capillaries, glomeruli, and tubular basement
66                   Although C4d deposition in peritubular capillaries has been identified as a strong
67 i1(+) kidney pericytes in the maintenance of peritubular capillary health, and the consequences of pe
68 d deposited prominently and diffusely in the peritubular capillaries in all AHR biopsies (16 of 16).
69 y-implicated in the progressive attrition of peritubular capillaries in areas of tubular atrophy and
70 ition and thrombosis in renal glomerular and peritubular capillaries in association with a fall in he
71 vealed significant pathologic alterations in peritubular capillaries in CKD.
72                     Endothelial cells of the peritubular capillaries in grafts with ABMR expressed fa
73                 We explored the integrity of peritubular capillaries in relation to expression of vas
74 s are also related to the presence of C4d in peritubular capillaries in TG biopsies.
75  saturation of hemoglobin, and blood flow in peritubular capillaries in vivo.
76 of the basement membranes (glomerular and/or peritubular capillaries) in milder forms of injury.
77 of microvascular inflammation (glomerulitis, peritubular capillary infiltrates; P values 0.001) and s
78 of microvascular inflammation (glomerulitis, peritubular capillary infiltrates; p-values <=0.001) and
79 ated rejection manifests with glomerular and peritubular capillary inflammation and transplant glomer
80 ial, mononuclear cell infiltrates; prominent peritubular capillary inflammatory cell margination; pat
81  be the consequence of ischemia secondary to peritubular capillary injury and altered eNOS expression
82                            C4d deposition in peritubular capillaries is a specific marker for the pre
83                              C4d staining in peritubular capillaries is a well-established feature of
84        The relevance of C4d staining outside peritubular capillaries is not well understood.
85                    Ang-1 therapies attenuate peritubular capillary loss in adult models of tubulointe
86 al perfusion and oxygenation, and attenuated peritubular capillary loss, tubular injury, and fibrosis
87 nti-angiogenic effects leading to aggravated peritubular capillary loss.
88 jury as evidenced by reduced C4d staining in peritubular capillaries, microcirculation inflammation,
89       Glomerular CD68+ cells correlated with peritubular capillary multilamellation, and similarly, t
90                                              Peritubular capillary multilayering (PTCML) of basement
91 loss, the optimal cutoff for the fraction of peritubular capillaries needed to establish a positive s
92  was associated with a higher density of the peritubular capillary network in the corticomedullary ju
93                       Integrity of the renal peritubular capillary network is an important limiting f
94 alk by Vegfa is essential for maintenance of peritubular capillary networks in kidney.
95 e AKI, we measured a 40%+/-7.4% reduction in peritubular capillary number (P<0.05) and a 36%+/-4% dec
96  genetically labeled endothelia, we compared peritubular capillary number and size after moderate AKI
97 medullary descending vasa recta and cortical peritubular capillaries occurred near pericyte somata, a
98   Diffuse C4d deposition was detected in the peritubular capillaries of 6 of 48 (13%) biopsies.
99 ecificity the endothelium of the fenestrated peritubular capillaries of the kidney and those of the i
100 nduced an acute and significant reduction in peritubular capillary oxygen saturation of hemoglobin, c
101  In ACR, no more than trace C4d was found in peritubular capillaries (P < 0.0001 versus AHR), and no
102 etection of the C4d complement product along peritubular capillaries (PC) may indicate humoral reject
103  organ level, trametinib completely restored peritubular capillary perfusion in the kidney.
104 e increased pericytes around kidney cortical peritubular capillaries, perhaps an indirect consequence
105 calized E-selectin to the endothelium of the peritubular capillary plexus.
106 d by determining whether C4d is deposited in peritubular capillaries (PTC) and whether it correlates
107 r of layers of basement membrane (BM) around peritubular capillaries (PTC) can be used in a cohort of
108                            C4d deposition in peritubular capillaries (PTC) of renal allografts has be
109 graft function (DGF) and to an early loss of peritubular capillaries (PTC).
110 odies (DSA) and complement C4d deposition in peritubular capillaries (PTC).
111 inical and molecular significance of minimal peritubular capillary (PTC) and isolated glomerular C4d+
112 whether, on electron microscopy examination, peritubular capillary (PTC) basement membrane multilayer
113 for allograft dysfunction, were assessed for peritubular capillary (PTC) C4d and CD55 expression.
114 ediated rejection, which is characterized by peritubular capillary (PTC) deposition of C4d.
115                         Deposition of C4d in peritubular capillaries (PTCs) has been shown to be a se
116  hyperuricemic rats, endothelial staining in peritubular capillaries (PTCs) was substantially decreas
117 nic kidney diseases, leads to rarefaction of peritubular capillaries (PTCs), promoting secondary isch
118  vehicle, P < 0.01), a threefold decrease in peritubular capillary rarefaction (P < 0.01), and a twof
119                                              Peritubular capillary rarefaction is hypothesized to con
120                                     As such, peritubular capillary rarefaction may be a consequence r
121                   We detected glomerular and peritubular capillary rarefaction, macrophage infiltrati
122                                              Peritubular capillary rarefaction, which refers to a dec
123 igger transient tubular injury and permanent peritubular capillary rarefaction.
124 rein, we report that the proximal tubule and peritubular capillary, rather than the glomerulus, serve
125 operfusion of L-arginine (10[-3] M) into the peritubular capillaries reduced the maximum TGF response
126                            The percentage of peritubular capillaries staining positively for C4d was
127 ata show that in long-term renal allografts, peritubular capillary staining for C4d occurs in approxi
128 ion, Nec-1 prevented RCM-induced dilation of peritubular capillaries, suggesting a novel role unrelat
129  of rejection had widespread C4d deposits in peritubular capillaries, suggesting a pathogenic role of
130 ted with preservation or accelerated loss of peritubular capillaries, suggesting no significant pro-a
131 anism for this epidemiologic link is loss of peritubular capillaries triggering chronic hypoxia.
132             In chronic kidney disease (CKD), peritubular capillaries undergo anatomic and functional
133 howed gp-Fy in the endothelium of glomeruli, peritubular capillaries, vasa recta, and the principal c
134                                       C4d in peritubular capillary walls distinguishes AHR from ACR,
135                        Fascin1 expression in peritubular capillaries was also induced in a rat model
136                       The presence of C4d in peritubular capillaries was an independent risk factor f
137 ation of polymorphonuclear leukocytes within peritubular capillaries was noted at the acute phase aft
138 ndothelial cell adhesion molecule-expressing peritubular capillaries was preceded by marked decreases
139 e higher in NRs, whereas Banff-C4d scores of peritubular capillaries were increased in the Rs.
140                               Glomerular and peritubular capillaries were increased with endothelial
141 sensitive AR, and widespread C4d deposits in peritubular capillaries were present in 18 of these 19 (
142 ficiency causes dysmorphogenesis of cortical peritubular capillaries, with adjacent cells expressing
143 scopy revealed dilation of renal tubules and peritubular capillaries within 20 minutes of RCM applica

 
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