ライフサイエンスコーパス: peritumoral

1 ass (intratumoral) or surrounding the tumor (peritumoral).

2 luation or SLN mapping (ex vivo, subserosal, peritumoral, 1% isosulfan blue dye) and ultrastaging wit

3 Peritumoral (50 micro g/tumor, twice a week) or i.p. (10

4 We observed peritumoral accumulation of CXCR6-associated lymphocytes

5 In vivo experiments confirm the presence of peritumoral acid gradients as well as cellular toxicity

6 hanger-1, both of which were associated with peritumoral acidosis.

7 y acinar cells, becoming highly prominent in peritumoral acini, and particularly high in acinar ducta

8 We compared gene-expression differences in peritumoral adipose tissue and tumour tissue in an addit

9 scriptomic differences in primary tumour and peritumoral adipose tissue between obese patients and th

10 with non-metastatic clear cell RCC (the MSK peritumoral adipose tissue cohort).

11 at a normal weight in the COMPARZ, TCGA, and peritumoral adipose tissue cohorts.

12 lial hyaluronan receptor-1 were found within peritumoral adipose tissue from melanoma-bearing K14-VEG

13 We found increased peritumoral adipose tissue inflammation in obese patient

14 erplay between the clear cell RCC tumour and peritumoral adipose tissue microenvironment might have c

15 vironment that vary by BMI in the tumour and peritumoral adipose tissue, which might contribute to th

16 hydrogel-based cell patch are developed for peritumoral administration for treating primary and meta

17 We show that peritumoral administration of these vectors results in t

18 Peritumoral administration of u-oligonucleotide results

19 mice, we found that SIX1 expression promoted peritumoral and intratumoral lymphangiogenesis, lymphati

20 In this study, peritumoral and intratumoral radiomics was used to ident

21 reveal higher WNT5A and GdECs expression in peritumoral and recurrent GBMs relative to matched intra

22 ADV.RSV-tk vector and HSV1-tk expression in peritumoral and remote liver tissue at higher doses.

23 addition, we identified CARD11 mutations in peritumoral and sun-exposed skin, suggesting that CARD11

24 n an area </= 500 mum from the tumor border (peritumoral) and was correlated with recurrence, metasta

25 es by tofacitinib inhibited STAT3 signaling, peritumoral angiogenesis, and cellular scattering.

26 un therapy with interleukin-12 cDNA into the peritumoral area of immunocompetent 129/J mice with life

27 f monocytes/macrophages, particularly in the peritumoral area, since we found no evidence for monocyt

28 sed on intratumoral regions, overlooking the peritumoral area.

29 he tumor mass; (3) decreases astrogliosis in peritumoral area; and (4) reduces glioma cell infiltrati

30 al analysis of chronic pancreatitis (CP) and peritumoral areas in PDAC tissues showed that AGR2 was p

31 and averaged across the homologues of these peritumoral areas in the contralateral hemisphere.

32 etastatic nodules were associated with focal peritumoral areas of infiltrating inflammatory cells and

33 levels: global average, averaged across the peritumoral areas, and averaged across the homologues of

34 ells are clustering around the mature DCs in peritumoral areas, thus resembling the DC-T cell cluster

35 ascular permeability specifically in tumoral/peritumoral areas, which resulted in fast and sustained

36 y increased vascular permeability in tumoral/peritumoral areas, without interfering with drug plasma/

37 he brain by infiltrative cell migration into peritumoral areas.

38 reg) cells in the intratumoral compared with peritumoral areas.

39 ls, whereas mature DCs adhere selectively to peritumoral areas.

40 ), present in 20/32 samples, are confined to peritumoral areas.

41 to quantify tracer uptake in the tumoral and peritumoral areas.

42 in endothelial cells of tumor-associated and peritumoral arteries.

43 tumor and increases NF-kappaB activation in peritumoral astrocytes.

44 When measured using histomorphometry, peritumoral BIO administration improved bone quality at

45 lation zone while transiently disrupting the peritumoral blood-brain barrier (BBB).

46 radioactive colloid injection also received peritumoral blue dye injection.

47 hasone in an experimental model of vasogenic peritumoral brain edema.

48 ain tumors, cancer cells infiltrate into the peritumoral brain structures which results in inevitable

49 s all carried a Braf V545E mutation, whereas peritumoral brain tissue of either strain had the wild-t

50 A decrease in the water content of tumor and peritumoral brain tissue was observed with proton-densit

51 ociated necrosis and possibly to seizures in peritumoral brain tissue.

52 ogies are used to analyze the changes in the peritumoral brain zone (PTZ).

53 The microanatomic distribution of benign peritumoral Ca++ in relation to the mass is detailed.

54 liferation of phytohaemagglutinin-stimulated peritumoral CD4+ T cells was shown after the addition of

55 ly suppressed phytohaemagglutinin-stimulated peritumoral CD4+ T-cell proliferation (p=0.005, n=10), p

56 ssion in MCPyV-negative tumors and increased peritumoral CD8+ T lymphocytes surrounding MCPyV-positiv

57 l CD4+ T-cell proliferation (p=0.005, n=10), peritumoral CD8+ T-cell proliferation (p=0.015, n=9), an

58 nation, the lesion periphery showed moderate peritumoral changes.

59 ic signal was significantly increased in the peritumoral ciliary body compared with the nonperitumora

60 -40-positive lymphatics were detected in the peritumoral ciliary body.

61 Therapeutic interventions aimed at reducing peritumoral clot formation and enhancing NK cell functio

62 amined pathophysiological alterations in the peritumoral cortex of patients undergoing tumor resectio

63 cells from mesial temporal lobe epilepsy or peritumoral cortex tissue expressed P2Y12 receptors.

64 l temporal lobe epilepsy or cortical glioma (peritumoral cortex).

65 unity following cryoablation with or without peritumoral CpG injection were conducted using two HER2/

66 arative proteomic profiling was performed on peritumoral cyst fluid and serum.

67 CNS peritumoral cyst formation is initiated by increased tum

68 tunity to examine the pathophysiology of CNS peritumoral cyst formation.

69 Because they commonly produce peritumoral cysts and because serial magnetic resonance

70 occurrence and frequent clinical effects of peritumoral cysts in the central nervous system (CNS), t

71 lial growth factor levels were determined in peritumoral cysts.

72 ociated with the appearance and evolution of peritumoral cysts.

73 he generation of MDA-MB-231-stable clones or peritumoral delivery in MDA-MB-231 xenografted mice, str

74 ared to systemic and intratumoral injection, peritumoral delivery of MSLN CAR-T cells using the TSPs

75 CM showed a significantly higher intra- and peritumoral density of proliferating lymphatics than did

76 near signal intensity representing selective peritumoral deposition in macrophages.

77 c hepatic parenchymal changes, which include peritumoral desmoplastic reaction, inflammatory cell inf

78 related with a thick tumor border containing peritumoral desmoplastic reaction, peritumoral inflammat

79 Peritumoral Dice similarity coefficients for Glu/tCr and

80 Peritumoral diffusion-tensor MR imaging metrics enable t

81 One single peritumoral dose of poly A:U was sufficient to induce IF

82 he most common locations of Ca++ were benign peritumoral ducts (62%) and ductal carcinoma in situ (54

83 L-R2 in sections of PDACs and non-neoplastic peritumoral ducts from patients.

84 Moreover, E2 remodeled peritumoral ECM architecture in WT animals, modifying al

85 As recurrence typically arises from the peritumoral edema adjacent to the resected bulk tumor, t

86 Moreover, variants in the peritumoral edema included unique cancer driver mutation

87 MRI clearly showed peritumoral edema that developed and slowly and progress

88 to enhancing necrotic portions of tumor and peritumoral edema were drawn, and quantitative image fea

89 argins, heterogeneous enhancement, prominent peritumoral edema, and bone destruction.

90 he necrotic and nonenhancing tumor core, the peritumoral edema, and the contrast-enhancing tumor-were

91 patients involve the management of seizures, peritumoral edema, medication side effects, and venous t

92 mably caused by tumor cells infiltrating the peritumoral edema.

93 n among the noncancerous brain tissue of the peritumoral edema.

94 neation of white matter tracts in regions of peritumoral edema.

95 ed to segment three multiclass tissue types (peritumoral edematous, infiltrated, or treatment-changed

96 features were associated with grade III STS: peritumoral enhancement (odds ratio [OR], 3.4; P = .003)

97 tures including necrosis, heterogeneity, and peritumoral enhancement of soft-tissue sarcomas were ass

98 In the late arterial phase, peritumoral enhancement or the presence of daughter nodu

99 or size, nonsmooth tumor margins, TTPVI, and peritumoral enhancement were significantly related to th

100 adiologic features (nonsmooth tumor margins, peritumoral enhancement, and TTPVI) was associated with

101 as tumor dimension, nonsmooth tumor margins, peritumoral enhancement, and TTPVI, have high accuracy i

102 ypoattenuating halo, tumor-liver difference, peritumoral enhancement, and tumor margin.

103 number of lesions, tumor margins, TTPVI, and peritumoral enhancement.

104 e to those at a normal weight, especially in peritumoral fat near the tumour.

105 rcumscribed mass, perinephric fat stranding, peritumoral fat planes obscured, retroperitoneal fluid (

106 rom the lungs associated with an increase in peritumoral fibrin and platelet clot formation was obser

107 The expression of SPARC by peritumoral fibroblasts portends a poorer prognosis for

108 The demonstration that peritumoral fibroblasts synthesize most of the MMPs in h

109 s of human intrahepatic CCA, including dense peritumoral fibrosis, increased inducible nitric oxide s

110 In contrast, TLR5 activation by peritumoral flagellin treatment substantially increased

111 Increased peritumoral fluorocholine uptake is a distinguishing cha

112 Tumoral gas, peritumoral gas, and PVG dissipated within 20 minutes af

113 ign cause of transient PVG, tumoral gas, and peritumoral gas.

114 Peritumoral gene gun introduction of interleukin-12 may

115 MYCN(A)RB1(+/+) retinoblastomas showed peritumoral hemorrhage (in 17 of 21 children; specificit

116 AZ and that deletion of Yap and Taz in these peritumoral hepatocytes accelerated tumor growth.

117 on was increased in HCC and benign-appearing peritumoral hepatocytes compared with remote benign hepa

118 sely, experimental hyperactivation of YAP in peritumoral hepatocytes triggered regression of primary

119 Additionally, the CNN captures the peritumoral heterogeneity better than conventional textu

120 icant, and the difference in FA decreases in peritumoral hyperintense regions between these tumors ap

121 Mean FA values in peritumoral hyperintense regions were 0.178 (43% of norm

122 Mean ADCs in peritumoral hyperintense regions were 1.309 x 10(-3) mm2

123 guided SLN mapping and lymphadenectomy after peritumoral ICG injection.

124 racellular pH, 7.18 +/- 0.03) (P = .008) and peritumoral immune cell exclusion.

125 suppressive microenvironment, different from peritumoral immune hotspots, warranting further study in

126 regulatory cells in intratumoral but not in peritumoral immune hotspots, with tertiary lymphoid stru

127 creased Dukes stage was associated with less peritumoral infiltrate (Jass criteria: P < 0.001, Klintr

128 The results of the present study suggest low peritumoral infiltrate (Klintrup criteria) and increased

129 d neutrophil count (P < 0.001) and low-grade peritumoral infiltrate (P < 0.05, Klintrup criteria).

130 Jass and Klintrup criteria for peritumoral infiltrate were directly associated (P < 0.0

131 according to Jass and Klintrup criteria for peritumoral infiltrate.

132 nic tumor because they lacked endotumoral or peritumoral infiltrates almost entirely.

133 lasia was associated with significantly less peritumoral inflammation (rho - 0.43, p < 0.0001).

134 essive phenotype of cervical cancer, reduced peritumoral inflammation, and inferior survival.

135 ontaining peritumoral desmoplastic reaction, peritumoral inflammation, and vascular proliferation at

136 Both localized peritumoral inflammatory cell infiltrate and the host sy

137 y and diameter and with impaired drainage of peritumoral injected liposomes specific for lymph vessel

138 ET/CT lymphoscintigraphy was performed after peritumoral injection of (89)Zr-nanocolloidal albumin.

139 ar injection of technetium is as accurate as peritumoral injection of blue dye.

140 In 560, peritumoral injection of isosulfan blue dye was also per

141 Peritumoral injection of isosulfan blue dye was performe

142 Peritumoral injection of lidocaine before breast cancer

143 the axilla (MOVA) started immediately after peritumoral injection of Millipore-filtered 99mTc-sulfur

144 Peritumoral injection of radioactive colloid has been us

145 dermal injection compared with subdermal or peritumoral injection of radioactive colloid.

146 Upon direct peritumoral injection of the hydrogel and with the treat

147 reast cancer and melanoma, a single low dose peritumoral injection of the therapeutic hydrogel promot

148 is being revisited via the strategy of intra/peritumoral injection with the idea of stimulating the p

149 rial would drain to the same lymph node as a peritumoral injection, regardless of the location of the

150 ive- to sevenfold more radioactive than with peritumoral injection, which simplifies SLN localization

151 the dermal injection technique compared with peritumoral injection.

152 erative lymphatic mapping was performed with peritumoral injections of blue dye alone or in combinati

153 uid flow velocity, we used a simple model of peritumoral interstitial fluid flow to calculate the flu

154 f radioactivity distribution was observed in peritumoral liver tissue in animals given injections of

155 tion of the biomechanical environment in the peritumoral liver tissue.

156 , we used samples of human blood, normal and peritumoral liver, and hepatocellular carcinoma (HCC) to

157 activity accumulation was more pronounced in peritumoral liver, which was confirmed by reverse transc

158 of primary human HCCs compared with matching peritumoral livers.

159 The peritumoral LNR exceeded 2.0 in seven high-grade gliomas

160 In addition, peritumoral lymph node-like structures were observed in

161 w that miR-221-3p is closely correlated with peritumoral lymphangiogenesis and lymph node (LN) metast

162 have documented a strong correlation between peritumoral lymphangiogenesis and tumor dissemination.

163 ed heparanase levels significantly increased peritumoral lymphangiogenesis in vivo and promoted the t

164 Peritumoral lymphangiogenesis was present in the ciliary

165 As a result of peritumoral lymphangiogenesis, metastases in lymph nodes

166 primary tumor but also in lymph nodes, with peritumoral lymphatic vessel density reduced in SK1-I-tr

167 Peritumoral lymphatic vessels connect the primary tumor

168 Hot spots of proliferating intratumoral and peritumoral lymphatic vessels were detected in a large n

169 ine T241 fibrosarcomas induced the growth of peritumoral lymphatic vessels, which occasionally penetr

170 ular extension, and as such, the presence of peritumoral lymphatics is not recommended as a prognosti

171 IFNgamma) ELISPOT assays were used to assess peritumoral lymphocyte function in vitro.

172 a positive correlation was observed between peritumoral lymphocyte ratio and (68)Ga-FAPI PET/CT-to-(

173 expressed in TILs (81% vs 28%; P < .001) and peritumoral lymphocytes (90% vs 28%; P < .001) of POLE a

174 The numbers of peritumoral lymphocytes and macrophages increased during

175 a (GCLS) is characterized by dense intra-and peritumoral lymphocytic infiltration and a high rate of

176 tumors that are locally invasive and exhibit peritumoral lymphovascular invasion.

177 Peritumoral MD and FA values indicated no statistically

178 garding intraaxial tumors, the measured mean peritumoral MD of metastatic lesions, 0.733 x 10(-3) mm(

179 egative mammary fat pad (MFP) tumors; and by peritumoral MFP injection of the targeted imaging probe

180 scribe the distinct physical features of the peritumoral microenvironment and link their relationship

181 inhibition, but how hyperexcitability in the peritumoral microenvironment evolves in an immunocompete

182 re novel insight about underlying biology of peritumoral microstructural heterogeneity, providing pot

183 as defined by (18)F-FDG imaging, but also in peritumoral microvasculature.

184 direct evidence that intratumoral as well as peritumoral monocytes/macrophages act to limit tumor siz

185 pressing OSM-induced cellular scattering and peritumoral neovascularization of orthotopic xenografts.

186 mporal volumetric change of the habitats and peritumoral/nodal tissue between baseline and midtreatme

187 n, n=10 tumours) and 4-1BB (12.6%, n=9) than peritumoral non-regulatory T cells and Tregs from periph

188 coefficient (ADC) measurements compared with peritumoral nonenhancing biopsy regions (P < .01).

189 3 adult patients yielded 16 enhancing and 14 peritumoral nonenhancing specimens.

190 board-approved study, multiple enhancing and peritumoral nonenhancing stereotactic neurosurgical biop

191 sue, Moussai et al. show that macrophages in peritumoral nonlesional skin near squamous cell carcinom

192 to remain proliferative and migrate into the peritumoral normal tissue producing the invasive phenoty

193 ate protons will diffuse from the tumor into peritumoral normal tissue subjecting nontransformed cell

194 This chronic exposure of peritumoral normal tissue to an acidic microenvironment

195 The difference in FA decreases in peritumoral normal-appearing WM between gliomas and meni

196 Mean FA values for peritumoral normal-appearing WM were 0.375 (83% of norma

197 Mean ADCs in peritumoral normal-appearing WM were 0.723 x 10(-3) mm2/

198 rawn from the tumoral centre, periphery, and peritumoral oedema (3 ROIs for each) followed by normali

199 hase II clinical testing of intratumoral and peritumoral ONYX-015 injection in 37 patients with recur

200 Two women each were given peritumoral or circumareolar injections of 100, 200, 300

201 ected directly into the tumor, as opposed to peritumoral or distant sites.

202 me results were obtained when recruitment of peritumoral or intratumoral monocytes/macrophages was bl

203 ficial (intradermal or periareolar) or deep (peritumoral or intratumoral) injections were performed.

204 es the SLN identification rate compared with peritumoral or subdermal injection.

205 Dermal, subdermal, peritumoral, or subareolar radioactive colloid injection

206 odium bicarbonate was sufficient to increase peritumoral pH and inhibit tumor growth and local invasi

207 In every case, the peritumoral pH was acidic and heterogeneous and the regi

208 In the current work, tumor invasion and peritumoral pH were monitored over time using intravital

209 ging the intratumoral pHe in relation to the peritumoral pHe can provide a novel readout of therapeut

210 The peritumoral physical microenvironment consists of comple

211 Here, we show that peritumoral progenitor-like GBM cells activate transcrip

212 Intratumoral and peritumoral proliferating lymphatic vessels were detecte

213 site-specific probe for detecting a secreted peritumoral protease expressed by cancer cells and the s

214 Proteases responsible for the increased peritumoral proteolysis associated with cancer represent

215 and extension of ablation zones into aerated peritumoral pulmonary parenchyma, possibly forming the e

216 Glu to total choline was higher in the peritumoral region as well (median, 3.44; tumoral: media

217 ased Glu and Gln metabolic ratios within the peritumoral region compared with NAWM of patients with g

218 The peritumoral region featured elevated lipid metabolism an

219 hat can be subsequently used to evaluate the peritumoral region in glioblastoma.

220 sured relative cerebral blood volumes in the peritumoral region in high-grade gliomas and metastases

221 A lower FA in the peritumoral region indicated more white matter tract dis

222 epolarization that arose frequently from the peritumoral region may provide a mechanism for transient

223 -to-creatine ratio was 2.28 +/- 1.24) in the peritumoral region of gliomas but not in metastases (cho

224 izing free water movement restriction in the peritumoral region using Diffusion Tensor Imaging (DTI)-

225 The peritumoral region was defined as the area in the white

226 Spectra from the enhancing tumor, the peritumoral region, and normal brain were obtained from

227 to create a map of heterogeneity within the peritumoral region, and the variance of this map served

228 essment of infiltrative heterogeneity in the peritumoral region, the area where biopsy or resection c

229 rentiated based on the microstructure of the peritumoral region.

230 ge the pHe gradient between intratumoral and peritumoral regions (DeltapHe) in both untreated and tem

231 f fractional anisotropy (FA) in nonenhancing peritumoral regions (NEPTRs) at baseline is associated w

232 dized uptake value (SUV(max)) for lesion and peritumoral regions was measured on PET images, and a le

233 rences in metabolic ratios between tumor and peritumoral regions, and assess associations of Glu and

234 fferences between infiltrative and vasogenic peritumoral regions, paving the way for its use in class

235 s and patterns in images, such as vessels or peritumoral regions, to enable clinical insights beyond

236 ted with increases in DC infiltration in the peritumoral regions.

237 eatures were extracted from intratumoral and peritumoral regions.

238 resident microglia were localized mainly to peritumoral regions.

239 Peritumoral-rim radiomic features were most relevant to

240 ous studies, dynamic in vivo observations of peritumoral rims demonstrated distended sinusoidal space

241 nation for previous histologic correlates of peritumoral rims.

242 160)Gd-labeled antibodies revealed localized peritumoral ring enhancement, which corresponded to gado

243 ecruit existing endothelial cells to promote peritumoral satellite lesions, which serve as a niche su

244 FOXP3(+)CD4 T(reg) cells in intratumoral and peritumoral sections of metastatic melanoma tumors and f

245 To investigate peritumoral seizure etiology, we implanted human-derived

246 dered as an adjuvant treatment to ameliorate peritumoral seizures associated with glioma in humans.

247 were calculated within the visible tumor and peritumoral shell, and Dice similarity coefficients were

248 sist in an immunosuppressive M2 state at the peritumoral site and promote the growth of gliomas.

249 investigated the ability of LCs from SCC and peritumoral skin to induce T-cell proliferation and pola

250 ation and IFN-gamma production than LCs from peritumoral skin.

251 ted the prognostic significance of tumor and peritumoral SPARC expression in patients with pancreatic

252 h the presence or absence of tumor SPARC and peritumoral SPARC status.

253 ion revealed that MTMs were localized in the peritumoral stroma and associated with eosinophils, whic

254 tion of fibronectin was also detected in the peritumoral stroma of HPV8-positive skin SCC.

255 ocess initiated by the local invasion of the peritumoral stroma.

256 erine cervix, the role of desmoplasia, i.e., peritumoral stromal remodeling characterized by fibrobla

257 er tumors contributing to HSC activation and peritumoral stromal transformation remain to be fully id

258 e 9.5%, 7.8%, and 6.5% (not significant) for peritumoral, subdermal, and dermal injection techniques,

259 Peritumoral, subdermal, or dermal injection of radioacti

260 Peritumoral, subdermal, or dermal injection of radioacti

261 d 0.98, respectively (95%CI, 0.96-1.00), for peritumoral SUV(max) and 94%, 88%, and 0.96, respectivel

262 ume, and total lesion glycolysis, as well as peritumoral SUV(max), SUV(mean), and their respective ra

263 d 0.96, respectively (95%CI, 0.92-1.00), for peritumoral SUV(mean) (all P <= 0.025).

264 Recent studies implicate peritumoral synaptic dysregulation as a driver of brain

265 In 16/19 (84%) cases, TIGIT+ peritumoral T lymphocytes showed also PD-1 expression.

266 n vivo by BCC tumor cells is associated with peritumoral T lymphocytes that are undergoing apoptosis.

267 s confirm co-expression of TIGIT and PD-1 in peritumoral T lymphocytes.

268 ified using the ratio of intratumoral versus peritumoral T-cell densities (I/P ratio).

269 ing tumor (ET), non-enhancing core (NC), and peritumoral T2 hyperintensity (namely, edema, ED) were u

270 nflammation potentially accounted for a high peritumoral tCho signal in CSI, as supported by histolog

271 If Ca++ were peritumoral, their distance from the tumor was measured.

272 the tumor as well as morphologically normal peritumoral tissue samples lacked the caspase-3 transcri

273 Conversely, peritumoral tissue was still intact 24 h after the combi

274 for its use in classifying and benchmarking peritumoral tissue with varying degrees of infiltration.

275 ons of interest (ROI)--enhancing tumor (ET), peritumoral tissue, and normal tissue on the contralater

276 that the extracellular water content of the peritumoral tissue, as captured by the free water volume

277 d miR-181a was also found in human cirrhotic peritumoral tissue, compared to normal liver.

278 patterns, with substantial leakage into the peritumoral tissue.

279 HRF1), was measured in human HCCs (n = 268), peritumoral tissues (n = 154), and HCC cell lines (n = 3

280 21 of hepatocellular carcinoma (both HCC and peritumoral tissues [PHCC]), and 10 controls (CONTR).

281 ents, whereas no toxicity to injected normal peritumoral tissues was demonstrated.

282 IP expression in HCC tumor and corresponding peritumoral tissues were determined by immunohistochemis

283 nd XBP1s were found expressed in CP and PDAC peritumoral tissues, but in contrast to AGR2, their expr

284 egulated in human HCC compared with adjacent peritumoral tissues.

285 res (88%), gas was also found in tumoral and peritumoral tissues.

286 was decreased respect to their correspondent peritumoral tissues.

287 Peritumoral toxicity developed 1-4 weeks after treatment

288 ctor Helios were expressed at high levels in peritumoral Tregs.

289 ue of Developmental Cell, Wu et al. identify peritumoral, uncommitted oligodendrocyte progenitor-like

290 e interval [95%CI], 0.67-0.85) and increased peritumoral uptake (94%, 84%, and 0.89, respectively; 95

291 ages were reviewed to qualitatively evaluate peritumoral uptake and to quantify tracer uptake in the

292 ening, lymph node involvement, and increased peritumoral uptake were more often present in patients w

293 microvasculature but not in the established peritumoral vessels.

294 ontralateral normal-appearing tissue and the peritumoral volume, with shorter-term survivors having l

295 tine in the intratumoral, contralateral, and peritumoral volumes of patients with recurrent GBM were

296 tine within intratumoral, contralateral, and peritumoral volumes were predictive of poor survivorship

297 Of major clinical relevance, we show that peritumoral WIF1 gene transfer reduces not only cancer g

298 nt of established mice tumor xenografts with peritumoral WIF1 gene transfer results in a significant

299 derpinnings of infiltrating GBM cells in the peritumoral zone remain underexplored in patients.

300 5 2.0), enabling non-invasive imaging of the peritumoral zone with high spatial-resolution, and accur