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1 s: periportal, intermediate or midzonal, and perivenous.
2  plot analysis of glutamine synthesis versus perivenous ammonia concentration revealed that TauT KO h
3 ated encephalomyelitis, the co-occurrence of perivenous and confluent demyelination in some individua
4 planted cells was shifted from periportal to perivenous areas by targeted hepatic ablations with carb
5                  Transplanted hepatocytes in perivenous areas exhibited inducible cytochrome P450 act
6 eriportal regions of hepatic acinus, whereas perivenous areas were weakly stained or were stain-free.
7 ransplanted cells shifted from periportal to perivenous areas.
8 d thoracic radiologists independently graded perivenous artifact and arterial enhancement.
9 ced iodine concentration appears to diminish perivenous artifact and to result in improved arterial e
10                                              Perivenous artifacts were statistically significantly re
11 case demonstrated striatal encephalitis with perivenous B- and T-lymphocytic infiltration.
12                In control livers, the distal perivenous cells contained 40% of the total glucokinase
13 f the lobule was taken up by the most distal perivenous cells; this distal perivenous uptake was grea
14  of the total glucokinase of the liver; this perivenous concentration of glucokinase was greatly redu
15                                Patients with perivenous demyelination (n = 13; median age 43 years, r
16 s demyelination cohort, 10 patients had only perivenous demyelination and three also had confluent de
17                                          The perivenous demyelination cohort was more likely than the
18                                    Among the perivenous demyelination cohort, 10 patients had only pe
19                                              Perivenous demyelination is associated with meningoencep
20                                              Perivenous demyelination is the pathological hallmark of
21            Although pathological evidence of perivenous demyelination may be superior to clinical cri
22                     All but one patient with perivenous demyelination only had a monophasic course, w
23 d cortical demyelination was found among six perivenous demyelination patients, all of whom had encep
24                      We investigated whether perivenous demyelination versus confluent demyelination
25 efined acute disseminated encephalomyelitis (perivenous demyelination), but misdiagnosed acute dissem
26                             In contrast with perivenous encephalomyelitis, in which demyelination was
27                                    Transient perivenous enhancement was consistently observed during
28 of mice and the expression of periportal and perivenous hepatocyte markers was determined by polymera
29                                              Perivenous hepatocyte-specific expression was confirmed
30 lations, suggesting that both periportal and perivenous hepatocytes are induced.
31 in kidney proximal tubule S3 segments and in perivenous hepatocytes, consistent with the sites of hig
32 e periportal pattern and the last downstream perivenous hepatocytes, respectively.
33             Basolateral RhBG is expressed by perivenous hepatocytes, where it may mediate ammonium up
34  P450 inducibility was originally greater in perivenous hepatocytes; however, periportal cells rapidl
35                       KO mice had attenuated perivenous hypoxia, suggesting disruption of the normal
36  that demyelination can occur independent of perivenous inflammatory changes and supports the presenc
37 taspline catheter in the basket pose creates perivenous linear lesions with gaps.
38       Surprisingly, in resting liver tissue, perivenous localization of the HIF hydroxylases was obse
39 mprove vein grafts and shows that preserving perivenous lymphatic vessels or promoting autologous lym
40 -derived venous samples, contributing to the perivenous macrophage accumulation and functional lympha
41  mice were initially treated with halothane, perivenous necrosis and an infiltration of CD11b(+) Gr-1
42 lidated clinical diagnostic criteria but its perivenous pathological findings distinguish it from mul
43 ibility occurs in the periportal (PP) and/or perivenous (PV) zones in response to hypoxia/reoxygenati
44 s in glucokinase expression in the immediate perivenous region.
45 F-injected fluorescent tracers can reach the perivenous space by passing through these arteriovenous
46 p-AQP4 drives overall convective flow toward perivenous space, i.e., glymphatics, whereas n-AQP4 may
47 omolecules can traverse from periarterial to perivenous spaces, with transfer occurring at sites of o
48 e ultimately exiting from the parenchyma via perivenous spaces.
49 However, HFD-fed TG mice developed prominent perivenous steatosis with periportal sparing.
50        Furthermore, a GLUL and Lgr5-positive perivenous subpopulation of zone 3 hepatocytes were refr
51 aging features associated with MS, including perivenous T2-weighted hyperintense lesions and focal le
52                                              Perivenous tunnels surrounding bridging veins terminate
53 periportal-type (wild-type beta-catenin) and perivenous-type (mutant beta-catenin), which expressed n
54 he most distal perivenous cells; this distal perivenous uptake was greatly diminished in maternal low
55 ointensity, periependymal brainstem lesions, perivenous white matter lesions, Dawson's fingers, curve
56 CCs display mutually exclusive periportal or perivenous zonation programs.
57  of hypoxia and lower gluconeogenesis in the perivenous zone as compared to the rest of the organ.
58 tion of GS in hepatocytes outside the normal perivenous zone was accompanied by a reduction in the ex
59 ntermediate, and the first few layers of the perivenous zone.
60 y fashion in the last 1-2 cell layers of the perivenous zone.
61 ributed downstream to midlobular (zone 2) or perivenous (zone 3) areas.