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1 ing diseases, such as multiple sclerosis and periventricular leukomalacia.
2 prematurity, necrotizing enterocolitis, and periventricular leukomalacia.
3 to have hypoxic brain injury in the form of periventricular leukomalacia.
4 time to surgery (P=0.028) than those without periventricular leukomalacia.
5 e rates of severe intracranial hemorrhage or periventricular leukomalacia.
6 se and severe intraventricular hemorrhage or periventricular leukomalacia.
7 ll as severe intraventricular hemorrhage and periventricular leukomalacia.
8 r hemorrhage grade of 3 or greater or cystic periventricular leukomalacia.
9 ks gestation, the period of highest risk for periventricular leukomalacia.
10 amate receptor blockade in a rodent model of periventricular leukomalacia.
11 supported by the simultaneous occurrence of periventricular leukomalacia.
12 severe intracranial hemorrhage and/or cystic periventricular leukomalacia.
13 f prematurity, intracerebral hemorrhage, and periventricular leukomalacia.
14 ce of severe intraventricular hemorrhage and periventricular leukomalacia (12.4 percent vs. 23.5 perc
15 incidence of intraventricular hemorrhage and periventricular leukomalacia (33.3 percent and 38.2 perc
16 ffuse white matter injury, previously called periventricular leukomalacia, a major form of brain inju
17 ffuse white matter injury, previously called periventricular leukomalacia, a major form of brain inju
18 7.5 percent vs. 23.9 percent, P=0.03) and of periventricular leukomalacia alone (5.2 percent vs. 9.0
19 ic white matter injury is thought to mediate periventricular leukomalacia, an age-dependent white mat
20 tial failure of recovery in insults, such as periventricular leukomalacia and multiple sclerosis.
21 n lesions of white matter disorders, such as periventricular leukomalacia and multiple sclerosis.
22 hogenesis of white matter disorders, such as periventricular leukomalacia and multiple sclerosis.
24 or translation as a therapeutic strategy for periventricular leukomalacia and that the mechanism of p
25 ncluding multiple sclerosis, cerebral palsy (periventricular leukomalacia), and spinal cord injury.
26 lasia, sepsis, intraventricular haemorrhage, periventricular leukomalacia, and necrotising enterocoli
27 d cerebral volume, thin corpus callosum, and periventricular leukomalacia, and quantitative volumetry
29 asia, severe intracranial hemorrhage, cystic periventricular leukomalacia, and/or severe retinopathy
30 eath; death, intraventricular hemorrhage, or periventricular leukomalacia; and death or necrotizing e
31 (n = 18), DFNB1 (n = 10), DFNB9 (n = 10) and periventricular leukomalacia associated with congenital
33 on, length of stay, intracranial hemorrhage, periventricular leukomalacia, chronic lung disease, pate
35 posite of any of stillbirth, neonatal death, periventricular leukomalacia, early respiratory morbidit
36 alence of structural brain abnormalities and periventricular leukomalacia, fetal and postnatal cerebr
37 eonatal outcome (bronchopulmonary dysplasia, periventricular leukomalacia > grade 1, intraventricular
38 epsis, intraventricular hemorrhage >grade 2, periventricular leukomalacia >grade 1, or necrotizing en
39 associated with BAS; however, neonates with periventricular leukomalacia had lower preoperative oxyg
40 al abnormality, congenital infection, cystic periventricular leukomalacia, hemorrhagic parenchymal in
41 plications with respect to the mechanisms of periventricular leukomalacia in infants and of persisten
42 se and severe intraventricular hemorrhage or periventricular leukomalacia in premature infants are as
43 severe intracranial hemorrhage and/or cystic periventricular leukomalacia in the neonatal period.
44 ith clinical potential for disorders such as periventricular leukomalacia in the preterm and neonatal
48 mes of myelination or remyelination, such as periventricular leukomalacia leading to cerebral palsy,
49 ntraventricular hemorrhage grade 3-4, cystic periventricular leukomalacia, necrotizing enterocolitis,
50 elination is also noted in children with the periventricular leukomalacia of cerebral palsy, another
51 the risk of intracranial hemorrhage, cystic periventricular leukomalacia, or other complications.
52 mbined end point of intracranial hemorrhage, periventricular leukomalacia, or ventriculomegaly (17.5
53 riventricular/intraventricular hemorrhage or periventricular leukomalacia (PIVH/PVL), retinopathy of
54 tributes to mental or physical impairment in periventricular leukomalacia (pre- or perinatal white ma
55 splasia, severe intraventricular hemorrhage, periventricular leukomalacia, proven sepsis, necrotizing
56 brain injuries, including the development of periventricular leukomalacia (PVL) and cerebral palsy (C
57 causes of perinatal brain injury leading to periventricular leukomalacia (PVL) and cerebral palsy.
61 al developing white matter, which results in periventricular leukomalacia (PVL), a condition associat
62 te of cerebral palsy in premature infants is periventricular leukomalacia (PVL), a disorder of the im
63 is generally thought to consist primarily of periventricular leukomalacia (PVL), a distinctive form o
64 lying cerebral palsy in premature infants is periventricular leukomalacia (PVL), a lesion of the imma
65 ellular models of Huntington's disease (HD), periventricular leukomalacia (PVL), and kidney dysfuncti
66 eriventricular white matter injury, that is, periventricular leukomalacia (PVL), the dominant form of
67 e infant leads to white matter injury termed periventricular leukomalacia (PVL), the leading cause of
68 d and one coinciding with the peak period of periventricular leukomalacia (PVL), the major disorder u
69 e matter is important in the pathogenesis of periventricular leukomalacia (PVL), the major pathologic
70 white-matter damage of immaturity, including periventricular leukomalacia (PVL), was the most common
71 ctor for multiple brain pathologies, notably periventricular leukomalacia (PVL), which is distinguish
75 schemic damage to the cerebral white matter [periventricular leukomalacia (PVL)] is a common and lead
76 o the periventricular cerebral white matter [periventricular leukomalacia (PVL)] results in cerebral
77 II or IV intraventricular hemorrhage, cystic periventricular leukomalacia, severe bronchopulmonary dy
79 lar hemorrhage grade of 3 or greater, cystic periventricular leukomalacia, surgical necrotizing enter
80 ral lesions (e.g., microcephaly, atrophy, or periventricular leukomalacia) that were consistently ide
83 associated with premature birth and typical periventricular leukomalacia, who had a dramatic improve