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1 an brain slices shows DAM with intracellular/phagocytic Abeta particles.
2 mice have functional defects, with decreased phagocytic ability and a decreased capacity to generate
3 l interfering RNA enhanced FcgammaR-mediated phagocytic ability but have little effect on cytokine pr
4 autoantigen presentation while retaining the phagocytic ability of homeostatic myeloid cells.
5 face expression of immunological markers and phagocytic ability were assessed by flow cytometry.
6 By assessing in vivo MRI of mononuclear cell phagocytic ability with (19)F nanoparticles, MRI with du
7  SIRPalpha in microglia and restricted their phagocytic ability.
8 lbicans-containing phagosomes independent of phagocytic ability.
9 ar macrophages with low pro-inflammatory and phagocytic ability.
10 or M2 phenotype, was reduced by DHA, but the phagocytic activation was not altered.
11  human macrophages with CARs to direct their phagocytic activity against tumors.
12    Macrophages from KO mice showed increased phagocytic activity and elevated expression of LDL recep
13                  These include modulation of phagocytic activity and responses to injury.
14 n of TNF-alpha and interleukin-10, inhibited phagocytic activity and stimulated production of ROS.
15 ults suggested that astrocytes may engage in phagocytic activity during extended wake, but direct evi
16 ich cells remove cell debris when microglial phagocytic activity is impaired.
17                      Additionally, increased phagocytic activity of differentiating microglia was obs
18 ouse orthologs of many AD risk genes and the phagocytic activity of mouse microglial cells.
19 we investigated whether increased microglial phagocytic activity that clears amyloid can also cause p
20  invariably followed by a sharp reduction in phagocytic activity that could persist for days.
21                               The microglial phagocytic activity toward Abeta was restored after adip
22 hondrial superoxide dismutase), (2) enhanced phagocytic activity toward red blood cells (an in vitro
23                      Immune cell numbers and phagocytic activity were evaluated by flow cytometry and
24  appears to occur as a result of an enhanced phagocytic activity within the lung and an associated al
25  as inflammatory cytokines, NOS 2, increased phagocytic activity, and a greater osteoclast differenti
26 on of a K(+) conductance, the UDP-stimulated phagocytic activity, and the ATP-dependent laser lesion-
27 etabolism under conditions of chronic myelin phagocytic activity, as TREM2 LOF causes pathogenic lipi
28  NK cells poorly and drove moderate monocyte phagocytic activity, both likely compromised because of
29 KO mice was associated with perturbations in phagocytic activity, defective transition of macrophages
30 exercise regimen displayed higher macrophage phagocytic activity, enhanced RvD1 levels, and earlier n
31 duction, phagocytic receptor expression, and phagocytic activity, enhancing parasite clearance by mac
32 y evoked membrane currents, stimulated basal phagocytic activity, evoked intracellular Ca(2+) transie
33                             Gene expression, phagocytic activity, production of reactive oxygen speci
34 of myocytes to stimulated endothelial cells, phagocytic activity, uptake of oxidized low-density lipo
35 tokines with LPS stimulation and showed more phagocytic activity.
36 o exhibited higher levels of active RhoA and phagocytic activity.
37 lting in killing of encapsulated bacteria by phagocytic activity.
38  the aortic wall by enhanced proteolytic and phagocytic activity; however, the cells do not transdiff
39                       Here, we show that the phagocytic adaptor protein Gulp1 regulates EphB/ephrinB
40 actor (TNF), and Abeta peptides enhance both phagocytic and antifungal activity from BV-2 cells.
41 ly derived cardiac tissue macrophages play a phagocytic and antigen presenting role.
42  showed transcriptional evidence of abnormal phagocytic and cell-survival programs.Conclusions: Our f
43 mplex class II that was coupled with reduced phagocytic and chemotactic activity compared to controls
44                         Altogether, both the phagocytic and immunomodulatory functions of Mer regulat
45       This assay can also be used with other phagocytic and non-phagocytic cells to study the individ
46 he recruitment and differentiation of highly phagocytic and stimulatory macrophages within tumours.
47 r TIM4(lo) and TIM4(-) cells, which were not phagocytic and were more abundant than Kupffer cells.
48 yed a TM cell-like genotypic profile, became phagocytic, and responded to dexamethasone stimulation,
49 ith primate microglia, including complement, phagocytic, and susceptibility genes to neurodegeneratio
50  to as SYNB1891, targets STING-activation to phagocytic antigen-presenting cells (APCs) in the tumor
51                               Six subsets of phagocytic APC (HLA-DR(+)) were consistently observed.
52  method made it possible not only to perform phagocytic assays with functionalized droplets and beads
53 ive anticipatory responses against impending phagocytic attack and promote host colonisation.
54  interactions resulting in microbial growth, phagocytic attack and secretion of host antimicrobial fa
55 altering the actomyosin cytoskeleton affects phagocytic behavior.
56  released were temporally correlated with OS phagocytic burst after light onset.
57         P2X7L-transfected HEK 293 cells have phagocytic but not channel, pore, or membrane-blebbing f
58        Marrow-derived macrophages are highly phagocytic, but whether they can also traffic into solid
59 ages to an active M1 phenotype, and superior phagocytic capabilities as compared to individual drug t
60                                    Thus, the phagocytic capability of cells that present self-peptide
61 demonstrate a dramatic and lasting change in phagocytic capacity after serum exposure.
62 astrocytes are accompanied by alterations in phagocytic capacity and effects on neuronal calcium sign
63 eostatic to a functional state with enhanced phagocytic capacity but reduced motility and synapse eng
64 reased Iba1 and C1q expression with enhanced phagocytic capacity in Pten(m3m4/m3m4) microglia, indica
65                                    Defective phagocytic capacity in Slc2a1(M-/-) BMDMs may have contr
66  macrophage marker expression, and augmented phagocytic capacity of human MDMs stimulated with LPS or
67 n were not different between groups, and the phagocytic capacity of Kupffer cells was preserved follo
68 ains of F. nucleatum significantly increased phagocytic capacity of neutrophils.
69 globulin levels and a modest decrease in the phagocytic capacity of pulmonary macrophage populations
70  of host-pathogen interactions, for instance phagocytic capacity or phenoloxidase activation, or at t
71 targeting lung macrophages to increase their phagocytic capacity, enhance their ability to make retin
72 ntrols, both AH monocyte subsets had greater phagocytic capacity, enhanced ability to drive memory T
73 vealed impeded M2 polarization and decreased phagocytic capacity.
74 to purified SL-1 enhanced the trafficking of phagocytic cargo to lysosomes.
75                                              Phagocytic cargo, upon internalization, follows a define
76                      We conclude that highly phagocytic, CD138(+) SPM-like cells with an anti-inflamm
77 ammatory cells to the peritoneum, or improve phagocytic cell killing of pathogens.
78                              GS-9620 induced phagocytic cell maturation and improved effector-mediate
79 d raises the fundamental question of how the phagocytic cell optimizes the acidification process to p
80                                              Phagocytic cell reduction in nitric oxide production dur
81 s replace CCR2(+) macrophages as the primary phagocytic cell.
82 is their rapid recognition and engulfment by phagocytic cells (a process called efferocytosis).
83                                However, many phagocytic cells also act as antigen-presenting cells an
84                              Neutrophils are phagocytic cells and are vital components in the human i
85 d light on the interactions between NETs and phagocytic cells and provide new insights regarding the
86 plicate in professional and non-professional phagocytic cells and subvert immune responses for chroni
87 hogens encounter numerous insults, including phagocytic cells designed to degrade bacteria, while est
88  suggesting that the pathogen may not target phagocytic cells for intracellular growth during natural
89 n of LRRK2 and its substrate Rab10 occurs in phagocytic cells in the immune system.
90                    Previously, we found that phagocytic cells ingest bacteria directly from the cytos
91                                Microglia are phagocytic cells involved in homeostasis of the brain an
92             Clodronate-mediated depletion of phagocytic cells markedly prolonged the serum half-life
93                                              Phagocytic cells of the innate immune system appear to b
94 ssion analysis by RNA sequencing of F4/80(+) phagocytic cells revealed activation of the activating t
95 totic tissue is recognized for engulfment by phagocytic cells such as macrophages.
96 n also be used with other phagocytic and non-phagocytic cells to study the individual or collective r
97 d the effect of this mutation on mononuclear phagocytic cells was examined.
98    Bacterial ascent and the cell dynamics of phagocytic cells were assessed by biophotonic imaging an
99                         METH interferes with phagocytic cells' phagosomal maturation, resulting in im
100 isease (CGD) is a rare inherited disorder of phagocytic cells(1,2).
101 ractions of live B. bacteriovorus with human phagocytic cells, determining the uptake mechanisms, per
102 icroglia (MG), a heterogeneous population of phagocytic cells, play important roles in central nervou
103 ated kinetoplastid parasites that parasitize phagocytic cells, principally macrophages, of vertebrate
104 the capacity to concentrate drug delivery to phagocytic cells, significantly reducing off-target toxi
105 the clearance of cell debris and bacteria by phagocytic cells, the role of CRP in additional immunolo
106 ar survival of S. aureus in non-professional phagocytic cells, via modulation of MazEF and Agr.
107 and molecular functions and may be deadly to phagocytic cells, which may result in increased suscepti
108 ighly immunogenic C. albicans hyphae attract phagocytic cells, which rapidly engulf adherent S. aureu
109 h apoptotic cells are cleared from tissue by phagocytic cells.
110  polarization, and activation of mononuclear phagocytic cells.
111 the ultimate fate of B. bacteriovorus inside phagocytic cells.
112 by liver-, spleen-, and bone marrow-resident phagocytic cells.
113 the interactions between A. pittii and human phagocytic cells.
114 everal cell types, including mitotic and non-phagocytic cells.
115 amentous actin and altered actin dynamics in phagocytic cells.
116 lammasome, Th17 signaling and recruitment of phagocytic cells.
117 isingly, feedback inhibition is abrogated in phagocytic cells.
118  cells evade the immune system by disrupting phagocytic clearance and the effect of phagocytosis chec
119        We sought to test the significance of phagocytic clearance by resident and recruited phagocyte
120 ction produces elongated hyphae resistant to phagocytic clearance compelling alternative neutrophil e
121 L expression in the adult brain promotes the phagocytic clearance of Abeta and ameliorates Abeta-depe
122 myloidogenesis is accelerated due to reduced phagocytic clearance of amyloid seeds despite reduced pl
123 re currently being explored to stimulate the phagocytic clearance of apoptotic cells; however, these
124                                              Phagocytic clearance of apoptotic germ cells by Sertoli
125                             In addition, the phagocytic clearance of apoptotic tumor cells (efferocyt
126  and the discovery of cellular immunity, the phagocytic clearance of cellular debris has been conside
127 ses from glia, including glial migration and phagocytic clearance of damaged neurons.
128      During the progression of the disorder, phagocytic clearance of dead photoreceptor cell bodies h
129 interactions, which are known to mediate the phagocytic clearance of degenerating neurons.
130 ating parasite sequestration to host organs, phagocytic clearance of parasites, and regulation of imm
131 stasis in the central nervous system through phagocytic clearance of protein aggregates and cellular
132 ins to constitute a functional LC3-dependent phagocytic complex.
133  leading to the extension and closure of the phagocytic cup around the target.
134 nvolving membrane-cytoskeletal crosstalk for phagocytic cup closure.
135 se and induce actin polymerization to form a phagocytic cup for engulfment of the bead.
136 over that EhActn2 plays an important role in phagocytic cup formation and might constitute a new drug
137       Cdc42 is not critical for filopodia or phagocytic cup formation, but plays a key role in drivin
138  role in phagocytic processing lies near the phagocytic cup in a myosin-independent fashion.
139  Cdc42-deficient macrophages exhibited rapid phagocytic cup kinetics, but reduced particle clearance,
140   Focal complex-like adhesions formed in the phagocytic cup that contained beta(2) integrins, focal a
141 y, CD82 organized Dectin-1 clustering in the phagocytic cup.
142 ith RBCs EhRab35 is recruited to the site of phagocytic cups as well as to the nascent phagosomes tha
143 Microglia first extend processes that end in phagocytic cups at the motoneuron surface, then they clo
144 days after injury, characterized by frequent phagocytic cups, high phagosome content and CD68 upregul
145 with the interior of both macropinocytic and phagocytic cups.
146                     Mfge8 partly rescued the phagocytic defect of Smad3 null macrophages, without aff
147  release was not dramatically regulated, yet phagocytic defects hindered MPhi function in chronic dis
148  cells, as well as rescue the cells from the phagocytic dysfunction induced by cholesterol crystals a
149  Axl and Mertk, which partially explains the phagocytic dysfunction.
150                       CD22 mediates the anti-phagocytic effect of alpha2,6-linked sialic acid, and in
151 enzymatic means is shown to markedly enhance phagocytic efficiency.
152 ealing subcellular force patterns throughout phagocytic engulfment and force dynamics in the cytotoxi
153 om the recognition of dying cells through to phagocytic engulfment and homeostatic resolution, and hi
154 d a prorepair phenotype, and increase of the phagocytic engulfment of neutrophils by macrophages.
155 ithin the gels is used to measure successful phagocytic engulfment.
156 s are associated with a decreased success of phagocytic engulfment.
157  targets are embedded, impact the success of phagocytic engulfment.
158 orne cholera disease, uses the T6SS to evade phagocytic eukaryotes, cause intestinal inflammation, an
159    Cytochrome c oxidase dysfunction enhances phagocytic function and osteoclast formation in macropha
160                    Herein, we describe a new phagocytic function for the nucleoside diphosphate kinas
161 proinflammatory cytokine production, reduced phagocytic function of alveolar macrophages, and consequ
162 rt that high dose atorvastatin increased the phagocytic function of ARPE-19 cells, as well as rescue
163 lease from hepatocytes and/or modulating the phagocytic function of macrophages represents a promisin
164                          Taken together, the phagocytic function of Muller glia is responsible for re
165 ower levels of serum cytokines, and improved phagocytic function relative to wild-type (WT) mice.
166 r macrophages with more pro-inflammatory and phagocytic function respectively, and large alveolar mac
167 more, that defective MCI macrophages recover phagocytic function via omega-3.
168 O dysfunction affects inflammatory pathways, phagocytic function, and osteoclastogenesis.
169 e, an increase in cell motility, and gain of phagocytic function.
170 -associated genes, but also showed decreased phagocytic gene expression and increased epithelial-asso
171 d that IDOL inhibition upregulated lysosomal/phagocytic genes in microglia.
172 isit to the glial cytoplasm, indicating that phagocytic glia act as obligatory intermediates in aggre
173   These findings expand our understanding of phagocytic glia as double-edged players in neurodegenera
174 rophils constitute the largest population of phagocytic granulocytes in the blood of mammals.
175 acrophages quickly differentiated toward non-phagocytic, high-SIRPalpha TAMs.
176 thogenesis, including its survival inside of phagocytic host cells.
177    This process involves corralling, wherein phagocytic immune cells become confined to the necrotic
178 ole for Fmr1 in regulating the activation of phagocytic immune cells both in the body and the brain.
179 riments, we also define specific subtypes of phagocytic immune cells in An. gambiae, providing insigh
180 use of a chemical-based treatment to deplete phagocytic immune cells in Anopheles gambiae, demonstrat
181                                              Phagocytic immune cells such as microglia can engulf and
182  order of magnitude larger than neutrophils, phagocytic immune cells that densely surround aggregates
183 s catabolize chitin released from hemocytes (phagocytic immune cells) that traffic into the light-org
184 M nanoemulsion was intrinsically taken up by phagocytic immune cells, particularly macrophages, with
185 , cell death, estrogen receptor pathways and phagocytic immune responses.
186 tly regulate actin assembly to ensure proper phagocytic internalization, and translocate phagosomes o
187 ion, slower adhesion turnover, and deficient phagocytic internalization.
188      In particular, whether microglia become phagocytic is controversial.
189 easing complement-mediated attack, improving phagocytic killing activity of neutrophils, and preventi
190  activity of AB307.30 but failed to increase phagocytic killing of capsule-positive strains.
191      We demonstrated decreased resistance to phagocytic killing of GAS lacking EndoS in vitro and dec
192  nutritional immunity, protects them against phagocytic killing, and activates immune evasion strateg
193 eumococcal nasopharyngeal carriage, reducing phagocytic killing, and resulting in increased inflammat
194  AMP, reduce antibiotic efficacy and enhance phagocytic killing.
195                                    Thanks to phagocytic leucocytes and other host defenses, the vast
196 ecting the function of neutrophils and other phagocytic leukocytes are notable for an increased susce
197  surrounded by the PV membrane, suggesting a phagocytic-like process for vesicle engulfment.
198                 Additional components of the phagocytic machinery are needed, but phagocytosis of pLL
199 ate that the majority of adult hemocytes are phagocytic macrophages (plasmatocytes) from the embryoni
200 d-derived immune cell populations, including phagocytic macrophages and microglia.
201 holestasis, which stalled the recruitment of phagocytic macrophages and the removal of cellular corps
202                                              Phagocytic macrophages expressed CD206, displayed blunte
203 ulatory secondary necrosis by binding to the phagocytic marker phosphatidylserine on dying tumour cel
204 lia in the embryonic retina are enriched for phagocytic markers and observe engulfment of nonapoptoti
205 ate synapse elimination through a novel, non-phagocytic mechanism in the retinogeniculate pathway dur
206 ed after spinal cord injury (SCI), but their phagocytic mechanisms and link to neuroprotection remain
207                                              Phagocytic microglia engulf more viable newborn cells in
208 Finally, we discovered that the secretome of phagocytic microglia limits the production of new neuron
209 ed the extent of amyloid plaques, oligomers, phagocytic microglia, and infiltrated T cells by 23.7%,
210 ed predominantly of a novel subset of highly phagocytic MPhi resembling small peritoneal MPhi (SPM) t
211 onses via TNF-alpha, which are essential for phagocytic myelin debris clearance and for oligodendroge
212  show a pathway for direct activation of the phagocytic NOX2 in hepatocytes by p52Shc binding and act
213 examining human peptides and the unicellular phagocytic organism Dictyostelium discoideum reveal that
214 sphoinositide signaling is critical both for phagocytic particle engulfment and subsequent phagosomal
215 k identifies a conserved crucial step in the phagocytic pathway of immune cells and provides a potent
216                   Contravening the canonical phagocytic pathway, tPCs mature by fusing with endosomes
217 t macrophages, by mediating acquisition of a phagocytic phenotype and by contributing to anti-inflamm
218                               They develop a phagocytic phenotype as early as 3 days after injury, ch
219 satory nerve terminal sprouts and to adopt a phagocytic phenotype on denervated NMJs in SOD1(G37R) mi
220                 RhoA inhibition restored the phagocytic phenotype to that seen in control macrophages
221  molecular patterns that are engulfed during phagocytic process and danger-associated molecular patte
222               For the biggest particles, the phagocytic process involves a massive reorganization of
223 is depleted during septal thinning through a phagocytic process provides a mechanism for the clearanc
224 terized by inflammatory processes, including phagocytic processes as well as complement cascade activ
225  further highlight that Vamp7 also regulates phagocytic processing and locomotion but uses pathways d
226 ml is needed for both hemocyte migration and phagocytic processing at distinct subcellular localizati
227 tility at the cell rear, whereas its role in phagocytic processing lies near the phagocytic cup in a
228  may have emerged during evolution to couple phagocytic processing to cell migration and facilitate s
229 nohistochemistry to analyze their repair and phagocytic properties.
230 elimited cell surface domains may enable the phagocytic pruning of these 'locally dead' domains by ma
231 adenine dinucleotide phosphate/NADPH levels, phagocytic reactive oxygen species production, neutrophi
232 tion technique to directly probe the role of phagocytic receptor "colocalization" in innate immune re
233 iously identified Jedi-1 (PEAR1/MEGF12) as a phagocytic receptor expressed by satellite glia in the D
234 regulated B1 cell expansion, IgM production, phagocytic receptor expression, and phagocytic activity,
235 ssed, or excess neurons and synapses via the phagocytic receptor Mer tyrosine kinase (MerTK).
236  of cardiac macrophages or deficiency in the phagocytic receptor Mertk resulted in defective eliminat
237  that selectively inhibited efferocytosis by phagocytic receptor MerTK.
238 ody antagonist of CD47 (CD47nb)(12), an anti-phagocytic receptor that is commonly overexpressed in se
239                             However, the pro-phagocytic receptor(s) responsible for tumour cell phago
240                         Thus, in addition to phagocytic receptor-mediated phagocytosis, neutrophils a
241 ant GPCR/Gi signaling works independently of phagocytic receptor/tyrosine kinase signaling to promote
242 rasite clearance mediated by upregulation of phagocytic receptors and increased IgM production, and d
243 tionship between the spatial organization of phagocytic receptors and innate immune responses.
244 s and neutrophils expressed higher levels of phagocytic receptors and showed enhanced phagocytosis of
245 ns forming adducts with SecoA identified the phagocytic receptors CD206 and CD64.
246 ase bacteria through chemotaxis and then use phagocytic receptors coupled with tyrosine kinases to de
247 ic receptors, which were the main astrocytic phagocytic receptors for cell debris in the above experi
248            Blocking phagocytosis or specific phagocytic receptors may alleviate synapse loss and axon
249 components on the macrophage surface shrouds phagocytic receptors, curtailing their ability to engage
250 ic cells relies on homeostatic engagement of phagocytic receptors, in particular, receptors of the ty
251 ever, the contribution of precursor monocyte phagocytic receptors, which are the first to interact wi
252 ven in a healthy CNS, astrocytes express TAM phagocytic receptors, which were the main astrocytic pha
253  due to the inaccessibility of intracellular phagocytic receptors.
254 plex signaling network initiated by distinct phagocytic receptors.
255 he formation of oxysterol-protein adducts on phagocytic receptors.
256  insights into the cellular determinants for phagocytic recognition and uptake of P. aeruginosa.
257                               This prevented phagocytic removal of dead cells, prevented maturation o
258  within Mt to microglia, where it promotes a phagocytic/reparative phenotype.
259 led by efferocytosis of dying hepatocytes by phagocytic resident liver macrophages and infiltrating G
260 ated with increased antibiotic tolerance and phagocytic resistance.
261                  In addition, the microglial phagocytic response and elevation of Trem2, both necessa
262                             Furthermore, the phagocytic response of BV2 cells to stressed neurones wa
263 globoid cells in nerves, suggesting that the phagocytic response of healthy macrophages, rather than
264  is via a miR-124-dependent reduction in the phagocytic response of microglia.
265  primary microglia cultures, DHA reduced the phagocytic response to myelin, which was associated with
266 tivation and robust apoptotic and microglial phagocytic responses that extensively eliminate cells wi
267 c cells (stretched cells) during their brief phagocytic role in nurse-cell engulfment.
268 tain lineage-labeled particles, suggesting a phagocytic role in the clearance of AMFs.
269 verse roles, with TPCs providing a universal phagocytic signal for a wide range of particles and TRPM
270 data reveal CD24 as a highly expressed, anti-phagocytic signal in several cancers and demonstrate the
271        Our study shows that pro-inflammatory phagocytic signaling is required for myelin debris degra
272 s, YopE and YopT, inactivate RhoA to disrupt phagocytic signaling.
273 at endo-lysosomes act as platforms for a new phagocytic signalling pathway in which FcgammaR activati
274  as the levels of PA found on phagosomes and phagocytic sites as shown with the PA probe Spo20p-GFP.
275 ession of cytokines which promote microglial phagocytic states, such as IL-6 and IL-4, and increased
276 crophages through the overexpression of anti-phagocytic surface proteins called 'don't eat me' signal
277                                       At the phagocytic synapse, SIRPA inhibited integrin activation
278 ion, positioning SIRPA for activation at the phagocytic synapse.
279 specific cell corpse/phagocyte interactions (phagocytic synapses) that impinge on host immunity, with
280 udies using engineered microenvironments and phagocytic targets have demonstrated how altering the ac
281                                Engulfment of phagocytic targets requires the activity of myosins, act
282 phagocytosis exist that utilize a variety of phagocytic targets with different combinations of recept
283 articles with spatially patterned ligands as phagocytic targets, we can decouple the receptor pair, D
284 ns that coat pathogenic as well as malignant phagocytic targets.
285 tivated in vitro by the venom, becoming more phagocytic; these results confirm that this cell is a ta
286                             In liver, highly phagocytic TIM4(hi) cells shared many transcripts with m
287 microbial pathogens and dead cells and their phagocytic uptake by specialized immune cells are essent
288 ication of bacterial peptidoglycans restored phagocytic uptake in the lysosomal degradation-defective
289 cally activates the PIP(3)-pathway to induce phagocytic uptake into primary or immortalized murine ce
290 eceptor TIM4, encoded by TIMD4, mediates the phagocytic uptake of apoptotic cells.
291                      Filopodia supported the phagocytic uptake of bacterial (Escherichia coli) partic
292  to investigate the influence of size on the phagocytic uptake of macrophages.
293 elled PFCs was able to outcompete the strong phagocytic uptake of PFCs by circulating monocytes.
294 ent inhibited mannosidase-II recruitment and phagocytic uptake of serum-coated or -uncoated latex bea
295  feedback inhibition in these cells requires phagocytic uptake of the bacteria, but cannot be explain
296 phage-mediated bacterial clearance relies on phagocytic uptake of the pathogen, subsequent phagolysos
297 ecruitment of mannosidase-II vesicles during phagocytic uptake required Ca(2+) from both extra- and i
298  role of Golgi-derived secretory vesicles in phagocytic uptake, the key innate defense function.
299 uce permeabilization of Escherichia coli and phagocytic uptake.
300                              Eosinophils are phagocytic white blood cells with a variety of roles in

 
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