ライフサイエンスコーパス: phagocytosis

1 ogical destruction (enzymes, metabolism, and phagocytosis).

2 jury, including active tissue remodeling and phagocytosis.

3 ons, like respiratory burst, chemotaxis, and phagocytosis.

4 promise Fcgamma-receptor binding or in vitro phagocytosis.

5 cell activation, myeloid cell migration, and phagocytosis.

6 ific myosins function at individual steps of phagocytosis.

7 one drives phenotypes like cell crawling and phagocytosis.

8 interactions needed for normal outer segment phagocytosis.

9 ing and corresponded with reduced macrophage phagocytosis.

10 echolamines promoted monocyte and neutrophil phagocytosis.

11 amebic trogocytosis and cell killing but not phagocytosis.

12 s and amebic cytotoxicity but did not impair phagocytosis.

13 which caused persistently elevated synaptic phagocytosis.

14 ariety of functions at distinct steps during phagocytosis.

15 glial cellular function such as beta-Amyloid phagocytosis.

16 rden in the airways by increasing macrophage phagocytosis.

17 ic support of neurons, glutamate uptake, and phagocytosis.

18 AB, which promotes S. aureus survival during phagocytosis.

19 ter activation of PMNs, along with defective phagocytosis.

20 ks CD47 tumor cell surface marker to promote phagocytosis.

21 ne is inefficient in stimulating glioma cell phagocytosis.

22 SF1Ri) to monitor the change of immune cells phagocytosis.

23 ine to internal organs and for inhibition of phagocytosis.

24 e mechanisms, including impairing macrophage phagocytosis.

25 eceptor/tyrosine kinase signaling to promote phagocytosis.

26 pression, previously shown to be integral to phagocytosis.

27 ne kinases to destroy opsonized bacteria via phagocytosis.

28 Nearly 50% of Muller glia engaged in phagocytosis.

29 ated protein-4 (MTMR4), regulates macrophage phagocytosis.

30 d on macrophages and limits inflammation and phagocytosis.

31 genes that play crucial roles in microglial phagocytosis.

32 with diminished clonogenicity and augmented phagocytosis.

33 alpha (SIRPalpha) on macrophages to prevent phagocytosis.

34 oeboid mode of movement as well as augmented phagocytosis.

35 for the first time that FPRs govern opsonic phagocytosis.

36 ellular traps and entered mast cells through phagocytosis.

37 synthesis, and reversed the defects in Abeta phagocytosis.

38 ch was supported by concomitant increases in phagocytosis.

39 complement or to undergo neutrophil-mediated phagocytosis.

40 idal activity, but as a result of inhibiting phagocytosis.

41 phocyte proliferation but increased monocyte phagocytosis.

42 zed particles decorated with CD200 decreased phagocytosis activities of THP-1 macrophages.

43 rrespective of CD56 expression, have reduced phagocytosis activity and low expression of genes involv

44 we could substantiate Ab-dependent cellular phagocytosis (ADCP) and Ab-dependent neutrophil phagocyt

45 als, as opposed to the Ab-dependent cellular phagocytosis (ADCP) and virus capture responses, which w

46 An antibody dependent cellular phagocytosis (ADCP) assay revealed substantial ADCP acti

47 Macrophage antibody (Ab)-dependent cellular phagocytosis (ADCP) is a major cytotoxic mechanism for b

48 gocytosis (ADCP) and Ab-dependent neutrophil phagocytosis (ADNP) activities in the lesions of the UCL

49 ce, whether the overall effect of microglial phagocytosis after brain injury in vivo is neuroprotecti

50 yte lineage regulates PH through complement, phagocytosis, Ag presentation, and chemokine/cytokine pa

51 in alpha, a negative regulator of macrophage phagocytosis allowing repression of rat macrophages by h

52 ble newborn cells in males; in females, less phagocytosis allows more astrocytes to survive to the ju

53 ses, including autophagy, endocytic sorting, phagocytosis, anabolic responses and cell division.

54 ograms macrophages for enhanced Pneumocystis phagocytosis and also suppresses PcP-related immunopatho

55 l DNA (CpG DNA) and mitochondrial DNA impair phagocytosis and attenuate phagocytosis-induced apoptosi

56 responses that stimulated robust neutrophil phagocytosis and complement activity.

57 on molecule in gout facilitating MSU crystal phagocytosis and contributing to the pathogenesis of gou

58 ectives: To investigate the effect of HRV on phagocytosis and cytokine response to bacteria by alveol

59 cells are known to exhibit varying levels of phagocytosis and cytokine secretion, and are differentia

60 f established AD risk variants on microglial phagocytosis and debris processing via the endolysosomal

61 , antagonize phagocyte recruitment, diminish phagocytosis and decrease production of iNOS and cytokin

62 munity of LOAD patients is malfunctioning in phagocytosis and degradation of Abeta and LOAD patients'

63 mpartment to maintain the high efficiency of phagocytosis and degradation of photoreceptor outer segm

64 These findings link TNC to CD47-driven phagocytosis and demonstrate that TNC affects the antitu

65 , hypophagia had no effect on Ab-independent phagocytosis and did not alter macrophage viability.

66 onstitutive- and PAMP-dependent LC3-assisted phagocytosis and does not induce expression of inflammat

67 ophages from knockout mice exhibited reduced phagocytosis and enhanced migration.

68 ow bacteria to become symbionts by surviving phagocytosis and exploiting the eukaryotic intracellular

69 ization method to standard ways of assessing phagocytosis and find it to perform better, exhibiting i

70 ify functional deficits of CB-LDG concerning phagocytosis and generation of neutrophil extracellular

71 active state, in which they display impaired phagocytosis and glutamate uptake and fail to support ne

72 aeruginosa and accelerates their removal by phagocytosis and increased production of bactericidal re

73 -antigen density, enhanced antibody-mediated phagocytosis and increased serum susceptibly.

74 ished release of induced cytokines, elevated phagocytosis and increased vulnerability to cellular str

75 cytes, have been intricately associated with phagocytosis and innate immune signaling.

76 hat ethanol affects activation, recruitment, phagocytosis and killing functions of neutrophils, causi

77 uence subsequent processes such as bacterial phagocytosis and killing.

78 venger receptor, resulting in an increase in phagocytosis and lipid uptake.

79 Vertebrate vision relies on the daily phagocytosis and lysosomal degradation of photoreceptor

80 Macrophages clear pathogens by phagocytosis and lysosomes that fuse with phagosomes are

81 opsonizes Pneumocystis organisms for greater phagocytosis and may also mask antigens that drive immun

82 ar CCL2 expression and regulating macrophage phagocytosis and migration.

83 esponse can be elicited by pLL too large for phagocytosis and nonetheless requires actin dynamics, Sy

84 as on single membranes during LC3-associated phagocytosis and other LC3-lipidation events.

85 at function at the early stages of microbial phagocytosis and phagosome formation.

86 In a cell system, FTY720 decreased phagocytosis and production of reactive oxygen species b

87 diated endogenous mechanism that impairs PMN phagocytosis and prolongs inflammation, and demonstrate

88 atherosclerotic plaque, reactivate lesional phagocytosis and reduce the plaque burden in atheroprone

89 icroglia phenotype characterized by enhanced phagocytosis and reduced proinflammatory responses.

90 of microglia revealed that genes related to phagocytosis and signaling (IL-6, IL-8, acute phase resp

91 of hippocampal synapses resulted in synaptic phagocytosis and subsequent cognitive decline.

92 hages (CAR-Ms) demonstrated antigen-specific phagocytosis and tumor clearance in vitro.

93 enes that are implicated in the recognition, phagocytosis and/or endocytosis, and maintenance of alga

94 ace plasmon resonance, Ab-dependent cellular phagocytosis, and Ab-dependent cellular cytotoxicity ass

95 immune functions, including neutralization, phagocytosis, and Ab-dependent cellular cytotoxicity.

96 assical monocytes play the canonical role of phagocytosis, and account for the majority of circulatin

97 ions, including NK cell activation, monocyte phagocytosis, and complement activity, all of which have

98 e immune cell chemoattraction, survival, and phagocytosis, and diminished pro-inflammatory immune cel

99 neuroinflammation, an increase in microglial phagocytosis, and improved memory.

100 tes host defense mechanisms, notably impairs phagocytosis, and increases TB drug metabolism.

101 , monocyte-derived macrophages showed intact phagocytosis, and lymphocyte counts and proliferative ca

102 IL-1RA and reduced neutrophil degranulation, phagocytosis, and NETosis.

103 , preserve C5aR expression, restore impaired phagocytosis, and redirect human PMNs to apoptosis.

104 3SS to inhibit neutrophil respiratory burst, phagocytosis, and release of inflammatory cytokines.

105 n etiological role in microglial activation, phagocytosis, and synaptic pruning, creating avenues for

106 ell functions from adhesion to migration and phagocytosis are mediated by integrin adhesion complexes

107 er innate cellular immune responses, such as phagocytosis, are drastically decreased in cavefish.

108 y-dependent cellular cytotoxicity (ADCC) and phagocytosis, are mediated by FcgammaRs, which upon cros

109 1 Env IgG3, antibody engagement of FcRs, and phagocytosis as potential mechanisms for HIV-1 preventio

110 a contact-dependent mechanism and macrophage phagocytosis, as was demonstrated by using in vitro mode

111 re required for both amebic trogocytosis and phagocytosis, as well as cell killing.

112 in vitro and these were added to a platelet phagocytosis assay.

113 ch is easily incorporated into most existing phagocytosis assays and allows for reproducible results

114 s reduced in affected cells (p = 0.015), and phagocytosis assays in MPO-deficient mice and human cell

115 putative Tp0126 surface-exposed loops, while phagocytosis assays showed that postimmunization sera op

116 g L. tropica specific antibody (Ab)-mediated phagocytosis assays, we could substantiate Ab-dependent

117 databases for fast quantitative analysis of phagocytosis assays.

118 eased myeloid cell superoxide production and phagocytosis associated with increased ACE expression.

119 cule potentiates antibody-dependent cellular phagocytosis beyond that of SIRPalpha blockade alone.

120 by suppressing interleukin 1beta, CD68, and phagocytosis but not CD208, following exposure to endoto

121 cystic fibrosis (CF) was normal during early phagocytosis, but it was diminished at later time points

122 TREM2 senses lipids and mediates myelin phagocytosis, but its role in microglial lipid metabolis

123 reased sensitivity to complement killing and phagocytosis by alveolar macrophages.

124 Tumour cell phagocytosis by antigen presenting cells (APCs) is criti

125 on't eat me" signal that protects cells from phagocytosis by binding and activating its receptor SIPR

126 yers of macrophages and their targets hinder phagocytosis by both steric and electrostatic means.

127 lyphosphate increased E. coli survival after phagocytosis by D. discoideum and macrophages.

128 e, and have relatively better survival after phagocytosis by D. discoideum or macrophages.

129 r polyphosphate and have poor survival after phagocytosis by Dictyostelium discoideum or human macrop

130 tosis ("eat me" signals), while others avoid phagocytosis by displaying inhibitory ligands ("don't ea

131 lular cytotoxicity and Ab-dependent cellular phagocytosis by Fc interactions with FcgammaRs and compl

132 Thus, the CD47-SIRPA axis suppresses phagocytosis by inhibiting inside-out activation of inte

133 Phagocytosis by macrophages and neutrophils is the prima

134 Phagocytosis by macrophages represents a fundamental pro

135 C. albicans cells had reduced survival upon phagocytosis by macrophages.

136 , a transmembrane receptor, in mediating MSU phagocytosis by macrophages.

137 i-GMP concentrations generated after initial phagocytosis by macrophages.

138 s engineered with SA-CD47 were refractory to phagocytosis by mouse macrophages.

139 n contrast, F(ab')2-only fragments increased phagocytosis by murine macrophages independent of SLAMF7

140 he site of inflammation, increased bacterial phagocytosis by neutrophils and macrophages, and acceler

141 However, cancer cells can evade phagocytosis by upregulating anti-phagocytosis molecule

142 sulted in increased EV release and decreased phagocytosis by VSMCs.

143 Macrophage phagocytosis can be triggered by diverse receptor-ligand

144 (nAb) responses, antibody-dependent cellular phagocytosis, CD4+ polyfunctionality responses, and CD4+

145 pe/subclass, PPD-specific antibody-dependent phagocytosis, cellular cytotoxicity, and natural killer

146 led that some key canonical pathways such as phagocytosis, chemotaxis, antigen presentation, and cell

147 produce IFN-gamma, which worsened macrophage phagocytosis, clearance of secondary Candida infection,

148 uding antibody-dependent neutrophil/monocyte phagocytosis, complement activation and natural killer c

149 resistant BC is caused by CD47-mediated anti-phagocytosis conjugated with HER2-prompted proliferation

150 ction to help reduce parasite burden through phagocytosis, cytokine production, and antigen presentat

151 dized mitochondrial DNA release, chemotaxis, phagocytosis, degranulation, ability to harm the endothe

152 irs multiple neutrophil functions, including phagocytosis, degranulation, leukotriene, and reactive o

153 Experimental results of macrophage phagocytosis demonstrated that more carbohydrate-decorat

154 th in vivo and in vitro In addition, chronic phagocytosis disruption in mice deficient for receptors

155 for a large variety of processes, including phagocytosis, drug delivery, and the effects of small mi

156 es in endocannabinoid tone promote microglia phagocytosis during a critical period of amygdala develo

157 clearance expose ligands that initiate their phagocytosis ("eat me" signals), while others avoid phag

158 th temozolomide results in a significant pro-phagocytosis effect due to the latter's ability to induc

159 mouse phagocytes, MaR1 (0.01-10 nM) enhanced phagocytosis, efferocytosis, and phosphorylation of a pa

160 iring actin dynamics, Syk, and PI3K, but not phagocytosis, elicits both NLRP3-dependent and NLRP3-ind

161 heir clearance of harmful substances involve phagocytosis, endocytosis, adhesion, and signaling.

162 Antibody-dependent cellular phagocytosis, Env-specific IgG3, tier 1A neutralising ac

163 delivery hurdles including renal clearance, phagocytosis, enzymatic degradation, protein absorption,

164 Many ways of assaying phagocytosis exist that utilize a variety of phagocytic

165 mechanism for the suppression of macrophage phagocytosis following ozone exposure through the genera

166 er a multitude of innate responses including phagocytosis, fungal killing, and antigen presentation w

167 a method for determining how the success of phagocytosis, here defined as the complete engulfment of

168 actin filaments drive crawling motility and phagocytosis in animal cells and slime molds.

169 egionally specific role of nerve fiber layer phagocytosis in areas of active disease.

170 layer in the coupling of cell locomotion and phagocytosis in hemocytes, the Drosophila macrophage-lik

171 mplement receptor 3) is a major receptor for phagocytosis in macrophages.

172 Blocking complement-dependent phagocytosis in males increases astrocyte survival and p

173 criptomic analysis of the changes induced by phagocytosis in microglia in vitro and identified genes

174 of MSU exposure and ECD-shedding reduced MSU phagocytosis in murine and human macrophages.

175 3 DPA, including the regulation of bacterial phagocytosis in neutrophils.

176 ulator of complement expression and synaptic phagocytosis in the brain, linking circadian proteins to

177 ciation with neuronal cell bodies and myelin phagocytosis in the optic tectum.

178 Antibodies that inhibited platelet phagocytosis in vitro also all ameliorated ITP in vivo.

179 t the antibody's ability to prevent platelet phagocytosis in vitro predicted its ability to ameliorat

180 Antibodies which supported erythrocyte phagocytosis in vitro successfully ameliorated ITP in vi

181 ndrial DNA impair phagocytosis and attenuate phagocytosis-induced apoptosis in human PMNs through Tol

182 terial and antiviral responses downstream of phagocytosis, interferon (IFN)-gamma, lipopolysaccharide

183 ns on electrodes, vesicles in exocytosis and phagocytosis, intracellular vesicle transport, and analy

184 , our findings suggest that non-professional phagocytosis is a general feature of normal tissue cells

185 Phagocytosis is a receptor-mediated, actin-dependent pro

186 To study how phagocytosis is affected by specific changes to the targ

187 -parameter studies (e.g., to investigate how phagocytosis is affected by specific receptor-ligand int

188 synaptic elimination via microglial-mediated phagocytosis is complement dependent and associated with

189 Here, we report that CD47-mediated anti-phagocytosis is concurrently upregulated with HER2 in ra

190 Remarkably, phagocytosis is driven by these local endo-lysosomal Ca(

191 Phagocytosis is measured as a functional outcome in many

192 mple, we demonstrate that antibody-dependent phagocytosis is more efficient for targets with fluid me

193 Here, we propose that phagocytosis is not merely passive corpse removal but ha

194 FcgammaR-mediated phagocytosis is regulated by the concerted actions of ac

195 Phagocytosis is the key mechanism for host control of Ps

196 ry of Mtb into macrophages, which occurs via phagocytosis, its molecular mechanism of action is unkno

197 with polyclonal antiserum modestly increased phagocytosis/killing by an oxidative burst of murine neu

198 previously demonstrated that LC3-associated phagocytosis (LAP), a noncanonical autophagic process de

199 Cd44(-/-) BMDMs showed reduced MSU phagocytosis, LDH release, IL-1beta expression and produ

200 ken up by S. aureus-infected macrophages via phagocytosis leading to enhanced delivery of mupirocin i

201 es internalize the MHC II-CD4 complexes in a phagocytosis-like mechanism and in the process eat the h

202 septation, chromosome translocation, and the phagocytosis-like process of engulfment, and also the ke

203 suggest that inhibition of in vitro platelet phagocytosis may prove to be a valuable tool for determi

204 but still living, cells, and this 'murder by phagocytosis' may be a common phenomenon.

205 report that blockade of MerTK-mediated phagocytosis mobilizes anti-tumor immunity through a mec

206 tumours and support further investigation of phagocytosis modulation as a strategy to enhance cancer

207 can evade phagocytosis by upregulating anti-phagocytosis molecule CD47.

208 in addition to phagocytic receptor-mediated phagocytosis, neutrophils also utilize the chemoattracta

209 Here we show that phagocytosis of apoptotic cells triggers a coordinated t

210 Phagocytosis of apoptotic cells via the receptor MerTK i

211 ependent increase in neutrophil and monocyte phagocytosis of bacteria and a decrease in the diurnal a

212 o upregulate cyclic adenosine monophosphate, phagocytosis of bacteria, and efferocytosis.

213 lasma membrane damage response occurs during phagocytosis of bacteria.

214 signal on cancer cells, prevents macrophage phagocytosis of cancer cells.

215 hat FcgammaRIIIA is also a major mediator of phagocytosis of CD20-opsonized beads by FcgammaRIIIB WT

216 TNC knockdown also inhibited phagocytosis of CD47(-/-) tumor cells in cocultures.

217 METH inhibits antibody-mediated phagocytosis of cryptococci by macrophages and microglia

218 n this Review, we present an overview of the phagocytosis of dead and dying cells, a process known as

219 ted ER stress genes, and promoted macrophage phagocytosis of debris, resulting in suppression of HCC

220 lphav integrin in the myeloid line to reduce phagocytosis of dying cells by CD103(+) dendritic cells,

221 vitro ability to support macrophage-mediated phagocytosis of erythrocytes.

222 receptors (Fpr1/2(-/-)) are defective in the phagocytosis of Escherichia coli and the chemoattractant

223 in an APP/PS1 mouse model through microglial phagocytosis of extracellular Abeta plaques and suppress

224 d spinal cord, microglia are crucial for the phagocytosis of infectious agents, apoptotic cells and s

225 f bacterial infections critically depends on phagocytosis of invading pathogens by polymorphonuclear

226 the IGFBP7-induced type II rosetting hampers phagocytosis of IRBC by host phagocytes.

227 eletion reduces microglial survival, impairs phagocytosis of key substrates including APOE, and inhib

228 particles and TRPML1 being only required for phagocytosis of large targets.

229 To better elucidate the phagocytosis of light chain fibrils, a potential method

230 underwent local cell death due to excessive phagocytosis of lysosomes.

231 utophagy has expanded roles, particularly in phagocytosis of mature myelin and in generating the vast

232 C5a decreased neutrophil killing, and phagocytosis of MRSA.

233 a biologically significant role in mediating phagocytosis of MSU and downstream inflammation and is a

234 This contribution of PTX3 to the phagocytosis of MSU crystals and consequent production o

235 Phagocytosis of mutant uromodulin by cultured dendritic

236 e MerTK expressed by myeloid cells regulates phagocytosis of myelin debris and apoptotic cells that c

237 mine the ability of each antibody to inhibit phagocytosis of platelets, the antibodies were used to s

238 of the phagocytic machinery are needed, but phagocytosis of pLL particles is not required.

239 ed with a significant increase in macrophage phagocytosis of Pneumocystis Both passive antibody and s

240 There was also a decrease in phagocytosis of postsynaptic elements by microglia expre

241 7 and PD-L1 reversed fibrosis, by increasing phagocytosis of profibrotic fibroblasts and by eliminati

242 rophils and, consequently, increased opsonic phagocytosis of S aureus and other pathogens.

243 ential protective epitopes of importance for phagocytosis of S. pyogenes cells are localized.

244 T cell proliferation are exclusively due to phagocytosis of stimulation beads used in cocultures and

245 REV-ERBalpha deletion increased microglial phagocytosis of synapses and synapse loss in the CA3 reg

246 ynapses directed ongoing microglia-dependent phagocytosis of synapses for at least 30 d after stroke,

247 PUFA deficiency increases microglia-mediated phagocytosis of synaptic elements in the rodent developi

248 ration of neutrophils, followed by extensive phagocytosis of the light chain fibrils by macrophages,

249 ceptors and participates in their renewal by phagocytosis of the tips of the photoreceptors.

250 GSDME expression enhances the phagocytosis of tumour cells by tumour-associated macrop

251 ting ZIKV VTx to the fetus, possibly through phagocytosis of virus or virus-infected cells.

252 at exogenous IFN-gamma suppressed macrophage phagocytosis of zymosan in vivo, and antibody blockade o

253 n-3 (MAC-2), a marker of glial axonal debris phagocytosis, on NMJ denervation in SOD1 mice.

254 No apparent defect in phagocytosis or bacterial killing was found.

255 known as macropinocytosis, without affecting phagocytosis or clathrin-mediated endocytosis.

256 Engulfment of extracellular material by phagocytosis or macropinocytosis depends on the ability

257 ate as well as to probe their environment by phagocytosis or macropinocytosis.

258 eutrophil reactive oxygen species formation, phagocytosis, or cytokine secretion, and neutrophils tre

259 reduces Mac-1 activation, cellular adhesion, phagocytosis, oxidized low-density lipoprotein uptake, a

260 these studies suggest that the NWA entry and phagocytosis pathways overlap.

261 mediated effects on both Ag presentation and phagocytosis pathways.

262 including those for Toll, melanization, and phagocytosis pathways.

263 nd female mice chronically deficient for two phagocytosis pathways: the purinergic receptor P2Y12, an

264 lement system, immune cell infiltration, and phagocytosis; pathways that may drive tubal injury.

265 with antiviral functions (neutralization and phagocytosis) predominantly mediated by the IgG fraction

266 through PLCgamma2 to mediate cell survival, phagocytosis, processing of neuronal debris, and lipid m

267 mulating microglia' (LDAM), are defective in phagocytosis, produce high levels of reactive oxygen spe

268 Increased phagocytosis promotes killing of S aureus and interleuki

269 ses neurogenesis, suggesting that microglial phagocytosis provides a negative feedback loop that is n

270 providing functional macrophages capable of phagocytosis, rather than by supplying GALC for Schwann

271 xin in the tissue-basement membranes and the phagocytosis receptor Eater on the hemocytes physically

272 s provided evidence of activated immune- and phagocytosis-related pathways with LPS and cytokines as

273 ad to hypercapsule production, which confers phagocytosis resistance, enhanced dissemination and incr

274 In vivo studies of macrophage phagocytosis reveal increased bacterial ingestion follow

275 survival in the neurogenic niche through the phagocytosis secretome, thereby supporting the long-term

276 ack of induction was not due to differential phagocytosis, subcellular location, or changes in endoso

277 Increased tumour cell phagocytosis subsequently enhances antigen cross-present

278 ect key PMN inflammatory functions including phagocytosis, superoxide release and apoptosis.

279 B cell receptor, as a negative regulator of phagocytosis that is upregulated on aged microglia.

280 Following phagocytosis, the growth of C. neoformans is inhibited b

281 To quantify phagocytosis, the reconstituted target particles are inc

282 ers PXN/LPXN limit myeloid cell motility and phagocytosis, thereby providing an important immune regu

283 endent lamellar protrusions for motility and phagocytosis, this work supports an evolutionarily ancie

284 ay induce dendritic cell tolerance following phagocytosis through an unfolded protein/endoplasmic ret

285 venger N-acetylcysteine, which also reverted phagocytosis to baseline levels.

286 ulf a few bacteria at a time if it is to use phagocytosis to clear the infection.

287 chemoattractant GPCR/Gi signaling to mediate phagocytosis to fight against invading bacteria.

288 Functional assays (killing, phagocytosis, transmigration, and respiratory burst) wer

289 This daily variation in microglial synaptic phagocytosis was abrogated by global REV-ERBalpha deleti

290 This astrocytic phagocytosis was also observed in Irf8-deficient mice, i

291 Such diminution of phagocytosis was examined to be associated with downregu

292 During phagocytosis, we find that disruption of RGBARG causes a

293 onal pathways for Fcgamma receptor dependent phagocytosis were among shared enriched pathways.

294 ithelial cell binding, and antibody-mediated phagocytosis were measured.

295 omega-3 drink SMF in vivo rescued macrophage phagocytosis when glycolysis or glycosylation were block

296 osomes contact nascent phagosomes to promote phagocytosis, whereas endo-lysosome immobilization preve

297 riation in the degree of microglial synaptic phagocytosis which was antiphase to REV-ERBalpha express

298 light on the physical barriers that modulate phagocytosis, which have been heretofore underappreciate

299 s underwent a brief burst (<1 hour) of rapid phagocytosis, which was then invariably followed by a sh

300 exerted by a macrophage during FcyR-mediated phagocytosis while performing both sequential two-color,