ライフサイエンスコーパス: phalloidin

1 C (calphostin C), and actin destabilization (phalloidin).

2 ), microglia (CD11b), and filamentous actin (phalloidin).

3 alpha on the actin cytoskeleton by rhodamine-phalloidin.

4 ls treated with the microfilament stabilizer phalloidin.

5 sing extracellular applications of rhodamine-phalloidin.

6 cofilin-induced partial release of rhodamine phalloidin.

7 bbit psoas fiber were labeled with rhodamine-phalloidin.

8 x at branching sites that were stabilized by phalloidin.

9 polymerization in the presence of MgCl2 and phalloidin.

10 ilamentous actin with fluorescein-conjugated phalloidin.

11 on was studied after staining with rhodamine phalloidin.

12 toskeletal actin when stained with rhodamine-phalloidin.

13 winding in both the presence and absence of phalloidin.

14 s jasplakinolide and 4-fold more active than phalloidin.

15 observe polymerization in real time, without phalloidin.

16 probing new actin assembly with fluorescent phalloidin.

17 yofibrillar proteins, and FITC- or rhodamine phalloidin.

18 hate (PtdIns(3,4,5)P3) was also prevented by phalloidin.

19 ntibody and host cell F-actin with rhodamine-phalloidin.

20 nce microscopy after labeling with rhodamine-phalloidin.

21 of changes in actin bundles were blocked by phalloidin.

22 chalasin B with or without pretreatment with phalloidin.

23 alogues, KCl concentration, and the level of phalloidin.

24 tin in the CL was labeled with conjugates of phalloidin.

25 sensitized emission by tetramethyl-rhodamine phalloidin.

26 by calcineurin inhibitors, staurosporine, or phalloidin.

27 cytochalasin D, and by the inability to bind phalloidin.

28 xtension when stained with rhodamine-labeled phalloidin.

29 ected by the microtubule polymerizing agent, phalloidin.

30 ared thicker after staining with fluorescent phalloidin.

31 mentous actin organization was visualized by phalloidin.

32 ere visualized with AlexaFluor488-conjugated phalloidin.

33 determined using Alexa Fluor 488-conjugated phalloidin.

34 F-actin cytoskeleton was visualized with phalloidin.

35 half-sarcomere was labeled with fluorescent phalloidin.

36 this mutant was sensitive to nocodazole and phalloidin.

37 Actin filaments were detected with phalloidin.

38 on was visualized by staining with Texas-red phalloidin.

39 n was quantified using fluorescently labeled phalloidin.

40 t a solid-phase synthetic approach to [Ala7]-phalloidin (1).

41 dialysis with the actin filament stabiliser phalloidin (10 microM) prevented KATP channel activation

42 Microsomes prepared in the presence of phalloidin (a toxin that binds to F actin and stabilizes

43 peptide (pHLIP)-facilitated translocation of phalloidin, a cell-impermeable polar toxin, inhibits the

44 he intensity of staining with Alexa-Fluor488-phalloidin, a compound that permits visualization and qu

45 Staining with BODIPY phalloidin, a fluorescent dye selective for F-actin, sho

46 protein that binds and severs filaments, and phalloidin, a fungal toxin that binds and stabilizes F-a

47 Phalloidin, a fungal toxin which stabilizes F-actin and

48 oduce the cell-impermeable bi-cyclic peptide phalloidin, a specific marker for actin filaments, into

49 nt acid phosphatase (TRAP), Oregon Green 488-phalloidin, a stain for cytoskeletal proteins, and count

50 The anisotropy of phalloidin-actin changed rapidly at first and was follow

51 luorescent nucleotide was similar to that of phalloidin-actin.

52 mbly and MMP secretion and pretreatment with phalloidin again retarded actin disassembly and MMP secr

53 n was investigated by visualizing actin with phalloidin-Alexa 488 after infection or transfection of

54 Dialysis with phalloidin also prevented hypo-osmotically activated Cl-

55 inject nanomolar concentrations of rhodamine phalloidin (an impermeable dye molecule for staining fil

56 ostsynaptic role for actin, latrunculin B or phalloidin, an actin filament stabilizer, was perfused i

57 s stained with fluorescent rhodamine-labeled phalloidin, an actin stain, that the transient viscoelas

58 In contrast, phalloidin, an agent that prevents depolymerization of a

59 ation by confocal fluorescein isothiocyanate-phalloidin analysis.

60 ed by the findings that the actin stabilizer phalloidin and a cofilin inhibitory peptide each blocked

61 1, 3, or 7 days, matrices were labeled with phalloidin and a nucleic acid dye, and were imaged using

62 The actions of phalloidin and actin were reversed by coapplication of g

63 actin cytoskeleton with rhodamine-conjugated phalloidin and analysis by confocal fluorescence microsc

64 body and anti-CSP24 antibody, or fluorescent phalloidin and anti-CSP24 antibody showed that CSP24 is

65 ons was determined by labeling cultures with phalloidin and anti-talin or ILK antibodies, respectivel

66 tics (anti-Ki-67, anti-p16), stratification (phalloidin and anti-ZO-1), and differentiation (anti-K3,

67 ECs were subjected to immunostaining to FITC-phalloidin and antibodies to different junction componen

68 ed cells by using fluorescein isothiocyanate-phalloidin and antibodies to phosphotyrosine and cortact

69 Buttons were single and double labeled using phalloidin and antibodies to ZO-1, Ki67, fibronectin, al

70 te here that together, but not individually, phalloidin and cofilin co-rescue the polymerization of c

71 The NRG 1beta effect was also inhibited by phalloidin and cytochalasin D.

72 ities were noticed, including attenuation of phalloidin and cytoplasmic active beta-catenin staining,

73 ctin polymerization were revealed in reduced phalloidin and deficits in oligodendrocyte cellular bran

74 Significantly, the unique actin inhibitors, phalloidin and DNase I, also inhibit synthesis of poly P

75 ocked by concurrent whole-cell dialysis with phalloidin and DNase, respectively.

76 or 15 to 120 min were stained with rhodamine phalloidin and examined with a confocal microscope.

77 actin) content by using rhodamine-conjugated phalloidin and flow cytometry showed an elevated F-actin

78 Rhodamine-phalloidin and flow cytometry were used to measure chang

79 vitro cultures were fixed, stained with FITC-phalloidin and hair cells were counted.

80 uld be restored to wild type actin levels by phalloidin and improved greatly through copolymerization

81 h this hypothesis, the actin modifying drugs phalloidin and jasplakinolide enhanced secretion, while

82 erates nuclear actin filaments-detectable by phalloidin and live-cell actin probes-with three charact

83 pletely resistant to hepatoxicity induced by phalloidin and microcystin-LR.

84 We found that a combination of phalloidin and NSF/GluR2 inhibitor reduced the response

85 he association rate constant is low for both phalloidin and rhodamine phalloidin because the filament

86 ease is prevented by the F-actin stabilizers phalloidin and to a lesser extent beryllium fluoride.

87 urons with fluorescently labeled tubulin and phalloidin and used fluorescence time-lapse imaging to a

88 After 24 hours, constructs were labeled with phalloidin and were imaged using fluorescent and reflect

89 ssed by confocal microscopy with fluorescein phalloidin and were not prevented by staurosporine or ca

90 Further, abnormal phalloidin and wheat germ agglutinin (WGA) staining of T

91 ing peptide nucleic acids, a cyclic peptide (phalloidin), and organic compounds.

92 compared (-)-doliculide with jasplakinolide, phalloidin, and chondramide C to gain insight into a pos

93 ting F-actin specific fluorescent conjugate, phalloidin, and high-content image analysis of the compl

94 ained with antibodies or fluorescent-labeled phalloidin, and viewed with a confocal microscope.

95 us were evaluated by staining with rhodamine-phalloidin, anti-paxillin, and anti-phosphotyrosine anti

96 e branches in samples treated with rhodamine-phalloidin arises from multiple influences of the peptid

97 Using rhodamine-phalloidin as a probe, the individualization complex is

98 concentrations of microinjected fluorescent phalloidin as a tracer for actin filaments, we found tha

99 er donor and anti-actin tetramethylrhodamine phalloidin as an acceptor.

100 High-affinity fluorescent phalloidin as well as immunocytochemistry using anti-act

101 gelsolin and stains intensely with rhodamine-phalloidin, as does the zebrafish extraocular muscle.

102 We observe differences in the phalloidin association kinetics between muscle alpha- an

103 s of wild-type actin, beryllium fluoride, or phalloidin at room temperature, although at 4 degrees C

104 ant is low for both phalloidin and rhodamine phalloidin because the filaments must undergo conformati

105 The rates of rhodamine phalloidin binding are independent of the pH, ionic stre

106 At equilibrium, rhodamine phalloidin binding generates a positive entropy change (

107 ces in actin filaments, we have examined the phalloidin binding kinetics and the bulk rheologic prope

108 mpens these conformational fluctuations, and phalloidin binding kinetics are inhibited.

109 ional changes (i.e. "breathe") to expose the phalloidin binding site.

110 ctin) in pollen were quantified by measuring phalloidin binding sites, a sensitive assay that had not

111 the kinetics and thermodynamics of rhodamine phalloidin binding to actin purified from rabbit skeleta

112 The cyclic peptide phalloidin binds and stabilizes actin filaments.

113 Here we show that rhodamine-phalloidin binds both Arp2/3 complex and the VCA domain

114 Mg(2+)-containing filaments with phalloidin bound also displayed increased rotational mot

115 escued by filament seeds added together with phalloidin but not with cofilin.

116 ngly and reversibly the release of rhodamine phalloidin by cofilin.

117 Fluorescein-labeled phalloidin confirmed marked disruption of filamentous ac

118 d that fibroblasts stained for f-actin using phalloidin conjugated with common fluorophores display d

119 All three mutants display abnormal phalloidin cytoskeletal staining.

120 FRET) between GFP-tagged Hsp27 and rhodamine phalloidin-decorated actin, minimal interaction was foun

121 flexibility of the filament, the binding of phalloidin decreased the torsional flexibility of all fi

122 1(-/-);Rac2(-/-) RBCs stained with rhodamine-phalloidin demonstrated F-actin meshwork gaps and aggreg

123 g of Kv1.5-GFP and retrospective labeling of phalloidin demonstrated motility of Kv1.5 vesicles on ac

124 stained with saturating levels of rhodamine-phalloidin demonstrated that changes in the level of F-a

125 es also increased KATP channel activity in a phalloidin-dependent manner.

126 plakinolide, readily displaced a fluorescent phalloidin derivative from actin polymer.

127 ctin polymer nor was it able to displace the phalloidin derivative from polymer.

128 did not inhibit the binding of a fluorescent phalloidin derivative to actin polymer nor was it able t

129 ulin (Tmod) or probes such as phallacidin (a phalloidin derivative).

130 Addition of phalloidin did not alter cooperative effects between bou

131 Phalloidin dissociates from all mammalian actin isoforms

132 heir binding sites are distinct, cofilin and phalloidin do not bind simultaneously to F-actin.

133 es not lock actin in filaments (as rhodamine-phalloidin does), possibly allowing for its use in inves

134 However, in contrast to jasplakinolide and phalloidin, dolastatin 11 did not inhibit the binding of

135 In neurons coinjected with rhodamine-phalloidin, F-actin was observed in dynamic cortical pat

136 ANP quenching of rhodamine phalloidin fluorescence showed that neither Ca(2+) nor S

137 We show that SIF causes the intensity of phalloidin fluorescence to increase 4-5 fold and its flu

138 This increase in cortical phalloidin fluorescence was inhibited by latrunculin B a

139 The distribution of the increasing phalloidin fluorescence was uniform with respect to the

140 during recovery; but when adjusted for total phalloidin fluorescence, FRET between Hsp27 and F-actin

141 ation of permeabilized strips with 50 microM phalloidin for 1 h, the increases in isometric force and

142 version method based in the high affinity of phalloidin for filamentous (F)-actin.

143 tant and lower binding affinity of rhodamine phalloidin for S. cerevisiae actin filaments provide a q

144 -actin yet synergize in promoting release of phalloidin from filaments, suggesting that Crn1/Cof1 co-

145 tabilization of F-actin by microinjection of phalloidin had no effect on GVBD.

146 Phalloidin had no effect on Vmax during steady-state iso

147 horylated GAP-43 inhibit binding of actin to phalloidin, implying a lateral interaction with filament

148 Stabilizing filaments with phalloidin in most experiments eliminated any contributi

149 xpressed GFP-mTn co-localized with rhodamine-phalloidin in permeabilized tobacco BY-2 suspension cell

150 angement of actin filaments (Alexa Fluor 568-phalloidin) in primary cultures of IMCD cells.

151 Phalloidin increased both filamentous actin (F-actin) an

152 F-actin, as measured by binding of rhodamine-phalloidin, increased transiently during phagocytosis, a

153 the hypothesis that F-actin stabilization by phalloidin increases tension cost (i.e. ATP hydrolysis r

154 reasing the rigidity of actin filaments with phalloidin increases the extent of depletion, whereas sh

155 epithelium stains very weakly with rhodamine-phalloidin, indicating little F-actin in the cytoplasm.

156 these cells coincided with that of rhodamine-phalloidin, indicating that GFP-ABD specifically binds f

157 ifferential salt precipitation or binding to phalloidin-induced actin filaments, had no effect on ves

158 otein kinase and allows entry of fluorescent phalloidin into the cytoplasm of epithelial cells.

159 n the cell after introduction of fluorescent phalloidin into the medium, and the cytokinetic ring was

160 his indicates that the stabilizing effect of phalloidin is achieved mainly through constraining struc

161 or association and dissociation of rhodamine phalloidin is dominated by entropic changes (delta S++).

162 om temperature, although at 4 degrees C only phalloidin is effective.

163 The affinity of NFA filaments for rhodamine phalloidin is lower than that of native actin filaments,

164 for actin marked by microinjected rhodamine phalloidin is very similar, 0.033 +/- 0.013 s(-1), sugge

165 Like phalloidin, jasplakinolide stabilizes F-actin and promot

166 proximately 1-micrometer stripe in rhodamine phalloidin-labeled actin appears stable up to at least 3

167 ectrodes elevated above a surface, rhodamine-phalloidin-labeled actin filaments were attracted to the

168 Rhodamine-phalloidin-labeled actin filaments were visualized glidi

169 velocity matches measurements with rhodamine-phalloidin-labeled actin.

170 the fluorescence lifetime (tau) of Alexa488-phalloidin-labeled actin.

171 fluorescence microscopy was used to identify phalloidin-labeled CLANs and to ascertain the presence o

172 ng actin-core dimensions of stereocilia from phalloidin-labeled mouse cochleas, we demonstrated that

173 scopy (FPM) has been used to study rhodamine phalloidin-labeled red cell ghosts.

174 Three days after laser injury, phalloidin-labeled RPE cells and isolectin-labeled endot

175 rdiac troponin and tropomyosin and rhodamine-phalloidin-labeled skeletal actin and skeletal heavy mer

176 e stretching stiffness of a single rhodamine-phalloidin-labeled, 1-microm-long F-actin is 34.5 +/- 3.

177 unts of conjunctiva prepared using rhodamine-phalloidin labeling followed by confocal microscopy.

178 Phalloidin labeling intensified in the ciliary stalk wit

179 while the loss of: 1) membrane integrity; 2) phalloidin labeling of F-actin; and 3) TO-PRO-1 labeling

180 BDNF caused no changes in phalloidin labeling of filamentous actin (F-actin) when

181 6 d, because calmodulin immunoreactivity and phalloidin labeling of filamentous actin are retained.

182 Phalloidin labeling of filamentous actin revealed profou

183 ions of spines in these areas indicated that phalloidin labeling was restricted to the largest and mo

184 scopy and immunohistochemistry together with phalloidin labeling, we systematically characterize both

185 n-activating mAb AP-5 and were identified by phalloidin labeling.

186 Rhodamine-phalloidin labelling was used wherever possible, and rev

187 The delivery construct carries phalloidin linked to its inserting C terminus via a disu

188 Yeast actin filaments bind rhodamine phalloidin more rapidly, suggesting that perhaps they ar

189 aments of S. cerevisiae actin bind rhodamine phalloidin more weakly than Acanthamoeba and rabbit skel

190 conjugate quantum dot (Qdot) binding biotin-phalloidin on actin.

191 at 25 degrees C but will in the presence of phalloidin or beryllium fluoride.

192 Interestingly, phalloidin or the presence of wild type actin reversed t

193 Pretreatment with phalloidin or treatment with MMP inhibitors partly preve

194 onent was found by using an actin inhibitor (phalloidin) or an inhibitor of NSF (N-ethylmaleimide-sen

195 ropelled actin filaments is similar (without phalloidin) or slightly lower (with phalloidin) than tha

196 eys were fluorescently labeled with lectins, phalloidin, or antibody.

197 LS-WH2 can bind to but fails to depolymerize phalloidin- or jasplakinolide-bound actin filaments.

198 Stabilization of actin filaments with phalloidin partially prevents the rundown, whereas colla

199 omic force microscopy, F-actin staining with phalloidin, passage of FITC-conjugated dextran through a

200 cybe albipes that cleaves a synthetic 22-mer phalloidin peptide to release the mature toxin peptide (

201 ofibrils were labeled lightly with rhodamine-phalloidin, placed on coverslips coated with SIF, illumi

202 t the plasmalemma, actin polymerization into phalloidin-positive stress fibers, and finally CAM endoc

203 Both were retarded by phalloidin pretreatment.

204 Phalloidin prevented pressure-stimulated adhesion.

205 Stabilization of F-actin with phalloidin prevented the Ca(2+) response in AF and TZ ce

206 tabilizing actin filaments with jaspamide or phalloidin prevented vesicle release induced by ischaemi

207 binding to regulated alpha-actin affects the phalloidin-probe distance.

208 We purified an enzyme from the phalloidin-producing mushroom Conocybe albipes that clea

209 Not only does phalloidin promote nucleation of pure actin monomers but

210 Conversely, phalloidin protects the L267C and L269C filaments and in

211 Although the numbers of synapsin and phalloidin puncta do not differ from WT, preCGG neurons

212 ucleus and actin distribution using DAPI and phalloidin respectively.

213 actin were identified by DNase and rhodamine phalloidin, respectively.

214 ls, the induction of actin polymerization by phalloidin resulted in the incorporation of both IQGAP a

215 CCV nucleocapsid (N) monoclonal antibody and phalloidin revealed a colocalization of the BCCV N prote

216 analysis of filaments rescued by cofilin and phalloidin revealed a dense contact between opposite str

217 Quantitative immunostaining with phalloidin revealed a significant increase in axonal F-a

218 ng of 3T3 fibroblasts with anti-vinculin and phalloidin revealed clear cytoskeletal filaments and foc

219 HT29 cells stained with fluorescein-labeled phalloidin revealed contraction of the cytoskeleton and

220 ining of the cells with rhodamine-conjugated phalloidin revealed rapid disassembly of actin filaments

221 Coprecipitation using phalloidin revealed that tau interacts with the most pre

222 T-treated monolayers (stained with rhodamine-phalloidin) revealed diminished and flocculated staining

223 stigated using vital staining with rhodamine phalloidin (RP).

224 s countered by filament stabilizing factors, phalloidin, S1, and tropomyosin.

225 ctron microscopy and staining with rhodamine-phalloidin showed that these lamellipodia displayed ruff

226 ated the architectural dynamics of rhodamine-phalloidin stabilized filamentous actin (F-actin) networ

227 orce microscopy (cryo-AFM) was used to image phalloidin-stabilized actin filaments adsorbed to mica.

228 Phalloidin-stabilized actin filaments were used in order

229 sin inhibited the disulfide cross-linking of phalloidin-stabilized F-actin.

230 Phalloidin stabilizes actin polymers and DNase inhibits

231 s B and D, but was not affected by 10 microM phalloidin (stabilizes actin filaments) or 50 microM col

232 Whereas both anti-MYC and Rho-phalloidin stained intra-Z-band F-alpha-actin oligomers,

233 bution, quantitative fluorescence imaging of phalloidin-stained cells, and ultrastructural studies in

234 ted thick actin assemblies seen in rhodamine phalloidin-stained GG cells.

235 Confocal microscopy of rhodamine phalloidin-stained infected Vero cells revealed the typh

236 ments of F-actin by immunoblot and rhodamine phalloidin staining after ultracentrifugation.

237 ular cytoskeleton, visualized by fluorescein phalloidin staining and confocal microscopy.

238 Cholestatic hepatocytes were analyzed by phalloidin staining and electron microscopy.

239 n reorganization was examined by fluorescent-phalloidin staining and Rac activity assay.

240 Furthermore, phalloidin staining and reactive oxygen species estimati

241 With fluorescent phalloidin staining and serial thin sections, we show th

242 ng and reduces actin filaments determined by phalloidin staining and Western blotting of Triton X-100

243 ent by fluorescein isothiocyanate-conjugated phalloidin staining as well as by indirect immunolabelin

244 al-stromal communications was evaluated with phalloidin staining as well as electron microscopy.

245 Immunofluorescence combined with phalloidin staining demonstrated the colocalization of t

246 g 3T3L1 adipocyte differentiation, rhodamine-phalloidin staining demonstrated the formation of a cort

247 Fluorescent phalloidin staining indicated that a well defined fringe

248 Rhodamine phalloidin staining indicates that ATPgammaS treatment i

249 was accompanied by a reduction of rhodamine-phalloidin staining most prominent in the growth cone pe

250 site of attachment, as visualized by either phalloidin staining of fixed cells or the active recruit

251 BODIPY-phalloidin staining of four different stably transfected

252 Phalloidin staining of hepatocytes showed a prominent sh

253 In contrast, phalloidin staining of infected cells showed that the vi

254 Rhodamine phalloidin staining of karst mutant ovaries similarly re

255 Rhodamine-labeled phalloidin staining of morphologically differentiated 3T

256 Phalloidin staining revealed dispersed, shorter and diso

257 Rhodamine-phalloidin staining revealed increase stress fiber forma

258 Rhodamine phalloidin staining reveals that the actin-based hexagon

259 Phalloidin staining showed that FTI treatment did not re

260 morphologic studies, AlexaFluor 488-labeled phalloidin staining was used to examine actin filament,

261 Annexin-V and phalloidin staining were used to detect apoptosis and th

262 cts of lovastatin on F-actin reorganization (phalloidin staining), focal adhesion formation (paxillin

263 By phalloidin staining, a colocalization of pyrin with acti

264 ured and analyzed by histology, immunoblots, phalloidin staining, immunohistochemistry, electron micr

265 hanges in actin cytoskeletal organization by phalloidin staining, MMP-2 activation by gelatin zymogra

266 at this later stage as well and, based upon phalloidin staining, we propose that the second half of

267 Phalloidin staining, which labels actin filaments of ste

268 leton (thickening of fibrils) as assessed by phalloidin staining, with more pronounced effects at 20

269 Actin structures were visualized by phalloidin staining.

270 n) was confirmed by the absence of rhodamine-phalloidin staining.

271 h an anti-acetylated tubulin antibody and by phalloidin staining.

272 sualized by light microscopy and fluorescent-phalloidin staining.

273 actin cytoskeletal network was visualized by phalloidin staining.

274 ve-muscle cultures, as revealed by rhodamine-phalloidin staining.

275 Actin was visualized by phalloidin staining.

276 migration using inserts, wound healing, and phalloidin staining; and cell synthesis using ELISA and

277 n fibers within 30 minutes, as determined by phalloidin stainings of the polymerized actin and tropon

278 Computational modeling reveals how bound phalloidin stiffens actin filaments and inhibits the rel

279 In contrast, although phalloidin strongly stabilizes F-actin, it causes little

280 n the rat central nervous system (CNS) using phalloidin tagged with the fluorophore eosin followed by

281 thalene-1-sulfonic acid at Cys-190 of Tm and phalloidin-tetramethylrhodamine B isothiocyanate bound t

282 (without phalloidin) or slightly lower (with phalloidin) than that of HMM-free filaments observed in

283 With beryllium fluoride, but not phalloidin, this polymerization rescue is cold-sensitive

284 actin and/or competition between cofilin and phalloidin to alter F-actin symmetry.

285 r and extracellular application of rhodamine-phalloidin to conventional hippocampal slices to test wh

286 ion, consistent with the expected binding of phalloidin to F actin, stabilizing the filaments against

287 telets were fixed and stained with Alexa-488-phalloidin to label the actin cytoskeleton.

288 n gelation activity, even in the presence of phalloidin, to stabilize actin filaments against debranc

289 n, to assist with actin nucleation, and with phalloidin, to stabilize the elongating filament segment

290 Phalloidin treatment altered actin polymerization, as de

291 erization defect can be partially rescued by phalloidin treatment, which stabilizes F-actin.

292 immunoreactivity with F-actin (labeled with phalloidin) was observed at the apices and bases of RPE

293 ystrophin antibody and tetramethyl-rhodamine phalloidin were present.

294 incubating the cells overnight in 10 microM phalloidin, which binds to actin filaments (n = 5).

295 Branches were stabilized by phalloidin, which inhibits phosphate dissociation from A

296 Phalloidin, which is known to stabilize actin filaments,

297 dual filaments after treatment with the drug phalloidin, which perturbs filament dynamics.

298 Rhodamine-phalloidin, which selectively binds to polymerized actin

299 Direct application of phalloidin, which stabilizes actin, preserved low PO and

300 However, preapplication of phalloidin, which stabilizes the actin polymerization, s