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1 etaldehyde, 4-hydroxyphenylacetaldehyde, and phenylethylamine.
2  and identified it to be a biogenic amine, 2-phenylethylamine.
3 ive sensory neurons that also responded to 2-phenylethylamine.
4 s the anti-Markovnikov addition product beta-phenylethylamine.
5  monoamines such as serotonin, tyramine, and phenylethylamine.
6  of serotonin, norepinephrine, dopamine, and phenylethylamine.
7 ffects for para-substituted benzylamines and phenylethylamines.
8 (*) from three chiral benzylic substrates (1-phenylethylamine, 1-phenylethanol, and alpha-vinylbenzyl
9 t study, this is exemplified by subjecting 1-phenylethylamine (1a) and analogous benzylic amines to D
10 iety of Phe-derived metabolites, including 2-phenylethylamine, 2-phenylethanol, and 2-phenylethyl-bet
11                                 The simplest phenylethylamine, 2-phenylethylamine itself, is without
12 6 receptors a series of compounds, including phenylethylamines 6, pyrroloethylamine 7, and phenylpipe
13                          In our study, alpha-phenylethylamine (alpha-PEA) allowed an efficient 1-3 st
14 nd rTAAR1, respectively) insensitive to beta-phenylethylamine, amphetamine (AMPH), and methamphetamin
15                 N-methyl-4-phenylpyridinium, phenylethylamine, amphetamine, and methylenedioxymethamp
16 methyl N-Boc-phosphoramidates derived from 1-phenylethylamine and 1,2,3,4-tetrahydronaphthalen-1-ylam
17 enzymatic functions and converted Phe into 2-phenylethylamine and 2-phenylacetaldehyde, respectively.
18 ens) resulted in decreased accumulation of 2-phenylethylamine and 2-phenylethyl-beta-d-glucopyranosid
19 ontrols the herbivore-induced formation of 2-phenylethylamine and 2-phenylethyl-beta-d-glucopyranosid
20 nt amine, followed by putrescine, histamine, phenylethylamine and cadaverine.
21 mine, oleoyl aminovaleric acid, a-linolenoyl phenylethylamine and caproyl tryptamine).
22                                  Tryptamine, phenylethylamine and histamine (with the single exceptio
23 evels of monomeric phenols, methylxanthines, phenylethylamine and lower levels of the putrefactive am
24                           The formation of 2-phenylethylamine and phenylacetaldehyde in mixtures of p
25 hanism because the Ea of formation of both 2-phenylethylamine and phenylacetaldehyde remained unchang
26 e that the benzenesulfonamide portion of the phenylethylamine and phenylpiperazine analogues can be "
27 revalence of tyramine followed by histamine, phenylethylamine and putrescine; and total amine levels
28                                         beta-phenylethylamine and tryptamine are biogenic amines (BA)
29 ontribute to establish legal limits for beta-phenylethylamine and tryptamine in food.
30 ent knowledge regarding the toxicity of beta-phenylethylamine and tryptamine is limited; more informa
31 study describes a real-time analysis of beta-phenylethylamine and tryptamine toxicity on a human inte
32 lative affinity for indoleethylamines versus phenylethylamines and temperature-activity relationships
33  indole, the neurotransmitter precursor beta-phenylethylamine, and a variety of peroxidase and laccas
34     The decrease in the content of tyramine, phenylethylamine, and dopamine in laboratory-scale white
35 mitters including serotonin, norepinephrine, phenylethylamine, and dopamine.
36 adienyl)ethanol, and the aromatized product, phenylethylamine, and that the two pathways do not cross
37 e former was the main mode of action of beta-phenylethylamine, and the latter the main mode of action
38 ytryptamine, a decreased preference for beta-phenylethylamine, and, similar to MAO A, was more sensit
39 do not exhibit aggression and only levels of phenylethylamine are increased.
40          The developed approach utilizes (S)-phenylethylamine as a chiral auxiliary handle for CIDT,
41 get compounds using Shi epoxidation or (R)-1-phenylethylamine as a source of chirality.
42 not a requirement for the binding of certain phenylethylamines at 5-HT(2A) receptors, the presence of
43                The discovery of novel uracil phenylethylamines bearing a butyric acid as potent human
44 ation of biogenic and dietary amines such as phenylethylamine, benzylamine, dopamine, and tyramine.
45 lycine and agmatine and neuromodulators beta-phenylethylamine (beta-PEA) and taurine are important bi
46                                         beta-Phenylethylamine (betaPEA) is a trace amine present in t
47 -aryl[or (1S,2S)-1-alkyl]-2-(phenylseleno)-2-phenylethylamines by reaction with t-BuLi and subsequent
48 spermidine, putrescine, histamine, tyramine, phenylethylamine, cadaverine and serotonin) were determi
49                                        Thus, phenylethylamine can be used as a carbon and nitrogen so
50  reactions of QHNDH with chiral deuterated 2-phenylethylamines demonstrated that the enzyme abstracts
51                                       During phenylethylamine-dependent growth, aromatic amine dehydr
52      The binding of dopamine, a hydroxylated phenylethylamine derivative, displays the same pH depend
53                  Measurements indicated that phenylethylamine-fed cells generate H2O2 at 30 times the
54 n studies involving enzymatic depletion of 2-phenylethylamine from a carnivore odor indicate it to be
55 ective in releasing tyramine, spermidine and phenylethylamine from conjugates.
56 amino function (isoamylamine, tyramine, beta-phenylethylamine), in general, showed a weak retention b
57 ptamine), norepinephrine, dopamine, and beta-phenylethylamine; in humans and mice, these neurochemica
58  of serotonin, norepinephrine, dopamine, and phenylethylamine increased, and serotonin metabolite 5-h
59            One such cue, carnivore-derived 2-phenylethylamine, is a key component of a predator odor
60             The simplest phenylethylamine, 2-phenylethylamine itself, is without significant central
61       The phenylalkylamine, particularly the phenylethylamine, moiety is a common structural feature
62 r mTAAR9-G(olf) trimers in complex with beta-phenylethylamine, N,N-dimethylcyclohexylamine or spermid
63   Two prey species, rat and mouse, avoid a 2-phenylethylamine odor source, and loss-of-function studi
64  without significant changes of the K(m) for phenylethylamine or dopamine or the IC(50) for deprenyl
65 0-fold slower in catalyzing the oxidation of phenylethylamine or of serotonin.
66 are synthesized in one step from allenes and phenylethylamines or benzylamines, respectively.
67 , spermine, spermidine), aromatic (tyramine, phenylethylamine) or heterocyclic (histamine, tryptamine
68 ependent biosynthesis of the TPQ cofactor in phenylethylamine oxidase (PEAO) and histamine oxidase fr
69 ling of the C2, C4, and C5 oxygens of TPQ in phenylethylamine oxidase (PEAO) from Arthrobacter globif
70 y of the topa quinone biogenesis reaction in phenylethylamine oxidase from Arthrobacter globiformis (
71                                      Second, phenylethylamine oxidase is an unusual catabolic enzyme
72 ect, the periplasmic compartmentalization of phenylethylamine oxidase serves the same purpose as the
73 atic that E. coli can synthesize a catabolic phenylethylamine oxidase that stoichiometrically generat
74 thway that is required for the catabolism of phenylethylamine (PEA) and its hydroxylated derivatives
75 roanalytical and physicochemical analysis of phenylethylamine (PEA), allowing the determined of the f
76 ntinuous intravenous infusion over 30 min of phenylethylamine (PEA), an endogenous substrate of MAOB,
77                                     Chiral 1-phenylethylamine (PEA)-hybridized perovskite PEA/CsPbBr(
78 ), whereas MAOB preferentially oxidizes beta-phenylethylamine (PEA).
79 inatorial library of 10 structurally related phenylethylamines (PEAs) is investigated for chiral sepa
80 ss 38 mammalian species indicates enriched 2-phenylethylamine production by numerous carnivores, with
81 ncluding serotonin, histamine, tryptamine, 2-phenylethylamine, putrescine, and agmatine.
82 The quantity of biogenic amines (tryptamine, phenylethylamine, putrescine, cadaverine, histamine, ser
83 ight biogenic amines (cadaverine, histamine, phenylethylamine, putrescine, spermine, spermidine, tyra
84  apparent affinities for a preferred ligand, phenylethylamine, rivaling those seen with mammalian phe
85 alyzed, one-pot synthesis of 1,2,3-triazolyl phenylethylamine scaffolds is reported.
86 n to afford 1,4-disubstituted 1,2,3-triazole phenylethylamine scaffolds.
87  amine dehydrogenase-catalyzed reaction with phenylethylamine shows complex pressure and temperature
88                          AMDA does contain a phenylethylamine skeleton within a tricyclic ring system
89     Putrescine, cadaverine, tryptamine, beta-phenylethylamine spermidine, spermine were analysed by U
90 d to a significant decrease in tryptamine, 2-phenylethylamine, spermidine, and spermine formation.
91 xtent, also by degradation of phenylalanine, phenylethylamine, styrene, and creatinine.
92 of the achiral parent ligand with a chiral 1-phenylethylamine substituent on the open face of the com
93 to perpendicular to the aromatic ring of the phenylethylamine substrate or cyclohexadiene ring of CHD
94                                      Certain phenylethylamines, such as 1-(4-bromo-2,5-dimethoxypheny
95  which exhibits a higher preference for beta-phenylethylamine than for the MAO A preferred substrate
96  roles in the neurotoxicity produced by this phenylethylamine, the primary source of DA responsible f
97                     This suggests that for 2-phenylethylamine, the rate-limiting step in the single-t
98 s was prepared from N-(o-nitrophenylsulfenyl)phenylethylamines through BINOL-phosphoric acid [(R)-TRI
99 tivity (from 0.02mgkg(-1) for spermidine and phenylethylamine to 0.2mgkg(-1) for spermine) when compa
100 imethyl N-Boc-phosphoramidate derived from 1-phenylethylamine to the N-Boc alpha-aminophosphonate pre
101 , in enclosed laboratory cultures, growth on phenylethylamine triggered induction of the OxyR H2O2 st
102 28 psychoactive compounds that belong to the phenylethylamine, tryptamine, and cannabinoid chemical c
103                               Tyramine, beta-phenylethylamine, tryptamine, and octopamine are biogeni
104 of six biogenic amines (histamine, tyramine, phenylethylamine, tryptamine, putrescine, and cadaverine
105 cted during substrate (either benzylamine or phenylethylamine) turnover, by anaerobic incubation with
106                                              Phenylethylamine, tyramine, and dopamine are oxidized by
107                       Neither tryptamine nor phenylethylamine was detected in the monitored samples;
108 model compound, first (S)- and/or (RS)-alpha-phenylethylamine was used, and their diastereomeric salt
109 ramine, putrescine, cadaverine, agmatine and phenylethylamine were higher in chocolate with <= 20 % U
110                               Tryptamine and phenylethylamine were not detected in pressure-treated s
111                              Methylamine and phenylethylamine were not determined in any vinegar.
112 permine, histamine, tyramine, tryptamine and phenylethylamine) were determined.
113 es, tyramine, histamine, tryptamine and beta-phenylethylamine, were detected in practically all ferme
114 ion of histamine, tyramine, tryptamine and 2-phenylethylamine, which eliminates the purification step
115               In contrast, the reaction of 2-phenylethylamine with QHNDH does not exhibit a significa

 
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