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1  and stereoselectivity of various classes of phosphoramides.
2 rmed with chiral (R)-BINOL-derived N-triflyl phosphoramides.
3  the presence of catalytic amounts of chiral phosphoramides.
4 , direct multiple a-C(sp(3))-H alkylation of phosphoramides and thiophosphoramides is demonstrated un
5 rkably, di- and tri-C(sp(3))-H alkylation of phosphoramides and thiophosphoramides using an acridiniu
6 ncluding sulfonamides, amides, sulfinamides, phosphoramides, and carbamates.
7 subjected to aldolization in the presence of phosphoramides, and the intrinsic selectivity of these e
8  N-triflylphosphoramide produces hydrazonium-phosphoramide anion complexes.
9                                   All of the phosphoramides are saturated with silicon tetrachloride
10                                        A new phosphoramide based on a 2,2'-bispyrrolidine skeleton ha
11 anistic pathways involving either one or two phosphoramides bound to a siliconium ion organizational
12 nistic studies reveal that monoalkylation of phosphoramides by Eosin-Y follows the HAT mechanism, whe
13 ired by battery technology [tris(pyrrolidino)phosphoramide] can allow for multigram-scale synthesis o
14 o hydrolase activity toward phosphodiesters, phosphoramides, carboxyl esters, or sulfoesters.
15                                       Chiral phosphoramide catalyzed-enantioselective aldol addition
16                            In all cases, the phosphoramide-catalyzed aldol addition of E-trichlorosil
17                                              Phosphoramide-catalyzed aldol additions lacked substrate
18                         The mechanism of the phosphoramide-catalyzed enantioselective aldol additions
19           Herein, we report chiral N-triflyl phosphoramide-catalyzed enantioselective pinacol rearran
20 d to DCP vapor, the conversion of FLA into a phosphoramide causes a rapid and intense fluorescence in
21 catalyst for mono-a-C(sp(3))-H alkylation of phosphoramide derivatives.
22 duct library, into a biodegradable, cationic phosphoramide-derived LNP formulation.
23         A thorough examination of a range of phosphoramides has established empirical structure-activ
24 the reaction was found to likely involve two phosphoramides in both the rate and stereochemistry dete
25 bination of silicon tetrachloride and chiral phosphoramides is a competent catalyst for highly select
26 sight that more than one Lewis basic moiety (phosphoramide) is involved in the rate- and stereochemis
27  by binding of a strongly Lewis basic chiral phosphoramide, leading to in situ formation of a chiral
28   The reaction is catalyzed by a BINAM-based phosphoramide Lewis base catalyst which assists in the h
29 bination of silicon tetrachloride and chiral phosphoramide Lewis bases.
30                             Different chiral phosphoramide moieties were connected by tethers of meth
31        The more active acyclic 4-nitrobenzyl phosphoramide mustard (7) showed 167 500x selective cyto
32       4-HC generates two active metabolites, phosphoramide mustard (PM) and acrolein.
33 iques were used to study the partitioning of phosphoramide mustard (PM) and its aziridinium ions amon
34 amide (4-HDC), a progenitor of acrolein, and phosphoramide mustard (PM).
35 ed to provide a pathway for expulsion of the phosphoramide mustard alkylating agent.
36                 Cyclic and acyclic nitroaryl phosphoramide mustard analogues were activated by E. col
37 t is metabolized by cytochrome P450 to yield phosphoramide mustard and acrolein, which alkylate DNA a
38        CPA undergoes metabolic activation to phosphoramide mustard and nornitrogen mustard (NOR) whic
39 y bound intermediates of mechlorethamine and phosphoramide mustard assumed thermodynamically stable c
40 ts in the highest quartile of o-carboxyethyl-phosphoramide mustard exposure had a 5.9-fold higher ris
41 ontrol compounds 5 and 15, which lack in the phosphoramide mustard group.
42                          Mechlorethamine and phosphoramide mustard induced intrastrand crosslinks bet
43 ter than 1) and/or excellent cytotoxicity of phosphoramide mustard released.
44 diamidates have been designed as prodrugs of phosphoramide mustard requiring bioreductive activation.
45 eduction by DTD followed by expulsion of the phosphoramide mustard substituent from the hydroquinone.
46 her crosslinking agents including cisplatin, phosphoramide mustard, and 1,2,3,4-diepoxybutane.
47 amide and 4-hydroperoxyifosfamide but not to phosphoramide mustard, ifosfamide mustard, melphalan, or
48 ere cross-resistant to other OAPs but not to phosphoramide mustard, ifosfamide mustard, melphalan, or
49 rticularly for the metabolite o-carboxyethyl-phosphoramide mustard, whose area under the curve varied
50 base-catalyzed beta-elimination of cytotoxic phosphoramide mustard.
51 ntracellular concentrations of the cytotoxic phosphoramide mustard.
52 resulted in rapid expulsion of the cytotoxic phosphoramide mustard.
53 s treated with melphalan, mechlorethamine or phosphoramide mustard.
54 icancer prodrugs that liberate the cytotoxic phosphoramide mustards (PM, IPM, and tetrakis-PM) via be
55 unds were more potent than the corresponding phosphoramide mustards against V-79 Chinese hamster lung
56 t all these compounds spontaneously liberate phosphoramide mustards with half-lives in the range of 0
57                   The effects of Lewis basic phosphoramides on the aggregate structure of t-BuLi have
58 sphorylates nucleophiles with elimination of phosphoramide OP(NC(4)H(8))(3).
59 +2] photocycloaddition catalyzed by a chiral phosphoramide organocatalyst.
60 ulted in identification of the corresponding phosphoramide prodrug (29) with an improved solubility a
61  of SiCl4 and the catalytic action of chiral phosphoramide (R,R)-5, silyl ketene imines undergo extre
62 o date with the silicon tetrachloride-chiral phosphoramide system.
63 rosilanes to benzaldehyde promoted by chiral phosphoramides to give the enantioenriched homoallylic a
64 lyltrichlorosilane promoted by the bidentate phosphoramides was found to be highly dependent on the t
65 nantioselective allylation, bidentate chiral phosphoramides were developed.
66 , biotin-HDAAMP (adenosine 5'-(6-aminohexyl) phosphoramide; where HDA is 1,6-hexanediamine), is chemi