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1 l cortex during times of high energy demand (photic stimulation).
2 K1 were colocalized in SCN cell nuclei after photic stimulation.
3 f postsynaptic responsiveness to 5-HT and by photic stimulation.
4 ors and bipolar cells, both in dark and with photic stimulation.
5 oretinographically determined sensitivity to photic stimulation, and the rhodopsin levels in the reti
6 ulate leaflet cells that express c-Fos after photic stimulation appear to represent a functionally-de
7 ssion tomography to measure perfusion during photic stimulation at four different rates.
8                 These findings indicate that photic stimulation can lead to a suppression or down-reg
9                At phases near the PER2 peak, photic stimulation causes little phase shift but enhance
10          Since the intensity and duration of photic stimulation determine the magnitude of these cone
11                      In contrast with CRTC1, photic stimulation did not affect the subcellular locali
12 tained images of the mouse brain response to photic stimulation during a period between deep anesthes
13                Recent work has revealed that photic stimulation during the night triggers rapid activ
14                            Here we show that photic stimulation during the subjective night stimulate
15                                 Furthermore, photic stimulation during the subjective night, but not
16 imary visual cortex in response to binocular photic stimulation in 16 healthy, young subjects (eight
17 e the regulation of REM sleep in response to photic stimulation in albino rats.
18 evoked by striatal forskolin infusion and by photic stimulation in HD animals.
19 re responsive to late-night than early-night photic stimulation, indicating that MSK1 may differentia
20  is an abnormal EEG reaction to intermittent photic stimulation (IPS), consisting of spikes, spike-wa
21 g the vascular response in one retina to the photic stimulation of the contralateral eye in health an
22 nal neuroimaging, which included fMRI during photic stimulation through closed eyelids, to measure fu
23                            In the absence of photic stimulation, transient disruption of MAPK signali
24                       Phase delays following photic stimulation were reduced in the old mice at all l