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1 abidopsis thaliana) that strongly influences photomorphogenesis.
2 ies and the degradation of PIFs to establish photomorphogenesis.
3 f phyB, events which are required for proper photomorphogenesis.
4 odulates multiple hormone pathways to affect photomorphogenesis.
5 eby facilitate functional equilibrium during photomorphogenesis.
6 numerous developmental processes, including photomorphogenesis.
7 as a negative regulator of various facets of photomorphogenesis.
8 ggesting that the mutant PIF1 is suppressing photomorphogenesis.
9 light-induced degradation of PIF1 to promote photomorphogenesis.
10 (HY5), a master transcriptional regulator of photomorphogenesis.
11 orylation and degradation of PIFs to promote photomorphogenesis.
12 : HY5 and HFR1, which play critical roles in photomorphogenesis.
13 red and far-red light conditions to promote photomorphogenesis.
14 h the ubi/26S proteasomal pathway to promote photomorphogenesis.
15 ive activity upon light exposure, initiating photomorphogenesis.
16 ontrol for light-regulated genes involved in photomorphogenesis.
17 light regulation of the circadian clock and photomorphogenesis.
18 tes peroxisome proliferation during seedling photomorphogenesis.
19 rphogenic state and regulating the switch to photomorphogenesis.
20 c degradation might be sufficient to promote photomorphogenesis.
21 relieves this negative regulation to promote photomorphogenesis.
22 egulator of the transcriptional cascades for photomorphogenesis.
23 anisms that use light for photosynthesis and photomorphogenesis.
24 ts role in splicing of pre-mRNAs involved in photomorphogenesis.
25 teasomes in the circadian clock and in early photomorphogenesis.
26 families of the photoreceptors and promotes photomorphogenesis.
27 se two domains are sufficient for repressing photomorphogenesis.
28 ansduced through several pathways to promote photomorphogenesis.
29 essing 35S-HFR1(DeltaN) display constitutive photomorphogenesis.
30 jasmonate signaling, flower development, and photomorphogenesis.
31 nd other phy-interacting factors to optimize photomorphogenesis.
32 these two genes act redundantly to modulate photomorphogenesis.
33 ed for post-translational degradation during photomorphogenesis.
34 the BAS1 gene, inactivates BRs and modulates photomorphogenesis.
35 signalosome (CSN) in Arabidopsis to repress photomorphogenesis.
36 ubiquitylation-promoting factor to regulate photomorphogenesis.
37 r ZTL in the control of circadian period and photomorphogenesis.
38 master regulator of transcription to promote photomorphogenesis.
39 d how phyB modulates this process to promote photomorphogenesis.
40 tinct and overlapping roles throughout plant photomorphogenesis.
41 ted in brassinosteroid catabolism as well as photomorphogenesis.
42 tion of key cellular regulators that promote photomorphogenesis.
43 ins first identified as a repressor of plant photomorphogenesis.
44 tor, CRY1, and COP1, a negative regulator of photomorphogenesis.
45 mplex initially identified as a repressor of photomorphogenesis.
46 quire phytochrome A for function in seedling photomorphogenesis.
47 is a bZIP transcription factor that promotes photomorphogenesis.
48 xyl terminal module conferring repression of photomorphogenesis.
49 pment, consistent with its role in promoting photomorphogenesis.
50 ling and circadian clock pathways to promote photomorphogenesis.
51 tion factors that are required for promoting photomorphogenesis.
52 opmental modulator involved in repression of photomorphogenesis.
53 er phyA or phyB is required for UV-B-induced photomorphogenesis.
54 anscription factor that positively regulates photomorphogenesis.
55 family, and they may interact in regulating photomorphogenesis.
56 velopment and that it acts as a repressor of photomorphogenesis.
57 signaling, acting as a central repressor of photomorphogenesis.
58 tunes its stability and activity to regulate photomorphogenesis.
59 lation of mRNAs encoding proteins needed for photomorphogenesis.
60 nphosphorylated form of HY5 display enhanced photomorphogenesis.
61 is a key transcription factor that promotes photomorphogenesis.
62 al and posttranscriptional levels to promote photomorphogenesis.
63 version to Pfr and is hyperactive in driving photomorphogenesis.
64 biological activity of PIF3, thus promoting photomorphogenesis.
65 are degraded in response to light to promote photomorphogenesis.
66 others) are degraded in the dark to prevent photomorphogenesis.
67 nts and enhanced far-red light/phyA-mediated photomorphogenesis.
68 t-induced transcriptional network central to photomorphogenesis.
69 s, antagonistically regulate PIFs to promote photomorphogenesis.
70 induce rapid degradation of PIFs to promote photomorphogenesis.
71 in chromatin and nuclear architecture during photomorphogenesis.
72 regulators of Arabidopsis thaliana seedling photomorphogenesis.
73 y for the subsequent effective completion of photomorphogenesis.
74 y interact in an additive manner to suppress photomorphogenesis.
75 ters expression patterns of genes underlying photomorphogenesis.
76 protein that acts as a negative regulator of photomorphogenesis.
77 ediated connection between BR catabolism and photomorphogenesis.
78 ng an adaptive advantage until emergence and photomorphogenesis.
79 riptional changes that drive a transition to photomorphogenesis.
80 nt and provide a revised mechanistic view of photomorphogenesis.
81 nt photosynthesis-associated proteins during photomorphogenesis.
82 There are two stages in photomorphogenesis.
83 at BBX25 is a negative regulator of seedling photomorphogenesis.
84 berellin (GA) hormones across both stages of photomorphogenesis.
85 ctor, central for the regulation of seedling photomorphogenesis.
86 HFR1 physically interacts with Constitutive Photomorphogenesis 1 (COP1) and that COP1 exhibits ubiqu
87 ade-avoidance responses require CONSTITUTIVE PHOTOMORPHOGENESIS 1 (COP1) but the mechanisms of action
88 cells by using the Arabidopsis Constitutive photomorphogenesis 1 (COP1) protein as a model system.
92 , we have identified COP1, REPRESSOR OF UV-B PHOTOMORPHOGENESIS 1 and 2 (RUP1 and RUP2), and the SUPP
97 explored the role of a negative regulator of photomorphogenesis, a B-box-containing protein (BBX19),
99 ivation of energy signaling, both targets of photomorphogenesis action, but the organ developmental o
100 ted to CSN subunits--including repression of photomorphogenesis, activation of JUN, and activation of
101 s in the nucleus are induced by light during photomorphogenesis, allowing plants to establish photoau
106 including deetiolated2 (det2), constitutive photomorphogenesis and dwarfism (cpd), brassinosteroid i
109 plays a critical role in promoting seedling photomorphogenesis and in balancing the shade-avoidance
110 important environmental signal that induces photomorphogenesis and interacts with endogenous signals
111 or of phyA-105"), functions in repression of photomorphogenesis and is required for normal photosenso
112 regulate Arabidopsis (Arabidopsis thaliana) photomorphogenesis and multiple aspects of root developm
114 fined genetically as a positive regulator of photomorphogenesis and recently has been shown to encode
117 plant phytochromes are master regulators of photomorphogenesis and the shade avoidance response.
118 cription factor HY5, a positive regulator of photomorphogenesis, and promotes its proteasome-mediated
119 eals an additional way in which HY5 promotes photomorphogenesis, and provides an insight into the reg
120 cumulated in darkness and repressed seedling photomorphogenesis, and that PIFs linked different photo
121 nuclear protein DET1, involved in repressing photomorphogenesis, and the cry1 gene for the blue light
122 ry for rapid light-induced expression of the photomorphogenesis- and circadian-related PSEUDO-RESPONS
124 lorophyll production, thylakoid stacking and photomorphogenesis are also restored in PORA-overexpress
127 plant photoreceptors with critical roles in photomorphogenesis are phytochrome B (phyB), a red/far-r
128 osure to light, developing seedlings undergo photomorphogenesis, as illustrated by inhibition of hypo
129 lorate-resistant mutant cr88 is defective in photomorphogenesis, as shown by the phenotypes of long h
130 expressing UVR8(W285A) exhibit constitutive photomorphogenesis associated with constitutive activati
131 OP9 signalosome (CSN), a suppressor of plant photomorphogenesis, associated with multiple cullins and
132 Here we show that PIF1 negatively regulates photomorphogenesis at the seedling stage under blue ligh
133 AM7 acts synergistically with HY5 to promote photomorphogenesis at various wavelengths of light.
135 anscription factors that negatively regulate photomorphogenesis both in the dark and in the light in
138 hat it also plays a role in phy assembly and photomorphogenesis but the ho2 mutant phenotype is more
141 hese data suggest a combinatorial control of photomorphogenesis by bHLH proteins in response to light
142 ion suggests that phyA may regulate seedling photomorphogenesis by direct targeting of light signals
143 s (HECs), function as positive regulators of photomorphogenesis by directly interacting and antagoniz
144 n darkness, COP1 functions as a repressor of photomorphogenesis by promoting the ubiquitin-mediated p
145 ining DnaJ-like domain, plays a dual-role in photomorphogenesis by stabilizing the BR receptor, BRI1,
146 cr88 and mutants of two other loci affecting photomorphogenesis, CONSTITUTIVE PHOTOMORPHOGENIC1 (COP1
148 xpression results in plants with exaggerated photomorphogenesis, dark green leaves, and elevated frui
149 tenoid acted in parallel to the repressor of photomorphogenesis, DEETIOLATED1 (DET1), to transcriptio
151 d in terms of an organ-autonomous feature of photomorphogenesis directed by the red/blue light absorb
152 suggested that COP1, a negative regulator of photomorphogenesis, directly interacts with nuclear HY5
153 l tested loci, chromatin condensation during photomorphogenesis does not detectably rely on DNA methy
154 n plays a critical role in the repression of photomorphogenesis during Arabidopsis seedling developme
155 radation of PIF1 prevents over-activation of photomorphogenesis during early seedling development.
157 h of the encoded proteins also have roles in photomorphogenesis, especially in the absence of HY1.
158 deposition and allowed the plants to undergo photomorphogenesis even when they were grown in the dark
160 ng development research, particularly in the photomorphogenesis field, by replacing many tedious, err
162 , which targets HY5, a positive regulator of photomorphogenesis, for degradation via the proteasome p
163 e survival by regulating shade avoidance and photomorphogenesis genes to outgrow submergence and by p
172 gether, our results show that DET1 represses photomorphogenesis in darkness in part by reducing the a
173 Arabidopsis COP1 acts as a repressor of photomorphogenesis in darkness, and light stimuli abroga
174 COP1 acts within the nucleus to repress photomorphogenesis in darkness, while light depletes COP
179 te the circadian rhythm, flowering time, and photomorphogenesis in higher plants as responses to blue
180 activity is necessary for normal skoto- and photomorphogenesis in juvenile seedlings as well as long
182 This allowed us to examine UV-B-induced photomorphogenesis in photoreceptor deficient plants and
183 biological processes, such as repression of photomorphogenesis in plants and protein subcellular loc
184 romes are blue light receptors that regulate photomorphogenesis in plants and the circadian clock in
191 rabidopsis thaliana seedlings that initiates photomorphogenesis in response to a FR-enriched environm
197 transcriptional networks are active early in photomorphogenesis in the aerial parts of dicotyledon se
198 tein complex that mediates the repression of photomorphogenesis in the dark in Arabidopsis through th
199 , and PIF5 act as constitutive repressors of photomorphogenesis in the dark, action that is rapidly a
200 , 35S::MIF1 seedlings underwent constitutive photomorphogenesis in the dark, with root growth similar
204 tterns according to their light environment: photomorphogenesis in the light and etiolation or skotom
205 terns depending on ambient light conditions, photomorphogenesis in the light and skotomorphogenesis o
207 tomorphogenesis) to light-grown development (photomorphogenesis) in part by rapid modulation of brass
208 me interacting factors (PIFs), repressors of photomorphogenesis, in response to environmental light s
209 as a set of negative regulators of seedling photomorphogenesis, including DET1, that appear to act d
210 lso altered in several aspects of vegetative photomorphogenesis, including hypocotyl elongation.
211 raction studies suggest that BBX31 regulates photomorphogenesis independent of HY5 We found no eviden
212 motif of CRY2 abolished the CRY2 activity in photomorphogenesis, indicating the importance of VP.
213 light conditions, suggesting that cotyledon photomorphogenesis involves a transition from globally q
222 identified in Arabidopsis as a repressor of photomorphogenesis, is composed of multiple subunits inc
223 PHYA (SPA), one of the central repressors of photomorphogenesis, is critical for maintaining skotomor
227 s exhibit chromophore-dependent constitutive photomorphogenesis, light-independent phyB(Y276H) nuclea
229 that in the processes of endoreplication and photomorphogenesis, LIP1 acts downstream of the red and
230 n to suppressing red and blue light-mediated photomorphogenesis, LIP1 is also required for light-cont
231 ight responses, including inhibited seedling photomorphogenesis, loss of thylakoid organization, and
233 ound that elongated, a previously identified photomorphogenesis mutant, enhances high-light phototrop
235 nd inhibition of hypocotyl elongation during photomorphogenesis of Arabidopsis thaliana seedlings.
237 n of Arabidopsis cryptochrome 2 in the early photomorphogenesis of seedlings was studied by using tra
239 osition CR88 in the genetic hierarchy of the photomorphogenesis pathway, we determined that CR88 acts
241 1 restores the seed germination and seedling photomorphogenesis phenotypes of max2 but does not affec
242 processes in plants, such as photosynthesis, photomorphogenesis, photoprotection, and development.
244 nction redundantly as negative regulators of photomorphogenesis, possibly by influencing the turnover
245 y) as light-dependent negative regulators of photomorphogenesis, possibly in a downstream signaling o
246 matic light suggested that CYP72B1 modulates photomorphogenesis primarily through far-red light and t
248 idence supporting that HFR1, which encodes a photomorphogenesis-promoting bHLH transcription factor,
250 or of photomorphogenesis that interacts with photomorphogenesis-promoting factors such as HY5 to prom
251 ible for targeted degradation of a number of photomorphogenesis-promoting factors, including HY5, LAF
252 ion through targeted degradation of multiple photomorphogenesis-promoting transcription factors in th
253 inds and targets for degradation a number of photomorphogenesis-promoting transcription factors, incl
254 ger E3 ubiquitin-protein ligase constitutive photomorphogenesis protein 1 (COP1) for degradation via
256 nteracts in yeast with the REPRESSOR OF UV-B PHOTOMORPHOGENESIS proteins, RUP1 and RUP2, which are ne
257 specific expression profiles during seedling photomorphogenesis provide genome-level evidence for div
260 accumulation of genes encoding phytochromes, photomorphogenesis-repressor factors, and plastid divisi
261 eaves, reduced apical dominance, and altered photomorphogenesis, resembling brassinosteroid-deficient
262 arget transcription factors, which initiates photomorphogenesis, resulting in dramatic changes of the
263 ptochrome alleles that are non-functional in photomorphogenesis retain the capacity to induce ROS-res
264 UVR8-interacting proteins repressor of UV-B photomorphogenesis (RUP)1 and RUP2 mediate UVR8 redimeri
265 ight receptors mediate many aspects of plant photomorphogenesis, such as suppression of hypocotyl elo
266 es the degradation of positive regulators of photomorphogenesis, such as the transcription factor HY5
267 id fragment of HFR1 (CT161) display enhanced photomorphogenesis, suggesting an autonomous function of
268 st development, revealing a key regulator of photomorphogenesis that acts across cell compartments.
270 bidopsis COP1 is a constitutive repressor of photomorphogenesis that interacts with photomorphogenesi
272 ning protein that functions to repress plant photomorphogenesis, the light-mediated programme of plan
273 nscription factors that act as repressors of photomorphogenesis; their inhibition by PHYs leads to su
274 which encodes a bHLH protein that regulates photomorphogenesis through modulating phytochrome and cr
275 photoreceptors are major regulators of plant photomorphogenesis through their unique ability to photo
277 ese results suggest that phyC is involved in photomorphogenesis throughout the life cycle of the plan
281 at PIF1 functions as a negative regulator of photomorphogenesis under blue light conditions and that
282 at PIF3 plays a prominent role in repressing photomorphogenesis under continuous blue light condition
283 switch for the seedling developmental fates: photomorphogenesis under light conditions and skotomorph
285 In a screen for mutants showing altered photomorphogenesis under red light, we identified a muta
287 cate that it acts as a negative regulator of photomorphogenesis under white light but is a positive r
289 icing (AS) of etiolated seedlings undergoing photomorphogenesis upon exposure to blue, red, or white
290 nt of the mechanism by which light initiates photomorphogenesis upon first exposure of dark-grown see
291 y steps of the transition between skoto- and photomorphogenesis upon light exposure and to complete t
292 icating that SUMOylation of PIF3 also alters photomorphogenesis via the regulation of phyB levels.
294 analyze further the role that CR88 plays in photomorphogenesis, we investigated the genetic interact
295 ytochrome requirement for certain aspects of photomorphogenesis, we tested whether SHY2/IAA3 and rela
296 latory context in which DET1 acts to repress photomorphogenesis, we used a simple morphological scree
297 darkness into the light environment undergo photomorphogenesis, which enables autotrophic growth wit
298 ling in the control of cell expansion during photomorphogenesis, which is supported by physiological
299 Arabidopsis COP1 is a negative regulator of photomorphogenesis, which targets HY5, a positive regula
300 umulation of transcription factors promoting photomorphogenesis; yet, the mechanism by which they ina