ライフサイエンスコーパス: pistil

1 xpanding leaves (about 3.5-fold less than in pistils).

2 ing specific interactions between pollen and pistil.

3 rabidopsis (Arabidopsis thaliana) pollen and pistil.

4 an effective pollen tube guidance within the pistil.

5 ion involves interactions between pollen and pistil.

6 ecifically, in the transmitting tract of the pistil.

7 oral tissues to access the ovules within the pistil.

8 male gametophyte) that is encased within the pistil.

9 xpressing pollen tubes elongating within the pistil.

10 hat express specificities in common with the pistil.

11 tches either S-allele present in the diploid pistil.

12 re carried in the pollen grain to the female pistil.

13 recise communications between the pollen and pistil.

14 e-specific rejection of "self" pollen by the pistil.

15 is induced in pollen tubes by growth in the pistil.

16 pollinations with Col and RIL pollen on Col pistils.

17 promoters from genes expressed primarily in pistils.

18 and anthers, TsYUC6 in anthers and TsBAHD in pistils.

19 1, GACO2, GACO3) were isolated from geranium pistils.

20 was similarly impaired in both are and VF36 pistils.

21 development, resulting in the persistence of pistils.

22 tically marked Col-0 and RIL pollen on Van-0 pistils.

23 marked Col-0 pollen and Van-0 pollen on RIL pistils.

24 with Col and Landsberg erecta pollen on RIL pistils.

25 stils but unilaterally compatible (UC) on SC pistils(1,4-6).

26 loret in each spikelet on the ear includes a pistil abortion process that requires the action of the

27 M1 gene appears to perform a similar role in pistil abortion.

28 Binding between NaPCCP and NaSBP1 and the pistil AGPs may contribute to signaling and trafficking

29 ollen tube gene products that respond to the pistil and are required for reproductive success; moreov

30 arch across disciplines, for example, pollen-pistil and graft incompatibility interactions and plant

31 communication between female tissues of the pistil and paternal pollen tubes imposes hybridization b

32 re tightly regulated and expressed solely in pistil and pollen cells, respectively.

33 In Papaver rhoeas, cognate pistil and pollen S-determinants, PrpS, a pollen-express

34 nflata S-locus F-box (Pi SLF), determine the pistil and pollen specificity, respectively.

35 ering plants, pollen grains germinate on the pistil and send pollen tubes down the transmitting tract

36 tion, corolla tube structure, nectar volume, pistil and stamen length) remains poorly understood.

37 S-RNase is produced in the pistil and taken up by pollen tubes in a non-S-haplotype

38 otein that is expressed predominantly in the pistils and anthers of Brassica flowers late in flower d

39 moderate levels in leaves, pedicels, sepals, pistils and petals, and at very low levels in roots.

40 Message levels of PhETR1 and PhETR2 in pistils and receptacles are unaffected by self-pollinati

41 n, and nitrogen allocation to female whorls (pistils and sepals) decreased under high density, wherea

42 in floral organ size including elongation of pistils and shortened stamen filaments that resulted in

43 processes (e.g., pollen tube penetration of pistils) and disease progression (e.g., cancer metastasi

44 ated hypocotyls (about 2.5-fold less than in pistils) and in young expanding leaves (about 3.5-fold l

45 ally similar tissues such as leaves, anther, pistil, and embryo, while orthologs that are highly expr

46 carboxylase2 (ODC2) and HT-A/-B genes in the pistil, and farnesyl pyrophosphate synthase2 (FPS2), ui6

47 owth, similar to what normally occurs in the pistil, and this ability correlates with the accumulatio

48 ot density, delayed sepal opening, elongated pistils, and reduced fertility in the primary infloresce

49 TTS) and 120-kDa glycoprotein (120K) are two pistil arabinogalactan proteins (AGPs) that share a cons

50 uidance on extracellular matrices within the pistil are essential processes that convey the pollen tu

51 sms of guidance for pollen tubes through the pistil are not known, the female tissues play a critical

52 len tubes are reduced when NaStEP-suppressed pistils are pollinated with either compatible or incompa

53 y regulated to ensure that anthers reach the pistil at the correct developmental stage.

54 ecific antibody first detects the protein in pistils at one day prior to flowering, with higher level

55 nd weakened (or absent) interspecific pollen-pistil barriers.

56 control the enhanced style elongation of pro pistils, because its expression was not higher in pro st

57 llen is unilaterally incompatible (UI) on SI pistils but unilaterally compatible (UC) on SC pistils(1

58 minish pollen tube length in vitro or in the pistil, but it reduces ovule targeting by twofold.

59 LeSTIG1, a small cysteine-rich protein from pistil, can bind the extracellular domains of both LePRK

60 The pistil candidate gene, TsBAHD, differs from that of Prim

61 17; Chi2;1) identified by screening a tomato pistil cDNA library has been found to encode a protein s

62 results suggest that cytokinin can determine pistil cell fate during maize floret development.

63 ncluding deterioration and death in specific pistil cell types.

64 Ill-fated pistil cells undergo a cell death process associated wit

65 d1 and tasselseed2 are required for death of pistil cells.

66 for the accumulation of TASSELSEED2 mRNA in pistil cells.

67 The establishment of pollen-pistil compatibility is strictly regulated by factors de

68 developmental timing of variation in pollen-pistil compatibility.

69 f non-functional S-haplotypes with disrupted pistil component (stylar-S) and/or pollen component (pol

70 t, nonfunctional S-haplotypes with disrupted pistil component (stylar-S) and/or pollen component (pol

71 ncompatibility, employs ribonucleases as the pistil component.

72 e with S3-, S5-, S7-, S11-, and S13-carrying pistils, confirming that other SLF proteins are responsi

73 In addition, cbl10 mutant pistils could not sustain the growth of wild-type pollen

74 naling between the male (pollen) and female (pistil) counterparts where the interplay between several

75 ollen tube growth assays in vitro and in the pistil demonstrate that hydroxyl free radicals are likel

76 Aniline blue staining of pollinated pistils demonstrated that pollen tube growth was affecte

77 S-locus, which contains S-RNase encoding the pistil determinant and 16-20 S-locus F-box (SLF) genes c

78 y interactions: the S-RNase gene encodes the pistil determinant and the previously unidentified S-gen

79 The S-RNase gene encodes the pistil determinant, whereas the pollen determinant gene,

80 tains two separate genes encoding pollen and pistil determinants in SI interactions.

81 t positive regulators or effectors of SI and pistil development are regulated by ta-siRNA(s).

82 We show that ARF3, a regulator of pistil development that is expressed in the vascular tis

83 stigma and ovary during the early stages of pistil development that marks style differentiation.

84 nts show increased sterility due to abnormal pistil development with about half of the plants develop

85 CO1, and leafy, genes regulating anther and pistil development, and stress-related transcription fac

86 ed expression during certain stages of early pistil development, Cel4 mRNA was also detected at high

87 y pathway and the dual role of SRK in SI and pistil development, our study provides a molecular expla

88 hat LFR is important for proper filament and pistil development, resembling the function of SYD.

89 for PCP in pollen-pistil interactions or in pistil development.

90 D9-1 mutants are unable to block GA-induced pistil development.

91 iator of cross-talk between SI signaling and pistil development.

92 fic genes that respectively suppress female (pistil) development and are necessary for male (anther)

93 hich are normally expressed very late in the pistil developmental pathway and function in the final s

94 efore, it could function in the transport of pistil ECM proteins in the pollen tube endomembrane syst

95 receptor kinase (SRK) gene further enhances pistil elongation and stigma exsertion in this mutant ba

96 are essential for pollen tube elongation in pistil, especially, free Ca(2+) providing a concentratio

97 ection response, the identification of three pistil essential modifier genes unlinked to the S-locus

98 ucuronidase staining occurred throughout the pistil, except in the stigma.

99 ted predominantly in the stigma and style of pistils excised from open flowers; much lower levels of

100 ic expression of S-linked genes, including a pistil-expressed candidate gene for style length, are ma

101 expressed transmembrane protein, and PrsS, a pistil-expressed secreted protein, interact to trigger a

102 We characterized mating system and pistil-expressed SI factors in 20 populations of the wil

103 nte crossing barrier1-s haplotype contains a pistil-expressed, potential speciation gene, encoding a

104 TS) protein, 120 kDa glycoprotein (120K) and pistil extensin-like protein III (PELP III) are stylar g

105 ovary, they are in constant contact with the pistil extracellular matrix (ECM).

106 interaction between pollen proteins and the pistil extracellular matrix.

107 We isolated an Arabidopsis pistil extract fraction that stimulates Arabidopsis poll

108 ompound that is specifically present in this pistil extract fraction.

109 ect of pistils on pollen germination and the pistil factors that stimulate pollen germination remain

110 A model is proposed for the control of pistil fate by the action of the ts1-ts2-sk1 pathway.

111 ncoding highly polymorphic pollen (male) and pistil (female) S-determinants that control whether self

112 s firmness, enzymatic activity was higher in pistil followed by equatorial and peduncle zones.

113 ordia express TASSELSEED2 RNA but functional pistils found in ear spikelets are protected from cell d

114 highest levels of transcript accumulation in pistils from flowers at anthesis.

115 mutations in pollen-S that reduce the set of pistils from which the pollen accepts inhibition and dis

116 wing pollination, the upper and lower floret pistils fuse, producing a connated kernel with two genet

117 Most pro pistils had exserted stigmas, thus preventing self-polli

118 o be correlated with rapid cell expansion in pistils, hypocotyls and leaves.

119 The pistil in each floret was fertile, but the spikelet prod

120 m pollen tubes to penetrate farther into the pistil in HT suppressed plants, but not to reach the ova

121 PRKs) control pollen tube growth through the pistil in response to extracellular signals, and regulat

122 stamen in ear spikelets and the abortion of pistils in both the tassel spikelets and in the secondar

123 in plantacyanin levels in the overexpression pistil, including the transmitting tract.

124 y involves separate genes for the pollen and pistil incompatibility recognition processes.

125 center of spikelets replacing the stamen and pistil, indicating that they synergistically determine s

126 -carboxylic acid (ACC), to the flower or the pistil induced overall deterioration in the entire flowe

127 Pollen and pistil interact to kill invading pollen from an incompat

128 specific gene ontology classes (e.g., pollen-pistil interaction) in apomicts implies that gene enrich

129 oncurrent developmental timing of the pollen-pistil interaction, suggests these incompatibilities hav

130 nsduction' pathway is enhanced during pollen-pistil interaction.

131 MP) mating systems, and characterized pollen-pistil interactions among S. habrochaites populations an

132 The high degree of specificity in pollen-pistil interactions and the precision of directional pol

133 Pollen-pistil interactions are critical early events regulating

134 understanding the molecular basis of pollen-pistil interactions as reproductive isolating barriers.

135 Pollen-pistil interactions establish interspecific/intergeneric

136 Initial pollen-pistil interactions in the Brassicaceae are regulated by

137 genes in compatible and incompatible pollen-pistil interactions is discussed.

138 stent with a possible role for PCP in pollen-pistil interactions or in pistil development.

139 Pollen-pistil interactions serve as important prezygotic reprod

140 l compatibility results from multiple pollen-pistil interactions with additive effects.

141 ial signal transduction components of pollen-pistil interactions, and isolated two structurally relat

142 evelopment is dependent on successful pollen-pistil interactions.

143 and LePRK2 are involved in mediating pollen-pistil interactions.

144 ties of AGPs in ovule development and pollen-pistil interactions.

145 sses that are required for compatible pollen-pistil interactions.

146 olarized secretion, in the context of pollen-pistil interactions.

147 e further evaluated mechanisms at the pollen-pistil interface contributing to outcross failure and le

148 Pollen tube elongation in the pistil is a crucial step in the sexual reproduction of p

149 Although the pistil is a great facilitator of pollen function, it can

150 imination of self and non-self pollen by the pistil is controlled by a single polymorphic locus, the

151 ation between self and nonself pollen by the pistil is controlled by the highly polymorphic S-RNase g

152 elf recognition mechanism between pollen and pistil is controlled by two polymorphic genes at the S-l

153 elf-/non-self-recognition between pollen and pistil is regulated by the pistil-specific S-RNase gene

154 combination with either HT-A or HT-B in the pistil is sufficient to cause rejection of pollen from a

155 determinants of S-allele specificity in the pistil, it is not known how allele-specific information

156 expression patterns in subepidermal cells of pistils just before abortion.

157 brary enriched in transcripts present in the pistil late in flower development - potentially encodes

158 the pollen tube nucleus during growth in the pistil leads to pollen tube differentiation required for

159 nic lines (NILs) for two dimensional traits, pistil length and corolla size.

160 locus genes, which control anther position, pistil length and pollen size in pin and thrum flowers,

161 The NILs refined two pistil length QTLs, only one of which has tightly correl

162 long stamen length, short stamen length, and pistil length) in a cosmopolitan sample of 15 ecotypes.

163 of reproductive and immune responses of the pistil makes it a prime system in which to study the con

164 llen rejection; therefore, NaStEP is a novel pistil-modifier gene.

165 To date, two essential pistil-modifier genes, 120K and High Top-Band (HT-B), ha

166 ponents are taken up during growth, and some pistil molecules exert their effect inside the pollen tu

167 subtle pleiotropic effects on both sepal and pistil morphology.

168 The pistil of 28-5 ap2-1 mutant flowers shows a structure si

169 c self-incompatibility mechanism enables the pistil of a plant to reject self-pollen and therefore pr

170 sm between the pollen of one species and the pistil of another.

171 nsmitting tissue extracellular matrix of the pistil of tobacco.

172 s in S. pennellii LA0716 are incompatible on pistils of all tested S. pennellii and some Solanum habr

173 d in leaf and flower abscission zones and in pistils of fully open flowers.

174 y expressed in both abscission zones and the pistils of mature flowers.

175 -RNase, the determinant of SI specificity in pistils of Nicotiana alata.

176 ith higher levels of the protein seen in the pistils of open flowers.

177 Pistils of self-incompatible (SI) species/populations ty

178 hat lack S-RNase, a key protein expressed in pistils of SI Solanum species.

179 .1, are required for pollen compatibility on pistils of SI species or hybrids.

180 ction often follows the 'SI x SC' rule, i.e. pistils of SI species reject the pollen of SC (self-comp

181 Pistils of Solanum pennellii LA0716 (SC, no S-RNase) rej

182 ible (SC) red-fruited species is rejected on pistils of the predominantly self-incompatible (SI) gree

183 Pistils of the wild tomato relative Solanum lycopersicoi

184 ns during pollination between pollen and the pistil on which it lands.

185 However, in Arabidopsis, the effect of pistils on pollen germination and the pistil factors tha

186 ary analysis of mutations that affect either pistil or pollen specificity indicates that natural sele

187 on typically favored increased allocation to pistils (or stamens) but decreased allocation to other w

188 s exhibit normal growth and guidance in pop2 pistils, perhaps by degrading excess GABA and sharpening

189 ple aspects of development (e.g., height and pistil persistence in tassel florets).

190 CE1 mRNA accumulated specifically within the pistil, petals, and stamen filaments.

191 Wild-type pistils pollinated with a limited number of single and d

192 tic self-incompatibility (GSI) system in the pistil predates speciation.

193 ) has been shown to be identical to a tomato pistil-predominant EGase cDNA, TPP18.

194 All pistil primordia express TASSELSEED2 RNA but functional

195 Abortion of pistil primordia in staminate florets is controlled by a

196 Elimination of pistil primordia occurs in the primary and secondary flo

197 blocks tasselseed-induced cell death in the pistil primordia of primary ear florets.

198 Our results show that Arabidopsis pistils promote germination by producing azadecalin-like

199 rmination by restricting the function of the pistil-protecting factor, silkless1, from the apical inf

200 In self-incompatible Solanaceae, the pistil protein S-RNase contributes to S-specific pollen

201 use specific interactions between pollen and pistil proteins as "self" recognition and/or rejection m

202 The pistil provides everything the pollen requires for succe

203 ences between male pollen release and female pistil receptivity (dichogamy), and self-pollen rejectio

204 nia possesses self-incompatibility, by which pistils reject self-pollen but accept non-self-pollen fo

205 intra-specific reproductive barrier by which pistils reject self-pollen to prevent inbreeding and acc

206 Pollen-pistil reproductive barriers can act as soon as pollen i

207 ce interactions in male (pollen) and female (pistil) reproductive tissues.

208 ts and that at least LePRK2 may mediate some pistil response.

209 PrpS is a single-copy gene linked to the pistil S gene (currently called S, but referred to herea

210 patible) pollen by interaction of pollen and pistil S locus components, and is subsequently inhibited

211 In Papaver rhoeas, the pistil S locus product is a small protein that interacts

212 /S-locus Protein11 (SCR/SP11) ligand and the pistil S Receptor Kinase (SRK).

213 r rhoeas), interaction of cognate pollen and pistil S-determinants triggers programmed cell death (PC

214 multiallelic S locus, comprising pollen and pistil S-determinants.

215 ceae, the S-specific interaction between the pistil S-RNase and the pollen S-Locus F-box protein cont

216 The protein products of S alleles in the pistil, S proteins, were initially identified based on t

217 ms, roots, and cotyledons) and reproductive (pistils, sepals, petals, stamens, and floral buds) organ

218 cific reproductive barriers or loss of known pistil SI factors.

219 ecific and interpopulation compatibility and pistil SI factors.

220 For example, the pistil SI proteins S-RNase and HT protein function in a

221 roteins S-RNase and HT protein function in a pistil-side IRB that causes rejection of pollen from sel

222 Expression of pistil-side UI is weakened in S. lycopersicum x S. lycop

223 When present in the pistil (silk and ovary) a number of maize genes discrimi

224 The class III pistil-specific extensin-like protein (PELPIII) was cons

225 acting with multiple allelic variants of the pistil-specific S-ribonucleases (S-RNases).

226 the polymorphic S-locus, which contains the pistil-specific S-RNase and multiple pollen-specific S-L

227 etween pollen and pistil is regulated by the pistil-specific S-RNase gene and by multiple pollen-spec

228 s a gene encoding an S-RNase, which controls pistil specificity, and multiple S-locus F-box (SLF) gen

229 ty in Petunia inflata; the S-RNase regulates pistil specificity, and multiple S-locus F-box (SLF) gen

230 LHS1 expression in pistils, stamens, and lodicules varies among the cereals

231 Expression in the pistil steadily increases during flower development and

232 hat stigma, style, and ovules in Arabidopsis pistils stimulate pollen germination.

233 In many plants, pistils stimulate pollen germination.

234 enhance reproductive defects in lre-5/lre-5 pistils, suggesting that LLG1 function is not redundant

235 cloned from tomato (Lycopersicon esculentum) pistils, than to any other reported EGases.

236 we purified an additional molecule from the pistil that enhances pollen tube adhesion when combined

237 between pollen/pollen tubes and cells of the pistil that line their path.

238 must penetrate different tissues within the pistil, the female reproductive organ of a flower.

239 type pollen was used to pollinate the mutant pistil, the pollinated 28-5 silique became >10% longer a

240 fication of the pollen tube cell wall by the pistil, then, is likely a key mechanism for pollen rejec

241 As pollen tubes grow through the pistil they are thought to perceive and respond to diver

242 ems and roots) as well as in floral tissues (pistil tips, developing anthers and sepal vasculature).

243 Validation of selected genes on pistil tissue of the 26 single genotypes revealed that d

244 roper trajectory of pollen tubes through the pistil tissues to reach the ovules.

245 to a ca. 2.4 kb mRNA primarily expressed in pistil tissues.

246 e a pollen tube, which elongates through the pistil to deliver sperm cells to female gametes for doub

247 tube which embarks on a long journey in the pistil to deliver them to the female gametophyte for fer

248 n shown to indeed control the ability of the pistil to recognize and reject self pollen.

249 ompatibility (SI), which allows cells of the pistil to recognize and specifically inhibit "self" poll

250 cialized extracellular matrices (ECM) of the pistil to the ovule.

251 dings have revealed pearl millet and sorghum pistils to be equally sensitive to heat stress.

252 pollen-tube success and its variation among pistils to pollen receipt.

253 es that ovules are regularly arranged in the pistils to reduce competition for nutrients and space.

254 This enabled pistils to reject pollen expressing cognate PrpS.

255 a mutation in the S-RNase gene that enables pistils to reject the new pollen type.

256 ch allows the female reproductive organ, the pistil, to distinguish between self pollen and non-self

257 differentially expressed genes (DEGs) in the pistil transcriptomes of Arabidopsis thaliana and Arabid

258 of the angiosperms, pollen tubes grow in the pistil transmitting tract (TT) and are guided to the ovu

259 nting self-pollination, similar to wild-type pistils treated with GA(3) or auxins.

260 howed that LTP5 is present in pollen and the pistil TT in low levels.

261 Phosphorus allocation decreased by half in pistils under drought, while stamen phosphorus was unaff

262 Activity was not apparent in pistils until the flowers had opened and was confined to

263 uring cooler hours with increased pollen and pistil viability will overcome heat stress-induced damag

264 e and methylsalicylate emission, whereas the pistil was the main source of benzylbenzoate.

265 Ethylene production in geranium pistils was not autocatalytic.

266 mosaic virus 35S, and protein levels in the pistil were examined as well as the pollination process.

267 elf-compatible and self-incompatible apricot pistils were created using liquid chromatography coupled

268 ntrast to its non-expression in unpollinated pistils, where expression decreased after anthesis.

269 st number of DEGs was identified in infected pistils, where genes encoding regulators of cell divisio

270 he transcription of DL in stamen and SPW1 in pistil, which is crucial for sexual organ origination in

271 tubes through the transmitting tract of the pistil, which represents the longest segment of its grow

272 ID1: GID1A is expressed throughout the whole pistil, while GID1B is expressed in ovules, and GID1C is

273 c S locus where "self" pollen is rejected on pistils with matching S alleles.

274 significantly higher levels than HLP mRNA in pistils, with the opposite pattern in leaves.