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1 eleosts, tac3 expression was absent from the pituitary.
2 in the retina, hindbrain, hypothalamus, and pituitary.
3 ronal tracer DiI following implants into the pituitary.
4 ently high circulating levels of LH from the pituitary.
5 de in the developing and postnatal mammalian pituitary.
6 nctional role for the lncRNA in the anterior pituitary.
7 one and prolactin production in the anterior pituitary.
8 e and follicle-stimulating hormone) from the pituitary.
9 regulator of TGFbeta/Activin pathways in the pituitary.
10 tegrated development of the hypothalamus and pituitary.
11 nfundibulum - the precursor of the posterior pituitary.
12 ndocrine lobe [adenohypophysis (ADH)] of the pituitary, a master gland controlling growth, metabolism
15 22.2%]), trigeminal neuralgia (565 [11.5%]), pituitary adenomas (641 [13.1%]), haemangioblastoma (29
16 /secretion, cell viability and apoptosis) in pituitary adenomas (n = 74), and to compare with the res
19 Pituitary histopathology did not uncover any pituitary adenomas or somatotroph hyperplasia in either
21 ome sequencing of 159 prospectively resected pituitary adenomas showed that somatic copy number alter
25 lowing decompression, with ADCYAP1 (encoding pituitary adenylate cyclase activating peptide, PACAP) b
27 stress (CVS) has been shown to increase BNST pituitary adenylate cyclase activating polypeptide (PACA
29 revealed strong homology with the mammalian pituitary adenylate cyclase-activating peptide (PACAP).
30 e present structures of peptide and Gs-bound pituitary adenylate cyclase-activating peptide, PAC1 rec
31 gated one such brain stress response system, pituitary adenylate cyclase-activating polypeptide (PACA
33 own and beige gene programs and illustrate a pituitary-adipose signaling axis in the control of therm
34 tion between functioning of the hypothalamic pituitary adrenal (HPA) axis and cognitive capability at
35 and competition may involve the hypothalamic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonad
36 tream molecular consequences of hypothalamic-pituitary-adrenal (HPA) axis activation by exogenous adr
37 independent of their effects on hypothalamic pituitary-adrenal (HPA) axis activation, aversive condit
40 fective disturbance and promote hypothalamic-pituitary-adrenal (HPA) axis dysregulation, a key featur
42 Genetic variation within the hypothalamic-pituitary-adrenal (HPA) axis has been linked to risk for
47 s, predation risk activates the hypothalamic-pituitary-adrenal (HPA) axis, and there is growing evide
48 at is the primary output of the hypothalamus-pituitary-adrenal (HPA) axis, has been hypothesized to b
49 s affected by senescence of the hypothalamic-pituitary-adrenal (HPA) axis, leading to progressive dys
50 s, leading to activation of the hypothalamic-pituitary-adrenal (HPA) axis, the sympathetic nervous sy
51 derived cytokines stimulate the hypothalamic-pituitary-adrenal (HPA) axis, triggering endogenous gluc
56 rent increases in immobility and hypothalamo-pituitary-adrenal (HPA) output in male rats during tail
57 on immune, cardiometabolic, and hypothalamic-pituitary-adrenal (HPA) systems) in first-episode psycho
58 describe the physiology of the hypothalamic-pituitary-adrenal (HPA), hypothalamic-pituitary thyroid
60 he newborn decreases the limbic-hypothalamic-pituitary-adrenal (LHPA) axis activity in the offspring.
61 ne concentrations (a measure of hypothalamic-pituitary-adrenal [HPA] axis activation and the stress r
62 promotes the activation of the hypothalamic-pituitary-adrenal and hypothalamic-pituitary-thyroid axe
65 ed the hypothesis that maternal hypothalamic-pituitary-adrenal axis activity, measured by hair cortis
68 t, including alterations in the hypothalamic-pituitary-adrenal axis and inflammatory cytokines, which
69 kely mediated by inhibiting the hypothalamic-pituitary-adrenal axis and inflammatory responses to str
71 s through the activation of the hypothalamus-pituitary-adrenal axis and the sympathetic nervous syste
72 y effects of opioid drugs on the hypothalamo-pituitary-adrenal axis and their negative effects on bon
73 Second, we review the role of hypothalamic-pituitary-adrenal axis dysfunction in the neurobiology o
75 hanistically, we found impaired hypothalamic-pituitary-adrenal axis feedback, blunted sympathetic res
76 t in GABAergic, neurons induced hypothalamic-pituitary-adrenal axis hyperactivity and reduced fear- a
78 vation of catecholaminergic and hypothalamic-pituitary-adrenal axis leads to splenic atrophy and cont
80 health, such as by influencing hypothalamic-pituitary-adrenal axis regulation and cortisol dynamics,
81 important central component of hypothalamic-pituitary-adrenal axis regulation that prepares the orga
82 ted Glp1r knockdown had reduced hypothalamic-pituitary-adrenal axis responses to both acute and chron
84 howed a significantly increased hypothalamic-pituitary-adrenal axis stress response and impaired sens
85 sympathetic nervous system and hypothalamic-pituitary-adrenal axis) transcription factor activation.
87 rs physiological changes in the hypothalamic-pituitary-adrenal axis, reward processing in the brain,
88 luding on the regulation of the hypothalamic-pituitary-adrenal axis, thereby affecting an individual'
93 nd endocannabinoid systems; the hypothalamus-pituitary-adrenal axis; and adenosine and nitric oxide s
95 natal growth, cardiovascular development and pituitary-adrenal function of isolated chronic developme
96 that avBST inhibition augmented posttraining pituitary-adrenal output and enhanced the memory for inh
97 aint abolished their heightened hypothalamic-pituitary-adrenal responsivity and reduced stress-induce
98 the expression of genes in the hypothalamic-pituitary-adrenal/stress system (e.g., Crhr1) is one of
99 n animal models reveal that the hypothalamic-pituitary-adrenocortical (HPA) axis calibrates to the ha
102 neurons, which simultaneously project to the pituitary and forebrain regions involved in social behav
103 em translate to vesicular release toward the pituitary and identify how dopamine dynamics are control
104 both in thyrotropes and gonadotropes of the pituitary and in Leydig and germ cells in the testes, bu
106 lls co-localized with dendritic cells in the pituitary and produced increased levels of interferon-ga
107 ntrations, transcript abundance in the fetal pituitary and testes and circulating steroids, at day 75
108 al progesterone administration altered fetal pituitary and testicular function in ovine male fetuses.
109 TRH-immunoreactive elements in the brain and pituitary, and explored its role in regulation of hypoph
110 n factor BMAL2, in the pars tuberalis of the pituitary, and triggers summer biology through the eyes
111 The fish condition supports the vertebrate pituitary arising through interactions of an ancestral e
112 methods to measure the hypothalamic-adrenal-pituitary axis (N = 173), immune and inflammatory marker
114 ess are largely mediated by the hypothalamic-pituitary axis, a highly conserved neurohormonal cascade
116 been reported on the safety and efficacy of pituitary biopsy in the pediatric population for suspect
118 ocellular neurons that do not project to the pituitary but synapse onto magnocellular neurons, is pre
119 In situ hybridization showed two distinct pituitary cell populations, tshbetaa cells in the anteri
125 C) genes Asic1, Asic2, and Asic4 in anterior pituitary cells and Asic1 and Asic2 in immortalized GH3
130 ents increased the excitability of secretory pituitary cells, consistent with their potential physiol
135 ronic stress, hypothalamic activation of the pituitary changes from corticotropin-releasing hormone-d
139 Stimulation of cAMP in C57BL/6 mouse primary pituitary cultures using forskolin or a long-acting GH-r
142 on spaceflight was associated with increased pituitary deformation, augmented aqueductal cerebrospina
143 R-7 family, miR-7a2, is essential for normal pituitary development and hypothalamic-pituitary-gonadal
144 estigate the function of this pathway during pituitary development and in the regulation of the SOX2
146 is not successful and can be classified into pituitary-directed drugs, steroid synthesis inhibitors,
147 h an increasing number of suspected cases of pituitary diseases, there has been a paradigm shift in t
154 meningeal enhancement, sagging of the brain, pituitary enlargement, and subdural fluid collections.
163 e changes in circulating GH levels to adjust pituitary GH secretion within a narrow physiological ran
164 protein and localized a specific area of the pituitary gland (i.e., adenohypophysis) known to secrete
165 actin from lactotropic cells in the anterior pituitary gland and thus play a central role in prolacti
166 ocin (OT) originates from secretion from the pituitary gland into the circulation and from absorption
167 wing to organic disease of the hypothalamus, pituitary gland or testes has been treated with testoste
168 he hormone arginine vasopressin (AVP) in the pituitary gland or the hypothalamus, whereas nephrogenic
169 acellular compartment to the cell surface in pituitary gland somatotropes, concomitant with increasin
171 We observed that a reduced volume in the pituitary gland was associated with the slope of neuroti
172 ated by the pars tuberalis (PT) of the fetal pituitary gland, before the fetal circadian system and a
175 (GH), a pleiotropic hormone secreted by the pituitary gland, regulates immune and inflammatory respo
184 d by a 50% threshold for all lesions and the pituitary gland; and for (18)F-FDG (C)-RD of SUVs of the
185 ory granules were significantly decreased in pituitary glands of 4.1N(-/-) compared to 4.1N(+/+).
188 imilarity in the formation of the pineal and pituitary glands, and suggests that all CNS neuroendocri
189 known to regulate the mammalian hypothalamo-pituitary gonadal axis, may be involved in the seasonal
190 ic-pituitary thyroid (HPT), and hypothalamic-pituitary-gonadal (HPG) axes and review the evidence for
191 mic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonadal (HPG) axes in underlying some of these
192 al hormones are produced by the hypothalamic-pituitary-gonadal (HPG) axis and have been shown to dete
195 In women, suppression of the hypothalamic-pituitary-gonadal axis appears to be a principal contrib
197 of metabolic information to the hypothalamo-pituitary-gonadal axis is mediated by leptin receptors o
203 lucose utilization and GLUT1 expression in a pituitary gonadotrope cell model and in primary gonadotr
204 lated the Fshb expression levels obtained in pituitary gonadotrope cells perifused with varying GnRH
206 show normal sexual behavior but hypothalamic-pituitary-gonadotropin (HPG) axis dysregulation, likely
207 ) leads to a marked decrease in secretion of pituitary gonadotropins LH and FSH and impairment of rep
214 iates L-DOPA-induced dyskinetic movements in pituitary homeobox 3 (Pitx3)-deficient mice that lack of
218 ntibody that targets the beta-subunit of the pituitary hormone follicle-stimulating hormone (Fsh) inc
224 e (TIDA) neurons that control release of the pituitary hormone, prolactin, which triggers key materna
225 nhibited the expression/secretion of several pituitary hormones (specially GH/PRL), which was accompa
227 bernation, and metabolism, are controlled by pituitary hormones released in response to annual enviro
228 ropin subunit and GnRH receptor genes in rat pituitary in vitro and in vivo to clarify their expressi
231 ehaviour and functioning of the hypothalamic-pituitary-interrenal (HPI) axis, the fish equivalent of
232 by seasonal stimulation of the hypothalamic-pituitary-interrenal axis, and that relaxed selection on
233 on scores; RR, 6.74 [95% CI, 4.61 to 9.86]), pituitary irradiation (5-20 Gy: RR, 4.24 [95% CI, 1.98 t
234 (>= 15 Gy on >= 90% of their volume) without pituitary irradiation increased the RR of SAH by 4.62 (9
238 levels at e10.5 and e12.5 were comparable to pituitary levels from adult female mice at proestrus and
239 n the differential diagnosis of solid-cystic pituitary masses along with clinical correlation, which
241 anges in intracranial volumetric parameters, pituitary morphologic structure, and aqueductal cerebros
244 ntroduced to be exclusively expressed in the pituitary on the background of a global ERalpha-null (Pi
248 onate to adulthood, we investigated a common pituitary origin for hypothyroidism and macroorchidism,
252 rve transfer and highly purified lyophilized pituitary porcine GH treatment (0.6 mg/day); Group-3 (po
254 ption in neurochemical identity, however, as pituitary prolactin secretion is primarily under monoami
255 and female reproductive tracts, hypothalamic-pituitary regulation of reproduction and prenatal develo
257 higher expression of the Fshb subunit in the pituitary, resulting in elevated serum estrogen and high
258 Single-cell RNA sequencing of the adult pituitary reveals similar competency of endodermal and e
259 a scaling factor for translation capacity in pituitary secretory cells and that it directly binds ~75
260 nclude that the gonadotropes of the anterior pituitary sense glucose availability and integrate this
264 ESRRG), which results in stronger binding of pituitary-specific positive transcription factor 1 (Pit-
267 nohypophysis, and tshbetab cells near to the pituitary stalk, a location comparable to the pars tuber
269 nd regulated in other tissues, including the pituitary, suggesting that locally- and AT-produced adip
271 alamic-pituitary-adrenal (HPA), hypothalamic-pituitary thyroid (HPT), and hypothalamic-pituitary-gona
274 othalamic-pituitary-adrenal and hypothalamic-pituitary-thyroid axes, as well as a rise in systolic bl
275 ing hormone (TRH) regulates the hypothalamic-pituitary-thyroid axis in mammals and also regulates pro
277 beta leads to disruption of the hypothalamic-pituitary-thyroid axis with resistance to TH, while muta
279 f surgically resected tumors from forty five pituitary tumor patients [gonadotropic (LH/FSH-secreting
282 Growth hormone-secreting (GH-secreting) pituitary tumors are driven by oncogenes that induce cAM
285 iagnosis of various histological subtypes of pituitary tumors is made using serum based hormone panel
286 Prolactinomas are the most frequent type of pituitary tumors, which represent 10-20% of all intracra
290 In adult cohorts separately, rs1344110 in pituitary tumour-transforming 1 interacting protein (PTT
292 lation of the gene and protein expression of pituitary-type GHRH-receptor in prefrontal cortex of mic
297 Expression profiles of both paralogs in the pituitary were measured by qPCR throughout smoltificatio
298 ound that miR-7a2 is highly expressed in the pituitary, where it suppresses golgi glycoprotein 1 (GLG
299 he rat hypothalamus project to the posterior pituitary, where they secrete their products into the bl