ライフサイエンスコーパス: plasma osmolality

1 ta mRNA correlated inversely with changes in plasma osmolality.

2 onse to angiotensin II; and (c) reduction in plasma osmolality.

3 d-type (WT) mice while maintaining a similar plasma osmolality.

4 ipheral circulation in response to increased plasma osmolality.

5 ality failed to decrease at the same rate as plasma osmolality.

6 ncomitant with increases in fluid intake and plasma osmolality.

7 travenous infusion of HYPER saline increased plasma osmolality (294 +/- 3 to 316 +/- 5 mOsm kg(-1) H2

8 We also found an acute decrease in plasma osmolality after a vaginal, but not C-section bir

9 induced drinking, vasopressin (AVP) release, plasma osmolality and c-fos expression in the brain of c

10 During HYPER, plasma osmolality and copeptin increased (P < 0.05) and

11 'salt-loading' produces a large increase in plasma osmolality and depletes the pituitary content of

12 Loads of 900 and 1200 mOsm/kg both increased plasma osmolality and sodium concentration by 15 min aft

13 smotic stimulus, determined by assessment of plasma osmolality and sodium content, SON neurons exhibi

14 Except for the expected differences in plasma osmolality and sodium, basal measures were simila

15 Hydration status (plasma osmolality and urine specific gravity) and body c

16 ration, which in turn influence the osmotic (plasma osmolality) and blood volume-dependent compensato

17 atocrit, plasma protein concentration, total plasma osmolality, and plasma COP.

18 ydration status, including hematocrit, total plasma osmolality, and plasma protein concentration, fai

19 s generally accepted that dialysis may lower plasma osmolality at a faster rate than changes in ocula

20 rea bath decreased plasma urea by 21 mM, and plasma osmolality by 22 mosmol/kg H2O, and increased bra

21 e growth factor binding protein 3, urine and plasma osmolality, electrolytes, glucose, and insulin we

22 An elevation in plasma osmolality elicits a complex neurohumoral respons

23 measured oxytocin or vasopressin secretion, plasma osmolality, haematocrit or pituitary hormone cont

24 decrease in urinary osmolality, and rise in plasma osmolality in the OPC 31260-treated CHF rats as c

25 Plasma volume is expanded and plasma osmolality is decreased, yet vasopressin secretio

26 dration-induced anorexia in which increasing plasma osmolality leads to a centrally generated reducti

27 by the classical homeostatic, interosensory plasma osmolality negative feedback as well as by novel,

28 The decreased plasma osmolality of P14 rats was not modified by OPC-31

29 (P < .02), despite their consistently lower plasma osmolality (P < .007).

30 However, only plasma osmolality (P(osm)) showed statistical promise fo

31 ting these results with ongoing behavior and plasma osmolality points to the existence of brain netwo

32 l plasma sodium (WD(5)), the substitution of plasma osmolality (Posm) for sodium (WD(6)), and actual

33 hat individuals classified as DE have higher plasma osmolality (Posm), indicating suboptimal hydratio

34 w; however, plasma sodium concentrations and plasma osmolality remained low.

35 Increased plasma osmolality results in increased central as well a

36 , which produced a physiological increase in plasma osmolality to 299 +/- 1 mosmol (kg water)(-1), el

37 g neurones are activated during increases in plasma osmolality to elicit sympathoexcitation.

38 Plasma osmolality was consistently reduced by the chroni

39 Plasma osmolalities were significantly lower in pregnant

40 Cardiac output (CO) and plasma osmolality were significantly decreased and plasm

41 ared different equations used for predicting plasma osmolality when its direct measurement was not pr

42 to the LHA responded to a rapid increase in plasma osmolality with increased c-fos mRNA levels.