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1 d function, despite significant increases in plasma selenium.
3 the context of established relations between plasma selenium and risk of cancer and mortality, and re
4 telet glutathione peroxidase activity and in plasma selenium and selenoprotein P concentrations were
5 ly demonstrated a strong interaction between plasma selenium and the manganese superoxide dismutase (
6 igh levels of blood cadmium or low levels of plasma selenium and the NLRP3 haplotype C-A-A-C multipli
7 protein is <20 mg/L (plasma zinc), <10 mg/L (plasma selenium and vitamins A and D), or <5 mg/L (vitam
9 tions were positively associated with infant plasma selenium at 2 or 6 and 24 wk postpartum (P < 0.00
10 erved only for men with a deficient level of plasma selenium, but healthy dietary sources should be r
19 study was to investigate the association of plasma selenium concentration with subsequent prostate c
20 meta-analysis indicated that an increase in plasma selenium concentrations above 100 mug/L did not f
23 elenium and dietary intakes coupled with low plasma selenium concentrations in HIV infection could ha
24 nfant (55.6 +/- 16.3 to 61.0 +/- 15.4 mug/L) plasma selenium concentrations increased, whereas breast
25 -gene interaction was apparent when baseline plasma selenium concentrations were included in the regr
33 microg/mL) was not seen in any subjects, and plasma selenium in 244 HIV-positive subjects (0.120 +/-
36 diabetes in the highest tertile of baseline plasma selenium level (hazard ratio, 2.70 [CI, 1.30 to 5
37 adient was found across tertiles of baseline plasma selenium level, with a statistically significantl
42 nd young adults, we examined the relation of plasma selenium, whole-blood glutathione, and whole-bloo
43 ignificant differences in the association of plasma selenium with risk when analyzed by stage or grad