ライフサイエンスコーパス: plasmacytoid

1 Both plasmacytoid and IgE(+)FcepsilonRI(+) myeloid dendritic

2 dent cross-talk between the main DC subsets, plasmacytoid and myeloid DCs (mDCs) was necessary for in

3 o in vivo expansion of IRF8-dependent CD8a+, plasmacytoid, and CD103+ CD11b2 DCs.

4 Macrophages, plasmacytoid, and conventional dendritic cells were each

5 Plasmacytoid bladder cancer is an aggressive histologic

6 alterations in the CDH1 gene occur in 84% of plasmacytoid carcinomas and are specific to this histolo

7 ent with the aggressive clinical behavior of plasmacytoid carcinomas, which frequently recur locally,

8 ction; however, its regulatory mechanisms in plasmacytoid cells (pDCs) still remain unclear.

9 Depletion of either plasmacytoid DC (pDC) alone or the lymphoid-resident DC

10 l fate decision between interferon-producing plasmacytoid DC (pDC) and antigen-presenting classical D

11 and activity in freshly isolated monocytes, plasmacytoid DC (pDC) and in vitro-generated Langerhans

12 Pre-DC share surface markers with plasmacytoid DC (pDC) but have distinct functional prope

13 esulted in increased IFN-alpha production by plasmacytoid DC (pDC) from skin/tumor draining lymph nod

14 endritic cells (mDC) and macrophages but not plasmacytoid DC (pDC) had suppressed capacity to stimula

15 (+) DC, CD1c(+) DC, and, to a lesser extent, plasmacytoid DC (pDC) in the blood, spleen, and bone mar

16 and is physiologically important because in plasmacytoid DC (pDC) stimulated with Toll-like receptor

17 examined the number and activation of blood plasmacytoid DC (pDC), CD141(+), and CD1c(+) myeloid DC

18 two distinct subsets, myeloid DC (mDCs) and plasmacytoid DC (pDCs).

19 CD11c(high)CD103(high)) DC and plasmacytoid (plasmacytoid DC Ag-1(high)B220(+)CD11c(int)) DC (pDC) po

20 Irf8 expression is essential for plasmacytoid DC and CD8alpha(+) DC development, and this

21 HC acquisition by thymic CD8alpha(+) cDC and plasmacytoid DC but not SIRPalpha(+) cDC.

22 g plays a pivotal role whereby it suppresses plasmacytoid DC development while enhancing that of CD8a

23 eq studies, we show that Irf8 functions as a plasmacytoid DC epigenetic and fate-determining TF, regu

24 set of DCs; the relationship between blastic plasmacytoid DC neoplasia cells and healthy DCs; and cir

25 cluding the myeloid DC subset (mDCs) and the plasmacytoid DC subset (pDCs).

26 ss I and II from thymic epithelial cells but plasmacytoid DC were less efficient.

27 either CD11c as conventional DC or CD123 as plasmacytoid DC).

28 d monocyte-derived DC, Langerhans cells, and plasmacytoid DC.

29 culating myeloid DC, and at a lower level in plasmacytoid DC.

30 Plasmacytoid DCs (pDC) produce large amounts of type I I

31 the numbers of vaccine-resident CD8(+) DCs, plasmacytoid DCs (pDC), along with local interleukin (IL

32 We found that myeloid DCs (mDCs) and plasmacytoid DCs (pDCs) accumulate in the bone marrow du

33 ajor subsets, the interferon (IFN)-producing plasmacytoid DCs (pDCs) and antigen-presenting classical

34 rol of cell survival of two main DC subsets: plasmacytoid DCs (pDCs) and conventional DCs (cDCs) and

35 2 (BDCA2) is expressed exclusively on human plasmacytoid DCs (pDCs) and plays a role in Ag capture,

36 differential effect on DC subsets, including plasmacytoid DCs (pDCs) and the classical DC lineages cD

37 Although plasmacytoid DCs (pDCs) are a primary source of IFN, the

38 both conventional dendritic cells (cDCs) and plasmacytoid DCs (pDCs) are required for the crossprimin

39 Plasmacytoid DCs (pDCs) are typically thought to be key

40 a new DC subset that shares properties with plasmacytoid DCs (pDCs) but potently activates T cells,

41 isingly, IFN expression was not dependent on plasmacytoid DCs (pDCs) but rather was dependent on mDCs

42 Plasmacytoid DCs (PDCs) exposed to PSA do not produce pr

43 conventional DCs (cDC) 1 and cDC2 as well as plasmacytoid DCs (pDCs) in the peripheral blood of pigs.

44 we studied monocyte-derived DCs (moDCs) and plasmacytoid DCs (pDCs) in two HPS2 siblings.

45 Plasmacytoid DCs (pDCs) support antiviral immunity by li

46 rentially captured and internalized by human plasmacytoid DCs (pDCs) that express the TIM1 phosphatid

47 dian frequency of myeloid DCs (mDCs) but not plasmacytoid DCs (pDCs) was observed in the blood of SR

48 d DC1s (mDC1), BDCA3+(high) mDC2s and BDCA2+ plasmacytoid DCs (pDCs) were identified and expressions

49 of inflammatory cells, such as macrophages, plasmacytoid DCs (pDCs), and neutrophils, which were obs

50 Plasmacytoid DCs (pDCs), the major producers of type I i

51 HC complexes, along with increased PD-L1, on plasmacytoid DCs (pDCs).

52 istinct subsets: conventional DCs (cDCs) and plasmacytoid DCs (pDCs).

53 the blood DC antigen 2 (BDCA-2) receptor on plasmacytoid DCs (pDCs).

54 RNA for markers of myeloid DCs (mDCs; CD1c), plasmacytoid DCs (pDCs; CD303) and Langerhans cells (LCs

55 , whereas IDO2 is expressed in both mDCs and plasmacytoid DCs and is not modulated by PGE2.

56 tions to distinct DC lineages, consisting of plasmacytoid DCs and several subsets of classical DCs th

57 ent antigen to CD4(+) T cells and identified plasmacytoid DCs as an important antigen-presenting cell

58 , increasing the number of blood myeloid and plasmacytoid DCs by 16- and 60-fold, respectively.

59 th types of CRSwNP, but only myeloid DCs and plasmacytoid DCs from eosinophilic CRSwNP demonstrated a

60 At the functional level, both mDCs and plasmacytoid DCs generate T regulatory cells through an

61 Supernatant from plasmacytoid DCs harvested postinfection with BVDV or re

62 y expressed on activated primary myeloid and plasmacytoid DCs in human and mouse.

63 ations of monocytes, myeloid DCs (MDCs), and plasmacytoid DCs in the lung mucosa.

64 ytes, monocyte-derived DCs, macrophages, and plasmacytoid DCs in the skin, we addressed their dynamic

65 Aggregation of plasmacytoid DCs in the splenic perifollicular region, f

66 On the other hand, recognition of viruses by plasmacytoid DCs inhibits IL-1beta and IL-23 release, in

67 By contrast, the function of plasmacytoid DCs is largely innate and restricted to the

68 spond more effectively to TLR ligation, with plasmacytoid DCs making more IFN-alpha and both CD8alpha

69 FN production by DCs that lack TLR3, such as plasmacytoid DCs or CD8(-) DCs.

70 f alpha4 integrin-deficient conventional and plasmacytoid DCs to the CNS was unaffected, whereas alph

71 ha(+) conventional dendritic cells (DCs) and plasmacytoid DCs upon TLR-mediated activation and detect

72 Plasmacytoid DCs were not detectable during the whole co

73 d DCs to acute IM plasma resulted in loss of plasmacytoid DCs, and further studies with individual cy

74 as Tlr9 deficiency in dendritic cells (DCs), plasmacytoid DCs, and neutrophils had no discernable eff

75 onal dendritic cells (DCs) and CD11c1/CD1231 plasmacytoid DCs, and striking granulocytic hyperplasia.

76 Increased numbers of myeloid DCs, plasmacytoid DCs, and their activated subsets were found

77 r CD8 CTLs, and skewing of DC subsets toward plasmacytoid DCs, coupled with greater CD4 T follicular

78 L1B+ T-regulatory cells, IL1B+ DCs and IL1B+ plasmacytoid DCs, IL1B+ monocytes, and fewer group 1 inn

79 cal infiltrates composed of conventional and plasmacytoid DCs, with the subsequent induction of poten

80 ted an expansion of classical DCs (cDCs) and plasmacytoid DCs.

81 ucing DCs compared with conventional DCs and plasmacytoid DCs.

82 ells, IL1B+ dendritic cells (DCs), and IL1B+ plasmacytoid DCs.

83 We identified that transient plasmacytoid dendritic cell (pDC) depletion during prima

84 nscription factor is required for B cell and plasmacytoid dendritic cell (pDC) development, but its m

85 HIV-2 favored plasmacytoid dendritic cell (pDC) differentiation into c

86 The plasmacytoid dendritic cell (pDC) is vital to the coordi

87 expression of the monocyte marker, CD14; the plasmacytoid dendritic cell (pDC) marker, BDCA4, identif

88 Blastic plasmacytoid dendritic cell (PDC) neoplasm (BPDCN) is an

89 induced limited type I interferon (IFN) and plasmacytoid dendritic cell (pDC) responses.

90 immune complexes that amplify TLR9-mediated plasmacytoid dendritic cell (pDC)-hyperactivation and in

91 unopathology; others, including basophil and plasmacytoid dendritic cell depletion, correlate strongl

92 ell contact, and induction was suppressed by plasmacytoid dendritic cell depletion.

93 ification of CD38 as a critical regulator of plasmacytoid dendritic cell function in response to infl

94 his study, we report that DMF inhibits human plasmacytoid dendritic cell function through a mechanism

95 MyD88-independent manner, while we confirmed plasmacytoid dendritic cell IFN-I had inverse requiremen

96 IFN Regulatory Factor 5 (IRF5) in the human plasmacytoid dendritic cell line Gen2.2 prevented IFNbet

97 dent oncogenic regulatory network in blastic plasmacytoid dendritic cell neoplasm (BPDCN) and demonst

98 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare m

99 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is an aggre

100 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is an uncom

101 study of treatment of patients with blastic plasmacytoid dendritic cell neoplasm (BPDCN), an aggress

102 ing acute myeloid leukemia (AML) and blastic plasmacytoid dendritic cell neoplasm (BPDCN).

103 s of CD4 and memory B lymphocytes, decreased plasmacytoid dendritic cell numbers, and increased repre

104 0.0001) and increased regulatory T-cell and plasmacytoid dendritic cell percentages.

105 circuit blocks E-protein activity to exclude plasmacytoid dendritic cell potential and explains the s

106 enotype and gene expression revealed a novel plasmacytoid dendritic cell precursor preferentially mob

107 ed endothelial cells can promote myeloid and plasmacytoid dendritic cell transmigration across endoth

108 We characterized plasmacytoid dendritic cell, natural killer (NK), and T-

109 Thus, upon VSV infection, plasmacytoid dendritic cell-derived IFN-I primarily prot

110 atures reflect the extent of activation in a plasmacytoid dendritic cell-type I IFN-T/B lymphocyte ne

111 the developmental origin of conventional and plasmacytoid dendritic cells (cDCs and pDCs, respectivel

112 Plasmacytoid dendritic cells (DCs [pDCs]) develop from p

113 his study, we show that conventional but not plasmacytoid dendritic cells (DCs) are required for anti

114 significant loss of circulating myeloid and plasmacytoid dendritic cells (DCs) during acute IM, a lo

115 that a highly enriched population of bovine plasmacytoid dendritic cells (DCs) produced IFN in respo

116 factors as well as an increased frequency of plasmacytoid dendritic cells (DCs) that corresponded wit

117 on of the gut-homing receptor alpha4beta7 on plasmacytoid dendritic cells (p < 0.01) and the magnitud

118 Plasmacytoid dendritic cells (pDC) are specialized in se

119 vesicular stomatitis virus (VSV) particles, plasmacytoid dendritic cells (pDC) are triggered to moun

120 Plasmacytoid dendritic cells (pDC) are type I interferon

121 Plasmacytoid dendritic cells (pDC) constitute the body's

122 Here, we report that AGM plasmacytoid dendritic cells (pDC) express extremely low

123 Plasmacytoid dendritic cells (pDC) have been regarded as

124 s, cytokines produced in large quantities by plasmacytoid dendritic cells (pDC) in response to engage

125 edge, nothing is known about the survival of plasmacytoid dendritic cells (pDC) in such situation.

126 We show that plasmacytoid dendritic cells (pDC) of natural hosts disp

127 Following IAV infection, plasmacytoid dendritic cells (pDC) or CD8alpha(+) DC reg

128 Plasmacytoid dendritic cells (pDC) produce IFN-I in resp

129 se to the Toll-like receptor-9 agonist CpGA, plasmacytoid dendritic cells (pDC) produced type 1 IFNs,

130 Plasmacytoid dendritic cells (pDC) rapidly and massively

131 the alpha interferon (IFN-alpha) response of plasmacytoid dendritic cells (pDC) to MHV68 was reduced

132 Enzymatic IDO1 activity, TGF-beta, and plasmacytoid dendritic cells (pDC) were neutralized by 1

133 7 triggers rapid sensing of nucleic acids by plasmacytoid dendritic cells (pDC).

134 he microbiome alters TLR7-MyD88 signaling in plasmacytoid dendritic cells (pDCs) and blunts systemic

135 "auto"-regulatory feedback mechanism between plasmacytoid dendritic cells (pDCs) and Breg cells.

136 derived from mice and humans, as well as in plasmacytoid dendritic cells (pDCs) and CD141(+) DCs fro

137 d gnotobiotic conditions, notably increasing plasmacytoid dendritic cells (pDCs) and interferon signa

138 The numbers of plasmacytoid dendritic cells (pDCs) and naive T cells (T

139 Following the discovery of plasmacytoid dendritic cells (pDCs) and of their extraor

140 1 infection induces persistent activation of plasmacytoid dendritic cells (pDCs) and production of ty

141 Plasmacytoid dendritic cells (pDCs) and type I IFN drive

142 Plasmacytoid dendritic cells (pDCs) are a major source o

143 Plasmacytoid dendritic cells (pDCs) are a unique sentine

144 Plasmacytoid dendritic cells (PDCs) are central in antiv

145 Plasmacytoid dendritic cells (pDCs) are considered to be

146 Plasmacytoid dendritic cells (pDCs) are important early

147 Plasmacytoid dendritic cells (pDCs) are important in ant

148 Plasmacytoid dendritic cells (pDCs) are key components o

149 Plasmacytoid dendritic cells (pDCs) are key players in t

150 Plasmacytoid dendritic cells (pDCs) are potent producers

151 Plasmacytoid dendritic cells (pDCs) are primary producer

152 Airway myeloid dendritic cells (mDCs) and plasmacytoid dendritic cells (pDCs) are professional ant

153 Plasmacytoid dendritic cells (pDCs) are professional typ

154 In chronic diseases, such as HIV infection, plasmacytoid dendritic cells (pDCs) are rendered dysfunc

155 Plasmacytoid dendritic cells (pDCs) are robust producers

156 Plasmacytoid dendritic cells (pDCs) are the major produc

157 Plasmacytoid dendritic cells (pDCs) are the major source

158 Plasmacytoid dendritic cells (pDCs) are the major type I

159 Plasmacytoid dendritic cells (pDCs) are the most powerfu

160 Plasmacytoid dendritic cells (pDCs) are vital to antivir

161 of antigen-presenting cells, in particular, plasmacytoid dendritic cells (pDCs) around each MTZ form

162 Plasmacytoid dendritic cells (pDCs) bridge innate and ad

163 the production of type I interferon (IFN) in plasmacytoid dendritic cells (pDCs) by binding to endoso

164 Plasmacytoid dendritic cells (pDCs) efficiently produce

165 Plasmacytoid dendritic cells (pDCs) express Toll like re

166 rferon (IFN-I) signaling and IFN-I-producing plasmacytoid dendritic cells (pDCs) facilitate the diffe

167 Plasmacytoid dendritic cells (pDCs) from females produce

168 f the JCI, Mitchell and authors investigated plasmacytoid dendritic cells (pDCs) from the blood of in

169 nocytes, myeloid dendritic cells (mDCs), and plasmacytoid dendritic cells (pDCs) from three individua

170 Although plasmacytoid dendritic cells (pDCs) have been shown to p

171 Plasmacytoid dendritic cells (pDCs) highly populate lung

172 bsets, we found that BCMA was transcribed in plasmacytoid dendritic cells (pDCs) in both blood and ly

173 Bone marrow plasmacytoid dendritic cells (pDCs) in patients with mul

174 The potential contribution of plasmacytoid dendritic cells (pDCs) in the presentation

175 of metaflammation by recruiting circulating plasmacytoid dendritic cells (pDCs) into VAT through che

176 ryptococcus neoformans; however, the role of plasmacytoid dendritic cells (pDCs) is unknown.

177 cluding IFN-alpha, and sortilin depletion in plasmacytoid dendritic cells (pDCs) led to a reduction o

178 myeloid dendritic cells (MDDCs/mDCs), and by plasmacytoid dendritic cells (pDCs) of human and murine

179 Human plasmacytoid dendritic cells (pDCs) play a major role in

180 type I IFN response by a small percentage of plasmacytoid dendritic cells (pDCs) present in the monoc

181 Plasmacytoid dendritic cells (pDCs) produce abundant typ

182 Plasmacytoid dendritic cells (pDCs) produce large amount

183 Plasmacytoid dendritic cells (pDCs) produce type I inter

184 Plasmacytoid dendritic cells (pDCs) produced FasL in res

185 Plasmacytoid dendritic cells (pDCs) rapidly produce larg

186 g protective antiviral immune responses, and plasmacytoid dendritic cells (pDCs) represent a major so

187 Plasmacytoid dendritic cells (pDCs) require a particular

188 ell TLR7 expression or temporal depletion of plasmacytoid dendritic cells (pDCs) slow progression; ho

189 Plasmacytoid dendritic cells (pDCs) that secrete large a

190 Ab response against HIV-1 p24 that activates plasmacytoid dendritic cells (pDCs) through FcgammaRIIa

191 terized the immune response of monocytes and plasmacytoid dendritic cells (pDCs) to influenza A virus

192 RNA from human Huh-7 hepatoma cells to human plasmacytoid dendritic cells (pDCs) triggers pDC alpha/b

193 gp96 preferentially engages conventional and plasmacytoid dendritic cells (pDCs) under low and high d

194 denitis tissues but localization in CD123(+) plasmacytoid dendritic cells (pDCs) was found only in ly

195 Plasmacytoid dendritic cells (pDCs) were considered to b

196 Plasmacytoid dendritic cells (pDCs) were identified as t

197 Plasmacytoid dendritic cells (pDCs) were isolated from w

198 Plasmacytoid dendritic cells (pDCs), a primary source of

199 ic for double-stranded DNA (dsDNA) activated plasmacytoid dendritic cells (pDCs), a type of cell of t

200 f PTB in macaque lungs include the influx of plasmacytoid dendritic cells (pDCs), an Interferon (IFN)

201 BM demonstrated increased CD9(-)Siglec H(hi) plasmacytoid dendritic cells (pDCs), and depletion of pD

202 N-alpha production was primarily mediated by plasmacytoid dendritic cells (pDCs), and was significant

203 Ialpha, including mast cells, basophils, and plasmacytoid dendritic cells (pDCs), are regulated by Ig

204 Plasmacytoid dendritic cells (pDCs), B cells and NK cell

205 due to differences in number or function of plasmacytoid dendritic cells (pDCs), because these cells

206 both conventional dendritic cells (cDCs) and plasmacytoid dendritic cells (pDCs), but whereas cDCs pr

207 HIV-1-dependent reduction of ILC3s required plasmacytoid dendritic cells (pDCs), IFN-I, and the CD95

208 , particularly type I interferons (IFNs) and plasmacytoid dendritic cells (pDCs), in the pathogenesis

209 Plasmacytoid dendritic cells (pDCs), prominent cells of

210 ice enhances Fas ligand (FasL) expression on plasmacytoid dendritic cells (pDCs), resulting in apopto

211 Using isolated plasmacytoid dendritic cells (pDCs), we demonstrated tha

212 oduction upon incubation in vitro with human plasmacytoid dendritic cells (pDCs), whereas lentivirus

213 Here, we demonstrate that plasmacytoid dendritic cells (pDCs), which are prototypi

214 ritical virus-induced IFN-alpha responses of plasmacytoid dendritic cells (pDCs), which can be defici

215 This occurs via selective expansion of plasmacytoid dendritic cells (pDCs), which further augme

216 ce, we analyzed the role of this receptor in plasmacytoid dendritic cells (pDCs), which preferentiall

217 responses; therefore, we questioned whether plasmacytoid dendritic cells (pDCs), which produce IFN w

218 ive immunity and are massively produced from plasmacytoid dendritic cells (pDCs).

219 Type I IFN is mainly produced by plasmacytoid dendritic cells (pDCs).

220 synergistic response in both mouse and human plasmacytoid dendritic cells (pDCs).

221 able hematologic malignancy originating from plasmacytoid dendritic cells (pDCs).

222 mplex- and TLR7-mediated activation of human plasmacytoid dendritic cells (pDCs).

223 ompanied by increases in CD80(+) and CD86(+) plasmacytoid dendritic cells (pDCs).

224 A major source of IFN-alphabeta is plasmacytoid dendritic cells (pDCs).

225 IFN) produced by dendritic cells, especially plasmacytoid dendritic cells (pDCs).

226 SLE, a major source of type I IFNs being the plasmacytoid dendritic cells (pDCs).

227 ffectors predominantly produced by activated plasmacytoid dendritic cells (pDCs).

228 vitro upon priming naive CD4(+) T cells with plasmacytoid dendritic cells activated with oxidized mit

229 S-1/MAVS signaling and induced type I IFN in plasmacytoid dendritic cells and macrophages, with the l

230 by unique members of the microbiota regulate plasmacytoid dendritic cells and monocytes via TLR7 and

231 neutrophils promoted cleavage of FcgRIIA on plasmacytoid dendritic cells and monocytes, resulting in

232 ces of type I IFNs during IOE infection were plasmacytoid dendritic cells and monocytes.

233 by inflammatory monocytes, dendritic cells, plasmacytoid dendritic cells and NK cells in all tissues

234 ion by monocytes required TLR7 activation of plasmacytoid dendritic cells and secretion of type I IFN

235 ne caused by TLR7 induction of type I IFN by plasmacytoid dendritic cells and subsequent IFN stimulat

236 Upon adoptive transfer of wild-type plasmacytoid dendritic cells and subsequent VSV infectio

237 03 treatment led to pronounced activation of plasmacytoid dendritic cells and substantial increases i

238 nd established systemic lupus erythematosus, plasmacytoid dendritic cells are not effector cells, hav

239 ent type I interferons (IFN-alpha/beta) from plasmacytoid dendritic cells as well as the production o

240 1 receptor-associated kinase 1 expression in plasmacytoid dendritic cells both in vivo and in vitro,

241 In addition, plasmacytoid dendritic cells have a transcriptional sign

242 Plasmacytoid dendritic cells have been implicated in the

243 tiation of these cytokines is not clear, but plasmacytoid dendritic cells have been thought to contri

244 assical dendritic cells and CD11c+ low)B220+ plasmacytoid dendritic cells in both the heart and splee

245 n, and increased classic dendritic cells and plasmacytoid dendritic cells in peripheral blood and bon

246 to induce interferon-alpha in primary human plasmacytoid dendritic cells in response to hepatitis C

247 CXCL4 is the predominant protein secreted by plasmacytoid dendritic cells in systemic sclerosis, both

248 Plasmacytoid dendritic cells influenced by microbes migr

249 w that type I interferons (IFNs) produced by plasmacytoid dendritic cells inhibit the clearance of ap

250 nfluenza virus, the patient's leukocytes and plasmacytoid dendritic cells produced very little type I

251 that rCl-13(GPC/VGKS) infected fewer splenic plasmacytoid dendritic cells than rCl-13, yet the two vi

252 tologic cancer that is caused by transformed plasmacytoid dendritic cells that overexpress interleuki

253 gressive hematologic malignancy derived from plasmacytoid dendritic cells that typically involves the

254 munity, in which the differential ability of plasmacytoid dendritic cells to produce interferon alpha

255 cells, monocyte-derived dendritic cells, and plasmacytoid dendritic cells to vIL-10 suppresses multip

256 gered the production of type I interferon by plasmacytoid dendritic cells via activation of Toll-like

257 une lupus patients can stimulate B cells and plasmacytoid dendritic cells via Toll-like receptors 7 a

258 T cells, B cells, type I IFN, IFN-gamma, and plasmacytoid dendritic cells were contributed to efficie

259 ses, frequencies of tolerogenic CD103(+) and plasmacytoid dendritic cells were increased.

260 In addition, infiltrated plasmacytoid dendritic cells were the primary IL-10-secr

261 primary human epithelial cells, B cells, and plasmacytoid dendritic cells with dsDNA viruses induces

262 of pathogenic immune cells (plasmablasts and plasmacytoid dendritic cells).

263 Depletion of plasmacytoid dendritic cells, a major cellular source of

264 al infections, especially by memory T cells, plasmacytoid dendritic cells, and CD56(bright) NK cells.

265 mmune factors, such as natural killer cells, plasmacytoid dendritic cells, and the expression pattern

266 -hematopoietic cellular sources, rather than plasmacytoid dendritic cells, are responsible for interf

267 onventional, monocyte-derived, lymphoid, and plasmacytoid dendritic cells, as well as activated T-cel

268 of MHV68 in a subset of immune cells called plasmacytoid dendritic cells, as well as on the control

269 ells, natural killer cells, conventional and plasmacytoid dendritic cells, B cells and especially pla

270 ytokine production, including IFN-alpha from plasmacytoid dendritic cells, but only in the presence o

271 We reveal that some cells, including plasmacytoid dendritic cells, can express both CCR2 and

272 Activation of plasmacytoid dendritic cells, modulation of B-cell respo

273 e, memory and activated subsets, myeloid and plasmacytoid dendritic cells, monocytes, B lymphocytes,

274 ment were also related to the proportions of plasmacytoid dendritic cells, to distinct expression pat

275 cer, previously thought to be active only in plasmacytoid dendritic cells, was found to also be trans

276 s, demonstrating that IL-36 acts directly on plasmacytoid dendritic cells, where it potentiates toll-

277 ted Treg cell boost involved TNF, OX40L, and plasmacytoid dendritic cells, whereas in a condition of

278 Moreover, CCL22 induces an influx of plasmacytoid dendritic cells, which correlates with high

279 interplay of Tregs, invariant NKT cells, and plasmacytoid dendritic cells, which results in suppressi

280 LL37 licenses autoreactivity by stimulating plasmacytoid dendritic cells-(pDCs)-Type I interferon (I

281 cell (DC) differentiation, predominantly of plasmacytoid dendritic cells.

282 nduction of type 1 interferon responses from plasmacytoid dendritic cells.

283 ed CLEC4C or CD303) is uniquely expressed on plasmacytoid dendritic cells.

284 thought to initiate disease exacerbation via plasmacytoid dendritic cells.

285 monocytes, conventional dendritic cells, and plasmacytoid dendritic cells.

286 s, with lower expression on conventional and plasmacytoid dendritic cells.

287 nocytes of substances inhibitory or toxic to plasmacytoid dendritic cells.

288 of endothelial and myeloid cells, as well as plasmacytoid dendritic cells.

289 ve CD4 T cells and an increased frequency of plasmacytoid dendritic cells.

290 d interferon-alpha (IFN-alpha) production by plasmacytoid dendritic cells.

291 s again dependent on type I IFNs produced by plasmacytoid dendritic cells.

292 e expression, IFNalpha levels, and activated plasmacytoid dendritic cells; 2) higher proinflammatory

293 nate and innate-like cell subsets, including plasmacytoid dendritic cells; CD1c + myeloid DCs; neutro

294 ways induce IFN-I production in CMV-infected plasmacytoid dendritic cells; however, the initial burst

295 ified a CD34(+) subpopulation primed for the plasmacytoid dendritic lineage, suggesting an intrathymi

296 Blastic plasmacytoid dendritic-cell neoplasm (BPDCN) is an aggre

297 pure squamous, adenocarcinoma, sarcomatoid, plasmacytoid, or micropapillary disease.

298 ral killer (NK) cells and myeloid (mDCs) and plasmacytoid (pDCs) dendritic cells on control of virus

299 conventional (CD11c(high)CD103(high)) DC and plasmacytoid (plasmacytoid DC Ag-1(high)B220(+)CD11c(int

300 breast tumor cells induced the activation of plasmacytoid predendritic cells (pDC), a cell type criti