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1 ifically required for the differentiation of plasmatocytes.
2 d Ush in dpp mutants leads to hyperplasia of plasmatocytes.
3 ivates a class of insect immune cells called plasmatocytes.
4  causes crystal cells to be transformed into plasmatocytes.
5 ytes involved in insect cellular immunity is plasmatocytes.
6 e melanization response, and phagocytosis by plasmatocytes.
7 ify the embryonic functions of another - the plasmatocytes.
8 tide and that other peptidyl factors mediate plasmatocyte adhesion responses.
9 is that Ush participates in a switch between plasmatocyte and lamellocyte fate in a common precursor
10 asmatocyte production, but Dif controls both plasmatocyte and lamellocyte production.
11 phila melanogaster, the lymph gland produces plasmatocytes and crystal cells that are not released un
12  major types of D. melanogaster blood cells, plasmatocytes and crystal cells.
13  and induces myeloproliferative expansion of plasmatocytes and crystal cells.
14                          Here, we use insect plasmatocytes and hemocyte-like Drosophila S2 cells to c
15                                              Plasmatocytes are reduced in number, but still express t
16 d that inhibition of DNA damage signaling in plasmatocytes, as well as JNK or upd3 overactivation, re
17 , the peripodial epithelium and blood cells (plasmatocytes) associated with the developing retina.
18 alysis identified candidate genes regulating plasmatocyte behaviour: pan-plasmatocyte expression of o
19                    We further found that the plasmatocyte calcium burst is suppressed by G1 venom in
20         Finally, by genetically manipulating plasmatocyte calcium levels, we were able to alter fly i
21                                       Mature plasmatocytes can proliferate but it is not known if cry
22 kin in Drosophila larvae, blood cells called plasmatocytes can transform into other classes of blood
23 and a rate-limiting step in immune-activated plasmatocytes controlling JNK-mediated release of the pr
24  cells, whereas mature haemocytes comprising plasmatocytes, crystal cells and lamellocytes are periph
25 tages three types of hemocytes are produced, plasmatocytes, crystal cells, and lamellocytes, and thei
26 es of terminally differentiated blood cells: plasmatocytes, crystal cells, and lamellocytes.
27 itoid wasp venom proteins led us to identify plasmatocyte cytoplasmic calcium bursts as an important
28 clarify the role of these redundant genes in plasmatocyte development.
29 has previously been shown to be required for plasmatocyte development.
30                                              Plasmatocytes differentiate into adhesive lamellocytes t
31 at positively and cell-autonomously controls plasmatocyte differentiation.
32 present a transitory state in prohemocyte to plasmatocyte differentiation.
33                                              Plasmatocytes (Drosophila macrophages) are an important
34 genes regulating plasmatocyte behaviour: pan-plasmatocyte expression of one such gene (Calnexin14D) i
35 udens larvae caused a transient depletion of plasmatocytes from circulation.
36 ic peptide at concentrations >/=2 nM induced plasmatocytes from P. includens to spread on the surface
37  adult hemocytes are phagocytic macrophages (plasmatocytes) from the embryonic lineage that parallels
38 ional phagocytes with many functions, called plasmatocytes in Drosophila and granular cells in other
39 ytes lacking Stat fail to differentiate into plasmatocytes, indicating that Stat positively and cell-
40            The major blood cell type, called plasmatocyte, is small, non-adhesive and phagocytic.
41  have been characterized in the lymph gland: plasmatocytes, lamellocytes, and crystal cells, which ar
42 gland gives rise to three mature cell types: plasmatocytes, lamellocytes, and crystal cells.
43 st closely resemble mammalian myeloid cells: plasmatocytes (macrophage-like cells), crystal cells (in
44 hages, gcm cannot be the sole determinant of plasmatocyte/macrophage differentiation.
45 gcm has a role in the differentiation of the plasmatocyte/macrophage lineage of hemocytes.
46 hes of Drosophila hematopoiesis give rise to plasmatocytes/macrophages and crystal cells.
47  the Drosophila hematopoietic repertoire are plasmatocytes/macrophages and crystal cells.
48 of prohemocytes in the cortical zone induces plasmatocyte maturation in adjacent hemocytes.
49 Further analysis revealed that Wee1 inhibits plasmatocyte maturation through upregulation of Tiggrin
50 ressed in the LG, is a specific regulator of plasmatocyte maturation.
51 ingly, we found that fly immune cells termed plasmatocytes normally undergo a cytoplasmic calcium bur
52 ce mutation of gcm causes only a decrease in plasmatocyte numbers without changing their ability to c
53  like gcm, mutation of gcm2 leads to reduced plasmatocyte numbers.
54  other was active in hemocyte precursors and plasmatocytes only.
55         However, less is known about how the plasmatocyte pool of the LG is established and matures.
56 e required together for the proliferation of plasmatocyte precursors, the expression of Croquemort pr
57                      dl primarily stimulates plasmatocyte production, but Dif controls both plasmatoc
58 -1 and -2 are essential for crystal cell and plasmatocyte production, respectively.
59 ired for the production of crystal cells and plasmatocytes, respectively.
60 uggests that only a certain subpopulation of plasmatocytes responds to the peptide and that other pep
61           To identify residues important for plasmatocyte spreading activity, we bioassayed PSP mutan
62 tured domain of PSP and was found to have no plasmatocyte spreading activity.
63                                              Plasmatocyte spreading peptide (PSP) is a 23-amino acid
64     The structure of the recently identified plasmatocyte spreading peptide from the moth Pseudoplusi
65 d structured domains of PSP are required for plasmatocyte-spreading activity.
66                                              Plasmatocyte-spreading peptide (PSP) is a 23-amino acid
67                     This peptide, designated plasmatocyte-spreading peptide (PSP1), contains 23 amino
68 dentification of enhancer elements labelling plasmatocyte subpopulations, which vary in abundance acr
69  which selectively affects mitochondria-rich plasmatocyte survival and function, leading to melanotic
70 s indicated that this peptide induced 75% of plasmatocytes that were double-labeled by the monoclonal
71 tified distinct cellular responder states in plasmatocytes, the Drosophila macrophages, associated wi
72 ons is accompanied by high concentrations of plasmatocytes, the major hemocyte class in uninfected co
73 With this approach, we discriminate and sort plasmatocytes, the major hemocyte subset, from lamellocy
74 th mRNAs are expressed in granular cells and plasmatocytes, the primary classes of hemocytes involved
75  Ush is expressed in hemocyte precursors and plasmatocytes throughout embryogenesis and larval develo
76 -dependent manner, leading to the failure of plasmatocytes to become activated and migrate toward G1
77 on of Croquemort protein, and the ability of plasmatocytes to convert into macrophages.
78 sia includens that mediates the spreading of plasmatocytes to foreign surfaces.
79 nduces a class of insect immune cells called plasmatocytes to spread on foreign surfaces.
80 d, whereas plasma induced significantly more plasmatocytes to spread.
81 iginate through a Notch-dependent process of plasmatocyte transdifferentiation.
82 d is dominated by a macrophage-like lineage (plasmatocytes), until very recently these cells were con
83 rin mutants exhibit precocious maturation of plasmatocytes, whereas Tiggrin overexpression blocks thi
84 multiple lineages, including macrophage-like plasmatocytes, which comprise the vast majority of matur
85 pomorph larvae had a 35% decreased number of plasmatocytes with a 45% reduced active mitochondrial st