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1 ss a range of scales, but dynamic control of plasmodesmal aperture can change the possible domains of
2                                              Plasmodesmal aperture is controlled by specialised signa
3 describe the molecular characterization of a plasmodesmal-associated protein kinase (PAPK).
4                                              Plasmodesmal-associated protein kinases could play a cen
5 r, the molecular features that determine the plasmodesmal association of PDLP5 or other proteins rema
6 teins have a high affinity for the mesophyll plasmodesmal binding site(s).
7 ough plasmodesmata remains a key question in plasmodesmal biology.
8 ate movement of viral DNA across nuclear and plasmodesmal boundaries, respectively.
9 and PDLP6 results in the overaccumulation of plasmodesmal callose at different cell interfaces, indic
10 ous GLS treatment was sufficient to modulate plasmodesmal callose in WT plants.
11                                   Either the plasmodesmal channels involved in SE/CC unloading are ex
12 vum L.) grains to evaluate the dimensions of plasmodesmal channels involved in sieve element/companio
13 show that regulating callose accumulation at plasmodesmal channels is a common strategy to alter plas
14  interaction between such proteins/RNPCs and plasmodesmal chaperones/receptors.
15 e plasmodesmal plasma membrane, both execute plasmodesmal closure via callose synthesis at the plasmo
16 specially the way in which calcium regulates plasmodesmal closure.
17 restriction of cell-to-cell communication by plasmodesmal closure.
18  BURST OXIDASE HOMOLOGUE D (RBOHD) to induce plasmodesmal closure.
19 ng cylindrical symmetries will underestimate plasmodesmal conductivities.
20 Vd-I) strain from mature guard cells lacking plasmodesmal connections and from in vitro-cultivated me
21 er C(4) dicotyledons also show this enhanced plasmodesmal connectivity and so whether this is a gener
22 ts with callose synthesis inhibitors suggest plasmodesmal connectivity as a potential mechanism for t
23 microchannels, and provide direct proof that plasmodesmal dilation is a prerequisite for the cell-to-
24                                              Plasmodesmal enriched cell fractions and the contents of
25 le of interacting with proteins present in a plasmodesmal-enriched cell wall fraction.
26 e performed on a tobacco (Nicotiana tabacum) plasmodesmal-enriched cell wall protein preparation usin
27                                              Plasmodesmal flux is regulated by a variety of environme
28  interface of C(4) G. gynandra showed higher plasmodesmal frequency compared with closely related C(3
29 o lines previously reported to have impaired plasmodesmal function as well as in wild-type seedlings
30 US6 and CALS7 in the vasculature to regulate plasmodesmal function.
31 pathway in conjunction with the regulator of plasmodesmal gating Plasmodesmata-located protein5.
32            A Cm-PP16 interaction partner, Nt-PLASMODESMAL GERMIN-LIKE PROTEIN1 (Nt-PDGLP1) was identi
33                                          Two plasmodesmal-localized beta-1,3 glucanases (PdBGs) were
34 lin, but not fungal chitin, is mediated by a plasmodesmal-localized Ca(2+) -binding protein Calmoduli
35 feedback circuit that regulates the level of plasmodesmal-localized callose in order to locally downr
36 lysis of GFP mobility, callose staining, and plasmodesmal marker lines in Arabidopsis thaliana and Ni
37 ation of, and protein translocation through, plasmodesmal microchannels, and provide direct proof tha
38 act with a motif involved in the dilation of plasmodesmal microchannels.
39  allows these Hsc70 chaperones to engage the plasmodesmal non-cell-autonomous translocation machinery
40 n-accumulating regions of sxd1 leaves due to plasmodesmal occlusion at the bundle sheath-vascular par
41 xpression of GAT1 in mature leaves increased plasmodesmal permeability and led to a delay in senescen
42 ich different environmental stressors affect plasmodesmal permeability are not well understood.
43                                    Moreover, plasmodesmal permeability is strongly altered by applied
44 esmal channels is a common strategy to alter plasmodesmal permeability under both pathogen infection
45 e regulatory effect of MP phosphorylation on plasmodesmal permeability was host dependent, occurring
46 TED PROTEIN 5 (PDLP5), a potent regulator of plasmodesmal permeability, generates feed-forward or fee
47 r exchange between plant cells determined by plasmodesmal permeability.
48 specialized immune signaling cascades in the plasmodesmal plasma membrane, both execute plasmodesmal
49                               In plants, the plasmodesmal PM is a discrete microdomain that hosts spe
50 in signaling, we found that responses in the plasmodesmal PM require the LysM receptor kinases LYK4 a
51 , but only LYM2 and LYK4 are detected in the plasmodesmal PM.
52 ion protein was located in the centre of the plasmodesmal pore, between paired callose platelets.
53             A 34-kD protein, isolated from a plasmodesmal preparation, exhibits calcium-independent k
54                   The mutations in genes for plasmodesmal proteins have provided valuable genetic too
55 opsis line (pdko3) mutated in genes encoding plasmodesmal proteins is defective in some, but not all,
56 ontrols the spatiotemporal expression of the plasmodesmal regulator PDLP5 in cells overlying LRP, cre
57 at may be transported, and also (co-)defines plasmodesmal resistance to diffusion and convective flow
58 equired for chitin-, flg22- and SA-triggered plasmodesmal responses and PDLP-mediated activation of c
59 red signaling, we profiled the dependence of plasmodesmal responses triggered by different elicitors
60 ction of flowering may represent a change in plasmodesmal selectivity at this time or that a period o
61 -NHL3 complex acts as an integrating node of plasmodesmal signaling cascades, transmitting multiple i
62 iggered by different elicitors on a range of plasmodesmal signaling machinery.
63                                              Plasmodesmal size exclusion limit increased to greater t
64                This system likely involves a plasmodesmal switch that would prevent the dissipation o
65 els of calreticulin severely interfered with plasmodesmal targeting of TMV MP, which, instead, was re
66                                     Notably, plasmodesmal targeting signals exhibit little sequence c
67 nd no protein motifs have been identified as plasmodesmal targeting signals.
68 hese features appear to be a common theme in plasmodesmal targeting.
69 ween cells via PD, can act on PD to regulate plasmodesmal trafficking capacity.
70 t involves supracellular control achieved by plasmodesmal trafficking of informational molecules, her
71                 Callose deposition modulates plasmodesmal transport in vivo, but little is known abou
72                   As the threshold value for plasmodesmal transport of phloem sap proteins falls with
73 netic approach to identify mutants affecting plasmodesmal transport.