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1 domains together with previous studies on a plasmodium CDPK suggests a model whereby even at normall
2 yses of ncRNA from bacteria, human cells and plasmodium-infected reticulocytes, as well as a viral RN
3 popolysaccharide (LPS) for typhoid fever and plasmodium lactate dehydrogenase (pLDH) for malaria infe
4 is question further, we introduced human and plasmodium native and length-matched artificial linkers
7 tudies: 1) comparative genomic study mapping plasmodium gene sequences to corresponding human and mos
12 , by plasmodial coalescence, into an S-phase plasmodium, failed to start another round of DNA synthes
13 second pregnancy) and maternal or placental plasmodium infection at delivery in multigravidae (third
14 review focuses on efforts to identify potent plasmodium-specific aaRS inhibitors using phenotypic scr
17 t immunization with plasmid DNA encoding the plasmodium yoelii circumsporozoite protein protected one
18 xaggerated accumulation of quinolines in the plasmodium digestive vacuole, and suggest that a quinoli
19 the slime mold Physarum polycephalum in the plasmodium state under different environmental condition
21 ile and may process local signals within the plasmodium to coordinate cell growth, metabolism, and re