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1 donor's blood samples (both whole blood and platelet-rich plasma).
2 imilar to that observed in vitro using mouse platelet rich plasma.
3 ergizes with soluble collagen in aggregating platelet-rich plasma.
4 bility to inhibit ADP-induced aggregation in platelet-rich plasma.
5 o study activation of isolated platelets and platelet-rich plasma.
6 counts for approximately 20% of total VWF in platelet-rich plasma.
7 DP-induced aggregation of human platelets in platelet-rich plasma.
8 extracellular protein substrates of ERp57 in platelet-rich plasma.
9 nic acid-induced platelet aggregation in rat platelet-rich plasma.
10 ntent of anandamide in either macrophages or platelet-rich plasma.
12 RPAI-1 inhibits in vitro the ADP-dependent platelet-rich plasma aggregation by collagen (COLL), TRA
14 ibition of aggregation of human platelets in platelet rich plasma, an IC50 of 6.8 nM for the inhibiti
15 orn trypsin inhibitor-treated whole blood or platelet rich plasma and subsequent array scanning via a
16 togenous bone and anorganic bovine bone with platelet-rich plasma and a bioabsorbable collagen membra
17 n of intra-articular cell therapies (such as platelet-rich plasma and bone marrow aspirate concentrat
18 telet aggregation in response to collagen in platelet-rich plasma and firm adhesion on VWF under arte
21 sional injections of autologous concentrated platelet-rich plasma and minoxidil showed some benefit i
22 ain antioxidants, anti-inflammatory drugs or platelet-rich plasma and new synthetic synovial fluid ad
23 arkedly enhanced thrombin generation in both platelet-rich plasma and platelet-poor plasma, indicatin
26 ibition of platelet aggregation in the human platelet rich plasma assay with IC(5)(0) values below 50
27 tivates human VWF and induces aggregation of platelet-rich plasma at submicromolar concentrations.
28 hat fails to inhibit platelet aggregation in platelet-rich plasma, blocked the inhibitory effect of t
32 itro Abeta40 also increased the stiffness of platelet-rich plasma clots in the presence of FXIIIa.
33 nt with a single injection of intratendinous platelet-rich plasma, compared with insertion of a subcu
34 llograft (ADM) to that of a CPT plus ADM and platelet-rich plasma (CPT/PRP) 4 months post-surgically.
37 groups: demineralized freeze dried bone with platelet-rich plasma (DFDB + PRP), DFDB alone, and no tr
38 se of autologous fibrin membrane and the eye platelet-rich plasma (E-PRP) clot could be considered as
39 ncoated stents were then immersed in porcine platelet-rich plasma for two min and the platelet cyclic
43 ated primary glomerular endothelial cells to platelet-rich plasma from patients, or patient platelet-
44 hibited when compared to the wild-type using platelet-rich plasma from the principal donor, but adhes
45 eatment of control neutrophils with COVID-19 platelet-rich plasma generated TF-bearing NETs that indu
46 erse events compared between patients in the platelet-rich plasma group vs the sham group were inject
47 luorescence spectroscopy) on whole blood and platelet-rich plasma indicate that exogenous alphaSyn ha
49 40 people from 24 sites assigned to either a platelet-rich plasma injection or a sham injection betwe
50 ment protocols, and clinician credentials of platelet-rich plasma injection therapy for erectile dysf
51 dinopathy, to assess the effects of a single platelet-rich plasma injection, compared with sham injec
58 of blood microparticles (MPs) obtained from platelet-rich plasma of healthy individuals was characte
59 anufacture of whole-blood-derived platelets (platelet-rich plasma or buffy coat intermediate steps) r
61 Shear-induced GPVI shedding also occurred in platelet-rich plasma or washed platelets isolated from a
64 nts or sample volumes can be used, either of platelet-rich plasma or whole blood from human subjects
65 exposure upon agonist-induced activation in platelet-rich plasma or whole blood whereas LPS(SM) and
67 ormed in platelets, platelet microparticles, platelet-rich plasma, platelet-poor plasma, and serum.
69 to assess the efficacy of the injections of platelet rich plasma (PRP) for the treatment of severe d
70 th PMN and PMN-RBC combinations suspended in platelet rich plasma (PRP) than in platelet poor plasma
71 played inhibition of platelet aggregation in platelet rich plasma (PRP) with an IC(50) value of 53 nM
75 as to evaluate the regenerative influence of platelet-rich plasma (PRP) added to xenogenic bone graft
76 ion of CD62P by wild-type mouse platelets in platelet-rich plasma (PRP) and caused their secretion of
77 against PAR4 activation by gamma-thrombin in platelet-rich plasma (PRP) and greater than 2500-fold se
80 lyze the content and specific effect of both platelet-rich plasma (PRP) and platelet-poor plasma (PPP
84 encouraging results reported with regards to Platelet-rich plasma (PRP) application in osteoarthritis
85 Autologous blood-derived products such as platelet-rich plasma (PRP) are widely used to treat musc
86 with an amniotic membrane graft (AMG) and a platelet-rich plasma (PRP) clot, and the anterior chambe
87 nhanced by 127% (P < 0.001) in study patient platelet-rich plasma (PRP) compared to control PRP and c
90 lcium sulfate hemihydrate (MGCSH) mixed with platelet-rich plasma (PRP) for extraction socket preserv
91 Most clinical guidelines do not recommend platelet-rich plasma (PRP) for knee osteoarthritis (OA)
93 rombin burst in response to tissue factor in platelet-rich plasma (PRP) from patients with mild or mo
96 e mineral (NBM) with and without addition of platelet-rich plasma (PRP) has been shown to result in s
98 ch surrounding intra-articular injections of platelet-rich plasma (PRP) has not produced clear eviden
99 hput microfluidic removal of leukocytes from platelet-rich plasma (PRP) in a continuous flow regime.
100 of autologous platelet-rich fibrin (PRF) and platelet-rich plasma (PRP) in the treatment of intrabony
101 fective nonsurgical interventions exist, but platelet-rich plasma (PRP) injections are widely used, w
102 al benefits provided by the incorporation of platelet-rich plasma (PRP) into a regenerative protocol
109 of recalcified platelet-free plasma (PFP) or platelet-rich plasma (PRP) supplemented with corn trypsi
110 al intrabony defects, the benefits of adding platelet-rich plasma (PRP) to a bone replacement graftin
111 plored through a spectrophotometric assay on platelet-rich plasma (PRP) treated with the thromboxane
116 aggregation tests (PATs) were performed with platelet-rich plasma (PRP), a shorter lag time was measu
118 Autologous bone marrow concentrate (BMC), platelet-rich plasma (PRP), and platelet lysate (PL) hav
122 tomorphometrically analyzes the influence of platelet-rich plasma (PRP), low-level laser therapy (LLL
123 ractionated to yield red blood cells (RBCs), platelet-rich plasma (PRP), platelet-poor plasma (PPP),
126 ng of recombinant human TPO (rhTPO) to human platelet-rich plasma (PRP), washed platelets (WP), and c
132 specifically in VAT, in addition to elevated platelet-rich-plasma (PRP) NPY, compared to control fema
133 ogic agent (EMD, platelet-rich fibrin [PRF], platelet-rich plasma [PRP] or rhPDGF-BB) played a signif
134 12-hydroxyeicosatetraenoic acid to P-12LO-/- platelet-rich plasma rescues the hyperresponsive phenoty
135 Platelet aggregation assays with citrated platelet-rich plasma reveal that the primary and seconda
136 d at baseline in increasing proportions with platelet-rich plasma sampled 4 hours after loading dose.
137 ation, including the use of gene therapy and platelet-rich plasma scaffolds, are also discussed.
138 ing, hydrogel-coated PVC membranes soaked in platelet-rich plasma showed less adhesion and activation
140 ctor Xa-inhibited (250 nM apixaban), diluted platelet rich plasma that had been loaded with the calci
141 ld be prevented by simply reducing the pH of platelet-rich plasma to about 6.5 prior to centrifugatio
142 erous modifications from whole blood-derived platelet-rich plasma to apheresis-derived platelet conce
143 Using gel-filtered platelets derived from platelet-rich plasma treated with alpha-tocopherol (500
144 r, second-wave aggregation induced by MDC in platelet-rich plasma was inhibited by aspirin, ADP scave
147 the coagulation cascade in platelet poor or platelet-rich plasma, when activation was initiated by t
149 roparticles in megakaryocyte cultures and in platelet-rich plasma, which are predominantly derived fr
150 atelets derived from apheresis compared with platelet-rich plasma whole-blood-derived platelets.
152 ma cofactor, because a 35-hour incubation of platelet-rich plasma with 125I-factor V showed no specif