ライフサイエンスコーパス: plead

1 d three response regulators, one of which is PleD.

2 protein that contains a GGDEF domain, called PleD.

3 onstrate that not only do these HTLs enhance PLED anode hole injection but they also exhibit signific

4 Covalent chemical bonding of TPDSi(2) to PLED anodes (e.g., indium tin oxide, ITO) and its self-c

5 1.2%, suggesting their potential for use in PLED applications.

6 PLED devices fabricated using these new HTLs exhibit com

7 PLED devices with luminance response times of microsecon

8 This plead for a systematic vascular axis imaging after venov

9 against the "classical") concept of SIBO and plead for caution in the application of breath tests, an

10 rization of pathways that regulate HNF1B and plead for considering individually tailored graft manage

11 We plead for more time for hard-pressed college professors

12 together with the presence of immune cells, plead for the existence of a HIV-1 reservoir in the ging

13 While some politicians pleaded for Americans to unite against political violenc

14 ith a poorer prognosis than paramyxoviruses, pleading for a broader and large-scale vaccination of in

15 This article pleads for scientific, health policy and editorial commu

16 International (London, United Kingdom), each pled guilty to one violation of the Prescription Drug Ma

17 ITO substrates precoated with a conventional PLED HTL, poly(3,4-ethylenedioxythiophene)-poly(styrenes

18 abrication of three generations of insoluble PLED HTLs: (1) self-assembled monolayers (SAMs) of TPDSi

19 y have tended to avoid nutritional problems, plead ignorance of nutrition principles, and delegate th

20 Altogether, these findings plead in favor of a broad age-promoting effect of ACBP a

21 fees, citizen juries, and the nature of tort pleading in the United States.

22 Prior to this study, the roles of PleC and PleD in the obligatory intracellular parasitism of A. ph

23 tion pathways mediated by PleC-DivK and DivJ-PleD in the regulation of chemotaxis as well as motility

24 raction, whereas it detected NtrY, NtrX, and PleD in the soluble fraction.

25 on, we find an experimental versus predicted pLED (lowest effective dose) R(2) = 0.79 for 35 bisphosp

26 t of pleC mutants is suppressed by a pleD301/pleD+ merodiploid and results in a similar, supermotile,

27 multiple transcription factors (RpoH, VisN, PleD, MucR, MocR and CtrA) at both transcriptional and/o

28 Analysis of the pleD mutant cell cycle demonstrates that disruption of t

29 essed by the sokA301 allele of ctrA and in a pleD mutant, both of which display a supermotile, stalkl

30 Analysis of PleC and PleD mutants also provides insights into the function of

31 The cohort of defendants who pleaded not criminally responsible in both the circuit a

32 imore City's circuit and district courts who pleaded not criminally responsible, Maryland's version o

33 nt regulatory systems (TCSs) designated PleC-PleD, NtrY-NtrX, and CckA-CtrA based on amino acid seque

34 be applied as a general strategy to enhance PLED performance.

35 A recombinant PleD (rPleD) has autokinase activity using phosphorylate

36 Recombinant E. chaffeensis PleD showed diguanylate cyclase activity as it generated

37 results suggest that the two-component PleC-PleD system is a diguanylate cyclase and that a c-di-GMP

38 ith mutations in the response regulator gene pleD that block the loss of motility.

39 We therefore plead to assess nontarget effects of the complete nanoca

40 smembrane helix of PilA to activate the PleC-PleD two-component signaling system, increase cellular c

41 he CoMFA predicted (rat) bone antiresorptive pLED values are in agreement with literature (human reco

42 were used as training sets for the predicted pLED values for two sets of 4 compounds which have an rm

43 factors by demonstrating that both pleC and pleD were expressed in an HGA patient.

44 in human promyelocytic HL-60 cells, PleC and PleD were synchronously upregulated at the exponential g

45 In the present study, we found that PleC and PleD were upregulated synchronously during exponential g

46 ase, PleC, and a cognate response regulator, PleD, which can produce c-di-GMP.