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1 e heparin-binding growth factors midkine and pleiotrophin.
2 evelopmentally regulated neurotrophic factor pleiotrophin.
3 cells expressed hepatocyte growth factor and pleiotrophin.
4 otentiated programmed cell death elicited by pleiotrophin.
5 rfering RNA-mediated knockdown of endogenous pleiotrophin.
6 ular matrix components and the growth factor pleiotrophin.
7    The identification of the novel biomarker pleiotrophin 151-166 demonstrates that our quantificatio
8                            Here we show that pleiotrophin, a neurite outgrowth factor with no known f
9 nase (ALK) as a tyrosine kinase receptor for pleiotrophin, a secreted growth factor that is highly ex
10                             Mechanistically, pleiotrophin activated phosphoinositide 3-kinase (PI3K)
11 e cancer cells by inducing EMT and promoting pleiotrophin activity through the syndecan-3 pathway.
12        Here we report on the contribution of pleiotrophin-ALK signaling to glioblastoma growth.
13                                              Pleiotrophin alone was also found to induce the formatio
14 ealed the neurite outgrowth-promoting factor pleiotrophin, along with required binding partners SPARC
15                                              Pleiotrophin also increased wound healing of injured typ
16 cord blood CD34(+)CDCD38(-)Lin(-) cells with pleiotrophin also substantially increased severe combine
17                                We found that pleiotrophin and its receptor, protein-tyrosine phosphat
18                                  The role of pleiotrophin and its receptors RPTPbeta/zeta and Syndeca
19 any published reports focused on the role of pleiotrophin and its receptors, receptor protein tyrosin
20      Elucidating the molecular mechanisms of pleiotrophin and midkine action in tumorgenesis and tumo
21                                              Pleiotrophin and midkine are both developmentally regula
22     The heparin-binding polypeptide homologs pleiotrophin and midkine are the only known members of a
23                   Constitutive expression of pleiotrophin and midkine in responsive cells support the
24                   Widespread deregulation of pleiotrophin and midkine is found in many known human ca
25                       Moreover, two markers, pleiotrophin and TNFRSF9, predict poor disease survival
26                             Mechanistically, pleiotrophin antagonizes IGF-1 associated Ser-473 phosph
27       We identify the secreted growth factor pleiotrophin as a new regulator of both HSC expansion an
28 ion of multiple signaling pathways including pleiotrophin, beta-catenin, and Notch pathways.
29 was identified as a downstream target of the pleiotrophin/beta-catenin pathway by endogenous dlk1 exp
30     Treatment of mouse bone marrow HSCs with pleiotrophin caused a marked increase in long-term repop
31                              We suggest that pleiotrophin could act as a key mesenchymally derived fa
32                                        Thus, pleiotrophin expression in NIH 3T3 cells is associated w
33                                              Pleiotrophin expression is strongly enriched in the SVZ,
34 re has a distant relationship to the midkine/pleiotrophin fold, particularly in the conservation of d
35 d to apparent homogeneity an 18 kDa protein, pleiotrophin, from the conditioned medium of a metanephr
36 OXA5, we initiated an investigation into the pleiotrophin gene by first cloning its promoter.
37                                              Pleiotrophin has previously been shown to induce express
38 iety of known UB branching morphogens (i.e., pleiotrophin, heregulin, FGF1 and GDNF) were found to ha
39 , WT1, BCL2, Homeobox protein A11, timeless, pleiotrophin, HGF, HNF3, BMP4, TGF-alpha, TGF-beta2, IGF
40 d resistance, mTOR in innate resistance, and pleiotrophin in both settings, suggesting their utility
41                                  The role of pleiotrophin in fetal lung development was investigated.
42 mportant for the expression of growth factor pleiotrophin in human choriocarcinoma cells.
43          In addition, MTA and HICR repressed pleiotrophin in PDL fibroblasts, while HICR repressed pe
44                                              Pleiotrophin, in complex with the binding partners, acti
45                                              Pleiotrophin increased the tyrosine phosphorylation of b
46                                 Knockdown of pleiotrophin influenced lung branching morphogenesis in
47              These observations suggest that pleiotrophin inhibition of PTPRZ1 contributes to the hom
48                                              Pleiotrophin is a development-regulated cytokine and gro
49                                              Pleiotrophin is a growth factor that induces carcinogene
50 nd progenitor cells in vivo, indicating that pleiotrophin is a regenerative growth factor for HSCs.
51                                              Pleiotrophin is directly angiogenic; it initiates an ang
52 ion and in human dilated cardiomyopathy that pleiotrophin is markedly up-regulated.
53 sociated with quiescence, and suppression of pleiotrophin is related to oncogenic transformation.
54                                 We show that pleiotrophin is released from cardiomyocytes in vitro in
55     Additionally, we found that the cytokine pleiotrophin is sufficient to induce VEGFR2 expression o
56 ression is strongly enriched in the SVZ, and pleiotrophin knock down starkly reduced glioma invasion
57  but was not associated with increased serum pleiotrophin levels.
58 urve = 0.96) was identified as a fragment of pleiotrophin located near the protein's C-terminus.
59 ition of syndecan-3 expression inhibited the pleiotrophin-mediated cell migration and attachment thro
60 agonism of PI3K or Notch signaling inhibited pleiotrophin-mediated expansion of HSCs in culture.
61 bition of RPTPbeta/zeta expression increased pleiotrophin-mediated migration and attachment through a
62 Pbeta/zeta knockdown initiates EMT, promotes pleiotrophin-mediated migration and attachment through S
63 trate that RPTPbeta/zeta counterbalanced the pleiotrophin-mediated syndecan-3 pathway.
64 stational ages 16-22 weeks, we asked whether pleiotrophin modulated the expansion of OPCs and, if so,
65 PZ was accompanied by that of its modulators pleiotrophin, NrCAM, tenascin, and the chondroitin sulfa
66                  To elucidate the effects of pleiotrophin on cardiac contractile cells, we employed p
67 sion, we have uncovered a novel function for pleiotrophin on heart cells following injury.
68              These latter data indicate that pleiotrophin potentiates cardiomyocyte cell death, at le
69 ithelial type II cells, we demonstrated that pleiotrophin promoted fetal type II cell proliferation a
70 ted that HOXA5 can specifically activate the pleiotrophin promoter.
71 can directly bind to one binding site on the pleiotrophin promoter.
72 hypoxia and that the addition of recombinant pleiotrophin promotes caspase-mediated genomic DNA fragm
73 7230]; P = 3.98 x 10(-)(1)(2)), the cytokine pleiotrophin (PTN [rs919581]; P = 5.38 x 10(-)(4)), the
74                                              Pleiotrophin (PTN the protein and Ptn the gene) is a lig
75                        In contrast, mRNA for pleiotrophin (PTN) and glial cell line-derived neurotrop
76 ecently demonstrated that the growth factors pleiotrophin (PTN) and midkine (MK) are ligands for ALK
77  CML stem cells upregulate the expression of pleiotrophin (PTN) and require cell-autonomous PTN signa
78  is inserted in the human growth factor gene pleiotrophin (PTN) between the 5' untranslated and the c
79                   The secreted growth factor pleiotrophin (PTN) can induce mitogenesis in cells that
80 y reported that the angiogenic growth factor pleiotrophin (PTN) coaxes monocytes to assume the phenot
81                                          The pleiotrophin (PTN) gene (Ptn) is a potent proto-oncogene
82 stitutively high levels of expression of the pleiotrophin (Ptn) gene are found in human breast cancer
83  element (HERV-E.PTN) into the growth factor pleiotrophin (PTN) gene generated a phylogenetically new
84 d 495 nucleotides is inserted into the human pleiotrophin (PTN) gene upstream of the open reading fra
85          Herein we use ribozyme targeting of pleiotrophin (PTN) in metastatic human melanoma cells to
86                                              Pleiotrophin (PTN) is a developmentally regulated protei
87                                              Pleiotrophin (PTN) is a heparin-binding, 18 kDa secretor
88                                              Pleiotrophin (PTN) is a multifunctional, cationic, glyco
89                                              Pleiotrophin (PTN) is a platelet-derived growth factor-i
90                                              Pleiotrophin (PTN) is a recently described 18- kDa hepar
91                                              Pleiotrophin (PTN) is a secreted growth factor that indu
92                                              Pleiotrophin (PTN) is an 18-kDa heparin-binding secretor
93                                              Pleiotrophin (PTN) is an angiogenic factor that is produ
94                                              Pleiotrophin (PTN) is an important developmental cytokin
95                   The secreted growth factor pleiotrophin (PTN) promotes glioblastoma migration and p
96 In this study, we analyzed the growth factor pleiotrophin (PTN) that was originally described as a de
97 Herein, we report that TAMs secrete abundant pleiotrophin (PTN) to stimulate glioma stem cells (GSCs)
98                                Growth factor pleiotrophin (PTN) was expressed at higher levels in non
99                                              Pleiotrophin (PTN) was found to regulate tyrosine phosph
100              We discovered that the mRNA for pleiotrophin (PTN) was highly up-regulated in acutely de
101                                  Among them, pleiotrophin (PTN) was identified as the paracrine effec
102                                We found that pleiotrophin (PTN), a neurotrophic cytokine, is a metast
103 was associated with loss of pericyte-derived pleiotrophin (PTN), a neurotrophic growth factor.
104 re, we found that systemic administration of pleiotrophin (PTN), a protein that is secreted by BM-der
105 ), brain-derived neurotrophic factor (BDNF), pleiotrophin (PTN), and NT-3 in asymmetrically guiding t
106                                     Midkine, pleiotrophin (PTN), and their receptors syndecan-3 and r
107 nase (ALK) and its ligand, the growth factor pleiotrophin (PTN), are highly expressed during the deve
108    Particularly, we found that the excessive Pleiotrophin (PTN), released by dysregulated qNSCs, is a
109 pression of a heparin binding growth factor, pleiotrophin (Ptn), was found to be up-regulated in Pten
110 trating that LV-neural progenitors secrete a pleiotrophin (PTN)-containing complex, which attracts gl
111 f several angiogenic growth factors, such as pleiotrophin (PTN).
112 atasezeta (RPTPzeta) and one of its ligands, pleiotrophin (Ptn).
113 ulated, secreted growth factor homologous to pleiotrophin (PTN).
114 rn overlapping but distinct from its homolog pleiotrophin (PTN).
115                                              Pleiotrophin (PTN, Ptn) is an 18-kDa secretory cytokine
116 amino acid secreted heparin-binding cytokine pleiotrophin (PTN, Ptn) stimulates both normal and patho
117 e the physiological receptor of the cytokine pleiotrophin (PTN, Ptn).
118          These results provide evidence that pleiotrophin regulates fetal type II cell proliferation
119   To investigate the mechanism through which pleiotrophin regulates tumor metastasis, we used two dif
120 argeting and demonstrated that this prevents pleiotrophin-stimulated phosphorylation of the anti-apop
121                             Midkine, but not pleiotrophin, stimulates tyrosine phosphorylation of sev
122                                 In contrast, pleiotrophin strongly potentiated the proliferation of C
123 eta/zeta (PTPRZ1) and its inhibitory ligand, pleiotrophin, suggesting the maintenance of an autocrine
124 d cell lines, and the molecular targeting of pleiotrophin to block its signaling in tumor cells has l
125                   Systemic administration of pleiotrophin to irradiated mice caused a pronounced expa
126 expression of the gene encoding the cytokine pleiotrophin was also found in Catnb(flox(ex3)/+);Amhr2(
127 bud morphogenesis during kidney development, pleiotrophin was found to localize to the basement membr
128                      In further experiments, pleiotrophin was undetectable or almost undetectable in

 
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