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1 ing hemorrhage, major polytrauma, burns, and poisoning).
2  chronic liver disease, and drug and alcohol poisoning.
3 ant isoleucyl tRNA synthase, preventing self-poisoning.
4 eam inflammatory and oxidative effects of CO poisoning.
5 old pesticide storage reduces pesticide self-poisoning.
6  patients with severe chloroquine or quinine poisoning.
7 oach for adults with calcium channel blocker poisoning.
8  a reduction in blood oxygen level and acute poisoning.
9  containers for prevention of pesticide self-poisoning.
10 ropriate flushing procedures to prevent lead poisoning.
11 ributing to the lack of heterocycle catalyst poisoning.
12  predictive of mortality in chloroquine self-poisoning.
13 or storage and prevention of accidental self-poisoning.
14 resenting an established plasma biomarker of poisoning.
15 d musculoskeletal diseases, and injuries and poisoning.
16 , driven by mental illness and injury and/or poisoning.
17 ate to treat or prevent organophosphate (OP) poisoning.
18 se as a first specific treatment for sulfide poisoning.
19 emission, raising the possibility of (210)Po poisoning.
20 pylobacter jejuni is the major cause of food poisoning.
21 pal neurogenesis after acute carbon monoxide poisoning.
22 , and no specific therapy exists for sulfide poisoning.
23 marine food webs and leads to ciguatera fish poisoning.
24 uring the first 7 days after carbon monoxide poisoning.
25  initiated urgently in the context of severe poisoning.
26 formation on both nanocatalysis and catalyst poisoning.
27 e in the treatment of patients with methanol poisoning.
28 icant advance for treating and preventing CO poisoning.
29  drug therapy against acute organophosphorus poisoning.
30 real removal in the case of severe metformin poisoning.
31  alone kills at 20 mM, CN must potentiate HP poisoning.
32 gnostic marker of death after acute paraquat poisoning.
33 ns for extracorporeal treatment in metformin poisoning.
34 e for bacterial infectious diseases and food poisoning.
35 ons for extracorporeal treatment in methanol poisoning.
36  episodes of opioid overdose and nerve agent poisoning.
37 se of extracorporeal membrane oxygenation in poisoning.
38  suggesting a complicated mechanism of water poisoning.
39  cytotoxic activity through topoisomerase II poisoning.
40 asive and rapid diagnostic tool for methanol poisoning.
41 rse the clinical and molecular effects of CO poisoning.
42  (CO) remains the most common cause of human poisoning.
43 more, Ir(1)/CN displays high tolerance to CO poisoning.
44 s of extracorporeal membrane oxygenation for poisoning.
45 lation and protection to FeS enzymes from Cu poisoning.
46  was notably reduced due to prevention of CO poisoning.
47 st genetic hyperoxaluria and ethylene glycol poisoning.
48 ere complications such as infection and food poisoning.
49 ed foods such as fish is known to cause food poisoning.
50 s the dilemma between reactivity and anti-CO poisoning.
51  activity, high overpotential, and severe CO poisoning.
52 nation was used for metabolic or hematologic poisonings.
53 to 19 years who were hospitalized for opioid poisonings.
54 le for thousands of accidental and malicious poisonings.
55 treatment is recommended in severe metformin poisoning (1D).
56 e-treated control, (2) acute carbon monoxide poisoning, (3) acute carbon monoxide poisoning followed
57 l membrane oxygenation than those with other poisonings (49% vs 72%; p = 0.004).
58 ent neurological and affective effects of CO poisoning, a portion of survivors still have substantial
59 d Ir) nanowires (NWs) with excellent anti-CO-poisoning ability and high activity.
60                                         Drug poisoning accounted for 59.4% of acts but only 13.5% of
61                               Pesticide self-poisoning accounts for 14-20% of suicides worldwide.
62 ited enhanced resistance to sintering and CO poisoning, achieving an order of magnitude increase in s
63  position for topoisomerase IB (Top1) enzyme poisoning activity and cytotoxicity to human cancer cell
64  the 3-nitro group with minimal loss of Top1 poisoning activity.
65                         Carbon monoxide (CO) poisoning affects 50,000 people a year in the United Sta
66 n barrier and triggers lethal phenethylamine poisoning after monoamine oxidase inhibitor administrati
67 : self-injury alone pound753 (SD 2061), self-poisoning alone pound806 (SD 1568), self-poisoning and s
68       1150 (70%) presentations were for self-poisoning alone, 367 (22%) for self-injury alone, and 13
69     Similarly, relative to self-harm by self-poisoning alone, attempted hanging or asphyxiation (adju
70 elative to hospital presentations after self-poisoning alone, presentations involving both self-injur
71                              Ethylene glycol poisoning also results in hyperoxaluria promoting acute
72                                      Mercury poisoning and "hot filtration" experiments ruled out lea
73 um perfringens enterotoxin (CPE) causes food poisoning and antibiotic-associated diarrhea.
74 rfringens enterotoxin (CPE) cause human food poisoning and antibiotic-associated diarrhea.
75 certain dietary exposures or ethylene glycol poisoning and are a well known cause of AKI.
76 h, promising for rapid screening of methanol poisoning and assessment of severity.
77 arrying enterotoxin A gene (sea) causes food poisoning and cannot be distinguished from non-pathogeni
78 idium perfringens is a leading cause of food-poisoning and causes avian necrotic enteritis, posing a
79 , low selectivity, poor durability, impurity poisoning and fuel crossover effects, and detrimental ef
80 ides new perspectives for the design of anti-poisoning and highly effective catalysts.
81 s an effective strategy to circumvent the CO poisoning and improve the performance of CO(2) reduction
82 V) in people who have recovered from arsenic poisoning and in patients with acute promyelocytic leuke
83 nal pain may be a presenting symptom of lead poisoning and is often difficult to diagnose.
84  Bacillus cereus a pathogen that causes food poisoning and life-threatening wound infections, secrete
85 tinction, condors remain compromised by lead poisoning and limited genetic diversity.
86 med to evaluate the association between lead poisoning and liver fibrosis as well as the change in th
87 ponders to distinguish methanol from ethanol poisoning and monitor in real time the subsequent hospit
88 Addition of detergent in milk can cause food poisoning and other complications.
89 ylactic therapeutics against organophosphate poisoning and other important targets.
90 a subclass of PFTs widely implicated in food poisoning and other related infections.
91 nal bicyclopentadione) mediate topoisomerase poisoning and possibly many other activities of curcumin
92  favour the adsorption of O2, alleviating CO poisoning and promoting the catalysis.
93 O(2) and NO movements averts irreversible NO poisoning and reductive inactivation of the enzyme.
94 (upper gastrointestinal haemorrhage, alcohol poisoning and related head injury, and a completed suici
95                                         Self-poisoning and self-injury are associated with a high ris
96 elf-poisoning alone pound806 (SD 1568), self-poisoning and self-injury pound987 (SD 1823; p<0.0001).
97 causes the symptoms of common bacterial food poisoning and several non-foodborne human gastrointestin
98  sulfate species responsible for the surface poisoning and shows how this product can be obtained fro
99 10)Po) gained widespread notoriety after the poisoning and subsequent death of Mr Alexander Litvinenk
100 e show a clear relationship between red kite poisoning and the decline of its breeding population in
101 on and blooms in people recovering from food poisoning and Vibrio cholerae infections.
102                      Increases in fatal drug poisonings and hepatitis C infections associated with th
103 of heterogeneous DNA damage that causes Top2 poisoning, and indicate that evaluation of Tdp2 status m
104 oints were highest for deaths due to injury, poisoning, and other external causes, in both men (7.89;
105 CN exhibits greatly enhanced tolerance to CO poisoning, and Rh atoms in SA-Rh/CN resist sintering aft
106 , recent history of intentional use for mass poisoning, and the absence of a known antidote raise pub
107 dministration as an antidote for (134/137)Cs poisoning, and use for (131/137)Cs radiotherapy (brachyt
108  saw increases in mortality from suicide and poisonings, and an overall increase in external cause mo
109                    Early symptoms of (210)Po poisoning are indistinguishable from those of a wide ran
110 re, curing is robust and not as sensitive to poisoning as other silicone elastomer formulations.
111 ratio=15.86, 95% CI=10.7-23.4; computed with poisoning as reference), and to a lesser extent after ev
112 edicinal use in the treatment of heavy metal poisonings as well as having potency for reducing inflam
113 ) catalyst that severely suffers from oxygen poisoning at high O(2) /C(3) H(8) ratios.
114          In adolescents aged 15 to 19 years, poisonings attributed to heroin were also identified.
115                  Main Outcomes and Measures: Poisonings attributed to prescription opioids were ident
116 ciated with several diseases, including food poisoning, bacterial arthritis, and toxic shock syndrome
117 e theoretical basis behind carbon and sulfur poisoning, before examining the strategies toward carbon
118                     In the absence of severe poisoning but if the methanol concentration is elevated
119 cyanide antidote in animal models of cyanide poisoning, but it is unstable in solution and poorly abs
120 riggered by Top2 engagement of DNA damage or poisoning by anticancer drugs.
121 s O2 , and Ag/Pt surface proximity disfavors poisoning by CO or oxidized species.
122 eaction will be inhibited because of surface poisoning by CO.
123                   HQNO induces specific self-poisoning by disrupting the flow of electrons through th
124 , moderate and severe acute organophosphorus poisoning by exposing zebrafish larvae to different conc
125 euRS), while the TM84-producer prevents self-poisoning by expressing a resistant LeuRS AgnB2.
126 ilizing these nanoscale Ni catalysts against poisoning by interactions with the BN substrate.
127 antagonists is most commonly associated with poisoning by or surreptitious ingestion of warfarin, war
128 emory impairment after acute carbon monoxide poisoning by preserving adult neurogenesis via an increa
129 ever no efficient and approved treatment for poisoning by ricin inhalation, although there have been
130             These materials are resistant to poisoning by small reactive gases (CO and H2S) that may
131       Pt is notorious for its sensitivity to poisoning by strong CO adsorption.
132  response to concerns for wide-scale mercury poisoning by these activities.
133  distribution across all other methods, self-poisoning (by medication) (25%) was significantly more l
134  all causes (by race and ethnicity) and drug poisoning, by county, for the periods of 2000-03 and 201
135  risk of future opioid dependence, abuse, or poisoning can be predicted in advance of an initial opio
136                                     Methanol poisoning can induce death and disability.
137                              Organophosphate poisoning can occur from exposure to agricultural pestic
138 s and those that result from ethylene glycol poisoning, can result in end-stage renal disease.
139 phimurium (ST), a major source of human food poisoning, caused inflammation of murine intestinal tiss
140 s/porous nanomaterials through intentionally poisoning certain MNPs using suitable modifiers.
141 butable to accidental deaths (primarily drug poisonings), chronic liver disease and cirrhosis, and su
142 d steps, leading to the abrupt desorption of poisoning CO from all crystal facets at the same tempera
143 es to prevent suicides due to pesticide self-poisoning, compared with a null comparator.
144 SAAs also offer reduced susceptibility to CO poisoning, cost savings from reduced precious metal usag
145 especially for young people and against self-poisoning, could potentially lower future costs at a tim
146 uicides and mischaracterization of most drug poisoning deaths as "accidents" on death certificates.
147 has been largely driven by increases in drug poisoning deaths.
148 e control group; incidence of pesticide self-poisoning did not differ between groups (293.3 per 100 0
149 entific questions can help avert future lead poisoning disasters.
150 ISPR/Cas9-mediated chromosome cutting, or by poisoning DNA decatenation resulted in mistiming of MapZ
151 ined all-cause mortality (ACM), drug-related poisoning (DRP) mortality, and mortality not attributabl
152 posed to hospital admissions for injuries or poisonings due to self-harm, interpersonal violence, or
153 e admitted to hospital following injuries or poisonings during their childhood.
154 ive role of electride support where the H(2) poisoning effect is absent during ammonia synthesis over
155 ed directing group capable of overriding the poisoning effect of a wide range of heterocycle substrat
156 ymer layer as a transducer did not show this poisoning effect.
157 tter tolerance for methanol crossover and CO poisoning effects, long-term durability than commercial
158  redispersion process removes impurities and poisoning elements, yet is able to maintain the integrit
159     Most suicides occur long after the index poisoning, emphasizing the importance of longitudinal, s
160 mental remediation to address childhood lead poisoning epidemic due to artisanal gold mining in Zamfa
161                                 A first self-poisoning episode is a strong predictor of subsequent su
162                         Patients with a self-poisoning episode were also more likely to die because o
163   For each individual with a deliberate self-poisoning episode, we randomly selected 1 control from t
164 ears) who were discharged after a first self-poisoning episode.
165 der age, male sex, multiple intervening self-poisoning episodes, higher socioeconomic status, depress
166 illed medication prescriptions, and nonfatal poisoning events during the 30 days and 12 months before
167                                     Based on poisoning experiments, we propose that the microbially m
168             The Extracorporeal Treatments in Poisoning (EXTRIP) workgroup sought to determine the eff
169 ns, especially in the setting of intentional poisoning, fire-related toxic gas exposures, and inhalat
170 onoxide poisoning, (3) acute carbon monoxide poisoning followed by 7-day hyperbaric oxygen treatment,
171 B-Fc) chimera, and (5) acute carbon monoxide poisoning followed by intracerebroventricular infusion o
172 ectively subvert innate immune-mediated zinc poisoning for systemic spread.
173 ive OD were identified using ICD-9 codes for poisoning from opioids or adverse effects from opioids.
174                        Whether cancers avoid poisoning from oxidized nucleotides by preventing incorp
175                           In this age group, poisonings from heroin increased from 0.96 (95% CI, 0.75
176  years or older, 611 cases of pesticide self-poisoning had occurred by 3 years in the intervention gr
177                   We developed a model of CO poisoning in anesthetized and mechanically ventilated mi
178 hot pellets constitutes the main cause of Pb poisoning in avifauna.
179 letal muscle cytochrome c oxidase to sulfide poisoning in ethylmalonic encephalopathy, resulting from
180 ance against both carbon build-up and sulfur poisoning in hydrocarbon fuels.
181 lated role in preventing magnetite formation poisoning in magnetotactic bacteria via Mn(2+) incorpora
182  is the leading cause of Paralytic Shellfish Poisoning in North and South America, Europe, Africa, Au
183 mary outcome was incidence of pesticide self-poisoning in people aged 14 years or older during 3 year
184 be-PVC-based membrane, suffered from sulfide poisoning in the deeper, sulfidic regions of the lake.
185 0027), and those who used methods other than poisoning in their index presentation (fatal and non-fat
186 lter Top2 chromatin binding and prevent Top2 poisoning in yeast.
187 ses addressed an unprecedented epidemic lead poisoning in Zamfara State, northern Nigeria.
188 ng practices and hospitalizations for opioid poisonings in adults.
189 exist describing hospitalizations for opioid poisonings in children and adolescents.
190 ics of hospitalizations attributed to opioid poisonings in children and adolescents.
191                       The consequences of CO poisoning include cardiac dysfunction, brain injury, and
192 or extracorporeal treatment: Severe methanol poisoning including any of the following being attribute
193                         Hospitalizations for poisonings increased after widowhood.
194                 Extracorporeal Treatments in Poisoning inhibitors and folic/folinic acid should be co
195 gments can be fully oxidized without CO as a poisoning intermediate.
196 ), an increase of 161% (P for trend, <.001); poisonings involving methadone increased from 0.10 (95%
197            Acetaminophen (APAP, paracetamol) poisoning is a leading cause of acute liver failure (ALF
198                  Agricultural pesticide self-poisoning is a major public health problem in rural Asia
199                                     Selenium poisoning is a significant health problem in parts of Pu
200                          Staphylococcal food poisoning is caused by enterotoxins excreted into foods
201 t universally fatal, survival following self-poisoning is common, providing an opportunity for second
202 EB) as a bacterial toxin causing severe food poisoning is of great importance.
203                                         Lead poisoning is often seen as a problem of the past.
204                                         Food poisoning is one of the leading causes of morbidity and
205                              Deliberate self-poisoning is the leading method of attempted suicide.
206 of suicide following a first episode of self-poisoning is unknown.
207 untermeasures against acute organophosphorus poisoning is urgently needed.
208  fishery products, responsible for scombroid poisoning, is essential to guarantee the safety of food.
209    Nanoparticles suffer from aggregation and poisoning issues (e.g., oxidation) that severely hinder
210 en reduction reaction, (iii) carbonate anion poisoning, (iv) unique challenges arising from the speci
211   Phototherapy administered during severe CO poisoning limited the blood COHb increase and improved t
212                            Furthermore, this poisoning may be cancer cell selective if this pathway i
213 ases, we suggest that the very nature of CS2 poisoning may be related to interrupting or overwhelming
214                      Here we show that metal poisoning may have caused these aberrant morphologies du
215 , rather than DNA damage resulting from TOP2 poisoning, may play a role in doxorubicin cardiotoxicity
216                                   This "self-poisoning" mechanism explains the need to mix CO into th
217 vel of the mutant in the cell, suggesting a 'poisoning' mechanism in which R402 mutant alpha-tubulin
218 es is a key step towards addressing catalyst poisoning mechanisms in fuel cells.
219 al activity of EDP on food spoilage and food poisoning microorganisms was evaluated through the measu
220 ed US trends in premature all-cause and drug poisoning mortality between 2000 and 2015 at the county
221                                         Drug poisoning mortality increased in counties throughout the
222                                Although drug poisoning mortality increased rapidly in counties with t
223                              Increasing drug poisoning mortality was not limited to poor white people
224 se, n = 6; viral infections, n = 3; mushroom poisoning, n = 5; indeterminate, n = 20) were compared.
225 ts can be obtained from others, stabbing and poisoning neighbouring cells, or colonising spaces while
226 LA(2)s) block neuro-muscular transmission by poisoning nerve terminals.
227 inically described as neurological shellfish poisoning (NSP).
228 dity index of 0 vs 2), diagnoses of overdose/poisoning (odds ratio, 1.35; 95% CI, 1.23-1.47) and diab
229  from a home marine aquarium involved in the poisoning of a whole family.
230 rent length scales for the analysis of metal poisoning of an individual catalyst particle.
231 s of splicing activators, thereby preventing poisoning of key transcripts and disruption to gene expr
232 ersibly store hydrogen prevents the hydrogen poisoning of Ru surfaces.
233                                          The poisoning of rural populations in South and Southeast As
234  0.1 under light, thereby suppressing oxygen poisoning of the Pt catalyst.
235 ct experimental support to the well-accepted poisoning of the TPRE and IIT mechanisms, but also provi
236                        However, IIT, TPRE or poisoning of the TPRE mechanism cannot be used to interp
237 mits formation of EPFRs due to inhibition or poisoning of the transition metal active sites necessary
238  reversal protects forks from breakage after poisoning of Topoisomerase 1.
239                On the other hand, phosphorus poisoning of zeolite-based catalysts, which are used for
240 ic deactivation due to hydrocarbon coking or poisoning of zeolites at an unprecedented spatial resolu
241 om problematic operation stability due to CO poisoning on surface.
242 elf-harm (intentional acts of non-fatal self-poisoning or self-injury) is common, particularly in you
243 onspecific ocular chemical burn and chemical poisoning or toxic effects were eligible for inclusion.
244                                     Methanol poisoning outbreaks after consumption of adulterated alc
245 ual incidence of hospitalizations for opioid poisonings per 100000 children aged 1 to 19 years rose f
246 s epitomize the serious threat that wildlife poisoning poses to biodiversity.
247                          Paralytic shellfish poisoning (PSP) is a serious human illness caused by the
248 veterinary drugs have caused severe wildlife poisoning, pushing the populations of several apex speci
249  suicides are attributable to pesticide self-poisoning, reaching a cost-effectiveness ratio of $75 pe
250                              Treatment of CO poisoning requires rapid removal of CO and restoration o
251 e evidence on population effects of wildlife poisoning rests on assessments conducted at an individua
252           Furthermore, acute carbon monoxide poisoning resulted in a delayed impairment of cognitive
253 years, hospitalizations attributed to opioid poisonings rose nearly 2-fold in the pediatric populatio
254  which are commonly ingested in acts of self-poisoning, seem to be effective in reducing pesticide-sp
255 cidental death, particularly accidental self-poisoning, should be considered to be as important as th
256  Palythoa- and Ostreopsis-related inhalatory poisonings showed many similarities including fever, res
257 rophylactic efficacy against organophosphate poisoning, showing its application potential.
258                        Acute carbon monoxide poisoning significantly suppressed adult hippocampal neu
259 n is critical for C. perfringens type A food poisoning since spores contribute to transmission and re
260 , a derivative of C. perfringens type A food-poisoning strain NCTC8798.
261             Four examined acute Three neonic poisoning studies reported two fatalities (n = 1,280 cas
262   Four examined acute exposure: Three neonic poisoning studies reported two fatalities (n = 1,280 cas
263 ignificant number of patients who survive CO poisoning suffer from long-term neurological and affecti
264 of potentially avoidable causes such as drug poisonings, suicide, and chronic liver disease and cirrh
265 increasing death rates from drug and alcohol poisonings, suicide, and chronic liver diseases and cirr
266 underestimated owing to misclassification of poisoning suicides and mischaracterization of most drug
267 tes a tightly regulated QS self-defense anti-poisoning system.
268 genate aminoalkenes and aminoalkynes without poisoning the catalyst through competitive amine ligatio
269 vity may prevent iron hydride formation from poisoning the P3(B)Fe system.
270 thway features high chemoselectivity without poisoning the surface.
271                     In the absence of severe poisoning, the decision to use extracorporeal treatment
272 ransiently reducing paralysis following BoNT poisoning, the precise mechanisms by which aminopyridine
273                                   Given that poisoning threatens the global populations of more than
274 no evidence of switching from pesticide self-poisoning to other forms of self-harm, with no significa
275                               By selectively poisoning under-coordinated sites with Pb, we have confi
276 ystericus, vocal cord dysfunction, scombroid poisoning, vasoactive amine intolerance, carcinoid syndr
277                                    Lead (Pb) poisoning via ingestion of shot pellets is a frequent ca
278                        Acute carbon monoxide poisoning was achieved by exposing the rats to carbon mo
279              Self-cutting combined with self-poisoning was also associated with increased suicide ris
280  of lung phototherapy administered during CO poisoning was also studied.
281 of adult neurogenesis by the carbon monoxide poisoning was appreciably alleviated by early treatment
282                         For this study, lead poisoning was defined as a blood lead level (BLL) greate
283           The risk of suicide following self-poisoning was markedly increased relative to controls (h
284                                     Thallium poisoning was suspected and investigated but ultimately
285 n method of suicide (125 [73%] of 171), self-poisoning was the main reason for presenting to hospital
286                            Azaspiracid (AZA) poisoning was unknown until 1995 when shellfish harveste
287 bioterrorism agent and a major cause of food poisoning, we developed a "class T" anti-SEB neutralizin
288 ntations involving both self-injury and self-poisoning were associated with higher suicide risk (adju
289                 Patients with acute paraquat poisoning were recruited.
290 04) hospitalizations for prescription opioid poisonings were identified.
291      Patients with metabolic and hematologic poisonings were less likely to survive following extraco
292 cle crashes, and suicide by means other than poisoning-were compared with a counterfactual estimate o
293                SEA is the main cause of food poisoning, while SEB is a common toxin manufactured as a
294 , poor durability, and susceptibility to gas poisoning with adverse environmental issues.
295  are often managed using similar tools, like poisoning with antibiotics or algicides.
296                                    Metformin poisoning with lactic acidosis appears to be amenable to
297 ical course prompted active consideration of poisoning with radioactive material, with the diagnosis
298 ns for extracorporeal treatments in cases of poisoning with these drugs.
299             The Extracorporeal Treatments in Poisoning workgroup aimed to develop evidence-based cons
300       Here, the Extracorporeal Treatments In Poisoning workgroup, comprising international experts re

 
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