ライフサイエンスコーパス: polar headgroup

1 ength and was dependent on the charge of the polar headgroup.

2 ttings into an average orientation of the PC polar headgroup.

3 dependent on the nature of the phospholipid polar headgroup.

4 izable nitroxide tag attached to the lipids' polar headgroup.

5 ctions between charged amino acids and lipid polar headgroups.

6 s as its electrostatic interactions with the polar headgroups.

7 cificity toward phospholipids with different polar headgroups.

8 oduct ions arising from the combined loss of polar headgroup and HNO2, [NO2-FA + H](+) and [NO2-FA -

9 omplex network of hydrogen bonds between the polar headgroup and the protein, while the 3-phenoxyphen

10 Both ceramide and cholesterol have small polar headgroups and relatively large nonpolar bodies.

11 ues of RB-005, in which the lipophilic tail, polar headgroup, and linker region were modified to exte

12 er PFAS classes as methods to activate their polar headgroups are identified.

13 routes enable the incorporation of different polar headgroups as well as nonpolar tails at late stage

14 ty also decreased the mobility of oleic acid polar headgroups, as well as the area/molecule of lipid.

15 low 60 muM preferentially interacts with the polar headgroups at the membrane-electrolyte interface,

16 mplexes have defined both the orientation of polar headgroups between the alpha1 and alpha2 helices o

17 y the lipid acyl length (bilayer thickness), polar headgroups (bilayer interfacial area), inclusion o

18 -and not each enzyme's catalytic residues or polar headgroup binding site-predominantly determines en

19 high-precision biological investigations of polar headgroup/biological target interactions of these

20 The diversity of potential fatty acyl and polar headgroup combinations in this complex saccharolip

21 reveal (i) different thermal regulations and polar headgroup compositions of membrane lipids between

22 g the nitroxide moiety directly to the lipid polar headgroup defines the location of the measured pot

23 uctural changes involved either altering the polar headgroup (e.g., 6-ketocholestanol) or eliminating

24 by double acyl chains and by the presence of polar headgroups facilitating the entrance/exit of proto

25 on to form one or two strong, characteristic polar headgroup fragments.

26 d membranes, and persists in the presence of polar headgroup heterogeneity (e.g., POPC/POPE).

27 shifts indicate that the conformation of the polar headgroup in these bicelles may be different from

28 The polar headgroup interactions are treated using the oppos

29 One population was at the polar headgroup level, but the second was deeply buried

30 Our results suggest that the polar headgroup of 1 resides at the lipid-water interfac

31 ith a nitrophenylazido group attached to the polar headgroup of cardiolipin (CL) via a linker contain

32 f LPA(3) form critical interactions with the polar headgroup of LPA.

33 a fatty acyl chain, and (4) attached to the polar headgroup of PE via a spacer group.

34 sured for dansyl groups: (1) attached to the polar headgroup of PE, (2) linked to an alkyl chain, (3)

35 rom the bilayer center) when attached to the polar headgroup of PE.

36 ns, but domain formation is dependent on the polar headgroup of the lipid.

37 d exhibits unfavorable interactions with the polar headgroups of DPPC, thereby inducing a dehydration

38 s between the OH groups of the sugar and the polar headgroups of DPPC.

39 he TM6-ICL3 junction by interacting with the polar headgroups of membrane phospholipids.

40 C, suggesting a conformational change in the polar headgroups of PLFE.

41 es 2 and 3 appear to be interacting with the polar headgroups of the phospholipids or constitute a di

42 We show that by placing polar headgroups on both ends of the diacetylene lipids

43 heir abundances do not depend on the type of polar headgroup or the number of double bonds of unsatur

44 ics simulations, and experimentally measured polar headgroup pK(a) values, are used to develop a coar

45 y lipids are oriented sideways so that their polar headgroups protrude laterally through a side porta

46 lipid/sHsp interactions are mediated by the polar headgroup region and that the proteins strongly af

47 se hydrophilic peptides do not penetrate the polar headgroup region of the membrane and that the bind

48 cated that the V338C site was located in the polar headgroup region of the membrane, approximately 1.

49 d state, the drug was found to locate in the polar headgroup region of the phospholipid bilayer, to i

50 that the Laurdan chromophore resides in the polar headgroup region of the PLFE liposomes, while the

51 ~ 2) and suggest a large contribution of the polar headgroup region to the dielectric response of the

52 all cases, the dansyl probes located in the polar headgroup region, 19-21 A from the bilayer center.

53 g tendency to seek a shallow location in the polar headgroup region.

54 icant decrease in water content in the lipid polar headgroup regions occurred during the first 1-2-h

55 their degree of acyl chain unsaturation and polar headgroup size using 1-palmitoyl-2-oleoyl-sn-glyce

56 of amphiphilic surfactants anchored by their polar headgroups; sliding occurs at the interface betwee

57 bility of ordered domains decreased with the polar headgroup structure of PO lipids in the order PE >

58 considered headless lipids because they lack polar headgroups that usually form the TCR epitope.

59 everal amphiphile molecules bind along their polar headgroup to the interface binding region (i-face)

60 show that bilayers containing steroids with polar headgroups undergo lateral phase separation, with

61 at cholesterol and ceramides both have small polar headgroups, we propose that ceramides and choleste

62 rfactants with a poly(ethylene glycol) (PEG) polar headgroup were synthesized.

63 e grafted to the aqueous interface via their polar headgroups, whereas the fatty acyl chains are in e

64 tants bearing azide-functionalized PEG-based polar headgroups, which spontaneously react when meeting