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1 is of other primary vasculitides, especially polyarteritis nodosa.
2 man in her 50s with a diagnosis of cutaneous polyarteritis nodosa.
5 henotype, as well as two young siblings with polyarteritis nodosa and one patient with small-vessel v
6 aphy findings led to a clinical diagnosis of polyarteritis nodosa, and immunosuppressive therapy was
7 Wegener's granulomatosis, Cogan's syndrome, polyarteritis nodosa, and systemic lupus erythematosus.
8 age, treating Kawasaki disease, and treating polyarteritis nodosa, but most reported feeling comforta
9 ith multiple cases of systemic and cutaneous polyarteritis nodosa, consistent with autosomal recessiv
10 implicated in the pathogenesis of cutaneous polyarteritis nodosa (cPAN) and early-onset vasculopathy
12 ication criteria for Wegener granulomatosis, polyarteritis nodosa, giant-cell arteritis, and hypersen
14 for vasculitis-related hospitalizations for polyarteritis nodosa, hypersensitivity vasculitis, Wegen
17 cytoplasmic antibody-associated vasculitis, polyarteritis nodosa, Kawasaki disease, giant cell arter
18 pathy (recurrent fevers, livedo reticularis, polyarteritis nodosa, lacunar ischemic strokes, and intr
19 ystemic vasculitides with neuropathy include polyarteritis nodosa, microscopic polyangiitis (MPA), rh
20 y in patients with Wegener's granulomatosis, polyarteritis nodosa, microscopic polyangiitis, and Chur
21 1 mutations, whereas the three patients with polyarteritis nodosa or small-vessel vasculitis were hom
22 (relapsing polychondritis, Sweet's syndrome, polyarteritis nodosa, or giant-cell arteritis) or a hema
23 anulomatosis, polymyositis, dermatomyositis, polyarteritis nodosa, or scleroderma who had an emergent
24 noch-Schonlein purpura (HSP) and macroscopic polyarteritis nodosa (PAN) as early manifestations of Ig
28 ment as a relatively common manifestation of polyarteritis nodosa (PAN), using the modern classificat
29 60% of Nppb-/- females developed mesenteric polyarteritis-nodosa (PAN)-like vasculitis in their life
30 's granulomatosis, inflammatory myopathy, or polyarteritis nodosa rather than rheumatoid arthritis, a
31 akayasu arteritis, giant cell arteritis, and polyarteritis nodosa) to atherosclerosis, display remark
32 extracellular adenosine deaminase, can cause polyarteritis nodosa vasculopathy with highly varied cli
33 of the first case of idiopathic vasculitis, polyarteritis nodosa, which even today is the basis of o